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The Journal of Nutritional Biochemistry, ISSN 0955-2863, 12/2012, Volume 23, Issue 12, pp. 1676 - 1684
Nonalcoholic steatosis is an important hepatic complication of obesity linked to mitochondrial dysfunction and insulin resistance. Furthermore, lipoic acid has... 
Obesity | Mitochondrial dysfunction | Oxidative phosphorylation | High-fat diet | Triglyceride synthesis | Energy homeostasis | Insulin resistance | Energy efficiency | Steatosis | OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | COMPLEX FORMULATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RAT-LIVER MITOCHONDRIA | NUTRITION & DIETETICS | INSULIN-RESISTANCE | ANTIOXIDANT STATUS | PPAR-GAMMA | HEPATIC STEATOSIS | ADIPOSE-TISSUE | Rats, Wistar | Mitochondria, Liver - metabolism | Body Weight - drug effects | Diet, High-Fat - adverse effects | Ion Channels - genetics | Male | Mitochondrial Proteins - genetics | Insulin - blood | Lipogenesis - genetics | Liver - drug effects | Protective Agents - pharmacology | Adenosine Triphosphate - metabolism | Phosphorylation - drug effects | Enzymes - metabolism | Liver - metabolism | Citric Acid Cycle - drug effects | Fatty Liver - prevention & control | Rats | Thioctic Acid - pharmacology | Triglycerides - metabolism | Gene Expression Regulation - drug effects | Mitochondria, Liver - drug effects | Organ Size - drug effects | Animals | Lipogenesis - drug effects | Lipid Metabolism - drug effects | Transaminases - blood | Uncoupling Protein 2 | Blood Glucose - metabolism | Fatty Liver - etiology | Prevention | Analysis | Triglycerides | Muscle proteins | Gene expression | Adenosine triphosphate | Index Medicus
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 11/2015, Volume 288, Issue 3, pp. 300 - 312
In this work, α-lipoic acid and cyclosporine demonstrated significant protection against acetic acid-induced ulcerative colitis in rats. We proposed that... 
MicroRNA | Cyclosporine | Cyclooxygenase-2 | Nitric oxide | Alpha-lipoic acid | Ulcerative colitis | CELLS | KAPPA-B ACTIVATION | MECHANISM | RAT | NITRIC-OXIDE-SYNTHASE | PERMEABILITY TRANSITION | MAINTENANCE | INFLAMMATORY-BOWEL-DISEASE | REACTIVE OXYGEN | PHARMACOLOGY & PHARMACY | TOXICOLOGY | EXPRESSION | Thioctic Acid - administration & dosage | Rats, Wistar | Glutathione - metabolism | Transforming Growth Factor beta1 - metabolism | Tumor Necrosis Factor-alpha - blood | Colon - drug effects | Male | MicroRNAs - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Dose-Response Relationship, Drug | C-Reactive Protein - metabolism | Cyclooxygenase 2 - genetics | Superoxide Dismutase - metabolism | Malondialdehyde - metabolism | Colon - pathology | Rats | Transforming Growth Factor beta1 - genetics | Acetic Acid - adverse effects | Catalase - metabolism | Thioctic Acid - adverse effects | Animals | Colitis, Ulcerative - chemically induced | Cyclosporine - adverse effects | Cyclooxygenase 2 - metabolism | Cyclosporine - administration & dosage | MicroRNAs - genetics | Nitric Oxide - metabolism | Colitis, Ulcerative - physiopathology | Index Medicus | CATALASE | SUPEROXIDE DISMUTASE | NITRIC OXIDE | RECTUM | RATS | ACETIC ACID | 60 APPLIED LIFE SCIENCES | GLOBULINS | OXYGEN | GROWTH FACTORS | POLYMERASE CHAIN REACTION | GLUTATHIONE | POLYMERASES | DNA | CYCLOSPORINE
Journal Article
Acta Poloniae Pharmaceutica - Drug Research, ISSN 0001-6837, 01/2015, Volume 72, Issue 1, pp. 49 - 52
The oxidation of cyanide (CN-) to a much less toxic thiocyanate (SCN-) is the main in vivo biochemical pathway for CN- detoxification. SCN- is excreted mainly... 
Sulfane sulfur | Lipoic acid | Thiocyanates (rhodanates) | Cyanides | Dihydrolipoic acid | RHODANESE | dihydrolipoic acid | sulfane sulfur | cyanides | PHARMACOLOGY & PHARMACY | lipoic acid | thiocyanates (rhodanates) | Animals | Rats, Wistar | Thiocyanates - urine | Rats | Male | Thioctic Acid - pharmacology | Potassium Cyanide - pharmacology | Index Medicus
Journal Article
Hypertension, ISSN 0194-911X, 04/2012, Volume 59, Issue 4, pp. 818 - 824
Journal Article
Fundamental & Clinical Pharmacology, ISSN 0767-3981, 08/2008, Volume 22, Issue 4, pp. 439 - 445
A number of experimental studies have found that reactive oxygen species are involved during morphine treatment or withdrawal. The aims of this study were to... 
latency times or pain threshold levels | fecal excretion | jumping | α‐lipoic acid | reactive oxygen species | malondialdehyde | morphine and morphine withdrawal | arachidonate metabolites | Arachidonate metabolites | Reactive oxygen species | Morphine and morphine withdrawal | Latency times or pain threshold levels | α-lipoic acid | Jumping | Fecal excretion | Malondialdehyde | OXIDATIVE STRESS | SUPEROXIDE | alpha-lipoic acid | LIPID-PEROXIDATION | PHYSICAL-DEPENDENCE | INVOLVEMENT | ARACHIDONIC-ACID | HEROIN | IN-VIVO | DOPAMINE | PHARMACOLOGY & PHARMACY | OPIOID WITHDRAWAL | Narcotic Antagonists - administration & dosage | Gas Chromatography-Mass Spectrometry | Substance Withdrawal Syndrome - etiology | Substance Withdrawal Syndrome - drug therapy | Male | Narcotics - adverse effects | Dose-Response Relationship, Drug | Drug Interactions | Malondialdehyde - blood | Pain Threshold - drug effects | Behavior, Animal - drug effects | Defecation - drug effects | Rats | Naloxone - therapeutic use | Thioctic Acid - pharmacology | Antioxidants - pharmacology | Rats, Sprague-Dawley | Narcotics - administration & dosage | Antioxidants - therapeutic use | Animals | Narcotic Antagonists - therapeutic use | Naloxone - pharmacology | Narcotic Antagonists - pharmacology | Oxidative Stress - drug effects | Naloxone - administration & dosage | Substance Withdrawal Syndrome - physiopathology | Morphine - administration & dosage | Morphine - adverse effects | Thioctic Acid - therapeutic use | Index Medicus
Journal Article