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Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 539 - 550
In contrast to microbially triggered inflammation, mechanisms promoting sterile inflammation remain poorly understood. Damage-associated molecular patterns... 
MEDICINE, RESEARCH & EXPERIMENTAL | DANGER SIGNALS | STERILE INFLAMMATION | CHROMATIN PROTEIN HMGB1 | IN-VIVO | SEPTIC SHOCK | MOBILITY GROUP BOX-1 | RECEPTOR | DEFICIENT MICE | CELL-DEATH | TRANSGENIC MICE | Acetaminophen - adverse effects | Inflammation - chemically induced | Leukocyte Elastase - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Tumor Necrosis Factor-alpha - genetics | Analgesics, Non-Narcotic - pharmacology | Hepatocytes - pathology | Hepatocytes - metabolism | Neutrophil Infiltration | fas Receptor - metabolism | Necrosis - pathology | HMGB1 Protein - genetics | Shock, Septic - metabolism | Inflammation - metabolism | Acetaminophen - pharmacology | Analgesics, Non-Narcotic - adverse effects | HMGB1 Protein - metabolism | fas Receptor - genetics | Chemical and Drug Induced Liver Injury - pathology | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Neutrophils - pathology | Shock, Septic - genetics | Shock, Septic - pathology | Lipopolysaccharides - toxicity | Macrophages - pathology | Shock, Septic - chemically induced | Leukocyte Elastase - genetics | Necrosis - metabolism | Chemical and Drug Induced Liver Injury - genetics | Mice, Knockout | Necrosis - chemically induced | Macrophages - metabolism | Animals | Chemical and Drug Induced Liver Injury - metabolism | Inflammation - genetics | Mice | Necrosis - genetics | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Physiological aspects | Causes of | Inflammation | Genetic aspects | Cell death | Necrosis | Chromosomal proteins | Mass spectrometry | Analysis | Proteins | Studies | Rodents | Mortality | Gangrene | Homeostasis | Mitochondrial DNA | Laboratory animals | Deoxyribonucleic acid--DNA | Apoptosis
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 09/2011, Volume 118, Issue 6, pp. 1032 - 1042
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2018, Volume 38, Issue 11, pp. 2638 - 2650
OBJECTIVE—Deep vein thrombosis results from a combination of risk factors including genetic conditions, obesity, drugs, pregnancy, aging, and malignancy. We... 
urokinase-type plasminogen activator | matrix metalloproteinase | tumor necrosis factor receptor p55 | thrombosis | tumor necrosis factor-α | MODEL | FACTOR-ALPHA | THROMBOEMBOLISM RISK | tumor necrosis factor | PLASMINOGEN-ACTIVATOR | IN-VIVO | PERIPHERAL VASCULAR DISEASE | MICE | TNF-ALPHA | TARGETED DELETION | VEIN | HEMATOLOGY | EXPRESSION
Journal Article
Journal of Periodontal Research, ISSN 0022-3484, 10/2009, Volume 44, Issue 5, pp. 598 - 608
Journal Article
2007, Advances in experimental medicine and biology, ISBN 9780387706290, Volume 597., xix, 206
It has been established that TNF receptor associated factors (TRAFs) are critical signaling mediators for not only the TNF receptor superfamily, but also the... 
Tumor necrosis factor | Receptors | Cellular signal transduction | Life Sciences | Developmental Biology | Cell Biology
Book
2004, Methods in molecular medicine, ISBN 1588292231, Volume 98, xi, 279
Because tumor necrosis factor-a (TNF-a) plays a pivotal role in the regulation of homeostasis and inflammatory immune responses, it offers valuable research... 
Receptors, Tumor Necrosis Factor | Tumor necrosis factor | Disease Models, Animal | Laboratory manuals | Oncology | Medicine & Public Health
Book