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1. Full Text The anthocyanin cyanidin-3- O-β-glucoside, a flavonoid, increases hepatic glutathione synthesis and protects hepatocytes against reactive oxygen species during hyperglycemia: Involvement of a cAMP–PKA-dependent signaling pathway
by Zhu, Wei and Jia, Qianju and Wang, Yun and Zhang, Yuhua and Xia, Min
Free Radical Biology and Medicine, ISSN 0891-5849, 2012, Volume 52, Issue 2, pp. 314 - 327
Enhanced oxidative stress due to high glucose contributes to pathological changes in diabetes-related liver complications. Reducing oxidative stress may... 
Anthocyanin | CREB | Free radicals | GSH | PKA | OXIDATIVE STRESS | METABOLIC SYNDROME | RISK-FACTORS | FATTY LIVER-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HUMAN ENDOTHELIAL-CELLS | RESPONSE ELEMENT | INSULIN-RESISTANCE | CELLULAR GLUTATHIONE | ENDOCRINOLOGY & METABOLISM | GLUTAMATE-CYSTEINE LIGASE | SUBUNIT GENE-EXPRESSION | Up-Regulation | Liver - pathology | Reactive Oxygen Species - metabolism | Oxidative Stress | Humans | Male | Hepatocytes - metabolism | fas Receptor - metabolism | Hyperglycemia - drug therapy | Glutathione - biosynthesis | MAP Kinase Kinase 4 - metabolism | fas Receptor - genetics | Anthocyanins - therapeutic use | Glucosides - therapeutic use | Glutamate-Cysteine Ligase - metabolism | Cyclic AMP - metabolism | Hepatocytes - drug effects | Cytoprotection | Cyclic AMP-Dependent Protein Kinases - metabolism | Cell Survival - drug effects | Anthocyanins - pharmacology | Gene Expression | Glucosides - pharmacology | Signal Transduction | Liver - metabolism | Fatty Liver - prevention & control | Thiobarbituric Acid Reactive Substances - metabolism | Antioxidants - pharmacology | Enzyme Activation - drug effects | Hep G2 Cells | Hyperglycemia - metabolism | Antioxidants - therapeutic use | Animals | Cyclic AMP Response Element-Binding Protein - metabolism | Protein Binding | Glutamate-Cysteine Ligase - genetics | Mice, Obese | Mice | Apoptosis | Index Medicus
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2. Full Text Cerium oxide nanoparticles modify the antioxidative stress enzyme activities and macromolecule composition in rice seedlings
by Rico, Cyren M and Morales, Maria I and McCreary, Ricardo and Castillo-Michel, Hiram and Barrios, Ana C and Hong, Jie and Tafoya, Alejandro and Lee, Wen-Yee and Varela-Ramirez, Armando and Peralta-Videa, Jose R and Gardea-Torresdey, Jorge L
Environmental Science and Technology, ISSN 0013-936X, 12/2013, Volume 47, Issue 24, pp. 14110 - 14118
Cerium oxide nanoparticles (nCeO2) have been shown to have significant interactions in plants; however, there are limited reports on their impacts in rice... 
Nanoparticles - chemistry | Oxidation-Reduction | Oxidative Stress | Cerium - chemistry | Oryza - metabolism | Antioxidants - metabolism | Stress, Physiological | Electrolytes - metabolism | Thiobarbituric Acid Reactive Substances - metabolism | Seedlings - enzymology | Oryza - enzymology | Amylose - metabolism | Hydrogen Peroxide - metabolism | Cerium - metabolism | Oryza - growth & development | Lignin - metabolism | Hydrogen Peroxide - analysis | Synchrotrons | Spectrometry, X-Ray Emission | Plant Roots - enzymology | Lipid Peroxidation | Fatty Acids - metabolism | Macromolecular Substances - metabolism | Seedlings - metabolism | Index Medicus
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3. Full Text Dietary polyunsaturated fatty acids and heme iron induce oxidative stress biomarkers and a cancer promoting environment in the colon of rats
by Guéraud, Françoise and Taché, Sylviane and Steghens, Jean-Paul and Milkovic, Lidija and Borovic-Sunjic, Suzana and Zarkovic, Neven and Gaultier, Eric and Naud, Nathalie and Héliès-Toussaint, Cécile and Pierre, Fabrice and Priymenko, Nathalie
Free Radical Biology and Medicine, ISSN 0891-5849, 06/2015, Volume 83, pp. 192 - 200
The end products of polyunsaturated fatty acid (PUFA) peroxidation, such as malondialdehyde (MDA), 4-hydroxynonenal (HNE), and isoprostanes (8-iso-PGF ), are... 
4-Hydroxynonenal | Polyunsaturated fatty acids | Colorectal cancer | Lipid peroxidation | Biomarkers | Heme iron | Malondialdehyde | LIPID-PEROXIDATION | COLORECTAL CARCINOGENESIS | HUMAN PLASMA | SOYBEAN OIL | TOXIC ALDEHYDE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MERCAPTURIC ACID | URINARY METABOLITE | ANIMAL-TISSUES | IN-VIVO | ENDOCRINOLOGY & METABOLISM | BEEF MEAT | Biomarkers - urine | Heme - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Diet - adverse effects | Antioxidants - metabolism | Rats, Inbred F344 | Colon - drug effects | Tumor Microenvironment | Colonic Neoplasms - metabolism | Lipid Peroxidation - drug effects | Female | Rectal Neoplasms - pathology | Tumor Cells, Cultured | Rectal Neoplasms - etiology | Colonic Neoplasms - etiology | Malondialdehyde - metabolism | Colon - pathology | Rats | Thiobarbituric Acid Reactive Substances - metabolism | Iron - metabolism | Colon - metabolism | Animals | Colonic Neoplasms - pathology | Fatty Acids, Unsaturated - adverse effects | Mice | Rectal Neoplasms - metabolism | Oxidative stress | Unsaturated fatty acids | Metabolites | Heme | Xenobiotics | Biological markers | Meat | Index Medicus | Life Sciences
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4. Full Text Thiobarbituric acid reactive substances (TBARS) is a state biomarker of oxidative stress in bipolar patients in a manic phase
by Tsai, Meng-Chang and Huang, Tiao-Lai
Journal of Affective Disorders, ISSN 0165-0327, 2014, Volume 173, pp. 22 - 26
Abstract Objectives Oxidative stress may contribute to the pathophysiology of bipolar disorder. The aim of this study was to investigate the serum levels or... 
Psychiatry | Mania | Bipolar | PCC | SOD | TBARS | GPx | LIPID-PEROXIDATION | PSYCHIATRY | DNA-DAMAGE | ANTIOXIDANT ENZYMES | SCHIZOPHRENIA | MECHANISMS | CLINICAL NEUROLOGY | TEARS | REDOX REGULATION | MOOD DISORDERS | NITRIC-OXIDE | ANIMAL-MODEL | SUPEROXIDE-DISMUTASE | Protein Carbonylation | Catalase - blood | Oxidative Stress | Glutathione Peroxidase - blood | Humans | Superoxide Dismutase - blood | Male | Thiobarbituric Acid Reactive Substances - metabolism | Biomarkers - blood | Bipolar Disorder - blood | Young Adult | Adult | Female | Oxidative stress | Bipolar disorder | Superoxide
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5. Full Text Elevated serum superoxide dismutase and thiobarbituric acid reactive substances in different phases of bipolar disorder and in schizophrenia
by Kunz, Maurício and Gama, Clarissa Severino and Andreazza, Ana Cristina and Salvador, Mirian and Ceresér, Keila Mendes and Gomes, Fabiano Alves and Belmonte-de-Abreu, Paulo Silva and Berk, Michael and Kapczinski, Flavio
Progress in Neuropsychopharmacology & Biological Psychiatry, ISSN 0278-5846, 2008, Volume 32, Issue 7, pp. 1677 - 1681
There is an increasing body of evidence suggesting that oxidative stress may play a role in the pathophysiology of both schizophrenia (SZ) and bipolar disorder... 
Lipid peroxidation | Schizophrenia | Oxidative stress | Bipolar disorder | Superoxide dismutase | LIPID-PEROXIDATION | PSYCHIATRY | DNA-DAMAGE | ANTIOXIDANT ENZYMES | FATTY-ACIDS | schizophrenia | lipid peroxidation | NEUROSCIENCES | CLINICAL NEUROLOGY | bipolar disorder | superoxide dismutase | NITRIC-OXIDE | MITOCHONDRIAL DYSFUNCTION | PHARMACOLOGY & PHARMACY | RATING-SCALE | EXPRESSION | oxidative stress | INCREASED OXIDATIVE STRESS | BRAIN | Analysis of Variance | Schizophrenia - blood | Humans | Middle Aged | Superoxide Dismutase - blood | Adult | Female | Male | Thiobarbituric Acid Reactive Substances - metabolism | Bipolar Disorder - blood | Lipid Peroxidation - physiology | Superoxide
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6. Full Text Increased oxidative stress in obesity and its impact on metabolic syndrome
by Furukawa, Shigetada and Fujita, Takuya and Shimabukuro, Michio and Iwaki, Masanori and Yamada, Yukio and Nakajima, Yoshimitsu and Nakayama, Osamu and Makishima, Makoto and Matsuda, Morihiro and Shimomura, Iichiro
The Journal of clinical investigation, ISSN 0021-9738, 12/2004, Volume 114, Issue 12, pp. 1752 - 1761
Obesity is a principal causative factor in the development of metabolic syndrome. Here we report that increased oxidative stress in accumulated fat is an... 
Body Weight | Reactive Oxygen Species | Antioxidants - chemistry | Oxidative Stress | Antioxidants - metabolism | Humans | Middle Aged | NADPH Oxidases - metabolism | Male | Metabolic Syndrome - metabolism | RNA, Messenger - metabolism | Immunoblotting - methods | Dose-Response Relationship, Drug | Hydrogen Peroxide - chemistry | Adipose Tissue - metabolism | Time Factors | Female | Cell Differentiation | Cytokines - metabolism | Oxidation-Reduction | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Thiobarbituric Acid Reactive Substances - metabolism | 3T3-L1 Cells | Reverse Transcriptase Polymerase Chain Reaction | Hydrogen Peroxide - metabolism | Obesity - metabolism | Obesity - pathology | Animals | Models, Biological | Adipocytes - metabolism | Mice, Obese | Aged | Mice | Metabolic Syndrome - pathology | Lipid Peroxidation
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7. Full Text Serum levels of thiobarbituric acid reactive substances predict cardiovascular events in patients with stable coronary artery disease: A longitudinal analysis of the PREVENT study
by Walter, Mary F and Jacob, Robert F and Jeffers, Barrett and Ghadanfar, Mathieu M and Preston, Gregory M and Buch, Jan and Mason, R. Preston and PREVENT study
Journal of the American College of Cardiology, ISSN 0735-1097, 11/2004, Volume 44, Issue 10, pp. 1996 - 2002
The objective of this study was to test the predictive value of an oxidative stress biomarker in 634 patients from the Prospective Randomized Evaluation of the... 
OXIDATIVE MODIFICATION HYPOTHESIS | CARDIAC & CARDIOVASCULAR SYSTEMS | CAROTID ATHEROSCLEROSIS | INCREASED PLASMA | LIPID-PEROXIDATION | ELEVATED LEVELS | LOW-DENSITY-LIPOPROTEIN | OXIDIZED LDL | SMOOTH-MUSCLE-CELLS | PEROXIDATION IN-VIVO | MALONDIALDEHYDE LEVELS | Myocardial Infarction - diagnosis | Myocardial Infarction - blood | Predictive Value of Tests | Myocardial Infarction - mortality | Humans | Middle Aged | Risk Factors | Male | Thiobarbituric Acid Reactive Substances - metabolism | Biomarkers - blood | Coronary Artery Disease | Randomized Controlled Trials as Topic | Sensitivity and Specificity | Aged, 80 and over | Adult | Female | Aged | Lipids | Heart attacks | Drug therapy | Angioplasty | Cholesterol | Chromatography
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8. Full Text Salicylic acid alleviates adverse effects of heat stress on photosynthesis through changes in proline production and ethylene formation
by Khan, M Iqbal R and Iqbal, Noushina and Masood, Asim and Per, Tasir S and Khan, Nafees A
Plant Signaling & Behavior, ISSN 1559-2316, 11/2013, Volume 8, Issue 11, p. e26374
We investigated the potential of salicylic acid (SA) in alleviating the adverse effects of heat stress on photosynthesis in wheat (Triticum aestivum L.) cv WH... 
proline | heat stress | salicylic acid | photosynthesis | ethylene | oxidative stress | Proline | Oxidative stress | Salicylic acid | Heat stress | Photosynthesis | Ethylene | BIOCHEMISTRY & MOLECULAR BIOLOGY | PLANT SCIENCES | Heat-Shock Response - drug effects | Nitrate Reductase - metabolism | Proline - metabolism | Triticum - physiology | Fluorescence | Phosphotransferases (Carboxyl Group Acceptor) - metabolism | Ethylenes - metabolism | Triticum - metabolism | Salicylic Acid - pharmacology | Plant Stomata - drug effects | Plant Leaves - drug effects | Proline Oxidase - metabolism | Triticum - enzymology | Plant Leaves - enzymology | Nitrogen - metabolism | Carbon Dioxide - metabolism | Water - metabolism | Thiobarbituric Acid Reactive Substances - metabolism | Chlorophyll - metabolism | Osmosis - drug effects | Hydrogen Peroxide - metabolism | Ribulose-Bisphosphate Carboxylase - metabolism | Photosynthesis - drug effects | Oxidative Stress - drug effects | Plant Stomata - physiology | Plant Leaves - physiology | Triticum - drug effects
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9. Full Text Reactive Oxygen Species-Induced Oxidative Stress in the Development of Insulin Resistance and Hyperandrogenism in Polycystic Ovary Syndrome
by González, Frank and Rote, Neal S and Minium, Judi and Kirwan, John P
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 01/2006, Volume 91, Issue 1, pp. 336 - 340
Context: Insulin resistance and chronic low level inflammation are often present in women with polycystic ovary syndrome (PCOS). Objective: The purpose of this... 
DIABETES-MELLITUS | WOMEN | LEUKOCYTES | ENDOCRINOLOGY & METABOLISM | MONONUCLEAR-CELLS | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | GENERATION | OBESE | THECA-INTERSTITIAL CELLS | ADIPOSE-TISSUE | NADPH Oxidases | Phosphoproteins - biosynthesis | Polycystic Ovary Syndrome - metabolism | Reactive Oxygen Species | Area Under Curve | Humans | Oxidative Stress - physiology | Androstenedione - blood | Monocytes - metabolism | Thiobarbituric Acid Reactive Substances - metabolism | Obesity - metabolism | Hyperglycemia - metabolism | Insulin Resistance - physiology | Lipid Peroxidation - drug effects | Glucose - metabolism | Adult | Female | Hyperandrogenism - metabolism | Testosterone - blood | Body Weight - physiology | Waist-Hip Ratio | Body Composition - physiology
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10. Full Text Chronic Restraint Stress Inhibits Hair Growth via Substance P Mediated by Reactive Oxygen Species in Mice
by Liu, Nan and Wang, Lin-Hui and Guo, Ling-Ling and Wang, Guo-Qing and Zhou, Xi-Ping and Jiang, Yan and Shang, Jing and Murao, Koji and Chen, Jing-Wei and Fu, Wen-Qing and Zhang, Guo-Xing
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e61574
Backgrounds: Solid evidence has demonstrated that psychoemotional stress induced alteration of hair cycle through neuropeptide substance P (SP) mediated immune... 
OXIDATIVE STRESS | LIPID-PEROXIDATION | MULTIDISCIPLINARY SCIENCES | MAST-CELLS | CATAGEN DEVELOPMENT | MOUSE MODEL | TISSUE-DAMAGE | SKIN-DISEASE | IMMOBILIZATION STRESS | FOLLICLE | TELOGEN STAGE | Reactive Oxygen Species - metabolism | Hair - metabolism | Male | Dermis - drug effects | Hair Follicle - pathology | Stress, Psychological - metabolism | Mast Cells - metabolism | Receptors, Neurokinin-1 - metabolism | Restraint, Physical | Weight Gain - drug effects | Corticosterone - blood | Neurokinin-1 Receptor Antagonists | Superoxide Dismutase - metabolism | Dermis - enzymology | Glutathione Peroxidase - metabolism | Stress, Psychological - blood | Mice, Inbred C57BL | Thiobarbituric Acid Reactive Substances - metabolism | Antioxidants - pharmacology | Hair Follicle - drug effects | Stress, Psychological - pathology | Animals | Hair Follicle - growth & development | Dermis - pathology | Substance P - metabolism | Hair - drug effects | Mice | Oxidative Stress - drug effects | Chronic Disease | Hair - growth & development | Antioxidants | Skin | Cellular signal transduction | Superoxide | Growth | Analysis | Brain | Regulations | Oxidative stress | Lipid peroxidation | Tempol | Lipids | Superoxide dismutase | Activation | Dermatitis | Cell activation | Rodents | Skin diseases | Physiology | Peroxidase | Clinical psychology | Glutathione | Peroxidation | Immune system | Hair | Psychological stress | Hypertension | Glutathione peroxidase | Enzymes | Oxygen | Immune response | Psoriasis | Baldness | Substance P | Inhibition (psychology) | Emotions | Laboratory animals | Hair loss
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11. Full Text Quercetin treatment ameliorates inflammation and fibrosis in mice with nonalcoholic steatohepatitis
by Marcolin, Eder and San-Miguel, Beatriz and Vallejo, Daniela and Tieppo, Juliana and Marroni, Norma and González-Gallego, Javier and Tuñón, María J
Journal of Nutrition, ISSN 0022-3166, 10/2012, Volume 142, Issue 10, pp. 1821 - 1828
We investigated whether quercetin protects from steatosis and limits the expression of proinflammatory and fibrogenic genes in C57BL/6J mice with nonalcoholic... 
GREEN TEA | CELLS | KAPPA-B ACTIVATION | NUTRITION & DIETETICS | REACTIVE OXYGEN | FATTY LIVER-DISEASE | GENE-EXPRESSION | RATS | FLAVONOIDS | MODEL | RECEPTOR 4 | Inflammation - pathology | Up-Regulation | Fibrosis - drug therapy | Liver - pathology | Fatty Liver - pathology | Collagen Type III - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | HMGB1 Protein - genetics | Collagen Type I - genetics | Cyclooxygenase 2 - genetics | Inflammation - drug therapy | HMGB1 Protein - metabolism | Lipid Peroxidation - drug effects | Non-alcoholic Fatty Liver Disease | Thiobarbituric Acid Reactive Substances - analysis | JNK Mitogen-Activated Protein Kinases - genetics | Interleukin-6 - metabolism | Disease Models, Animal | Methionine - deficiency | Collagen Type I - metabolism | Interleukin-6 - genetics | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Toll-Like Receptor 4 - genetics | Smad Proteins - genetics | Thiobarbituric Acid Reactive Substances - metabolism | Methionine - administration & dosage | Biomarkers - blood | Choline Deficiency - pathology | Collagen Type III - genetics | Toll-Like Receptor 4 - metabolism | Fatty Liver - drug therapy | Animals | Choline - administration & dosage | Diet | NF-kappa B - genetics | Quercetin - pharmacology | Cyclooxygenase 2 - metabolism | Mice | Fibrosis - pathology | Oxidative Stress - drug effects | Smad Proteins - metabolism | Prevention | Hepatitis | Fibrosis | Quercetin | Physiological aspects | Development and progression | Inflammation | Health aspects
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12. Full Text Elevation of Free Fatty Acids Induces Inflammation and Impairs Vascular Reactivity in Healthy Subjects
by Devjit Tripathy and Priya Mohanty and Sandeep Dhindsa and Tufail Syed and Husam Ghanim and Ahmad Aljada and Paresh Dandona
Diabetes, ISSN 0012-1797, 12/2003, Volume 52, Issue 12, pp. 2882 - 2887
Elevation of Free Fatty Acids Induces Inflammation and Impairs Vascular Reactivity in Healthy Subjects Devjit Tripathy , Priya Mohanty , Sandeep Dhindsa ,... 
PROTEIN-KINASE-C | MIGRATION INHIBITORY FACTOR | OBESE-PATIENTS | MACROPHAGE | ENDOCRINOLOGY & METABOLISM | MONONUCLEAR-CELLS | TROGLITAZONE | NECROSIS-FACTOR-ALPHA | HUMAN MUSCLE | I-KAPPA-B | INDUCED INSULIN-RESISTANCE | Reactive Oxygen Species - metabolism | Humans | Transcription Factor RelA | Male | Monocytes - metabolism | NF-kappa B - metabolism | Reference Values | Thiobarbituric Acid Reactive Substances - metabolism | Insulin - blood | Inflammation - etiology | Vasodilation | Triglycerides - blood | Adult | Fatty Acids, Nonesterified - blood | Female | Neutrophils - metabolism | Brachial Artery - physiology | Physiological aspects | Diabetes | Research | Fatty acid metabolism
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13. Full Text Hyperammonaemia‐induced skeletal muscle mitochondrial dysfunction results in cataplerosis and oxidative stress
by Davuluri, Gangarao and Allawy, Allawy and Thapaliya, Samjhana and Rennison, Julie H and Singh, Dharmvir and Kumar, Avinash and Sandlers, Yana and Wagoner, David R and Flask, Chris A and Hoppel, Charles and Kasumov, Takhar and Dasarathy, Srinivasan
The Journal of Physiology, ISSN 0022-3751, 12/2016, Volume 594, Issue 24, pp. 7341 - 7360
Key points Hyperammonaemia occurs in hepatic, cardiac and pulmonary diseases with increased muscle concentration of ammonia. We found that ammonia results in... 
portacaval anastamosis | mitochondria | reactive oxygen species | ammonia | cellular respiration | skeletal muscle | ATP | cirrhosis | PHYSIOLOGY | BLOOD AMMONIA | SUPEROXIDE-PRODUCTION | ANTIOXIDANT ENZYMES | ALPHA-KETOGLUTARATE | LIVER-DISEASE | NEUROSCIENCES | DEHYDROGENASE | NITROGEN-METABOLISM | MYOSTATIN | CYCLE | Cell Line | Hyperammonemia - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Humans | Middle Aged | Mitochondria, Muscle - metabolism | Male | Muscle, Skeletal - metabolism | Thiobarbituric Acid Reactive Substances - metabolism | Myosin Heavy Chains - metabolism | Rats, Sprague-Dawley | Animals | Cell Respiration | Adenosine Triphosphate - metabolism | Liver Cirrhosis - metabolism | Creatine Kinase - metabolism | Female | Aged | NAD - metabolism | Oxidative stress | Ammonia | Cytochrome b | Physiological aspects | Muscles | Respiratory tract diseases | Liver cirrhosis | Proteins | Musculoskeletal system | Acids | Rodents | Respiration | Index Medicus | Skeletal Muscle | Muscular Conditions, Disorders and Treatments | Research Paper | Muscle | Muscle Physiology
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