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The Journal of cell biology, ISSN 1540-8140, 2014, Volume 204, Issue 2, pp. 231 - 245
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2012, Volume 122, Issue 11, pp. 4094 - 4104
TNF, an inflammatory cytokine that is enriched in the tumor microenvironment, promotes tumor growth and subverts innate immune responses to cancer cells. We... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | INHIBITION | INFLAMMATION | MACROPHAGES | DEATH | EXPRESSION | CANCER | ANTITUMOR | T-CELLS | FACTOR-ALPHA | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Neoplasm Transplantation | Tumor Escape | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor-alpha - genetics | Caspase 8 - metabolism | Graft Rejection - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Receptors, Tumor Necrosis Factor, Type II - immunology | Caspase 8 - genetics | Signal Transduction - immunology | Neoplasms - genetics | T-Lymphocytes - metabolism | Myeloid Cells - immunology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Tumor Necrosis Factor-alpha - immunology | T-Lymphocytes - pathology | Graft Rejection - pathology | CASP8 and FADD-Like Apoptosis Regulating Protein - genetics | Signal Transduction - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | CASP8 and FADD-Like Apoptosis Regulating Protein - biosynthesis | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Caspase 8 - immunology | Animals | Neoplasms - immunology | Cell Line, Tumor | Myeloid Cells - metabolism | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | CASP8 and FADD-Like Apoptosis Regulating Protein - immunology | Graft Rejection - immunology | Myeloid Cells - pathology | Graft Rejection - genetics | Neoplasms - pathology | Tumor necrosis factor | Genetic aspects | Cellular signal transduction | Research | Properties | Gene expression | Apoptosis
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 10/2006, Volume 26, Issue 19, pp. 7046 - 7055
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
CANCER-CELLS | APO2L/TRAIL | DEATH RECEPTORS | ACTIVATION | PROTEIN | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RESISTANCE | CASPASE-8 | NF-KAPPA-B | EXPRESSION | CELL BIOLOGY | Receptors, Tumor Necrosis Factor - metabolism | Apoptosis Regulatory Proteins - pharmacology | Apoptosis - drug effects | Jurkat Cells | Humans | Membrane Glycoproteins - pharmacology | Cells, Cultured | GPI-Linked Proteins | Enzyme Activation - drug effects | Receptors, TNF-Related Apoptosis-Inducing Ligand | Death Domain Receptor Signaling Adaptor Proteins | Receptors, Tumor Necrosis Factor, Member 10c | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins - metabolism | Tumor Necrosis Factor Decoy Receptors | Tumor Necrosis Factor-alpha - pharmacology | Caspases - metabolism | Membrane Glycoproteins - antagonists & inhibitors | TNF-Related Apoptosis-Inducing Ligand | Membrane Microdomains - drug effects | Protein Binding - drug effects | Apoptosis Regulatory Proteins - antagonists & inhibitors | HeLa Cells | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Membrane Microdomains | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins | Biochemistry, Molecular Biology | Tumor Necrosis Factor-alpha | Life Sciences | Apoptosis Regulatory Proteins | Receptors, Tumor Necrosis Factor | Protein Binding | Caspases | Enzyme Activation | Membrane Glycoproteins | Hela Cells | Apoptosis
Journal Article
Nature reviews. Immunology, ISSN 1474-1741, 2009, Volume 9, Issue 7, pp. 491 - 502
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 488, Issue 7412, pp. 508 - 511
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2001, Volume 98, Issue 24, pp. 13960 - 13965
In vivo studies have shown T cells to be central to the mechanism by which estrogen deficiency induces bone loss, but the mechanism involved remains, in part,... 
T lymphocytes | Tumor necrosis factor receptors | Biological Sciences | Spleen cells | Estrogens | Bone marrow | Bones | Osteoclasts | Bone density | Mice | Bone marrow cells | Osteoporosis | Mouse | Ovariectomy | PQCT | FACTOR-RECEPTOR | NUDE-MICE | pQCT | FORMATION IN-VITRO | MULTIDISCIPLINARY SCIENCES | osteoporosis | mouse | TUMOR-NECROSIS-FACTOR | POSTMENOPAUSAL OSTEOPOROSIS | HISTOMORPHOMETRY | OSTEOPROTEGERIN LIGAND | GENE-EXPRESSION | OSTEOCLAST DIFFERENTIATION | ovariectomy | LYMPHOCYTES | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Tumor Necrosis Factor-alpha - genetics | Antigens, CD - genetics | Receptors, Tumor Necrosis Factor - physiology | Antigens, CD - metabolism | Osteoporosis - metabolism | T-Lymphocytes - metabolism | Receptors, Tumor Necrosis Factor, Type II | Female | Estrogens - metabolism | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Estrogens - physiology | Bone Marrow Cells - cytology | Mice, Inbred C57BL | Receptors, Tumor Necrosis Factor, Type I | Cells, Cultured | Mice, Knockout | Animals | T-Lymphocytes - cytology | Mice, Nude | Tumor Necrosis Factor-alpha - physiology | Antigens, CD - physiology | Tumor Necrosis Factor-alpha - biosynthesis | Bone Marrow Cells - metabolism | Physiological aspects | Research | T cells | Tumor necrosis factor | ovariectomy‖osteoporosis‖mouse‖pQCT
Journal Article
American Journal of Physiology: Cell Physiology, ISSN 1522-1563, 2007, Volume 292, Issue 5, pp. C1660 - C1671
Although p38 MAPK activation is essential for myogenesis, the upstream signaling mechanism that activates p38 during myogenesis remains undefined. We recently... 
Myosin heavy chain | Myogenin | Myocyte enhancer factor-2C | Mitogen-activated protein kinase | Akt | Tumor necrosis factor-α | p21 | myocyte enhancer factor-2C | myosin heavy chain | PROTEIN-KINASE PATHWAY | CYTOKINES | PHYSIOLOGY | myogenin | PHOSPHORYLATION | AUTOCRINE SECRETION | SATELLITE CELLS | MITOGEN | CELL BIOLOGY | tumor necrosis factor-alpha | mitogen-activated protein kinase | TUMOR-NECROSIS-FACTOR | GENE-EXPRESSION | TERMINAL DIFFERENTIATION | GROWTH-FACTOR-I | Tumor Necrosis Factor-alpha - metabolism | Cobra Cardiotoxin Proteins | Muscle, Skeletal - metabolism | Muscle Fibers, Skeletal - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Dose-Response Relationship, Drug | MAP Kinase Kinase 6 - metabolism | Receptors, Tumor Necrosis Factor, Type I - deficiency | Myoblasts - metabolism | Muscular Diseases - physiopathology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Muscle, Skeletal - drug effects | Cell Differentiation | p38 Mitogen-Activated Protein Kinases - metabolism | Muscular Diseases - chemically induced | Disease Models, Animal | Autocrine Communication | Cell Line | Muscular Diseases - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Tumor Necrosis Factor-alpha - pharmacology | Regeneration - drug effects | Animals | Muscle Development - drug effects | Muscle, Skeletal - physiopathology | Mice | Enzyme Activation | Muscle, Skeletal - pathology | tumor necrosis factor-α
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2014, Volume 124, Issue 4, pp. 1537 - 1551
Development of host protective immunity against Mycobacterium tuberculosis infection is critically dependent on the inflammatory cytokine TNF. TNF signals... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | BACILLUS-CALMETTE-GUERIN | TRANSMEMBRANE TNF | DEFICIENT MICE | BACTERIAL-INFECTION | HUMAN ALVEOLAR MACROPHAGES | DENDRITIC CELL MATURATION | FACTOR RECEPTOR | INDUCED GRANULOMA-FORMATION | FACTOR-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Tuberculosis, Pulmonary - microbiology | Tumor Necrosis Factor Decoy Receptors - genetics | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor Decoy Receptors - immunology | Dendritic Cells - immunology | Mycobacterium tuberculosis - immunology | Male | Receptors, Tumor Necrosis Factor, Type II - immunology | Host-Pathogen Interactions - immunology | CD4-Positive T-Lymphocytes - immunology | Mycobacterium tuberculosis - pathogenicity | Receptors, Tumor Necrosis Factor, Type I - deficiency | Signal Transduction - immunology | Female | Interleukin-12 Subunit p40 - metabolism | Tumor Necrosis Factor Decoy Receptors - deficiency | Lymphocyte Activation | Mice, Inbred C57BL | Solubility | Tuberculosis, Pulmonary - pathology | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Granuloma - pathology | Tuberculosis, Pulmonary - immunology | Animals | Mice | Prevention | Care and treatment | Immune response | Mycobacterium tuberculosis | Tumor necrosis factor | Physiological aspects | Drug targeting | Regulation | Research | Studies | Tuberculosis | Cytokines | Rodents | Mortality | Ligands | Infections
Journal Article
Neuroscience, ISSN 0306-4522, 2008, Volume 157, Issue 2, pp. 414 - 423
Abstract Recent studies suggest that tumor necrosis factor-alpha (TNF) sensitizes primary afferent neurons, and thus facilitates neuropathic pain. Here, we... 
Neurology | DRG | neuropathic pain | ectopic activity | cytokines | TNF receptor | TNF | PRIMARY AFFERENTS | ALPHA TNF-ALPHA | TACTILE ALLODYNIA | INCREASES | INJURY | DORSAL-ROOT GANGLIA | NEUROSCIENCES | CELL-DEATH | NEUROPATHY | HYPERALGESIA | UP-REGULATION | Hyperalgesia - chemically induced | Receptors, Tumor Necrosis Factor - classification | Receptors, Tumor Necrosis Factor, Type II - chemistry | Humans | Male | Ganglia, Spinal - cytology | Ligation - methods | Dose-Response Relationship, Drug | Nerve Fibers - physiology | Receptors, Tumor Necrosis Factor, Type I - chemistry | Time Factors | Reaction Time - drug effects | Receptors, Tumor Necrosis Factor, Type I - pharmacology | Pain Threshold - drug effects | Action Potentials - drug effects | Receptors, Tumor Necrosis Factor - genetics | Physical Stimulation | Receptors, Tumor Necrosis Factor - chemistry | Rats | Nerve Fibers - drug effects | Receptors, Tumor Necrosis Factor - administration & dosage | Mutation - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | Rats, Sprague-Dawley | Receptors, Tumor Necrosis Factor, Type II - genetics | Spinal Cord - surgery | Hyperalgesia - physiopathology | Animals | Receptors, Tumor Necrosis Factor, Type II - pharmacology | Sensory Receptor Cells - drug effects | Analysis of Variance | In Vitro Techniques | Pain Measurement | Neurosciences | Serum albumin | Tumor necrosis factor | Neurons | Tumors | Necrosis | Index Medicus
Journal Article