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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2013, Volume 110, Issue 43, pp. 17426 - 17431
The telomerase reverse transcriptase (TERT) promoter, an important element of telomerase expression, has emerged as a target of cancer-specific mutations.... 
Relapse | Urinary bladder neoplasms | Carcinoma | Urinary bladder | Alleles | Cell lines | Genetic mutation | Binding sites | Tumors | Cancer | Cancer genetics | Transcription factors | Noncoding mutations | TERT mutations | Reporter assays | noncoding mutations | WILD-TYPE P53 | CELLS | transcription factors | MULTIDISCIPLINARY SCIENCES | TUMOR | RISK | BREAST | reporter assays | cancer genetics | GENE-EXPRESSION | TRANSCRIPTIONAL REGULATION | EPIDEMIOLOGY | TELOMERE LENGTH | Luciferases - metabolism | Prognosis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Male | Promoter Regions, Genetic - genetics | Luciferases - genetics | Telomerase - genetics | Urinary Bladder Neoplasms - genetics | Base Sequence | DNA Mutational Analysis - statistics & numerical data | Urinary Bladder Neoplasms - pathology | Aged, 80 and over | Female | Carcinoma, Transitional Cell - genetics | Kaplan-Meier Estimate | Neoplasm Recurrence, Local | Proportional Hazards Models | Carcinoma, Transitional Cell - pathology | Cell Line, Tumor | Biomarkers, Tumor - genetics | Polymorphism, Single Nucleotide | Mutation | Promoters (Genetics) | Gene mutations | Patient outcomes | Physiological aspects | Genetic aspects | Research | Bladder cancer | Genetic polymorphisms | Biological Sciences | Cancer and Oncology | Cancer och onkologi
Journal Article
Aging Cell, ISSN 1474-9718, 08/2017, Volume 16, Issue 4, pp. 837 - 846
Summary Kallistatin, an endogenous protein, protects against vascular injury by inhibiting oxidative stress and inflammation in hypertensive rats and enhancing... 
sirtuin 1 | kallistatin | vascular senescence | aging | microRNA‐34a | oxidative stress | microRNA-34a | NECROSIS-FACTOR-ALPHA | TISSUE KALLIKREIN INHIBITOR | BINDING PROTEIN | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | ORGAN INJURY | CAENORHABDITIS-ELEGANS | PROTECTIVE ROLE | PROGENITOR-CELL SENESCENCE | ENDOTHELIAL-CELLS | RENAL INJURY | Sirtuin 1 - metabolism | Diabetes Mellitus, Experimental - drug therapy | Endothelial Progenitor Cells - cytology | MicroRNAs - antagonists & inhibitors | Streptozocin | Humans | Diabetes Mellitus, Experimental - genetics | NADPH Oxidases - metabolism | Caenorhabditis elegans Proteins - metabolism | Cellular Senescence - drug effects | Male | MicroRNAs - metabolism | Sirtuin 1 - genetics | Telomerase - genetics | Plasminogen Activator Inhibitor 1 - metabolism | Superoxides - metabolism | beta-Galactosidase - metabolism | NADPH Oxidases - genetics | Telomerase - metabolism | Diabetes Mellitus, Experimental - chemically induced | Superoxides - antagonists & inhibitors | Nitric Oxide Synthase Type III - metabolism | Plasminogen Activator Inhibitor 1 - genetics | Diabetes Mellitus, Experimental - metabolism | Sirtuins - genetics | Endothelial Progenitor Cells - metabolism | Cellular Senescence - genetics | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - genetics | Catalase - genetics | Endothelial Progenitor Cells - drug effects | Cells, Cultured | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Nitric Oxide Synthase Type III - genetics | Serpins - pharmacology | Catalase - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Animals | Caenorhabditis elegans - drug effects | Genistein - pharmacology | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Mice | MicroRNAs - genetics | beta-Galactosidase - genetics | Caenorhabditis elegans Proteins - genetics | Sirtuins - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Gene expression | MicroRNA | Oxidative stress | Senescence | Genistein | INK4a protein | Kinases | NAD(P)H oxidase | Catalase | Rodents | Aging | Aorta | Telomerase | Protein-tyrosine kinase | Tyrosine | Enzymes | Diabetes mellitus | p16 Protein | MiRNA | Superoxide | SIRT1 protein | Life span | Tumor necrosis factor | MicroRNAs | Stem cells | Nematodes | Diabetes | Original
Journal Article
Journal Article
Genes and Development, ISSN 0890-9369, 04/2016, Volume 30, Issue 7, pp. 812 - 826
Journal Article
Oncogene, ISSN 0950-9232, 06/2003, Volume 22, Issue 25, pp. 3911 - 3916
Journal Article
Molecular Aspects of Medicine, ISSN 0098-2997, 11/2015, Volume 45, pp. 87 - 96
The diffuse gliomas are primary central nervous system tumors that arise most frequently in the cerebral hemispheres of adults. They are currently classified... 
Biomarker | 1p/19q | Glioblastoma | ATRX | H3F3A | BRAF | IDH | EGFR | Oligodendroglioma | Glioma | TERT | TP53 | Astrocytoma | MEDICINE, RESEARCH & EXPERIMENTAL | PROMOTER HYPERMETHYLATION | IMMUNOHISTOCHEMICAL EXPRESSION | LOW-GRADE GLIOMAS | BRAIN-TUMORS | DNA-REPAIR GENE | IDH1 MUTATIONS | OLIGODENDROGLIAL DIFFERENTIATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | INTEGRATED GENOMIC ANALYSIS | HUMAN GLIOBLASTOMA | O-6-METHYLGUANINE-DNA METHYLTRANSFERASE | Receptor, Epidermal Growth Factor - genetics | Humans | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Receptor, Epidermal Growth Factor - metabolism | Telomerase - genetics | Glioblastoma - genetics | Telomerase - metabolism | Chromosomes, Human, Pair 1 - genetics | DNA Helicases - genetics | Proto-Oncogene Proteins B-raf - metabolism | Repressor Proteins - metabolism | Chromosome Deletion | PTEN Phosphohydrolase - genetics | Promoter Regions, Genetic | Glioblastoma - diagnosis | Tumor Suppressor Protein p53 - metabolism | PTEN Phosphohydrolase - metabolism | Repressor Proteins - genetics | Isocitrate Dehydrogenase - genetics | Receptor, Notch1 - metabolism | DNA-Binding Proteins - genetics | DNA Helicases - metabolism | Proto-Oncogene Proteins B-raf - genetics | Gene Rearrangement | Isocitrate Dehydrogenase - metabolism | Biomarkers, Tumor - genetics | Receptor, Notch1 - genetics | Diagnosis | Tumor proteins | Gliomas | Telomerase
Journal Article