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PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50683
Mitochondria are considered major generators of cellular reactive oxygen species (ROS) which are implicated in the pathogenesis of neurodegenerative diseases... 
APOPTOSIS | SUPEROXIDE | INHIBITION | ANTIOXIDANT | MULTIDISCIPLINARY SCIENCES | ANIMAL-MODELS | HUMAN SUBSTANTIA-NIGRA | HYDROGEN-PEROXIDE | 6-HYDROXYDOPAMINE | PARKINSONS-DISEASE | METALLOPORPHYRINS | Mitochondria - enzymology | RNA, Small Interfering - genetics | Mesencephalon - cytology | Peroxiredoxins - metabolism | RNA, Messenger - metabolism | Dopaminergic Neurons - cytology | Gene Knockdown Techniques | Cell Respiration - genetics | Oxidopamine - pharmacology | Cell Death - genetics | Dopaminergic Neurons - metabolism | Cell Respiration - drug effects | Dopaminergic Neurons - drug effects | Lentivirus - genetics | Cell Death - drug effects | Energy Metabolism - genetics | Paraquat - pharmacology | Auranofin - pharmacology | Cell Line | RNA, Messenger - genetics | Enzyme Inhibitors - pharmacology | Oxidative Stress - genetics | Rats | Mitochondria - metabolism | Thioredoxin Reductase 2 - antagonists & inhibitors | Mitochondria - drug effects | Mitochondria - pathology | Thioredoxin Reductase 2 - genetics | Hydrogen Peroxide - metabolism | Drug Synergism | Thioredoxin Reductase 2 - metabolism | Animals | Thioredoxin Reductase 2 - deficiency | Oxidative Stress - drug effects | Energy Metabolism - drug effects | Nervous system diseases | Biochemistry | Hydrogen peroxide | Thioredoxin | Cell death | Peroxiredoxin | Brain | Oxidative stress | Cell culture | Neurosciences | Basal ganglia | Parkinson's disease | Pathogenesis | Kinases | Proteins | Antioxidants | Mitochondria | Toxicants | Neurodegeneration | Rodents | Penicillin | Aging | Inhibition | Movement disorders | Dopamine receptors | Cellular manufacture | Medical research | Oxygen | Dopamine | Incubation | Neurodegenerative diseases | Mortality | 6-Hydroxydopamine | Central nervous system diseases | Oxygen consumption | Cultures | Pharmacology | Steady state | Substrates | Neurological diseases | Paraquat | Electron transport | Apoptosis | Reductase
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e71525
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2016, Volume 36, Issue 9, pp. 1891 - 1899
OBJECTIVE—Although the investigation on the importance of mitochondria-derived reactive oxygen species (ROS) in endothelial function has been gaining momentum,... 
Angiogenesis | Arteriogenesis | Reactive oxygen species | Inflammation | Thrombosis | Thioredoxin reductase 2 | CELLS | OXIDATIVE STRESS | angiogenesis | NITRIC-OXIDE SYNTHASE | ISCHEMIA | VASCULAR-DISEASE | arteriogenesis | thrombosis | inflammation | reactive oxygen species | PERIPHERAL VASCULAR DISEASE | thioredoxin reductase 2 | ENOS-KNOCKOUT MICE | HYPERTENSION | HEMATOLOGY | HEART-MITOCHONDRIA | Femoral Artery - pathology | Inflammation - pathology | Mitochondria - enzymology | Vascular Remodeling | Reactive Oxygen Species - metabolism | Ischemia - enzymology | Ischemia - genetics | Endothelial Progenitor Cells - enzymology | Endothelium, Vascular - enzymology | Vasodilation | Femoral Artery - enzymology | Ligation | Time Factors | Membrane Potential, Mitochondrial | Ischemia - pathology | Disease Models, Animal | Genetic Predisposition to Disease | Thrombosis - physiopathology | Oxidation-Reduction | Signal Transduction | Femoral Artery - physiopathology | Thrombosis - enzymology | Cells, Cultured | Endothelium, Vascular - physiopathology | Ischemia - physiopathology | Mitochondria - pathology | Thioredoxin Reductase 2 - genetics | Mice, Knockout | Thrombosis - pathology | Phenotype | Animals | Endothelial Progenitor Cells - pathology | Inflammation - genetics | Thioredoxin Reductase 2 - deficiency | Endothelium, Vascular - pathology | Thrombosis - genetics | Nitric Oxide - metabolism | Femoral Artery - surgery | Inflammation - enzymology | Neovascularization, Physiologic | Inflammation - physiopathology
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 07/2015, Volume 4, Issue 7
Background-Ubiquitous deletion of thioredoxin reductase 2 (Txnrd2) in mice is embryonically lethal and associated with abnormal heart development, while... 
heart failure | aging | thioredoxin reductase 2 | SYSTEM | CARDIAC & CARDIOVASCULAR SYSTEMS | TISSUE | ENERGETICS | AUTOPHAGY | SELENIUM | PLATFORM | GLUTATHIONE | DISEASE | STRESS | EXPRESSION | Blood Pressure | Mitochondria, Heart - ultrastructure | Reactive Oxygen Species - metabolism | Age Factors | Microtubule-Associated Proteins - genetics | Oxidative Stress | Ventricular Function, Left | Microtubule-Associated Proteins - metabolism | Heart Failure - enzymology | Heart Failure - physiopathology | RNA, Messenger - metabolism | Autophagy | Myocytes, Cardiac - enzymology | Time Factors | Ventricular Dysfunction, Left - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Ventricular Dysfunction, Left - enzymology | Disease Models, Animal | Myocardial Contraction | Genetic Predisposition to Disease | Lysosome-Associated Membrane Glycoproteins - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Gene Expression Regulation | Mitochondria, Heart - enzymology | Gene Expression Profiling - methods | Heart Failure - genetics | Heart Failure - pathology | Metabolomics - methods | Thioredoxin Reductase 2 - genetics | Ventricular Dysfunction, Left - physiopathology | Mice, Knockout | Stroke Volume | Phenotype | Animals | Energy Metabolism | Lysosome-Associated Membrane Glycoproteins - genetics | Thioredoxin Reductase 2 - deficiency | Myocytes, Cardiac - ultrastructure
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 08/2014, Volume 99, Issue 8, pp. E1556 - E1563
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 02/2012, Volume 52, Issue 4, pp. 803 - 810
Ribonucleotide reductase (RNR) activity requires an electron donor, which in bacteria, yeast, and plants is usually either reduced thioredoxin (Trx) or reduced... 
DNA replication | Hepatocyte | Mouse | Thioredoxin reductase | Glutathione | EARLY EMBRYONIC LETHALITY | THIOREDOXIN REDUCTASE 1 | MAMMALIAN THIOREDOXIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | RIBONUCLEOSIDE-DIPHOSPHATE REDUCTASE | RESPONSE ELEMENT | REDOX SYSTEMS | GENE | ENDOCRINOLOGY & METABOLISM | S-PHASE | HYDROGEN DONOR SYSTEM | Cell Proliferation | Liver - enzymology | Exons | Glutathione - metabolism | Molecular Sequence Data | Thioredoxin Reductase 1 - deficiency | Glutathione Disulfide - metabolism | Hepatocytes - metabolism | Liver - growth & development | Isoenzymes - metabolism | Base Sequence | Thioredoxins - metabolism | Transcription, Genetic | Hepatocytes - physiology | Isoenzymes - genetics | Liver - metabolism | Mice, Inbred C57BL | DNA Replication | Thioredoxin Reductase 2 - genetics | Glutathione - physiology | Thioredoxin Reductase 2 - metabolism | Animals | Heterozygote | Liver - cytology | Mice | Thioredoxin Reductase 1 - genetics | Histones - metabolism | Proliferating Cell Nuclear Antigen - metabolism | Hepatocytes - enzymology | Thioredoxin Reductase 1 - metabolism | Antigen-antibody reactions | Messenger RNA | Growth | Liver | DNA | Genes | Albumin | Genetic research | Thioredoxin | Cells | RNA | DNA polymerases | DNA biosynthesis | Enzymes | Thioredoxin-disulfide reductase | Buthionine sulfoximine | Hepatocytes | Ribonucleoside-triphosphate reductase | Glutaredoxin | Replication | mRNA | glutathione reductase | mouse | hepatocyte | glutathione | thioredoxin reductase
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e87300
Understanding how pathogenic fungi adapt to host plant cells is of major concern to securing global food production. The hemibiotrophic rice blast fungus... 
TRIGGERED IMMUNITY | OXIDATIVE STRESS | INVASION | FUNGUS | METABOLISM | MULTIDISCIPLINARY SCIENCES | ESCHERICHIA-COLI | SACCHAROMYCES-CEREVISIAE | PLANT INFECTION | NADPH OXIDASES | CELL-DEATH | Gene Expression Regulation, Fungal | Magnaporthe - enzymology | Reactive Oxygen Species - metabolism | Thioredoxin-Disulfide Reductase - genetics | Peroxiredoxins - deficiency | Plant Diseases - microbiology | Fungal Proteins - genetics | Glutathione Reductase - genetics | Thioredoxin-Disulfide Reductase - deficiency | Glutathione Reductase - deficiency | Host-Pathogen Interactions | Peroxiredoxins - genetics | Oryza - microbiology | Magnaporthe - pathogenicity | NADP - metabolism | Magnaporthe - genetics | Plant Leaves - microbiology | Fungal Proteins - metabolism | Fungi | Thiols | Genetic research | Peroxidase | Explosions | Thioredoxin | Rice blast disease | Gene expression | Oxidative stress | Food plants | Reactive oxygen species | Plant pathology | Disease | Genomics | Genes | Grain cultivation | Genomes | Infections | Glucose | Plant cells | Proteins | Leaves | Glutathione reductase | Nutrients | NADP | Lesions | Sugar | Glutathione | Rice | Cultivars | Growth conditions | Enzymes | Rice blast | Cell walls | Food production | Functional analysis | Metabolism | Plant diseases | Host plants | Thioredoxin peroxidase | Colonization | Growth stage | Reductase
Journal Article
Journal Article