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Journal of Biological Chemistry, ISSN 0021-9258, 2012, Volume 287, Issue 3, pp. 1847 - 1860
Obesity and insulin resistance are associated with chronic, low grade inflammation. Moreover, regulation of energy metabolism and immunity are highly... 
DIABETES-MELLITUS | SKELETAL-MUSCLE | HEPATIC GLUCONEOGENESIS | METABOLIC SYNDROME | INSULIN-RESISTANCE | NUCLEAR RECEPTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CALORIC RESTRICTION | HUMAN OBESITY | NECROSIS-FACTOR-ALPHA | ENDOPLASMIC-RETICULUM STRESS | Enzyme Activators - pharmacology | RNA-Binding Proteins - genetics | AMP-Activated Protein Kinases - metabolism | Liver - pathology | Receptors, Interleukin-15 - biosynthesis | Haptoglobins - biosynthesis | Caloric Restriction | Haptoglobins - genetics | Hepatocytes - pathology | Interleukin 1 Receptor Antagonist Protein - genetics | Male | Hepatocytes - metabolism | Obesity - genetics | Gene Knockdown Techniques | Receptors, Interleukin-15 - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Inflammation Mediators - metabolism | Trans-Activators - genetics | Mice, Inbred DBA | Fasting - metabolism | Interleukin 1 Receptor Antagonist Protein - biosynthesis | C-Reactive Protein - biosynthesis | Physical Conditioning, Animal | Gene Expression Regulation - genetics | Liver - metabolism | Metformin - pharmacology | Cells, Cultured | Rats | PPAR alpha - genetics | C-Reactive Protein - genetics | Transcription Factors - genetics | Hypoglycemic Agents - pharmacology | Obesity - metabolism | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Animals | Energy Metabolism | Insulin Resistance - genetics | Obesity - therapy | Trans-Activators - metabolism | Mice | PPAR alpha - metabolism | AMP-Activated Protein Kinases - genetics | RNA-Binding Proteins - metabolism | Index Medicus | IL15Rα | IL1Rn | Coactivator Transcription | Liver | Inflammation | Cellular Immune Response | Diabetes | Tweak | Metformin | Metabolism | Nuclear Receptors | necrosis-factor-alpha | skeletal-muscle | metabolic syndrome | nuclear receptors | hepatic gluconeogenesis | insulin-resistance | caloric restriction | diabetes-mellitus | endoplasmic-reticulum stress | human obesity
Journal Article
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 04/2013, Volume 133, Issue 4, pp. 1052 - 1062
Journal Article
Neuro-Oncology, ISSN 1522-8517, 09/2018, Volume 20, Issue 10, pp. 1321 - 1330
Abstract Background Glioblastoma (GBM) is a difficult to treat brain cancer that nearly uniformly recurs, and recurrent tumors are largely therapy resistant.... 
Fn14 | Temozolomide | Gliosarcoma | Migration | Glioblastoma | temozolomide | SURVIVAL | gliosarcoma | TWEAK | KAPPA-B | CLINICAL NEUROLOGY | ADJUVANT TEMOZOLOMIDE | WEAK INDUCER | MOLECULAR TARGETS | INVASION | glioblastoma | STEM-LIKE CELLS | EVOLUTION | ONCOLOGY | migration | RADIOTHERAPY | Index Medicus | Basic and Translational Investigations
Journal Article
Journal of Controlled Release, ISSN 0168-3659, 12/2017, Volume 267, pp. 144 - 153
The most common and deadly form of primary brain cancer, glioblastoma (GBM), is characterized by significant intratumoral heterogeneity, microvascular... 
Fibroblast growth factor-inducible 14 | Biodegradable nanoparticles | Glioblastoma | Targeted therapeutics | Invasive malignant glioma | Multiple particle tracking | Surface plasmon resonance | DRUG-DELIVERY | FN14 | CHEMISTRY, MULTIDISCIPLINARY | CHEMOTHERAPY | MALIGNANT GLIOMA | TRIAL | THERAPY | EXPRESSION LEVELS | PHARMACOLOGY & PHARMACY | GLIADEL WAFERS | BINDING | Nanoparticles - chemistry | Mice, Inbred C57BL | Antibodies, Monoclonal - pharmacokinetics | Drug Carriers - administration & dosage | Polyethylene Glycols - pharmacokinetics | Polyethylene Glycols - chemistry | Brain Neoplasms - drug therapy | Drug Carriers - chemistry | Polyethylene Glycols - administration & dosage | Glioma - metabolism | Brain - metabolism | Polyesters - chemistry | Polyesters - pharmacokinetics | TWEAK Receptor - metabolism | Animals | Antibodies, Monoclonal - administration & dosage | Polyesters - administration & dosage | Cell Line, Tumor | Drug Carriers - pharmacokinetics | Nanoparticles - administration & dosage | Antibodies, Monoclonal - chemistry | Extracellular Matrix Proteins - metabolism | Glioma - drug therapy | Drugs | Medical colleges | Drug delivery systems | Brain tumors | Polyols | Homeopathy | Materia medica and therapeutics | Vehicles | Nanoparticles | Gliomas | Polyethylene glycol | Analysis | Therapeutics | Exhibitions | Fibroblast growth factors | Health aspects | Index Medicus | invasive malignant glioma | glioblastoma | surface plasmon resonance | fibroblast growth factor-inducible 14 | multiple particle tracking | biodegradable nanoparticles | targeted therapeutics
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 1, pp. 111 - 120
Journal Article
Molecular Pharmacology, ISSN 0026-895X, 11/2012, Volume 82, Issue 5, pp. 948 - 957
Journal Article
The Journal of Pathology, ISSN 0022-3417, 12/2013, Volume 231, Issue 4, pp. 480 - 494
TWEAK , a member of the TNF superfamily, binds to the Fn14 receptor, eliciting biological responses. EGFR signalling is involved in experimental renal injury.... 
renal | ADAM17 | TWEAK | inflammation | EGFR | CELLS | ANGIOTENSIN-II | PROLIFERATION | PATHOLOGY | WEAK INDUCER | TUBULOINTERSTITIAL INFLAMMATION | INHIBITION | ONCOLOGY | KIDNEY INJURY | DISEASE | EGF RECEPTOR | MULTIFUNCTIONAL CYTOKINE | ADAM17 Protein | Erlotinib Hydrochloride | Receptor, Epidermal Growth Factor - genetics | Epithelial Cells - metabolism | ADAM Proteins - physiology | Humans | Transcriptional Activation - drug effects | NF-kappa B - metabolism | Nephritis - prevention & control | Nephritis - pathology | TWEAK Receptor | Intercellular Signaling Peptides and Proteins - metabolism | Receptor, Epidermal Growth Factor - metabolism | Inflammation Mediators - metabolism | Female | Nephritis - chemically induced | Phosphorylation - drug effects | Kidney Tubules - metabolism | Transforming Growth Factor alpha - metabolism | Tumor Necrosis Factors - pharmacology | Receptors, Tumor Necrosis Factor - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Silencing | Tumor Necrosis Factors - metabolism | Heparin-binding EGF-like Growth Factor | ADAM Proteins - metabolism | Cytokine TWEAK | Animals | Signal Transduction - drug effects | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Ligands | Nephritis - metabolism | Signal Transduction - physiology | Mice | Transcriptional Activation - physiology | Quinazolines - pharmacology | Transforming growth factors | Inflammation | Epidermal growth factor | Index Medicus
Journal Article
Basic Research in Cardiology, ISSN 0300-8428, 3/2013, Volume 108, Issue 2, pp. 1 - 13
Pulmonary arterial hypertension (PAH) is a fatal disease for which no cure is yet available. The leading cause of death in PAH is right ventricular (RV)... 
Right heart disease | Medicine & Public Health | Fibrosis | MAL | Cardiac fibroblasts | Fn14 | Cardiology | TRANSCRIPTION FACTORS | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | APOPTOSIS TWEAK | PULMONARY ARTERIAL-HYPERTENSION | SRF ACTIVITY | ENDOTHELIN SYSTEM | WEAK INDUCER | CARDIAC-HYPERTROPHY | CEREBRAL-ISCHEMIA | MOLECULAR-MECHANISMS | Immunohistochemistry | Tumor Necrosis Factors - genetics | Up-Regulation | Cell Proliferation | TWEAK Receptor | Receptors, Tumor Necrosis Factor - physiology | Hypertrophy, Right Ventricular - physiopathology | Trans-Activators - genetics | Apoptosis Regulatory Proteins - genetics | Ventricular Dysfunction, Right - physiopathology | Cell Differentiation | Membrane Proteins - metabolism | Endothelin-1 - physiology | Fibrosis - prevention & control | Hypertrophy, Right Ventricular - metabolism | Real-Time Polymerase Chain Reaction | Ventricular Dysfunction, Right - metabolism | Signal Transduction | Membrane Proteins - genetics | Tumor Necrosis Factors - metabolism | Rats | Familial Primary Pulmonary Hypertension | Myocardium - pathology | Transcription Factors - genetics | Rats, Sprague-Dawley | Blotting, Western | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Transcription Factors - metabolism | Ventricular Dysfunction, Right - prevention & control | Collagen - metabolism | Cytokine TWEAK | Animals | Fluorescent Antibody Technique | Hypertrophy, Right Ventricular - prevention & control | Trans-Activators - metabolism | Mice | Myofibroblasts | Hypertension, Pulmonary - complications | Heart | Endothelin | Collagen | Fibroblast growth factors | Heart diseases | Pulmonary hypertension | Index Medicus | Original Contribution
Journal Article