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1. Full Text Endothelial, but not smooth muscle, peroxisome proliferator-activated receptor β/δ regulates vascular permeability and anaphylaxis
by Wawrzyniak, Marta, PhD and Pich, Christine, PhD and Gross, Barbara, PhD and Schütz, Frédéric, PhD and Fleury, Sébastien, BSc and Quemener, Sandrine, MSc and Sgandurra, Marie, MSc and Bouchaert, Emmanuel and Moret, Catherine and Mury, Lionel and Rommens, Corinne and Mottaz, Hélène and Dombrowicz, David, PhD and Michalik, Liliane, PhD
Journal of allergy and clinical immunology, ISSN 0091-6749, 2014, Volume 135, Issue 6, pp. 1625 - 1635.e5
Allergy and Immunology | Peroxisome proliferator-activated receptor β/δ | smooth muscle cells | endothelium | anaphylaxis | vascular permeability | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Edema - genetics | Myocytes, Smooth Muscle - pathology | Capillary Permeability - drug effects | Capillary Permeability - immunology | Hypothermia - immunology | Intercellular Junctions - immunology | Hypothermia - pathology | Proto-Oncogene Proteins c-akt - genetics | Thrombin - pharmacology | Intercellular Junctions - pathology | Anaphylaxis - immunology | PPAR delta - immunology | Female | Mitogen-Activated Protein Kinases - immunology | Myocytes, Smooth Muscle - drug effects | Vascular Endothelial Growth Factor A - pharmacology | PPAR delta - genetics | Skin - pathology | Skin - immunology | PPAR delta - deficiency | Edema - immunology | Histamine - pharmacology | Signal Transduction | Anaphylaxis - pathology | Gene Expression Regulation | Mice, Transgenic | PPAR-beta - immunology | PPAR-beta - genetics | PPAR-beta - deficiency | Anaphylaxis - genetics | Endothelial Cells - immunology | Skin - blood supply | Animals | Proto-Oncogene Proteins c-akt - immunology | Intercellular Junctions - drug effects | Myocytes, Smooth Muscle - immunology | Mitogen-Activated Protein Kinases - genetics | Mice | Endothelial Cells - pathology | Edema - pathology | Hypothermia - genetics | Skin - drug effects | Endothelial Cells - drug effects | Index Medicus | Abridged Index Medicus | Mitogen-Activated Protein Kinases/genetics | Skin/blood supply | Endothelial Cells/drug effects | Endothelial Cells/immunology | Intercellular Junctions/immunology | PPAR delta/immunology | Skin/pathology | Intercellular Junctions/drug effects | Hypothermia/genetics | Capillary Permeability/drug effects | Life Sciences | PPAR delta/genetics | PPAR delta/deficiency | Hypothermia/immunology | Anaphylaxis/genetics | Histamine/pharmacology | PPAR-beta/immunology | Myocytes, Smooth Muscle/pathology | Anaphylaxis/pathology | Myocytes, Smooth Muscle/immunology | Endothelial Cells/pathology | Hypothermia/pathology | Vascular Endothelial Growth Factor A/pharmacology | Mitogen-Activated Protein Kinases/immunology | Myocytes, Smooth Muscle/drug effects | Edema/immunology | Proto-Oncogene Proteins c-akt/genetics | Edema/pathology | Skin/immunology | Intercellular Junctions/pathology | Proto-Oncogene Proteins c-akt/immunology | Edema/genetics | PPAR-beta/genetics | Skin/drug effects | PPAR-beta/deficiency | Thrombin/pharmacology
Journal Article
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2. Full Text Fibrinogen as a key regulator of inflammation in disease
by Davalos, Dimitrios and Davalos, Dimitrios and Akassoglou, Katerina and Akassoglou, Katerina
Seminars in immunopathology, ISSN 1863-2297, 1/2012, Volume 34, Issue 1, pp. 43 - 62
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | Pathology | Life Sciences & Biomedicine | Science & Technology | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Index Medicus | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
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3. Full Text Interaction between the coagulation and complement system
by Amara, Umme and Rittirsch, Daniel and Flierl, Michael and Bruckner, Uwe and Klos, Andreas and Gebhard, Florian and Lambris, John D and Huber-Lang, Markus
Advances in Experimental Medicine and Biology, ISSN 0065-2598, 2008, Volume 632, pp. 71 - 79
Fibrinolysin - immunology | Hemostasis - immunology | Factor Xa - immunology | Complement C1q - immunology | Humans | Complement System Proteins - immunology | Anaphylatoxins - immunology | Complement C2 - immunology | Complement C4 - immunology | Complement Activation - immunology | Factor XIa - immunology | Wound Healing - immunology | Complement C3 - immunology | Animals | Drug Interactions | Blood Coagulation - immunology | Models, Biological | Complement C5 - immunology | Serine Endopeptidases - immunology | Complement Factor B - immunology | Complement C3a - immunology | Complement C5a - immunology | Thrombin - immunology | Complement C4a - immunology | Index Medicus
Conference Proceeding
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4. Full Text Dust mite-derived Der f 3 activates a pro-inflammatory program in airway epithelial cells via PAR-1 and PAR-2
by Li, Bizhou and Zou, Zehong and Meng, Fanmei and Raz, Eyal and Huang, Yuye and Tao, Ailin and Ai, Yuncan
Molecular immunology, ISSN 0161-5890, 05/2019, Volume 109, pp. 1 - 11
Airway epithelial cell | House dust mite (HDM) | Inflammation | Protease-activated receptor (PAR) | Protease | Der f 3 | Biochemistry & Molecular Biology | Immunology | Life Sciences & Biomedicine | Science & Technology | Inflammation - chemically induced | Inflammation - pathology | Humans | NF-kappa B - immunology | Serine Endopeptidases - pharmacology | Th2 Cells - immunology | Extracellular Signal-Regulated MAP Kinases - genetics | MAP Kinase Signaling System - immunology | Extracellular Signal-Regulated MAP Kinases - immunology | Gene Knockdown Techniques | Receptor, PAR-2 - immunology | MAP Kinase Signaling System - genetics | Allergens - immunology | Respiratory Mucosa - immunology | Serine Endopeptidases - immunology | Calcium Signaling - immunology | Cytokines - genetics | Th2 Cells - pathology | Cytokines - immunology | Receptor, PAR-2 - genetics | Th17 Cells - pathology | Inflammation Mediators - immunology | A549 Cells | Gene Expression Regulation - immunology | Epithelial Cells - pathology | Inflammation - immunology | Arthropod Proteins - immunology | Receptor, PAR-1 - genetics | Receptor, PAR-1 - immunology | Gene Expression Regulation - drug effects | Arthropod Proteins - pharmacology | Animals | MAP Kinase Signaling System - drug effects | NF-kappa B - genetics | Calcium Signaling - drug effects | Epithelial Cells - immunology | Inflammation - genetics | Th17 Cells - immunology | Allergens - pharmacology | Calcium Signaling - genetics | Pyroglyphidae - immunology | Allergens | Allergy | Medical colleges | RNA | Biological control | Thrombin | Respiratory tract diseases | Genetic engineering | Pests | B cells | Allergic reaction | Index Medicus
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5. Full Text Endothelium-role in regulation of coagulation and inflammation
by van Hinsbergh, V.W.M
Seminars in immunopathology, ISSN 1863-2297, 2012, Volume 34, Issue 1, pp. 93 - 106
Endothelial cell | Biomedicine | Immunology | ADAMTS13 | von Willebrand factor | Antithrombotic | Internal Medicine | Inflammation | Activated protein C | Fibrinolysis | Von willebrand factor | Pathology | Life Sciences & Biomedicine | Science & Technology | Fundamental and applied biological sciences. Psychology | Fundamental immunology | Immunopathology | Biological and medical sciences | Medical sciences | Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis | Vasoconstrictor Agents - immunology | Blood Platelets - immunology | Platelet Aggregation - immunology | Receptor, PAR-1 - metabolism | Humans | Blood Proteins - immunology | Fibrinolysis - immunology | Inflammation - immunology | Blood Proteins - metabolism | Inflammation - blood | Receptor, PAR-1 - immunology | Leukocytes - immunology | Animals | Vasoconstrictor Agents - blood | Blood Platelets - metabolism | Endothelium, Vascular - metabolism | Leukocytes - metabolism | Endothelium, Vascular - immunology | Fibrin | Nitric oxide | Von Willebrand factor | Protein C | Thrombin | Endothelium | Index Medicus | prostacyclin | Coagulation | Proteinase-activated receptor 1 | Blood | Endothelial cells | Stress | plasminogen | Angiogenesis | Fluidity | Perfusion | Vasoactive agents | Platelets | fibrin | activated protein C | Review
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6. Full Text Complement and tissue factor-enriched neutrophil extracellular traps are key drivers in COVID-19 immunothrombosis
by Skendros, Panagiotis and Mitsios, Alexandros and Chrysanthopoulou, Akrivi and Mastellos, Dimitrios C and Metallidis, Simeon and Rafailidis, Petros and Ntinopoulou, Maria and Sertaridou, Eleni and Tsironidou, Victoria and Tsigalou, Christina and Tektonidou, Maria and Konstantinidis, Theocharis and Papagoras, Charalampos and Mitroulis, Ioannis and Germanidis, Georgios and Lambris, John D and Ritis, Konstantinos
The Journal of clinical investigation, ISSN 0021-9738, 11/2020, Volume 130, Issue 11, pp. 6151 - 6157
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Pandemics | Thrombosis - virology | Complement Membrane Attack Complex - metabolism | Extracellular Traps - metabolism | Humans | Middle Aged | Male | Peptides, Cyclic - pharmacology | Respiratory Distress Syndrome - immunology | Complement Membrane Attack Complex - immunology | Pneumonia, Viral - blood | Betacoronavirus - immunology | COVID-19 | SARS-CoV-2 | Extracellular Traps - immunology | Respiratory Distress Syndrome - virology | Coronavirus Infections - blood | Receptor, Anaphylatoxin C5a - immunology | Female | Thrombosis - blood | Neutrophils - metabolism | Thrombin - immunology | Complement Activation - drug effects | Thromboplastin - immunology | Respiratory Distress Syndrome - blood | Neutrophils - immunology | Thrombosis - immunology | Betacoronavirus - metabolism | Coronavirus Infections - immunology | Pneumonia, Viral - immunology | Receptor, Anaphylatoxin C5a - blood | Aged | Receptor, Anaphylatoxin C5a - antagonists & inhibitors | Thrombin - metabolism | Thromboplastin - metabolism | Neutrophils | Development and progression | Complement system | Thrombosis | Health aspects | Blood clot | Risk factors | Greece | Care and treatment | Immune response | Severe acute respiratory syndrome | Mortality | Thrombin | Research | Coronaviruses | Plasma | Thrombotic microangiopathy | Tissue factor | Leukocytes (neutrophilic) | Autophagy | Patients | Investigations | Endothelial cells | Severe acute respiratory syndrome coronavirus 2 | Complement activation | Complement component C3 | Aorta | Plasma levels | Platelets | Index Medicus | Abridged Index Medicus
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