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Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2015, Volume 125, Issue 5, pp. 1968 - 1978
Journal Article
The Journal of Physiology, ISSN 0022-3751, 07/2011, Volume 589, Issue 14, pp. 3641 - 3658
Non‐technical summary  Shivering is an involuntary somatic motor response that occurs in skeletal muscles to produce heat during exposure to cold environments... 
PHYSIOLOGY | NONSHIVERING THERMOGENESIS | 5-HT1A RECEPTORS | SYMPATHETIC PREMOTOR NEURONS | SPINAL-CORD | DORSOMEDIAL HYPOTHALAMUS | BROWN ADIPOSE-TISSUE | EXPRESSING PREOPTIC NEURONS | BODY-TEMPERATURE | NEUROSCIENCES | PROSTAGLANDIN EP3 RECEPTOR | RAPHE PALLIDUS | Skin Temperature - physiology | Neural Pathways - drug effects | Efferent Pathways - physiology | Fever - metabolism | Male | Preoptic Area - metabolism | Neural Pathways - physiology | Fever - physiopathology | N-Methylaspartate - pharmacology | Neurons - metabolism | Medulla Oblongata - physiology | Preoptic Area - physiology | Dinoprostone - pharmacology | Body Temperature Regulation - physiology | Shivering - physiology | Rats | Mediodorsal Thalamic Nucleus - drug effects | Tachycardia - metabolism | Raphe Nuclei - drug effects | Raphe Nuclei - physiology | Neural Pathways - metabolism | Adipose Tissue, Brown - drug effects | Receptors, GABA-A - metabolism | Muscimol - pharmacology | Rats, Wistar | Preoptic Area - drug effects | Sympathetic Nervous System - drug effects | GABA-A Receptor Antagonists - metabolism | Sympathetic Nervous System - metabolism | Raphe Nuclei - metabolism | Thermogenesis - physiology | Heart Rate - drug effects | Mediodorsal Thalamic Nucleus - metabolism | Sympathetic Nervous System - physiology | Shivering - drug effects | Neurons - physiology | Heart Rate - physiology | Blood Pressure - drug effects | Blood Pressure - physiology | Tachycardia - pathology | Mediodorsal Thalamic Nucleus - physiology | Neurons - drug effects | Receptor, Serotonin, 5-HT1A - metabolism | Cold Temperature | Medulla Oblongata - drug effects | Medulla Oblongata - metabolism | Adipose Tissue, Brown - physiology | 8-Hydroxy-2-(di-n-propylamino)tetralin - pharmacology | Efferent Pathways - metabolism | Animals | Thermogenesis - drug effects | GABA | Heart beat | Neurons | Analysis | Prostaglandins E | Heart rate | Skin | Cooling | Thermogenesis | Cold | Rodents | Index Medicus | Integrative
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0169743 - e0169743
Background Inhibitors of the renin angiotensin system and neprilysin (RAS-/NEP-inhibitors) proved to be extraordinarily beneficial in systolic heart failure.... 
NERVOUS-SYSTEM | OXIDATIVE STRESS | OMAPATRILAT | DIFFERENTIAL EXPRESSION | METABOLISM | MULTIDISCIPLINARY SCIENCES | PRESERVED EJECTION FRACTION | CARDIOMYOPATHIC HAMSTERS | DYSFUNCTION | ENALAPRIL | PATHOPHYSIOLOGY | Adaptation, Physiological | Mitochondria, Heart - ultrastructure | Rabbits | Mitochondria, Heart - metabolism | Heart Failure - physiopathology | Lipid Metabolism | Male | Heart Failure - metabolism | Thiazepines - pharmacology | Mitochondria, Heart - drug effects | Electron Transport Complex I - metabolism | Electron Transport Complex IV - metabolism | Animals | Neprilysin - antagonists & inhibitors | Glucose - metabolism | Renin-Angiotensin System - drug effects | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Heart Ventricles - metabolism | Pyridines - pharmacology | Heart Ventricles - drug effects | Heart failure | Usage | Analysis | Angiotensin | Research | Electron microscopy | Health aspects | Left ventricular function | Heart | Adaptations | Tricarboxylic acid cycle | Oxidative stress | Fat metabolism | Change detection | Cardiomyopathy | Electron transfer | Chains | Autophagy | Experiments | Proteins | Glucose metabolism | Mitochondria | Renin | Pathways | Enzymatic activity | Rodents | Physiology | Blood pressure | Inhibition | Heart diseases | Failure | Enzymes | Internal medicine | Metabolism | Medicine | Tachycardia | Hospitals | Transmission electron microscopy | Inhibitors | Medical prognosis | Neprilysin | Ventricle | Electron transport | Index Medicus
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 06/2012, Volume 21, Issue 12, pp. 2759 - 2767
Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an inherited arrhythmogenic disease so far related to mutations in the cardiac ryanodine... 
JUNCTIONAL SARCOPLASMIC-RETICULUM | CALSEQUESTRIN | GENE | RYANODINE RECEPTOR | CA2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | SKELETAL-MUSCLE TRIADIN | GENETICS & HEREDITY | MOLECULAR-CLONING | ISOFORM | C-TERMINAL ENDS | MUTATIONS | Calcium - metabolism | Humans | Cercopithecus aethiops | Endoplasmic Reticulum - metabolism | Family Health | Male | Death, Sudden, Cardiac | Protein Isoforms - metabolism | Muscle Proteins - metabolism | Tachycardia, Ventricular - metabolism | Tachycardia, Ventricular - genetics | Female | Cell Membrane - metabolism | Arrhythmias, Cardiac - genetics | Arrhythmias, Cardiac - metabolism | Genetic Predisposition to Disease - genetics | Rats | Reverse Transcriptase Polymerase Chain Reaction | Genes, Recessive | Blotting, Western | Mice, Knockout | Muscle Proteins - genetics | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Pedigree | Myocytes, Cardiac - metabolism | Tachycardia, Ventricular - pathology | Mice | Mutation | COS Cells | Protein Isoforms - genetics | Index Medicus | Heart | Tachycardia | Missense mutation | calsequestrin | Arrhythmia | Calcium | Nonsense mutation | Ryanodine receptors | Gene deletion | Cell Membrane | Arrhythmias, Cardiac | Endoplasmic Reticulum | Life Sciences | Muscle Proteins | Tachycardia, Ventricular | Carrier Proteins | Myocytes, Cardiac | Genetic Predisposition to Disease | Biochemistry, Molecular Biology | Protein Isoforms
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 11/2016, Volume 7, Issue 1, pp. 13344 - 13344
Diabetes mellitus (DM) encompasses a multitude of secondary disorders, including heart disease. One of the most frequent and potentially life threatening... 
ACTIVATION | MYOCARDIAL-INFARCTION | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | TOLL-LIKE RECEPTORS | NLRP3 INFLAMMASOME | SARCOPLASMIC-RETICULUM | HUMAN MONOCYTES | VENTRICULAR REPOLARIZATION | NALP3 INFLAMMASOME | CONVERTING-ENZYME | Potassium - metabolism | Toll-Like Receptor 2 - genetics | Calcium - metabolism | Myocytes, Cardiac - immunology | Receptors, Interleukin-1 - genetics | Caspase 1 - metabolism | Interleukin-1beta - genetics | Action Potentials | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Inflammasomes - antagonists & inhibitors | Interleukin-1beta - metabolism | Arrhythmias, Cardiac - immunology | Tachycardia, Ventricular - metabolism | Diabetes Mellitus, Experimental - complications | Antirheumatic Agents - pharmacology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Diabetes Mellitus, Experimental - metabolism | Macrophages - immunology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Arrhythmias, Cardiac - metabolism | Myocardial Contraction | Receptors, Interleukin-1 - immunology | NLR Family, Pyrin Domain-Containing 3 Protein - antagonists & inhibitors | Tachycardia, Ventricular - etiology | Interleukin-1beta - immunology | Mice, Transgenic | Arrhythmias, Cardiac - etiology | Diabetes Mellitus, Experimental - immunology | Animals | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Myocytes, Cardiac - metabolism | Toll-Like Receptor 2 - immunology | Mice | Receptors, Interleukin-1 - antagonists & inhibitors | Tachycardia, Ventricular - immunology | Index Medicus
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 10/2009, Volume 105, Issue 8, pp. 737 - 745
RATIONALE:Mutations in glycerol-3-phosphate dehydrogenase 1-like (GPD1-L) protein reduce cardiac Na current (INa) and cause Brugada Syndrome (BrS). GPD1-L has... 
Electrophysiology | Ion channels | Sudden death | Arrhythmias | PROTEIN-KINASE-C | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | SARCOPLASMIC-RETICULUM | VENTRICULAR MYOCYTES | SODIUM-CHANNEL | ion channels | sudden death | BRUGADA-SYNDROME | arrhythmias | electrophysiology | GATED K+ CHANNELS | PERIPHERAL VASCULAR DISEASE | XENOPUS OOCYTES | HEMATOLOGY | MODULATION | NICOTINAMIDE-ADENINE-DINUCLEOTIDE | Brugada Syndrome - genetics | Protein Kinase C - genetics | Superoxide Dismutase - genetics | Humans | Sodium - metabolism | Brugada Syndrome - metabolism | Cyclic AMP-Dependent Protein Kinases - genetics | Protein Kinase C - metabolism | Myocardium - metabolism | Muscle Proteins - metabolism | Sodium Channels - metabolism | Tachycardia, Ventricular - metabolism | Tachycardia, Ventricular - genetics | Antineoplastic Agents - pharmacology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | NAD - metabolism | NAV1.5 Voltage-Gated Sodium Channel | Superoxide Dismutase - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Cell Line | NAD - genetics | Oxidation-Reduction | Enzyme Inhibitors - pharmacology | Rats | Protein Kinase C - antagonists & inhibitors | Down-Regulation - drug effects | Benzophenanthridines - pharmacology | Down-Regulation - genetics | Muscle Proteins - genetics | Glycerolphosphate Dehydrogenase - metabolism | Animals | Mice | Sodium Channels - genetics | Mutation | Glycerolphosphate Dehydrogenase - genetics | Index Medicus
Journal Article
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2016, Volume 98, pp. 62 - 72
Abstract Diastolic calcium (Ca) leak via cardiac ryanodine receptors (RyR2) can cause arrhythmias and heart failure (HF). Ca/calmodulin (CaM)-dependent kinase... 
Cardiovascular | Dantrolene | Fluorescence resonance energy transfer | Calcium/calmodulin-dependent protein kinase II | Calmodulin | Ryanodine receptor | POLYMORPHIC VENTRICULAR-TACHYCARDIA | CARDIAC & CARDIOVASCULAR SYSTEMS | MALIGNANT HYPERTHERMIA | CA2+ RELEASE | CONTRACTILE DYSFUNCTION | HEART-FAILURE | CARDIAC RYANODINE RECEPTOR | AFFECT CALCIUM SPARKS | CELL BIOLOGY | INTERDOMAIN INTERACTIONS | CALMODULIN-BINDING | PROTEIN-KINASE-II | Phosphorylation | Calcium - metabolism | Protein Conformation - drug effects | Ryanodine Receptor Calcium Release Channel - metabolism | Tacrolimus Binding Proteins - metabolism | Tachycardia, Ventricular - metabolism | Sarcoplasmic Reticulum - metabolism | Calcium Signaling | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Disease Models, Animal | Rabbits | Calmodulin - metabolism | Tachycardia, Ventricular - drug therapy | Tachycardia, Ventricular - etiology | Heart Failure - metabolism | Dantrolene - pharmacology | Heart Failure - pathology | Permeability | Tachycardia, Ventricular - physiopathology | Animals | Ryanodine Receptor Calcium Release Channel - chemistry | Myocytes, Cardiac - metabolism | Protein Binding | Mice | Ion Channel Gating | Heart failure | Tachycardia | Index Medicus | fluorescence resonance energy transfer | dantrolene | calcium | ryanodine receptor | calmodulin-dependent protein kinase II | calmodulin
Journal Article