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Journal of Clinical Investigation, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, pp. 367 - 384
There is a considerable resurgence of interest in the role of aerobic glycolysis in cancer; however, increased glycolysis is frequently viewed as a consequence... 
MEDICINE, RESEARCH & EXPERIMENTAL | GENE-EXPRESSION SIGNATURE | SOLUBLE ADENYLYL-CYCLASE | VIRUS-TRANSFORMED CELLS | GLUCOSE-METABOLISM | PYRUVATE-KINASE M2 | EPIDERMAL-GROWTH-FACTOR | RECONSTITUTED BASEMENT-MEMBRANE | EXTRACELLULAR-MATRIX | HUMAN BREAST CELLS | MAMMARY EPITHELIAL-CELLS | Up-Regulation | Humans | Glucose Transporter Type 3 - metabolism | Acetylglucosamine - metabolism | Breast Neoplasms - metabolism | Receptor, Epidermal Growth Factor - metabolism | Guanine Nucleotide Exchange Factors - metabolism | Female | Membrane Proteins - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Telomere-Binding Proteins - metabolism | Oncogenes | Second Messenger Systems | Cell Line | Oxygen Consumption | Biosynthetic Pathways | Glycosylation | MAP Kinase Kinase Kinases - metabolism | Adenylyl Cyclases - metabolism | Cell Transformation, Neoplastic - metabolism | Integrin beta1 - metabolism | Phenotype | Carrier Proteins - metabolism | Thyroid Hormones - metabolism | Glucose - metabolism | Glycolysis | Breast Neoplasms - mortality | Protein Processing, Post-Translational | Complications and side effects | Care and treatment | Patient outcomes | Physiological aspects | Development and progression | Research | Carcinogenesis | Risk factors | Integrins | Medical research | Dehydrogenases | Metabolites | Cloning | Breast cancer | Glucose | Metabolism | Gene expression | Index Medicus | Abridged Index Medicus
Journal Article
Molecular Cell, ISSN 1097-2765, 09/2010, Volume 39, Issue 6, pp. 839 - 850
Journal Article
STEM CELLS, ISSN 1066-5099, 04/2005, Volume 23, Issue 4, pp. 594 - 603
Multipotential stem cells can be selected from the bone marrow by plastic adhesion, expanded, and cultured. They are able to differentiate not only into... 
Mesenchymal stem cell | Bone marrow | Transcription factors | Cell differentiation | Insulin | Pancreatic beta cell | Bonemarrow | PROGENITOR CELLS | mesenchymal stem cell | pancreatic beta cell | cell differentiation | insulin | VIVO | transcription factors | BETA-CELLS | NEUROENDOCRINE DIFFERENTIATION | bone marrow | PDX-1 GENE | CELL BIOLOGY | ISLET TRANSPLANTATION | DIABETES-MELLITUS | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | TELOMERASE ACTIVITY | EXPANSION | NUCLEAR FACTOR 3-BETA | HEMATOLOGY | Hepatocyte Nuclear Factor 3-beta - genetics | Coculture Techniques | Homeodomain Proteins - metabolism | Humans | Multipotent Stem Cells - metabolism | Adipocytes - cytology | RNA, Messenger - metabolism | Insulin - biosynthesis | Insulin-Secreting Cells - metabolism | Mesenchymal Stromal Cells - cytology | Trans-Activators - genetics | Adenoviridae - genetics | Cell Differentiation | Insulin-Secreting Cells - cytology | Telomere - metabolism | Trans-Activators - biosynthesis | Insulin - genetics | Biomarkers - metabolism | Hepatocyte Nuclear Factor 3-beta - biosynthesis | Homeodomain Proteins - biosynthesis | Bone Marrow Cells - cytology | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Transcription Factors - biosynthesis | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Homeodomain Proteins - genetics | Animals | Multipotent Stem Cells - cytology | Adipocytes - metabolism | Mice | Bone Marrow Cells - metabolism | Index Medicus
Journal Article
Science, ISSN 0036-8075, 5/2012, Volume 336, Issue 6081, pp. 593 - 597
The telomere end-protection problem is defined by the aggregate of DNA damage signaling and repair pathways that require repression at telomeres. To define the... 
Telomeres | Yeasts | Quantification | Lymphocytes | DNA | DNA damage | REPORTS | Cell cycle | Ataxia telangiectasia | Repression | Chromosomes | JOINING PATHWAY | POT1 PROTEINS | MAMMALIAN TELOMERES | SGS1 | MULTIDISCIPLINARY SCIENCES | DYSFUNCTIONAL TELOMERES | DOUBLE-STRAND BREAKS | HOMOLOGOUS RECOMBINATION | NHEJ | YEAST KU | Telomere - ultrastructure | Antigens, Nuclear - metabolism | Telomeric Repeat Binding Protein 1 - genetics | Telomeric Repeat Binding Protein 1 - metabolism | Homologous Recombination | DNA Breaks, Double-Stranded | DNA Ligases - metabolism | DNA-Binding Proteins - metabolism | Poly-ADP-Ribose Binding Proteins | Telomere-Binding Proteins - genetics | DNA End-Joining Repair | Telomere - metabolism | Telomere-Binding Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Tumor Suppressor Proteins - metabolism | Chromosomal Proteins, Non-Histone - metabolism | Signal Transduction | Cell Cycle Proteins - metabolism | Cells, Cultured | Ataxia Telangiectasia Mutated Proteins | Xenopus Proteins | DNA-Binding Proteins - genetics | Telomere Homeostasis | Mice, Knockout | Poly(ADP-ribose) Polymerases - metabolism | Animals | Antigens, Nuclear - genetics | Cell Cycle | Ku Autoantigen | DNA Repair | DNA Ligase ATP | Telomeric Repeat Binding Protein 2 - metabolism | Mice | Poly (ADP-Ribose) Polymerase-1 | Telomeric Repeat Binding Protein 2 - genetics | Tumor Suppressor p53-Binding Protein 1 | Proteins | Physiological aspects | Research | Health aspects | DNA repair | Telomerase | Index Medicus | Ataxia telangiectasia mutated protein
Journal Article
Molecular Cell, ISSN 1097-2765, 01/2013, Volume 49, Issue 1, pp. 172 - 185
The metabolism of glucose and glutamine, primary carbon sources utilized by mitochondria to generate energy and macromolecules for cell growth, is directly... 
TARGET | RAPTOR | TEL2 | TUMOR-SUPPRESSOR COMPLEX | RAG GTPASES | PROTEIN-KINASE | STABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | CELL-CYCLE PROGRESSION | NUTRIENT | CELL BIOLOGY | Glutamine - deficiency | Humans | Multiprotein Complexes | Protein Multimerization | Stress, Physiological | Breast Neoplasms - metabolism | Mechanistic Target of Rapamycin Complex 1 | Telomere-Binding Proteins - genetics | Ribosomal Protein S6 Kinases, 90-kDa - metabolism | Lysosomes - metabolism | Adenosine Triphosphate - metabolism | Adenylate Kinase - metabolism | Tumor Suppressor Proteins - genetics | Statistics, Nonparametric | Female | Telomere-Binding Proteins - metabolism | DNA Helicases - genetics | ATPases Associated with Diverse Cellular Activities | Tumor Suppressor Proteins - metabolism | Signal Transduction | Cells, Cultured | Citric Acid Cycle | Mice, Knockout | Protein Transport | Carrier Proteins - genetics | DNA Helicases - metabolism | Glucose - deficiency | Animals | Breast Neoplasms - genetics | Carrier Proteins - metabolism | Proteins - metabolism | Energy Metabolism | Monomeric GTP-Binding Proteins - metabolism | Protein Binding | Carcinoma - genetics | Mice | TOR Serine-Threonine Kinases | Carcinoma - metabolism | Proteins | Glucose metabolism | Physiological aspects | Stress (Physiology) | Mitochondrial DNA | Glucose | Cells | Dextrose | Glutamine | Index Medicus
Journal Article
Journal Article
Journal Article
Nature, ISSN 0028-0836, 02/2011, Volume 470, Issue 7334, pp. 359 - 365
Telomere dysfunction activates p53-mediated cellular growth arrest, senescence and apoptosis to drive progressive atrophy and functional decline in... 
STEM-CELLS | HEPATOCELLULAR-CARCINOMA | BIOGENESIS | PGC-1-ALPHA | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | LEADS | MICE | DAMAGE | P53 | COACTIVATORS | Cell Proliferation | Reactive Oxygen Species - metabolism | Hematopoietic Stem Cells - pathology | DNA, Mitochondrial - analysis | Adenosine Triphosphate - biosynthesis | Tumor Suppressor Protein p53 - genetics | RNA - genetics | Telomerase - deficiency | Cardiomyopathies - physiopathology | Telomerase - genetics | Myocardium - metabolism | Telomere - metabolism | Gluconeogenesis | Telomere - genetics | Liver - metabolism | Tumor Suppressor Protein p53 - metabolism | Cardiomyopathies - pathology | Transcription Factors - antagonists & inhibitors | Hematopoietic Stem Cells - metabolism | Mitochondria - metabolism | Mitochondria - pathology | Tumor Suppressor Protein p53 - deficiency | Doxorubicin - toxicity | Aging - pathology | Myocardium - cytology | Transcription Factors - metabolism | Telomere - pathology | Animals | Cardiomyopathies - metabolism | Telomere - enzymology | Liver - cytology | Mice | Aging - metabolism | Cardiomyopathies - chemically induced | Telomeres | Mitochondria | Genetic aspects | Chemical properties | Abnormalities | Liver | Cell division | Mitochondrial DNA | Biosynthesis | Biology | DNA repair | Proteins | Studies | Aging | Telomerase | Binding sites | Apoptosis | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 10/2007, Volume 449, Issue 7164, pp. 928 - 932
Journal Article
Journal Article