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Neuron, ISSN 0896-6273, 08/2012, Volume 75, Issue 4, pp. 618 - 632
Mitochondrial abnormalities have been documented in Alzheimer’s disease and related neurodegenerative disorders, but the causal relationship between... 
ALZHEIMERS-DISEASE BRAIN | DOMINANT OPTIC ATROPHY | MITOCHONDRIAL-FUNCTION | MOUSE MODEL | LIGHT-CHAIN | FRONTOTEMPORAL DEMENTIA | AXONAL-TRANSPORT | NEUROSCIENCES | DYNAMIN-RELATED PROTEIN | PHOSPHORYLATION SITES | TRANSGENIC MICE | Neurons - pathology | Microtubule-Associated Proteins - genetics | Tauopathies - genetics | Cytoskeletal Proteins - genetics | Gelsolin - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Actins - metabolism | Tauopathies - pathology | Cytoplasm - metabolism | MicroRNAs - metabolism | Green Fluorescent Proteins - genetics | Mitochondrial Proteins - genetics | Drosophila Proteins - metabolism | GTP-Binding Proteins - genetics | Nerve Degeneration - metabolism | Neurons - ultrastructure | tau Proteins - genetics | Cell Death - genetics | Mitochondria - genetics | Mitochondrial Proteins - metabolism | ATP Synthetase Complexes - metabolism | Cell Cycle Proteins - genetics | Tauopathies - complications | Cytoskeletal Proteins - metabolism | Myosins - metabolism | Cytoplasm - genetics | RNA Interference - physiology | Disease Models, Animal | In Situ Nick-End Labeling | Green Fluorescent Proteins - metabolism | Animals, Genetically Modified | Gene Expression Regulation - genetics | Drosophila | Cell Cycle Proteins - metabolism | Mitochondria - metabolism | Mitochondria - pathology | Mutation - genetics | Animals | GTP Phosphohydrolases - metabolism | Analysis of Variance | GTP Phosphohydrolases - genetics | Gelsolin - genetics | Mice | Drosophila Proteins - genetics | Nerve Degeneration - etiology | Voltage-Dependent Anion Channels - metabolism | GTP-Binding Proteins - metabolism | Nervous system diseases | Actin | Neurons | Utrophin | Myosin | Mitochondrial DNA | Alzheimer's disease | Proteins | Phosphorylation | Mitochondria | Neurotoxicity | Insects | Microscopy | Neurodegeneration | Pathogenesis | Morphology | Mutation | Defects | Neurodegenerative diseases | Tau protein | Cell death | Elongation
Journal Article
Neuron, ISSN 0896-6273, 02/2012, Volume 73, Issue 4, pp. 685 - 697
Journal Article
EMBO reports, ISSN 1469-221X, 11/2017, Volume 18, Issue 11, pp. 2051 - 2066
Endocytic processes are facilitated by both curvature‐generating BAR ‐domain proteins and the coordinated polymerization of actin filaments. Under... 
protein Bin1 | tau | BAR | Alzheimer's disease | actin binding | genetic risk factor | N-BAR protein Bin1 | PROTEIN | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CROSS-LINKING | FILAMENT TURNOVER | TAU PATHOLOGY | CELL BIOLOGY | SKELETAL-MUSCLE | STRUCTURAL BASIS | SYNAPTIC VESICLE ENDOCYTOSIS | MEMBRANE CURVATURE | SH3 DOMAIN | Tauopathies - genetics | Humans | Actins - metabolism | Tauopathies - pathology | tau Proteins - metabolism | Drosophila Proteins - metabolism | Drosophila melanogaster - genetics | Actins - genetics | Drosophila melanogaster - metabolism | Protein Isoforms - metabolism | tau Proteins - genetics | Tumor Suppressor Proteins - genetics | Cloning, Molecular | Escherichia coli - metabolism | Protein Interaction Domains and Motifs | Nuclear Proteins - genetics | Binding Sites | Actin Cytoskeleton - genetics | Disease Models, Animal | Recombinant Proteins - metabolism | Gene Expression | Tumor Suppressor Proteins - metabolism | Actin Cytoskeleton - metabolism | Genetic Vectors - chemistry | Gene Expression Regulation | Genetic Vectors - metabolism | Nuclear Proteins - metabolism | Recombinant Proteins - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Carrier Proteins - genetics | Tauopathies - metabolism | Animals | Carrier Proteins - metabolism | Escherichia coli - genetics | Adaptor Proteins, Signal Transducing - genetics | Protein Binding | Drosophila Proteins - genetics | Adaptor Proteins, Signal Transducing - metabolism | Protein Isoforms - genetics | Membranes | Neurodegenerative diseases | Drosophila | Health risks | Polymerization | Rods | Bundles | Risk factors | Membrane proteins | Bundling | Proteins | Depolymerization | Insects | Tau protein | Filaments | Neurodegeneration | Actin | Isoforms | BAR protein | Cytoskeleton | Alzheimers disease | Curvature | N‐BAR protein Bin1 | Neuroscience | Cell Adhesion, Polarity & Cytoskeleton
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2015, Volume 10, Issue 11, pp. e0142340 - e0142340
The perforant pathway projection from layer II of the entorhinal cortex to the hippocampal dentate gyrus is especially important for long-term memory... 
MAMMALIAN TARGET | IN-VITRO | STIMULATION | MULTIDISCIPLINARY SCIENCES | MEMORY IMPAIRMENT | DISEASE | TAU | ENTORHINAL CORTEX NEURONS | TOXICITY | AUTOPHAGY | PATHOLOGY | Microglia - metabolism | Entorhinal Cortex - metabolism | Humans | tau Proteins - metabolism | Male | Hippocampus - drug effects | Neurodegenerative Diseases - drug therapy | TOR Serine-Threonine Kinases - antagonists & inhibitors | Synapses - metabolism | Dentate Gyrus - drug effects | Perforant Pathway - metabolism | Neurons - metabolism | Phosphorylation - drug effects | Neurons - drug effects | Disease Models, Animal | Synapses - drug effects | Dentate Gyrus - metabolism | Microglia - drug effects | Alzheimer Disease - drug therapy | Axons - drug effects | Axons - metabolism | Memory, Long-Term - drug effects | Entorhinal Cortex - drug effects | Neurodegenerative Diseases - metabolism | Sirolimus - pharmacology | Hippocampus - metabolism | Tauopathies - metabolism | Animals | Perforant Pathway - drug effects | Alzheimer Disease - metabolism | Mice | Tauopathies - drug therapy | TOR protein | Brain | Phosphorylation | Toxicity | Medical services | Activation | mRNA | Kinases | Autophagy | Neurotoxicity | Neurodegeneration | Rodents | Long term memory | Degeneration | Inhibition | Alzheimer's disease | Neurodegenerative diseases | Neurons | Astrocytes | Rapamycin | Inflammation | Substrates | Microglia | Pathology | Cortex (entorhinal) | Dentate gyrus | Gliosis | Inhibitors | Tau protein | Hippocampus | Phagocytosis | Synapses | Index Medicus
Journal Article
Journal Article
Ageing Research Reviews, ISSN 1568-1637, 2017, Volume 36, pp. 50 - 63
Highlights • We reviewed the emerging tau PET literature across various neurodegenerative conditions and non-AD tauopathies. • Focal tau pathology has been... 
Neurology | Internal Medicine | Tauopathies | MRI | Neurodegeneration | Tau | PET | Dementia | Cognitive impairment | TAUOPATHY MOUSE MODEL | ALZHEIMERS-DISEASE | FRONTOTEMPORAL DEMENTIA | NEUROFIBRILLARY PATHOLOGY | MILD COGNITIVE IMPAIRMENT | LATE-LIFE DEPRESSION | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | POSITRON-EMISSION-TOMOGRAPHY | MEDIAL TEMPORAL-LOBE | PROGRESSIVE SUPRANUCLEAR PALSY | LEWY BODY DEMENTIA | Humans | tau Proteins - metabolism | Dementia - physiopathology | Temporal Lobe - diagnostic imaging | Dementia - metabolism | Neurofibrillary Tangles - metabolism | Tauopathies - diagnostic imaging | Cognitive Dysfunction - diagnostic imaging | Tauopathies - physiopathology | Neurodegenerative Diseases - diagnostic imaging | Biomarkers - metabolism | Dementia - diagnostic imaging | Neurofibrillary Tangles - pathology | Alzheimer Disease - physiopathology | Plaque, Amyloid - pathology | Cognitive Dysfunction - metabolism | Neurodegenerative Diseases - metabolism | Positron-Emission Tomography - methods | Cognitive Dysfunction - physiopathology | Temporal Lobe - metabolism | Tauopathies - metabolism | Temporal Lobe - physiopathology | Animals | Neurodegenerative Diseases - physiopathology | Plaque, Amyloid - physiopathology | Alzheimer Disease - metabolism | Fluorine Radioisotopes - metabolism | Plaque, Amyloid - metabolism | Alzheimer Disease - diagnostic imaging | Medicine, Experimental | Medical research | Nervous system diseases | PET imaging | Alzheimer's disease
Journal Article
Journal Article