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Journal of the American College of Cardiology, ISSN 0735-1097, 05/2017, Volume 69, Issue 20, pp. 2530 - 2537
Background It remains unclear whether radial access increases the risk of operator or patient radiation exposure compared to transfemoral access when performed... 
percutaneous coronary intervention | radiation dose | radial access | femoral access | SITE | PELVIC LEAD SHIELD | DESIGN | CARDIAC & CARDIOVASCULAR SYSTEMS | RATIONALE | ANGIOX | ANGIOGRAPHY | OPERATOR | ADVERSE HEMORRHAGIC EVENTS | INTERVENTION | SYSTEMIC IMPLEMENTATION | Catheterization, Peripheral - methods | Humans | Middle Aged | Fluoroscopy - methods | Male | Coronary Angiography - adverse effects | Radiography, Interventional - adverse effects | Coronary Angiography - methods | Time Factors | Percutaneous Coronary Intervention - methods | Occupational Exposure - prevention & control | Adult | Female | Catheterization, Peripheral - adverse effects | Risk Management - organization & administration | Needs Assessment | Radial Artery - surgery | Fluoroscopy - adverse effects | Radiation Protection - methods | Femoral Artery - diagnostic imaging | Radiography, Interventional - methods | Acute Coronary Syndrome - diagnosis | Radiation Exposure - prevention & control | Femoral Artery - surgery | Percutaneous Coronary Intervention - adverse effects | Radial Artery - diagnostic imaging | Transluminal angioplasty | Radiotherapy | Coronary heart disease | Analysis | Lithium compounds | Radiation | Radiation effects | Femur | Operators | Angiography | Angioplasty | Lithium fluoride | Health risks | Disorders | Lithium | Thorax | Fluoroscopy | Exposure | Patients | Radiation dosage | Wrist | Fluoride | Dosimetry | Acute coronary syndromes | Health risk assessment | Background radiation | α Radiation
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3264 - 3276
Radiation‐induced lung injury (RILI) is one of the most common and fatal complications of thoracic radiotherapy. It is characterized with two main features... 
radiation‐induced lung injury | free radicals | epithelial–mesenchymal transition (EMT) | polydatin (PD) | radiation-induced lung injury | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | SMAD3 | INDUCED FIBROSIS | CANCER CELLS | IRRADIATED MICE | DAMAGE | IMPAIRMENT | CELL BIOLOGY | epithelial-mesenchymal transition (EMT) | TH2-LIKE IMMUNE-RESPONSE | GROWTH | EXPRESSION | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - genetics | Epithelial Cells - metabolism | Epithelial-Mesenchymal Transition - radiation effects | Radiation Pneumonitis - pathology | Epithelial Cells - drug effects | Humans | Acute Lung Injury - genetics | Epithelial-Mesenchymal Transition - drug effects | Smad3 Protein - immunology | Stilbenes - pharmacology | Radiation Pneumonitis - prevention & control | NF-E2-Related Factor 2 - immunology | Smad3 Protein - genetics | Transforming Growth Factor beta1 - immunology | Acute Lung Injury - prevention & control | Radiation-Protective Agents - pharmacology | Female | Lung - radiation effects | NF-E2-Related Factor 2 - genetics | Lung - metabolism | Th1-Th2 Balance - drug effects | Disease Models, Animal | Cell Line | Epithelial Cells - radiation effects | Glucosides - pharmacology | Lung - pathology | Sirtuin 3 - genetics | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Epithelial Cells - pathology | Transforming Growth Factor beta1 - genetics | Acute Lung Injury - pathology | Radiation Pneumonitis - immunology | Sirtuin 3 - immunology | Animals | Th1-Th2 Balance - radiation effects | Lung - drug effects | Radiation Pneumonitis - genetics | Acute Lung Injury - immunology | Mice | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - immunology | Transforming growth factors | Lung diseases | Stem cells | Radiation | Cell culture | Radiation effects | Mesenchyme | Complications | Lymphocytes T | Smad3 protein | Thorax | Radiation therapy | Tissues | Signaling | Signal transduction | Pneumonitis | Beta irradiation | Lungs | Fibrosis | Radiation injuries | Irradiation | Inhibition | Injuries | Original
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 04/2017, Volume 26, Issue 11, pp. 563 - 582
Aims: Radiation-induced normal tissue toxicity is closely linked to endothelial cell (EC) damage and dysfunction (acute effects). However, the underlying... 
Original Research Communications | radioprotection | mesenchymal stem cell therapy | vascular dysfunction | Radiotherapy | TOTAL-BODY IRRADIATION | PRO-INFLAMMATORY CYTOKINES | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | TISSUE | INJURY | MECHANISMS | ENDOCRINOLOGY & METABOLISM | radiotherapy | IONIZING-RADIATION | UP-REGULATION | PULMONARY-FIBROSIS | Salicylates - pharmacology | Endothelial Cells - ultrastructure | Mesenchymal Stromal Cells - cytology | Radiation Injuries - genetics | Superoxide Dismutase-1 - metabolism | Lung - radiation effects | Lung - metabolism | Disease Models, Animal | Fibroblasts - metabolism | Radiation Injuries - therapy | Gene Expression | Lung - pathology | Endothelial Cells - metabolism | Endothelial Cells - radiation effects | Phenotype | Animals | Fibroblasts - radiation effects | Fibrosis | Superoxide Dismutase-1 - genetics | Radiation Injuries - pathology | Mice | Endothelial Cells - pathology | Radiation Injuries - metabolism | Radiation Injuries, Experimental | Mesenchymal Stem Cell Transplantation | Organometallic Compounds - pharmacology | Superoxide dismutase | Research | Lungs | Gene expression | Health aspects | Cellular therapy | Cell culture | Therapy | Mesenchyme | Toxicity | Radiation | Innovations | Stem cell transplantation | Paracrine signalling | Adoptive transfer | Antioxidants | Reduction | Bone marrow | Radiation injuries | Aorta | Acute effects | Radiation effects | Thorax | Radioprotection | Endothelial cells | Stem cells | Irradiation | Viability | Radiation damage | Index Medicus
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2013, Volume 61, Issue 23, pp. 2319 - 2328
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 10/2011, Volume 301, Issue 4, pp. 415 - 426
Molecular hydrogen (H2) is an efficient antioxidant that diffuses rapidly across cell membranes, reduces reactive oxygen species (ROS), such as hydroxyl... 
Peroxynitrite | Reactive oxygen species | Radioprotector | Free radical scavengers | Hydroxyl radical | Apoptosis | TOTAL-BODY IRRADIATION | CELLS | PHYSIOLOGY | peroxynitrite | BONE-MARROW-TRANSPLANTATION | apoptosis | free radical scavengers | RICH WATER | RAT MODEL | INTERSTITIAL PNEUMONITIS | hydroxyl radical | REACTIVE OXYGEN | RESPIRATORY SYSTEM | reactive oxygen species | radioprotector | IONIZING-RADIATION | PULMONARY-FIBROSIS | NORMAL TISSUE-INJURY | Immunohistochemistry | Hydrogen - pharmacology | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Apoptosis - radiation effects | Humans | Apoptosis Regulatory Proteins - analysis | Thorax - metabolism | Tomography, X-Ray Computed | Female | Lung - radiation effects | Lung - metabolism | Oxidative Stress - radiation effects | Hydroxyl Radical - antagonists & inhibitors | Apoptosis Regulatory Proteins - biosynthesis | DNA Damage - drug effects | In Situ Nick-End Labeling | Cell Line | Cell Survival - drug effects | Thorax - radiation effects | Hydroxyl Radical - metabolism | Pulmonary Fibrosis - pathology | Antioxidants - pharmacology | Radiation Dosage | X-Rays - adverse effects | Cell Survival - radiation effects | Blotting, Western | Reactive Oxygen Species - antagonists & inhibitors | Animals | Lung - drug effects | Pulmonary Fibrosis - prevention & control | Mice | Oxidative Stress - drug effects | Pulmonary Fibrosis - drug therapy | Thorax - drug effects | DNA Damage - radiation effects | Oxidative stress | Care and treatment | Hydrogen | Lung diseases | Research | Health aspects | Risk factors | Index Medicus
Journal Article
Dentomaxillofacial Radiology, ISSN 0250-832X, 02/2009, Volume 38, Issue 2, pp. 79 - 85
Journal Article
Health Physics, ISSN 0017-9078, 01/2014, Volume 106, Issue 1, pp. 56 - 72
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2016, Volume 94, Issue 4, pp. 808 - 815
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53628
Pneumonitis and fibrosis are major lung complications of irradiating thoracic malignancies. In the current study, we determined the effect of thoracic... 
TO-MESENCHYMAL TRANSITION | HEPATIC STELLATE CELLS | TGF-BETA | INDUCED PULMONARY-FIBROSIS | MULTIDISCIPLINARY SCIENCES | THORACIC IRRADIATION | GROWTH-FACTOR | HISTONE DEACETYLASE INHIBITORS | MOUSE LUNG | RAT LUNG | DECREASED EXPRESSION | Gamma Rays | Lung Injury - pathology | Pulmonary Alveoli - radiation effects | Cadherins - metabolism | Vimentin - metabolism | Transforming Growth Factor beta1 - metabolism | Epithelium - radiation effects | Time Factors | Pulmonary Alveoli - metabolism | Dose Fractionation | Female | Cell Line | Epithelium - pathology | Pulmonary Alveoli - pathology | Pulmonary Surfactant-Associated Protein C - metabolism | Radiation Injuries - complications | Mice, Inbred Strains | Animals | Aquaporin 5 - metabolism | Protein Transport - radiation effects | Survival Analysis | Lung Injury - complications | Radiation Injuries - pathology | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Dose-Response Relationship, Radiation | Complications and side effects | Pneumonia | Analysis | Collagen | Bacterial pneumonia | Radiation | Muscle proteins | Intermediate filament proteins | Vimentin | Oxidative stress | Chromatin | Cesium radioisotopes | Mesenchyme | Laboratories | Brain cancer | Smooth muscle | Oncology | Cesium isotopes | E-cadherin | Actin | Rodents | Hepatology | Fibroblasts | Radiation injuries | Alveoli | Localization | Drug dosages | Thickening | Edema | Radiation effects | Complications | Muscles | Protein C | Thorax | Inflammation | Radiation therapy | Epithelium | Morbidity | Medicine | Pneumonitis | Pulmonary fibrosis | Lungs | Fibrosis | Irradiation | Immunofluorescence
Journal Article
Radiation Research, ISSN 0033-7587, 11/2016, Volume 186, Issue 5, pp. 478 - 488
There is an ongoing and significant need for radiation countermeasures to reduce morbidities and mortalities associated with exposure of the heart and lungs... 
TGF-BETA | MYOCARDIAL-INFARCTION | LUNG INJURY | INDUCED CARDIOMYOPATHY | FACTOR-ALPHA | HEART | BIOPHYSICS | INDUCED PULMONARY-FIBROSIS | SIGNALING PATHWAY | BIOLOGY | ANIMAL-MODELS | THORACIC IRRADIATION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Radiation-Protective Agents - pharmacokinetics | Half-Life | Male | Cardiomyopathies - etiology | Radiation Injuries, Experimental - etiology | Radiation Injuries, Experimental - drug therapy | Myocardium - metabolism | Respiration - drug effects | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Radiation-Protective Agents - pharmacology | Female | Lung - radiation effects | Lung - metabolism | Signal Transduction - radiation effects | Radiation Injuries, Experimental - metabolism | Cardiomyopathies - drug therapy | Cardiomyopathies - pathology | Respiration - radiation effects | Radiation Injuries, Experimental - pathology | Collagen - metabolism | Animals | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Signal Transduction - drug effects | Cardiomyopathies - metabolism | Lung - drug effects | Survival Analysis | Radiation-Protective Agents - therapeutic use | Mice | Transforming Growth Factor beta - metabolism | Development and progression | Care and treatment | Enzyme inhibitors | Health aspects | Lung diseases | Radiation effects | Mortality | Fibrosis | Irradiation | Countermeasures | Exposure | Thorax | Survival
Journal Article