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1. Full Text CD4+ T-cell subsets in inflammatory diseases: Beyond the Th1/Th2 paradigm
by Hirahara, Kiyoshi and Nakayama, Toshinori
International immunology, ISSN 0953-8178, 04/2016, Volume 28, Issue 4, pp. 163 - 171
memory T | T-cell plasticity | pathogenic T | 2 cells | 1/T | 2 (T | 2 paradigm | epigenetics | 2) cells | T-Lymphocyte Subsets - immunology | Inflammation - microbiology | Epigenesis, Genetic | Humans | CD4-Positive T-Lymphocytes - metabolism | Gene Expression Regulation | Inflammation - immunology | Th2 Cells - immunology | Th1 Cells - immunology | Th2 Cells - metabolism | Host-Pathogen Interactions - immunology | CD4-Positive T-Lymphocytes - immunology | Th1 Cells - metabolism | Inflammation - metabolism | Animals | Host-Pathogen Interactions - genetics | T-Lymphocyte Subsets - metabolism | Inflammation - genetics | Immunologic Memory | Index Medicus | memory Th2 cells | pathogenic Th2 (Tpath2) cells | Th2 paradigm | Th1 | Invited Review
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2. Full Text CD301b+ Dermal Dendritic Cells Drive T Helper 2 Cell-Mediated Immunity
by Kumamoto, Yosuke and Linehan, Melissa and Weinstein, Jason S and Laidlaw, Brian J and Craft, Joseph E and Iwasaki, Akiko
Immunity (Cambridge, Mass.), ISSN 1074-7613, 10/2013, Volume 39, Issue 4, pp. 733 - 743
Life Sciences & Biomedicine | Immunology | Science & Technology | Ovalbumin - immunology | Dendritic Cells - immunology | Lectins, C-Type - immunology | Dendritic Cells - pathology | Th2 Cells - immunology | Nippostrongylus - immunology | Lectins, C-Type - genetics | Th2 Cells - drug effects | Th2 Cells - parasitology | Allergens - immunology | Strongylida Infections - parasitology | Strongylida Infections - immunology | Dendritic Cells - drug effects | Cell Differentiation | Interleukin-4 - genetics | Skin - pathology | Th2 Cells - pathology | Skin - immunology | Dendritic Cells - parasitology | Signal Transduction | Gene Expression Regulation | Skin - parasitology | Mice, Transgenic | Strongylida Infections - pathology | Interleukin-4 - immunology | Animals | Ovalbumin - administration & dosage | Mice | Alum Compounds - administration & dosage | Immunity, Cellular | Skin - drug effects | Proteins | Antigens | Lymphocytes | Rheumatic diseases | Software | Metabolic disorders | Immune system | Index Medicus | dendritic cell subset | T cell priming | Th2
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3. Full Text The pro‐Th2 cytokine IL‐33 directly interacts with invariant NKT and NK cells to induce IFN‐γ production
by Bourgeois, Elvire and Van, Linh Pham and Samson, Michel and Diem, Séverine and Barra, Anne and Roga, Stéphane and Gombert, Jean‐Marc and Schneider, Elke and Dy, Michel and Gourdy, Pierre and Girard, Jean‐Philippe and Herbelin, André
European journal of immunology, ISSN 0014-2980, 04/2009, Volume 39, Issue 4, pp. 1046 - 1055
Th1/Th2 cells | Inflammation | Natural killer cells | Cytokines | Natural killer T cells | Life Sciences & Biomedicine | Immunology | Science & Technology | Natural Killer T-Cells - drug effects | Spleen - immunology | Humans | Spleen - drug effects | Th2 Cells - immunology | Interferon-gamma - metabolism | Interleukin-4 - agonists | Th2 Cells - drug effects | Liver - immunology | Liver - drug effects | Natural Killer T-Cells - metabolism | Interleukins - immunology | Killer Cells, Natural - immunology | Female | Interleukins - pharmacology | Interleukin-33 | Interleukin-4 - metabolism | Liver - metabolism | Mice, Inbred C57BL | Interleukin-12 - metabolism | Interferon-gamma - agonists | Th2 Cells - metabolism | Mice, Knockout | Interleukin-4 - immunology | Animals | Spleen - metabolism | Interferon-gamma - immunology | Interleukin-12 - immunology | Killer Cells, Natural - drug effects | Mice | Mice, Inbred BALB C | Killer Cells, Natural - metabolism | Natural Killer T-Cells - immunology | Index Medicus
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4. Full Text IL‐33‐activated dendritic cells are critical for allergic airway inflammation
by Besnard, Anne‐Gaëlle and Togbe, Dieudonnée and Guillou, Noëlline and Erard, François and Quesniaux, Valérie and Ryffel, Bernhard
European journal of immunology, ISSN 0014-2980, 06/2011, Volume 41, Issue 6, pp. 1675 - 1686
Allergy | IL‐33 | Lung inflammation | Cytokines | DC | IL-33 | Life Sciences & Biomedicine | Immunology | Science & Technology | Interleukin-1 Receptor-Like 1 Protein | Pneumonia | Dendritic Cells - immunology | Th1-Th2 Balance | Dendritic Cells - pathology | Th2 Cells - immunology | Antigens, CD - genetics | Interleukins - metabolism | Antigens, CD - metabolism | Receptors, Interleukin - metabolism | Interleukins - immunology | Cell Differentiation | Lung - metabolism | Cytokines - genetics | Dendritic Cells - metabolism | Th2 Cells - pathology | Lung - pathology | Cytokines - metabolism | Receptors, Interleukin - immunology | Interleukin-33 | Lymphocyte Activation | Cells, Cultured | Myeloid Progenitor Cells - pathology | Respiratory Hypersensitivity - immunology | Receptors, Interleukin - genetics | Th2 Cells - metabolism | Mice, Knockout | Animals | Mice | Lung - immunology | Cell Movement | Rodents | Index Medicus | Dendritic Cells | Lung | Receptors, Interleukin | Myeloid Progenitor Cells | Th2 Cells | Life Sciences | Interleukins | Respiratory Hypersensitivity | Antigens, CD
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5. Full Text Asthma phenotypes and the use of biologic medications in asthma and allergic disease: The next steps toward personalized care
by Fajt, Merritt L., MD and Wenzel, Sally E., MD
Journal of allergy and clinical immunology, ISSN 0091-6749, 2014, Volume 135, Issue 2, pp. 299 - 310
Allergy and Immunology | Asthma phenotypes | biologic therapies | IgE | IL-4 | eosinophils | IL-5 | TH2/type 2 inflammation | IL-13 | 2/type 2 inflammation | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Asthma - metabolism | Eosinophils - metabolism | Eosinophils - drug effects | Anti-Asthmatic Agents - therapeutic use | Humans | Hypersensitivity - immunology | Th2 Cells - immunology | Eosinophils - immunology | Hypersensitivity - drug therapy | Th2 Cells - drug effects | Immunoglobulin E - immunology | Hypersensitivity - metabolism | Asthma - immunology | Hypersensitivity - diagnosis | Hypersensitivity - genetics | Cytokines - genetics | Neutrophils - metabolism | Precision Medicine | Asthma - diagnosis | Biomarkers - metabolism | Anti-Asthmatic Agents - pharmacology | Pharmacogenetics | Cytokines - metabolism | Neutrophils - drug effects | Neutrophils - immunology | Asthma - drug therapy | Th2 Cells - metabolism | Asthma - genetics | Biological Factors - therapeutic use | Phenotype | Signal Transduction - drug effects | Cytokines - antagonists & inhibitors | Biological Factors - pharmacology | Analysis | Immunoglobulin E | Respiratory agents | Genetic aspects | Drug therapy | Asthma | Studies | Medical research | Food allergies | Genotype & phenotype | Disease | Cytokines | Biomarkers | Bone marrow | Accreditation | Disclosure | Allergies | Index Medicus | Abridged Index Medicus
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6. Full Text Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells
by Bettelli, Estelle and Carrier, Yijun and Gao, Wenda and Korn, Thomas and Strom, Terry B and Oukka, Mohamed and Weiner, Howard L and Kuchroo, Vijay K
Nature, ISSN 0028-0836, 05/2006, Volume 441, Issue 7090, pp. 235 - 238
Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Cell physiology | Molecular and cellular biology | Cell differentiation, maturation, development, hematopoiesis | Th1 Cells - drug effects | Central Nervous System - metabolism | Cytokines - metabolism | Receptors, Interleukin-2 - metabolism | Th2 Cells - cytology | Th2 Cells - immunology | Interleukin-17 - biosynthesis | Th1 Cells - immunology | Th2 Cells - drug effects | Th2 Cells - metabolism | Central Nervous System - immunology | Th1 Cells - metabolism | Interleukin-17 - metabolism | Animals | Autoimmunity - immunology | Cell Differentiation - drug effects | Forkhead Transcription Factors - metabolism | Interleukin-6 - immunology | Mice | Th1 Cells - cytology | Interleukin-6 - metabolism | Transforming Growth Factor beta - metabolism | CD4 Antigens - metabolism | Cytokines - immunology | Immunology | Cytokines | Cellular biology | Rodents | Bacteria | T cell receptors | Autoimmune diseases | Inflammatory diseases | Index Medicus
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7. Full Text Differential glycosylation of TH1, TH2 and TH-17 effector cells selectively regulates susceptibility to cell death
by Toscano, Marta A and Bianco, Germán A and Ilarregui, Juan M and Croci, Diego O and Correale, Jorge and Hernandez, Joseph D and Zwirner, Norberto W and Poirier, Francoise and Riley, Eleanor M and Baum, Linda G and Rabinovich, Gabriel A
Nature immunology, ISSN 1529-2908, 08/2007, Volume 8, Issue 8, pp. 825 - 834
Th1 Cells - pathology | Galectin 1 - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Membrane Glycoproteins - metabolism | T-Lymphocyte Subsets - cytology | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Galectin 1 - genetics | Galectin 1 - metabolism | Immunoblotting | Adoptive Transfer | Th1 Cells - metabolism | Inflammation - metabolism | Flow Cytometry | T-Lymphocytes, Helper-Inducer - cytology | T-Lymphocytes, Helper-Inducer - pathology | Membrane Glycoproteins - immunology | Th2 Cells - pathology | In Situ Nick-End Labeling | T-Lymphocytes, Helper-Inducer - metabolism | Th2 Cells - cytology | Glycosylation | Inflammation - immunology | Polysaccharides - immunology | Th2 Cells - metabolism | Polysaccharides - metabolism | Cell Differentiation - immunology | Interleukin-17 - metabolism | Animals | Apoptosis - immunology | T-Lymphocyte Subsets - pathology | T-Lymphocyte Subsets - metabolism | Mice | Th1 Cells - cytology | Physiological aspects | Genetic aspects | Research | DNA binding proteins | T cells | Apoptosis | Index Medicus | T-Lymphocyte Subsets | T-Lymphocytes, Helper-Inducer | Inflammation | Th2 Cells | Life Sciences | Encephalomyelitis, Autoimmune, Experimental | Polysaccharides | Immunology | Th1 Cells | Cell Differentiation | Development Biology | Interleukin-17 | Membrane Glycoproteins | Galectin 1
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