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Science, ISSN 0036-8075, 5/2012, Volume 336, Issue 6081, pp. 593 - 597
The telomere end-protection problem is defined by the aggregate of DNA damage signaling and repair pathways that require repression at telomeres. To define the... 
Telomeres | Yeasts | Quantification | Lymphocytes | DNA | DNA damage | REPORTS | Cell cycle | Ataxia telangiectasia | Repression | Chromosomes | JOINING PATHWAY | POT1 PROTEINS | MAMMALIAN TELOMERES | SGS1 | MULTIDISCIPLINARY SCIENCES | DYSFUNCTIONAL TELOMERES | DOUBLE-STRAND BREAKS | HOMOLOGOUS RECOMBINATION | NHEJ | YEAST KU | Telomere - ultrastructure | Antigens, Nuclear - metabolism | Telomeric Repeat Binding Protein 1 - genetics | Telomeric Repeat Binding Protein 1 - metabolism | Homologous Recombination | DNA Breaks, Double-Stranded | DNA Ligases - metabolism | DNA-Binding Proteins - metabolism | Poly-ADP-Ribose Binding Proteins | Telomere-Binding Proteins - genetics | DNA End-Joining Repair | Telomere - metabolism | Telomere-Binding Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Tumor Suppressor Proteins - metabolism | Chromosomal Proteins, Non-Histone - metabolism | Signal Transduction | Cell Cycle Proteins - metabolism | Cells, Cultured | Ataxia Telangiectasia Mutated Proteins | Xenopus Proteins | DNA-Binding Proteins - genetics | Telomere Homeostasis | Mice, Knockout | Poly(ADP-ribose) Polymerases - metabolism | Animals | Antigens, Nuclear - genetics | Cell Cycle | Ku Autoantigen | DNA Repair | DNA Ligase ATP | Telomeric Repeat Binding Protein 2 - metabolism | Mice | Poly (ADP-Ribose) Polymerase-1 | Telomeric Repeat Binding Protein 2 - genetics | Tumor Suppressor p53-Binding Protein 1 | Proteins | Physiological aspects | Research | Health aspects | DNA repair | Telomerase | Index Medicus | Ataxia telangiectasia mutated protein
Journal Article
Journal Article
Molecular Cell, ISSN 1097-2765, 01/2013, Volume 49, Issue 1, pp. 172 - 185
The metabolism of glucose and glutamine, primary carbon sources utilized by mitochondria to generate energy and macromolecules for cell growth, is directly... 
TARGET | RAPTOR | TEL2 | TUMOR-SUPPRESSOR COMPLEX | RAG GTPASES | PROTEIN-KINASE | STABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | CELL-CYCLE PROGRESSION | NUTRIENT | CELL BIOLOGY | Glutamine - deficiency | Humans | Multiprotein Complexes | Protein Multimerization | Stress, Physiological | Breast Neoplasms - metabolism | Mechanistic Target of Rapamycin Complex 1 | Telomere-Binding Proteins - genetics | Ribosomal Protein S6 Kinases, 90-kDa - metabolism | Lysosomes - metabolism | Adenosine Triphosphate - metabolism | Adenylate Kinase - metabolism | Tumor Suppressor Proteins - genetics | Statistics, Nonparametric | Female | Telomere-Binding Proteins - metabolism | DNA Helicases - genetics | ATPases Associated with Diverse Cellular Activities | Tumor Suppressor Proteins - metabolism | Signal Transduction | Cells, Cultured | Citric Acid Cycle | Mice, Knockout | Protein Transport | Carrier Proteins - genetics | DNA Helicases - metabolism | Glucose - deficiency | Animals | Breast Neoplasms - genetics | Carrier Proteins - metabolism | Proteins - metabolism | Energy Metabolism | Monomeric GTP-Binding Proteins - metabolism | Protein Binding | Carcinoma - genetics | Mice | TOR Serine-Threonine Kinases | Carcinoma - metabolism | Proteins | Glucose metabolism | Physiological aspects | Stress (Physiology) | Mitochondrial DNA | Glucose | Cells | Dextrose | Glutamine | Index Medicus
Journal Article
Journal Article
Cell, ISSN 0092-8674, 05/2018, Volume 173, Issue 4, pp. 972 - 988.e23
Repair of damaged DNA is essential for maintaining genome integrity and for preventing genome-instability-associated diseases, such as cancer. By combining... 
DNA damage repair | proximity labeling | shieldin | proteomics | 53BP1 | PARP inhibitors | telomere maintenance | antibody class-switch recombination | NHEJ | BRCA1 | PATHWAYS | CELLS | RESECTION | DAMAGE-RESPONSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLASS-SWITCH RECOMBINATION | HOMOLOGOUS RECOMBINATION | DOUBLE-STRAND BREAK | TELOMERES | CELL BIOLOGY | Mad2 Proteins - metabolism | Humans | Ubiquitin-Protein Ligases - antagonists & inhibitors | DNA Breaks, Double-Stranded | Mad2 Proteins - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Telomere-Binding Proteins - genetics | RNA Interference | BRCA1 Protein - metabolism | Trans-Activators - genetics | Immunoglobulin Class Switching - drug effects | Nuclear Proteins - genetics | Telomere-Binding Proteins - metabolism | Tumor Suppressor p53-Binding Protein 1 - metabolism | DNA End-Joining Repair - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Mutagenesis, Site-Directed | Tumor Suppressor p53-Binding Protein 1 - genetics | Ubiquitin-Protein Ligases - metabolism | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Telomere-Binding Proteins - antagonists & inhibitors | Tumor Suppressor p53-Binding Protein 1 - antagonists & inhibitors | BRCA1 Protein - genetics | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | BRCA1 Protein - antagonists & inhibitors | Mad2 Proteins - genetics | Cell Line, Tumor | Trans-Activators - metabolism | Ubiquitin-Protein Ligases - genetics | RNA, Small Interfering - metabolism | DNA damage | Genomics | Antibodies | Genomes | DNA repair | Proteins | Viral antibodies | Telomeres | Evolutionary biology | Analysis | DNA | Genetic research | Mass spectrometry | Index Medicus
Journal Article
Journal Article
Molecular Cell, ISSN 1097-2765, 09/2010, Volume 39, Issue 6, pp. 839 - 850
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 2013, Volume 202, Issue 7, pp. 1023 - 1039
Chromosome pairing is an essential meiotic event that ensures faithful haploidization and recombination of the genome. Pairing of homologous chromosomes is... 
STRUCTURED ILLUMINATION | FISSION YEAST | ZEBRAFISH ZYGOTE | LINC COMPLEX | SPINDLE POLE BODY | ENVELOPE | ACTIN CYTOSKELETON | NUCLEAR-MEMBRANE PROTEIN | C. ELEGANS | BOUQUET FORMATION | CELL BIOLOGY | Meiosis - physiology | RNA, Small Interfering - genetics | Spermatocytes - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Molecular Sequence Data | Male | Immunoenzyme Techniques | Cell Cycle Proteins - antagonists & inhibitors | Telomere-Binding Proteins - genetics | Microtubules - metabolism | Cell Nucleus - metabolism | HEK293 Cells | Cell Cycle Proteins - genetics | Female | Membrane Proteins - metabolism | Nuclear Proteins - genetics | Telomere-Binding Proteins - metabolism | Real-Time Polymerase Chain Reaction | Cytoplasmic Dyneins - metabolism | Telomere - genetics | Protein Structure, Tertiary | Amino Acid Sequence | Membrane Proteins - genetics | RNA, Messenger - genetics | Cell Cycle Proteins - metabolism | Cells, Cultured | Cytoplasmic Dyneins - genetics | In Situ Hybridization, Fluorescence | Nuclear Proteins - metabolism | Microtubule-Associated Proteins - antagonists & inhibitors | Reverse Transcriptase Polymerase Chain Reaction | Telomere-Binding Proteins - antagonists & inhibitors | Blotting, Western | Sequence Homology, Amino Acid | Nuclear Envelope - metabolism | Animals | Cell Nucleus - genetics | Membrane Proteins - antagonists & inhibitors | Nuclear Proteins - antagonists & inhibitors | Cytoskeleton - metabolism | Spermatocytes - cytology | Mice | HeLa Cells | Chromosome Pairing | Chromosomes | Analysis | Dynein | Protein binding | Proteins | Membranes | Rodents | Zebrafish | Genomes | Cytoplasm | Index Medicus | s
Journal Article
Neuron, ISSN 0896-6273, 2009, Volume 64, Issue 2, pp. 173 - 187
Nuclear movement is critical during neurogenesis and neuronal migration, which are fundamental for mammalian brain development. Although dynein, Lis1, and... 
CELLBIO | DEVBIO | SKELETAL-MUSCLE | DYNEIN | MEMBRANE PROTEIN | ENVELOPE | DREIFUSS MUSCULAR-DYSTROPHY | LIS1 | CELL POLARIZATION | CENTRAL-NERVOUS-SYSTEM | ACTIN CYTOSKELETON | NEUROMUSCULAR-JUNCTION | NEUROSCIENCES | Cell Proliferation | Microtubule-Associated Proteins - metabolism | Nerve Tissue Proteins - deficiency | Neurons - cytology | Protein Transport - physiology | Cell Movement - genetics | Cell Movement - physiology | Neurogenesis - genetics | Exploratory Behavior - physiology | Membrane Proteins - deficiency | Cell Nucleus - metabolism | Neurons - ultrastructure | Nuclear Proteins - deficiency | Female | Membrane Proteins - metabolism | Bromodeoxyuridine - metabolism | Microtubule-Associated Proteins - deficiency | Telomere-Binding Proteins - metabolism | Dyneins - metabolism | Brain - cytology | Mice, Inbred C57BL | Cells, Cultured | Behavior, Animal - physiology | Mice, Transgenic | Nuclear Proteins - metabolism | Protein Structure, Tertiary - genetics | Telomere-Binding Proteins - deficiency | Electroporation - methods | Centrosome - metabolism | Nerve Tissue Proteins - metabolism | Pregnancy | Protein Interaction Mapping - methods | Animals | Neurogenesis - physiology | Mice | Proteins | Neurosciences | Neurons | Developmental biology | Dynein | Neurogenesis | Colleges & universities | Cell adhesion & migration | Index Medicus | Brain | nuclear envelope | UNC-84 | interkinetic nuclear movement | learning and memory | KASH domain | nucleokinesis
Journal Article
Journal Article
Cell, ISSN 0092-8674, 06/2013, Volume 153, Issue 6, pp. 1266 - 1280
Journal Article