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1. Full Text Tissue mechanics promote IDH1-dependent HIF1α-tenascin C feedback to regulate glioblastoma aggression
by Miroshnikova, Yekaterina A and Mouw, Janna K and Barnes, J. Matthew and Pickup, Michael W and Lakins, Johnathan N and Kim, Youngmi and Lobo, Khadjia and Persson, Anders I and Reis, Gerald F and McKnigh, Tracy R and Holland, Eric C and Phillips, Joanna J and Weaver, Valerie M
Nature Cell Biology, ISSN 1465-7392, 12/2016, Volume 18, Issue 12, pp. 1336 - 1345
Increased overall survival for patients with glioma brain tumours is associated with mutations in the metabolic regulator isocitrate dehydrogenase 1 (IDH1).... 
MALIGNANT GLIOMA | CELLS | GLIAL BRAIN-TUMORS | IDH1 | EXTRACELLULAR-MATRIX | MESENCHYMAL TRANSITION | HYPOXIA | EXPRESSION | CANCER | 2-HYDROXYGLUTARATE | CELL BIOLOGY | Signal Transduction | Neoplasm Invasiveness | Humans | Brain Neoplasms - pathology | Extracellular Matrix - metabolism | Isocitrate Dehydrogenase - genetics | MicroRNAs - metabolism | Mutation - genetics | Tenascin - metabolism | Brain Neoplasms - metabolism | Xenograft Model Antitumor Assays | Mechanotransduction, Cellular | Feedback, Physiological | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glioblastoma - pathology | Fluorescent Antibody Technique | Cell Line, Tumor | Glioblastoma - metabolism | Isocitrate Dehydrogenase - metabolism | Index Medicus
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2. Full Text VEGF-A and Tenascin-C produced by S100A4⁺ stromal cells are important for metastatic colonization
by Joyce T. O'Connell and Hikaru Sugimoto and Vesselina G. Cooke and Brian A. MacDonald and Ankit I. Mehta and Valerie S. LeBleu and Rajan Dewar and Rafael M. Rocha and Ricardo R. Brentani and Murray B. Resnick and Eric G. Neilson and Michael Zeisberg and Raghu Kalluri
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2011, Volume 108, Issue 38, pp. 16002 - 16007
Increased numbers of S100A4⁺ cells are associated with poor prognosis in patients who have cancer. Although the metastatic capabilities of S100A4⁺ cancer cells... 
Cell growth | Lungs | Stromal cells | Bone marrow | Fibroblasts | Breast cancer | Metastasis | Cancer | Tumors | Nodules | Metastasis-associated fibroblasts | Metastatic microenvironment | Stromal fibroblasts | Tumor microenvironment | BEVACIZUMAB | MULTIDISCIPLINARY SCIENCES | metastasis-associated fibroblasts | HUMAN BREAST-CANCER | stromal fibroblasts | metastatic microenvironment | MESSENGER-RNA | GENE | COLORECTAL-CANCER | P9KA | tumor microenvironment | PROGNOSTIC-SIGNIFICANCE | EXPRESSION | CALCIUM-BINDING PROTEIN | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Neoplasms - metabolism | Tenascin - genetics | Oligonucleotide Array Sequence Analysis | S100 Calcium-Binding Protein A4 | Humans | Male | Gene Expression Profiling | Green Fluorescent Proteins - genetics | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Tenascin - metabolism | Neoplasm Metastasis | S100 Proteins - genetics | Neoplasms - genetics | Stromal Cells - drug effects | Female | Ganciclovir - pharmacology | Extracellular Matrix Proteins - metabolism | Fibroblasts - metabolism | Tumor Microenvironment - drug effects | Green Fluorescent Proteins - metabolism | Thymidine Kinase - genetics | Extracellular Matrix Proteins - genetics | Stromal Cells - metabolism | Mice, Transgenic | S100 Proteins - metabolism | Mice, Knockout | Animals | Fibroblasts - drug effects | Thymidine Kinase - metabolism | Cell Line, Tumor | Mice | Neoplasms - pathology | Proteins | Rodents | Index Medicus | Biological Sciences
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3. Full Text Oct 4＋/Tenascin C＋ neuroblastoma cells serve as progenitors of tumor-derived endothelial cells
by Annalisa Pezzolo Federica Parodi Danilo MarimPietri Lizzia Raffaghello Claudia Cocco Angela Pistorio Manuela Mosconi Claudio Gambini Michele Cilli Silvia Deaglio Fabio Malavasi Vito Pistoia
细胞研究：英文版, ISSN 1001-0602, 2011, Volume 21, Issue 10, pp. 1470 - 1486
Neuroblastoma （NB）-associated endothelial microvesseis （EMs） may be lined by tumor-derived endothelial cells （TECs）, that are genetically unstable and... 
跨国公司 | 祖细胞 | 转录因子 | 血管内皮细胞 | 肿瘤细胞 | 无血清培养基 | 血管内皮生长因子 | 神经母细胞瘤 | neuroblastoma | progenitor cells | tumor-derived endothelium | Tenascin C | vascular mimicry | INITIATING CELLS | DIFFERENTIAL-DIAGNOSIS | SELF-RENEWAL | CANCER | CELL BIOLOGY | STEM-LIKE CELLS | GANGLIOSIDE GD2 | STEM/PROGENITOR CELLS | EXPRESSION | PERIVASCULAR NICHE | TENASCIN-C | Oncogene Proteins - genetics | Tenascin - genetics | Humans | Child, Preschool | Infant | Male | Transplantation, Heterologous | Tenascin - metabolism | Neovascularization, Pathologic - pathology | Octamer Transcription Factor-3 - genetics | Neoplasm Metastasis | Neoplastic Stem Cells - metabolism | Bone Marrow - metabolism | Neoplastic Stem Cells - pathology | Female | Cell Differentiation | N-Myc Proto-Oncogene Protein | Nuclear Proteins - genetics | Child | Neuroblastoma - pathology | Endothelial Cells - metabolism | Neuroblastoma - genetics | Oncogene Proteins - metabolism | Nuclear Proteins - metabolism | Drug Resistance, Neoplasm - genetics | Animals | Gene Amplification | Mice, Nude | Bone Marrow - pathology | Neuroblastoma - drug therapy | Octamer Transcription Factor-3 - metabolism | Mice | Neovascularization, Pathologic - metabolism | Neuroblastoma - metabolism | Endothelial Cells - pathology | Index Medicus | Original
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4. Full Text Staphylococcal superantigen-like protein 8 (SSL8) binds to tenascin C and inhibits tenascin C–fibronectin interaction and cell motility of keratinocytes
by Itoh, Saotomo and Yamaoka, Natsuko and Kamoshida, Go and Takii, Takemasa and Tsuji, Tsutomu and Hayashi, Hidetoshi and Onozaki, Kikuo
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 03/2013, Volume 433, Issue 1, pp. 127 - 132
► SSL8 bound to TNC. ► SSL8 inhibited TNC–FN interaction. ► SSL8 inhibited cell motility. Staphylococcal superantigen-like protein (SSL), a family of exotoxins... 
Tenascin C | Staphylococcal superantigen-like protein | Fibronectin | INDUCED DERMATITIS | MIGRATION | ATTACHMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | RECEPTOR | DEFICIENCY | ADHESION | BIOPHYSICS | EXPRESSION | REPEATS | DOMAINS | Tenascin - genetics | Humans | Molecular Sequence Data | Wound Healing | Tenascin - metabolism | Exotoxins - metabolism | Superantigens - metabolism | Base Sequence | Female | Protein Interaction Domains and Motifs | Milk, Human - metabolism | Staphylococcus aureus - metabolism | Recombinant Proteins - metabolism | Amino Acid Sequence | Cell Line | Recombinant Proteins - genetics | Staphylococcus aureus - pathogenicity | Fibronectins - metabolism | Keratinocytes - immunology | Protein Binding | Staphylococcus aureus - immunology | Fibronectins - genetics | Milk, Human - immunology | Cell Movement | Keratinocytes - physiology | Fibronectins | Physiological aspects | Fibrin | Epidermal growth factor | Fibrinogen | Protein binding | Index Medicus
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5. Full Text Tenascin-C in Cardiac Hypertrophy and Fibrosis: Friend or Foe?
by Park, Woo Jin and Jeong, Dongtak and Oh, Jae Gyun
Journal of the American College of Cardiology, ISSN 0735-1097, 09/2017, Volume 70, Issue 13, pp. 1616 - 1617
The protomeric heptad repeats are followed by multiple epidermal growth factor-like domains, a string of fibronectin type III repeats, and a carboxy-terminal... 
tenascin C | cardiac hypertrophy | cardiac fibrosis | pressure overload | angiotensin II | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | CARDIOMYOPATHY | Tenascin | Cardiomegaly | Inflammation | Bone Marrow | Humans | Fibrosis | Heart failure | Proteins | Heart attacks | Tenascin C | Cardiomyopathy | Rodents | Bone marrow | Extracellular matrix | Heart diseases | Hypertrophy | Index Medicus | Abridged Index Medicus
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6. Full Text Serum Tenascin-C as a Novel Predictor for Risk of Coronary Artery Lesion and Resistance to Intravenous Immunoglobulin in Kawasaki Disease　– A Multicenter Retrospective Study
by Okuma, Yoshiaki and Suda, Kenji and Nakaoka, Hideyuki and Katsube, Yasuhiro and Mitani, Yoshihide and Yoshikane, Yukako and Ichida, Fukiko and Matsushita, Takeji and Shichino, Hiroyuki and Shiraishi, Isao and Abe, Jun and Hiroe, Michiaki and Yoshida, Toshimichi and Imanaka-Yoshida, Kyoko
Circulation Journal, ISSN 1346-9843, 2016, Volume 80, Issue 11, pp. 2376 - 2381
Background:Tenascin-C (TN-C) is an extracellular matrix glycoprotein that is heavily upregulated at sites of inflammation. We conducted a retrospective study... 
Biomarkers | Kawasaki disease | Coronary artery lesions | Intravenous immunoglobulin (IVIG) resistance | Tenascin-C | DIAGNOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | BIOMARKER | PROGNOSIS | INTEGRIN | GAMMA-GLOBULIN | IMPACT | ANEURYSMS | RETREATMENT | Mucocutaneous Lymph Node Syndrome - drug therapy | Humans | Risk Factors | Child, Preschool | Immunoglobulins, Intravenous - administration & dosage | Infant | Male | Biomarkers - blood | Coronary Vessels - metabolism | Tenascin - blood | Female | Retrospective Studies | Child | Drug Resistance | Mucocutaneous Lymph Node Syndrome - blood | Index Medicus
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7. Full Text Amelioration of collagen antibody induced arthritis in mice by an antibody directed against the fibronectin type III repeats of tenascin-C: Targeting fibronectin type III repeats of tenascin-C in rheumatoid arthritis
by Mehta, Brij Bhushan and Tiwari, Anil and Sharma, Saniya and Shukla, Ashu and Sharma, Maryada and Vasishta, Rakesh K and Sen, Ramesh K and Sharma, Aman and Luthra-Guptasarma, Manni
International Immunopharmacology, ISSN 1567-5769, 05/2018, Volume 58, pp. 15 - 23
Tenascin-C (TN-C) levels are elevated in the synovial tissue and fluid, as well as cartilage of rheumatoid arthritis (RA) patients. In addition, the presence... 
Tenascin | Extracellular matrix | Inflammation | scFv antibody | Collagen antibody induced arthritis (CAIA) | Rheumatoid arthritis | Fibronectins | Rheumatoid factor | Arthritis | Collagen | Integrins | Immunoglobulins | Tenascin C | Destruction | Cytokines | Pathogenesis | Migration | Collagens | Patients | Fibronectin | Cartilage | Synovial fluid | Rodents | Fibrosis | Fibroblasts | Mice | Cell migration | Index Medicus
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8. Full Text Tenascin-C, a prognostic determinant of esophageal Squamous cell carcinoma
by Yang, Zhao-Ting and Yeo, So-Young and Yin, Yong-Xue and Lin, Zhen-Hua and Lee, Hak-Min and Xuan, Yan-Hua and Cui, Yan and Kim, Seok-Hyung
PLoS ONE, ISSN 1932-6203, 01/2016, Volume 11, Issue 1, pp. e0145807 - e0145807
Background Tenascin-C, an adhesion modulatory extracellular matrix molecule, is highly expressed in numerous human malignancies; thus, it may contribute to... 
EXPRESSION | MULTIDISCIPLINARY SCIENCES | GROWTH | Immunohistochemistry | Prognosis | Biomarkers, Tumor - analysis | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Kaplan-Meier Estimate | Proportional Hazards Models | Tenascin - analysis | Male | Fibroblasts - pathology | Esophageal Neoplasms - pathology | Carcinoma, Squamous Cell - diagnosis | Disease-Free Survival | Esophagus - pathology | Cell Line, Tumor | Esophageal Neoplasms - diagnosis | Female | Aged | Tumor Cells, Cultured | Esophageal Squamous Cell Carcinoma | Tenascin | Correlation | Platelet-derived growth factor | Lung cancer | Smooth muscle | Metastasis | Tissues | Macrophages | Carcinogenesis | Lymph nodes | Western blotting | Metastases | Proteins | Ethics | Carcinogens | Regression models | Actin | Fibroblasts | Extracellular matrix | Growth factors | Squamous cell carcinoma | Tenascin C | Committees | Muscles | Breast cancer | Regression analysis | Epithelium | Survival | Patients | Fibroblast activation protein | Esophagus | Studies | Medical prognosis | Isoforms | Cell lines | Hypoxia | Mutation | Esophageal cancer | Cancer | Tumors | Index Medicus
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9. Full Text PR336: The role of Prx2 transcription factor, tenascin‐C and TGF‐β1 in promoting scarless healing ability οf free soft tissue gingival grafts
Journal of Clinical Periodontology, ISSN 0303-6979, 06/2018, Volume 45, Issue S19, pp. 233 - 233
Tenascin
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10. Full Text The role of tenascin-C in tissue injury and tumorigenesis
by Midwood, Kim S and Orend, Gertraud
Journal of Cell Communication and Signaling, ISSN 1873-9601, 2009, Volume 3, Issue 3-4, pp. 287 - 310
The extracellular matrix molecule tenascin-C is highly expressed during embryonic development, tissue repair and in pathological situations such as chronic... 
Angiogenesis | Signaling | Wound healing | Oncogene | Cytokine | Tenascin-C | Extracellular matrix | Tumor | Arthritis | Inflammation | Fibronectin | Cancer | CELL BIOLOGY | Fibronectins | Heart failure | Embryonic development | Care and treatment | Universities and colleges | Tumors | Tenascin | Leukocyte migration | Tenascin C | Cytokines | C protein | Clinical trials | Drug delivery | Cell surface | Cell adhesion & migration | Embryogenesis | Tumorigenesis | Heart diseases | Cell migration
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11. Full Text MR targeted imaging for the expression of tenascin‐C in myocardial infarction in vivo
by Song, Jiacheng and Yu, Jing and Li, Yan and Lu, Shanshan and Ma, Zhanlong and Shi, Haibin
Journal of Magnetic Resonance Imaging, ISSN 1053-1807, 06/2017, Volume 45, Issue 6, pp. 1668 - 1674
PurposeTo investigate the presence of viable myocardium in mice with acute myocardial infarction (MI) using a molecular targeted probe. Materials and... 
Tenascin‐C | magnetic resonance image | SPIO | myocardial infarction | Tenascin-C | SALVAGE | THERAPY | TISSUE | MAGNETIC-RESONANCE | RISK | MODEL | INTERVENTION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Biomarkers - metabolism | Reproducibility of Results | Antibodies, Monoclonal - pharmacokinetics | Contrast Media - pharmacokinetics | Magnetic Resonance Imaging - methods | Molecular Imaging - methods | Myocardial Infarction - diagnostic imaging | Myocardial Infarction - metabolism | Tenascin - metabolism | Mice, Inbred ICR | Dextrans - pharmacokinetics | Myocardial Infarction - complications | Myocardial Stunning - metabolism | Animals | Sensitivity and Specificity | Female | Mice | Myocardial Stunning - etiology | Myocardial Stunning - diagnostic imaging | Magnetite Nanoparticles | Viral antibodies | Iron compounds | Magnetic resonance imaging | RNA | Ferric oxide | Antibodies | Heart attack | Nanoparticles | Tenascin | Myocardial infarction | Heart attacks | Tenascin C | Myocardium | Cardiomyocytes | Iron | Iron oxides | Gene expression | Index Medicus
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12. Full Text Breast cancer cells produce tenascin C as a metastatic niche component to colonize the lungs
by Oskarsson, Thordur and Acharyya, Swarnali and Zhang, Xiang H.-F and Vanharanta, Sakari and Tavazoie, Sohail F and Morris, Patrick G and Downey, Robert J and Manova-Todorova, Katia and Brogi, Edi and Massagué, Joan
Nature Medicine, ISSN 1078-8956, 07/2011, Volume 17, Issue 7, pp. 867 - 874
We report that breast cancer cells that infiltrate the lungs support their own metastasis-initiating ability by expressing tenascin C (TNC). We find that the... 
SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | SIGNALING PATHWAYS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NOTCH | FATE | CELL BIOLOGY | GROWTH | GENE-EXPRESSION | EXTRACELLULAR-MATRIX | PROGRESSION | IN-VITRO PROPAGATION | Receptors, G-Protein-Coupled - metabolism | STAT5 Transcription Factor - physiology | Tenascin - physiology | Receptors, Notch - physiology | Wnt Proteins - metabolism | Breast Neoplasms - physiopathology | Breast Neoplasms - metabolism | Animals | Lung Neoplasms - secondary | Breast Neoplasms - pathology | Female | Signal Transduction - physiology | Mice | Apoptosis - physiology | Gene Expression Regulation, Neoplastic - physiology | Physiological aspects | Breast cancer | Research | Lungs | Cancer cells | Metastasis | Tumors | Index Medicus
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