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Genes & cancer, ISSN 1947-6027, 2011, Volume 2, Issue 4, pp. 466 - 474
Inactivation of the p53 tumor suppressor is a frequent event in tumorigenesis. In most cases, the p53 gene is mutated, giving rise to a stable mutant protein... 
cancer | tumorigenesis | mutant | p53 | Monographs
Journal Article
Nature reviews. Cancer, ISSN 1474-175X, 02/2016, Volume 16, Issue 2, p. 70
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2015, Volume 125, Issue 1, pp. 42 - 46
Journal Article
Seminars in immunopathology, ISSN 1863-2297, 2012, Volume 35, Issue 2, pp. 229 - 244
Connection between inflammation and cancer is a rapidly developing field. Epidemiological data suggests that inflammation along with distinct arms of host... 
Inflammatory bowel disease | Biomedicine | Immunology | Colon cancer | Cytokines | Internal Medicine | Bacteria | Inflammation | INDUCED CYTIDINE DEAMINASE | SPONTANEOUS INTESTINAL TUMORIGENESIS | IMMUNOLOGY | PATHOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | DEXTRAN SODIUM-SULFATE | TUMOR-NECROSIS-FACTOR | MURINE ULCERATIVE-COLITIS | BETA-CATENIN ACTIVATION | PROMOTES COLON TUMORIGENESIS | NF-KAPPA-B | GENOME-WIDE ASSOCIATION | Colonic Neoplasms - prevention & control | Humans | Risk Factors | Colitis - complications | Colitis - pathology | Inflammatory Bowel Diseases - metabolism | Colonic Neoplasms - metabolism | Rectal Neoplasms - prevention & control | Rectal Neoplasms - microbiology | Animals | Colitis - microbiology | Colitis - therapy | Colonic Neoplasms - pathology | Inflammatory Bowel Diseases - pathology | Colitis - metabolism | Inflammatory Bowel Diseases - microbiology | Rectal Neoplasms - metabolism | Rectal Neoplasms - pathology | Inflammatory Bowel Diseases - complications | Rectal Neoplasms - etiology | Colonic Neoplasms - etiology | Inflammatory Bowel Diseases - therapy | Colonic Neoplasms - microbiology | Prevention | Gastrointestinal diseases | Colorectal cancer | Development and progression | Colitis | Risk factors | Tumors | Inflammatory bowel diseases | Molecular modelling | Intestine | Data processing | Tumorigenesis | Neoplasia | Immune system | Metastases | cytokines | bacteria | inflammation | colon cancer | inflammatory bowel disease
Journal Article
Chinese Medical Journal, ISSN 0366-6999, 11/2016, Volume 129, Issue 21, pp. 2646 - 2646
[This corrects the article DOI: 10.4103/0366-6999.185873.]. 
Tumorigenesis | Corrigendum
Journal Article
Carcinogenesis, ISSN 0143-3334, 1980
Journal
Molecular carcinogenesis, ISSN 1098-2744, 1988
Journal
by Gu, X and Xing, L and Shi, G and Liu, Z and Wang, X and Qu, Z and Wu, X and Dong, Z and Gao, X and Liu, G and Yang, L and Xu, Y
Cell Death and Differentiation, ISSN 1350-9047, 03/2012, Volume 19, Issue 3, pp. 397 - 405
Journal Article
The Journal of investigative dermatology, ISSN 0022-202X, 9/2018, Volume 138, Issue 9, pp. 1925 - 1934
Skin lipids (e.g. fatty acids) are essential for normal skin functions. Epidermal fatty acid-binding protein (E-FABP) is the predominant FABP expressed in skin... 
SOX2 | E-FABP | Keratinocyte | Skin tumorigenesis | p53
Journal Article
2018, ISBN 0674545397, xi, 271 pages
"Popular understanding holds that genetic changes create cancer. James DeGregori uses evolutionary principles to propose a new way of thinking about cancer's... 
Evolution | Genetic aspects | Etiology | Carcinogenesis | Cells | Cancer | Cells-Evolution | Cancer-Etiology
Book
Genes and Diseases, 03/2015, Volume 2, Issue 1, pp. 13 - 25
One of the greatest obstacles to current cancer treatment efforts is the development of drug resistance by tumors. Despite recent advances in diagnostic... 
Resistance | Therapy | Tumorigenesis | Insulin-like growth factor | Cancer
Journal Article
Cancer genomics & proteomics, ISSN 1109-6535, 2004
Journal
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2018, Volume 155, Issue 5, pp. 1593 - 1607.e12
Mutations at hotspots in GNAS, which encodes stimulatory G-protein, α subunits, are detected in approximately 60% of intraductal papillary mucinous neoplasms... 
Suppressor | Tumorigenesis | Signal Transduction | Oncogene | tumorigenesis | oncogene | signal transduction | suppressor
Journal Article
Genes & cancer, ISSN 1947-6027, 2010, Volume 1, Issue 6, pp. 597 - 604
Tumorigenesis is generally caused by genetic changes that activate oncogenes or inactivate tumor suppressor genes. The targeted inactivation of oncogenes can... 
Oncogene addiction | MYC | Tumorigenesis | oncogene addiction | tumorigenesis | Monographs
Journal Article