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Journal Article
PLoS ONE, ISSN 1932-6203, 12/2012, Volume 7, Issue 12, pp. e52682 - e52682
Pancreatic stellate cells (PSCs) play a crucial role in the aggressive behavior of pancreatic cancer. Although heterogeneity of PSCs has been identified, the... 
FIBROSIS | MATRIX SYNTHESIS | STIMULATING PROLIFERATION | CARCINOMA CELLS | DUCTAL ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | IDENTIFICATION | FIBROBLASTS | PROGRESSION | NERVE GROWTH-FACTOR | TUMOR-STROMAL INTERACTIONS | Neoplasm Transplantation | Pancreatic Neoplasms - metabolism | Prognosis | Coculture Techniques | Humans | Middle Aged | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | Male | Receptors, Nerve Growth Factor - metabolism | RNA, Messenger - metabolism | Aged, 80 and over | Adult | Carcinoma, Pancreatic Ductal - mortality | Female | Pancreatic Neoplasms - mortality | Tumor Cells, Cultured | Gene Expression | Pancreatic Neoplasms - pathology | RNA, Messenger - genetics | Stromal Cells - metabolism | Proportional Hazards Models | Cell Communication | Mice, SCID | Carcinoma, Pancreatic Ductal - pathology | Nerve Tissue Proteins - genetics | Receptors, Nerve Growth Factor - genetics | Nerve Tissue Proteins - metabolism | Animals | Aged | Mice | Cell Movement | Pancreatic Stellate Cells - metabolism | Immunohistochemistry | Flow cytometry | Severe combined immunodeficiency | Oncology | Tissues | Carcinogenesis | Carcinogens | Rodents | Surgery | Gastroenterology | Xenografts | Fibroblasts | Extracellular matrix | Growth factors | Stellate cells | Aggressive behavior | Melanoma | Gene expression | Polymerase chain reaction | Cytometry | Stromal cells | Pancreatic cancer | Medical prognosis | Tumors | Cancer | Index Medicus
Journal Article
Technology in Cancer Research & Treatment, ISSN 1533-0346, 10/2018, Volume 17, pp. 1533033818803632 - 1533033818803632
Pancreatic cancer is one of the most aggressive cancers with a 5-year patient survival rate of 8.2% and limited availability of therapeutic agents to target... 
BET | pancreatic cancer | tumor microenvironment | microengineered tumor stromal assay (µTSA) | stroma | CANCER-ASSOCIATED FIBROBLASTS | CONVERSION | INDUCE | microengineered tumor stromal assay (ATSA) | CULTURE | MICROENVIRONMENT | ONCOLOGY | GROWTH | ANIMAL-MODELS | EXPRESSION | Index Medicus
Journal Article
Biopolymers and Cell, ISSN 0233-7657, 2014, Volume 30, Issue 6, pp. 426 - 435
Intercellular communication is one of the most important factors involved in the maintenance of tissue homeostasis. The alteration of intercellular interaction... 
Tumor-stromal interaction | Exosome | exosome | tumor–stromal interaction
Journal Article
Journal Article
Endocrinology and Metabolism(구 대한내분비학회지), ISSN 2093-596X, 2016, Volume 31, Issue 2, p. 206
Bone metastasis is a frequent occurrence in breast cancer, affecting more than 70% of late stage cancer patients with severe complications such as fracture,... 
Tumor-stromal interaction | Bone metastasis | Transforming growth factor | Osteoclasts | Breast neoplasms | Osteoblasts
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2006, Volume 119, Issue 12, pp. 2750 - 2759
The hypoxic environment in tumor is reported to play an important role in pancreatic cancer progression. The interaction between stromal and cancer cells also... 
Tumor-stromal interaction | Hypoxia | c-Met | Hepatocyte growth factor | Pancreatic cancer | hypoxia | HEPATOCELLULAR-CARCINOMA CELLS | INDUCIBLE FACTOR-1-ALPHA | METASTASIS | ANGIOGENESIS | tumor-stromal interaction | FACTOR EXPRESSION | IDENTIFICATION | OVARIAN-CARCINOMA | MATRIX METALLOPROTEINASES | pancreatic cancer | INVASION | ONCOLOGY | hepatocyte growth factor | IN-VIVO | Proto-Oncogene Proteins c-met - metabolism | Cell Hypoxia - physiology | Matrix Metalloproteinases - genetics | Pancreatic Neoplasms - metabolism | Humans | Cell Communication - physiology | Culture Media, Conditioned - pharmacology | RNA, Messenger - metabolism | Cell Movement - physiology | Tissue Inhibitor of Metalloproteinases - genetics | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Hepatocyte Growth Factor - genetics | Fibroblasts - metabolism | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | RNA, Messenger - genetics | Stromal Cells - metabolism | Pancreatic Neoplasms - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Tissue Inhibitor of Metalloproteinases - metabolism | Hepatocyte Growth Factor - metabolism | Proto-Oncogene Proteins c-met - genetics | Cell Movement - drug effects | Matrix Metalloproteinase 2 - genetics | Signal Transduction - drug effects | Fibroblasts - drug effects | Cell Line, Tumor | Signal Transduction - physiology | Cell Proliferation - drug effects | Fibroblasts - cytology | Matrix Metalloproteinases - metabolism | Stromal Cells - cytology | Index Medicus
Journal Article
Journal Article
Cancer Microenvironment, ISSN 1875-2292, 12/2010, Volume 3, Issue 1, pp. 109 - 116
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 10/2011, Volume 22, Issue 20, pp. 3791 - 3800
Journal Article
Forum on Immunopathological Diseases and Therapeutics, ISSN 2151-8017, 2013, Volume 4, Issue 1, pp. 53 - 62
Journal Article
2007, AD-a470 594.
The tasks outlined in the Statement of Work for the first year of research on the action of ADAM9 isoforms in tumor-stromal interactions encompassed the... 
hypotheses | drugs | therapy | tissues(biology) | cells(biology) | tumor-stromal interactions | breast cancer | endogenous proteins | epidermal growth factor receptors | adam9 isoforms | metastasis | proteins | substrates | antibodies | metalloprotease activity | immunohistochemistry
Government Document
FASEB JOURNAL, ISSN 0892-6638, 04/2008, Volume 22, Issue 4, pp. 1144 - 1154
Extracellular matrix metalloproteinase inducer (EMMPRIN) is a cell surface glycoprotein enriched on tumor cells and normal epithelia. It is mainly known for... 
wound healing | TGF-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MMPs | METALLOPROTEINASE INDUCER | INDUCTION | BASIGIN | CELL BIOLOGY | BREAST-CANCER CELLS | GROWTH-FACTOR-BETA | epithelial-stromal interactions | BIOLOGY | GENE-EXPRESSION | EMMPRIN | DERMAL FIBROBLASTS | TGh beta | TUMOR-STROMAL INTERACTIONS
Journal Article
Oncogene, ISSN 0950-9232, 07/2005, Volume 24, Issue 32, pp. 5053 - 5068
Journal Article