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Blood, ISSN 0006-4971, 01/2008, Volume 111, Issue 2, pp. 613 - 623
Platelet a-granules constitute the major rapidly releasable reservoir of thrombospondin-1 in higher animals. Although some fragments and peptides derived from... 
ACTIVATION | CELL RESPONSES | FACTOR MULTIMER SIZE | L-ARGININE | GLYCOPROTEIN-IV | MONOCLONAL-ANTIBODY | TERMINAL PEPTIDE | KINASE-I | HEMATOLOGY | INTEGRIN-ASSOCIATED PROTEIN | VON-WILLEBRAND-FACTOR | Cyclic GMP - pharmacology | Immunologic Capping - physiology | Cell Adhesion Molecules - genetics | CD36 Antigens - genetics | Peptides - genetics | Secretory Vesicles - metabolism | Fibrinolytic Agents - metabolism | Phosphoproteins - metabolism | Platelet Adhesiveness - genetics | Secretory Vesicles - genetics | Peptides - metabolism | Thrombin - pharmacology | CD36 Antigens - metabolism | Blood Platelets - cytology | Arginine - genetics | Platelet Glycoprotein GPIIb-IIIa Complex - metabolism | Thrombospondin 1 - genetics | Immunologic Capping - drug effects | Microfilament Proteins - metabolism | Platelet Aggregation - drug effects | Platelet Glycoprotein GPIIb-IIIa Complex - genetics | Microfilament Proteins - genetics | CD47 Antigen - genetics | rap1 GTP-Binding Proteins - metabolism | Shear Strength | Nitric Oxide - antagonists & inhibitors | Platelet Aggregation - physiology | CD47 Antigen - metabolism | Phosphoproteins - genetics | Cell Adhesion Molecules - metabolism | Thrombin - genetics | Mice, Knockout | Peptides - pharmacology | Platelet Adhesiveness - drug effects | Fibrinolytic Agents - pharmacology | Animals | Cyclic GMP - metabolism | Blood Platelets - metabolism | Nitric Oxide - genetics | Cyclic GMP - genetics | Thrombospondin 1 - pharmacology | Mice | Thrombin - metabolism | Thrombospondin 1 - metabolism | Nitric Oxide - metabolism | rap1 GTP-Binding Proteins - genetics | Arginine - metabolism | Cyclic GMP - antagonists & inhibitors | Index Medicus | Abridged Index Medicus | Hemostasis, Thrombosis, and Vascular Biology
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2005, Volume 280, Issue 17, pp. 17286 - 17293
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2015, Volume 21, Issue 11, pp. 1307 - 1317
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 07/2008, Volume 100, Issue 1, pp. 101 - 109
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2007, Volume 27, Issue 12, pp. 2714 - 2721
OBJECTIVE—We previously showed that NAD(P)H oxidase deficiency significantly reduces atherosclerosis in apoE mice. The present study was designed to determine... 
Oxidized lipids | Signal transduction | Adhesion molecules | Thrombin | Reactive oxygen species | NADPH OXIDASE | thrombin | signal transduction | LOW-DENSITY-LIPOPROTEIN | MONOCYTES | ANION PRODUCTION | HYPERTROPHY | NAD(P)H OXIDASE | IN-VITRO | adhesion molecules | reactive oxygen species | PERIPHERAL VASCULAR DISEASE | MICE | oxidized lipids | HEMATOLOGY | EXPRESSION | Phosphorylation | Cell Proliferation | Apolipoproteins E - deficiency | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Endothelium, Vascular - injuries | Hyperplasia | NADPH Oxidases - metabolism | Aorta - metabolism | Endothelium, Vascular - enzymology | RNA, Messenger - metabolism | Apolipoproteins E - metabolism | Femoral Artery - enzymology | Femoral Artery - injuries | NADPH Oxidases - deficiency | Atherosclerosis - enzymology | Janus Kinase 2 - metabolism | Bone Marrow Transplantation | Dietary Fats - administration & dosage | Superoxides - metabolism | NADPH Oxidases - genetics | Aorta - enzymology | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Macrophages - pathology | Oxidation-Reduction | Signal Transduction | Cell Adhesion Molecules - metabolism | Atherosclerosis - metabolism | Macrophages - enzymology | Mice, Knockout | Macrophages - metabolism | Muscle, Smooth, Vascular - injuries | Muscle, Smooth, Vascular - pathology | Animals | Endothelium, Vascular - metabolism | Apolipoproteins E - genetics | Femoral Artery - metabolism | Endothelium, Vascular - pathology | Mice | Thrombin - metabolism | Muscle, Smooth, Vascular - enzymology | Atherosclerosis - prevention & control | Lipoproteins, LDL - blood | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
BMC Nephrology, ISSN 1471-2369, 01/2017, Volume 18, Issue 1, pp. 35 - 35
Background: During chronic kidney disease progression, kidney-specific risk factors for cardiovascular disease come into play. The present study investigated... 
Prothrombotic state | Hemostatic disorder | Indoxyl sulfate | von Willebrand factor | Tissue factor | Tryptophan derivatives | Uremic toxin | Cardiovascular disease | Chronic kidney disease | Monocytes activation | MORTALITY | KAPPA-B ACTIVATION | OXIDATIVE STRESS | RISK-FACTORS | KYNURENINE PATHWAY | ARYL-HYDROCARBON RECEPTOR | INFLAMMATION | ENDOTHELIAL-CELLS | UROLOGY & NEPHROLOGY | CARDIORENAL SYNDROME | EXPRESSION | Prevalence | C-Reactive Protein | Oxidative Stress | Tissue Plasminogen Activator - metabolism | Humans | Middle Aged | Male | Case-Control Studies | Renal Insufficiency, Chronic - epidemiology | Renal Insufficiency, Chronic - metabolism | Plasminogen Activator Inhibitor 1 - metabolism | Hemostasis | Lipoproteins - metabolism | Cardiovascular Diseases - epidemiology | Adult | Female | Prothrombin - metabolism | von Willebrand Factor - metabolism | Peptide Fragments - metabolism | Cardiovascular Diseases - metabolism | Inflammation | Hydrogen Peroxide - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Urokinase-Type Plasminogen Activator - metabolism | Indican - metabolism | Aged | Thrombin - metabolism | Thromboplastin - metabolism | Neopterin - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Physiological aspects | Toxins | Chronic kidney failure | Cardiovascular diseases | Health aspects | Index Medicus
Journal Article
Journal of Lipid Research, ISSN 0022-2275, 11/2013, Volume 54, Issue 11, pp. 3085 - 3097
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 09/2014, Volume 229, Issue 9, pp. 1292 - 1300
The plasma level of the regulatory metabolite adenosine increases during the activation of coagulation and inflammation. Here we investigated the effect of... 
ADHESION | PHYSIOLOGY | ACTIVATED PROTEIN-C | PERMEABILITY | SPECIFICITY | PATHWAY | INFLAMMATION | MECHANISMS | KAPPA-B | RECEPTORS | EXPRESSION | CELL BIOLOGY | Capillary Permeability | Human Umbilical Vein Endothelial Cells - metabolism | Coculture Techniques | Humans | Human Umbilical Vein Endothelial Cells - immunology | NF-kappa B - metabolism | rhoA GTP-Binding Protein - metabolism | Adenosine A2 Receptor Agonists - metabolism | Inflammation - blood | Dose-Response Relationship, Drug | Inflammation - metabolism | Thrombin - pharmacology | Transfection | Adenosine A2 Receptor Agonists - pharmacology | RNA Interference | Inflammation Mediators - metabolism | Receptor, Adenosine A2A - genetics | Cyclic AMP - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Cells, Cultured | E-Selectin - metabolism | Inflammation - immunology | Adenosine - pharmacology | Receptor, Adenosine A2A - metabolism | Cell Adhesion | Receptor, Adenosine A2B - metabolism | Protein Transport | Adenosine A2 Receptor Antagonists - pharmacology | Intercellular Adhesion Molecule-1 - metabolism | Blood Coagulation - drug effects | Signal Transduction - drug effects | Adenosine - metabolism | Enzyme Activation | Thrombin - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Thrombin | Adenosine | Analysis | Endothelium | Index Medicus | Adenosine receptor | Permeability
Journal Article
Brain Research, ISSN 0006-8993, 2016, Volume 1657, pp. 167 - 175
Graphical abstract 
Neurology | Calcium signaling | Blood-brain barrier | Cerebral microvasculature | Protease-activated receptor 1 | blood-brain barrier | L-NAME | nitric oxide | BBB | protease-activated receptor 1 | RBMVEC | NG-nitro-L-arginine methyl ester | concentration | units | rat brain microvascular endothelial cells | PAR1 | inositol 1,4,5-trisphosphate | Ca | cytosolic Ca | ENTRY | BARRIER PERMEABILITY | MITOCHONDRIA | RECEPTOR | CYTOSOLIC CALCIUM | NEUROSCIENCES | IN-VITRO | INDUCED INCREASE | PHARMACOLOGY | NITRIC-OXIDE | CHANNELS | Capillary Permeability - physiology | Thrombin - administration & dosage | Reactive Oxygen Species - metabolism | Calcium - metabolism | Receptor, PAR-1 - metabolism | Calcium Signaling - physiology | Humans | Actins - metabolism | Microvessels - metabolism | Capillary Permeability - drug effects | Endoplasmic Reticulum - metabolism | Nitric Oxide Synthase - antagonists & inhibitors | Cardiovascular Agents - metabolism | Inositol 1,4,5-Trisphosphate Receptors - metabolism | Endoplasmic Reticulum - drug effects | Tight Junctions - drug effects | Tight Junctions - metabolism | Cell Line | Cardiovascular Agents - administration & dosage | Receptor, PAR-1 - antagonists & inhibitors | Endothelial Cells - metabolism | Microvessels - drug effects | Rats | Mitochondria - metabolism | Mitochondria - drug effects | Blood-Brain Barrier - drug effects | Blood-Brain Barrier - metabolism | Animals | Calcium Signaling - drug effects | Nitric Oxide Synthase - metabolism | Thrombin - metabolism | Nitric Oxide - metabolism | Endothelial Cells - drug effects | Thrombin | Endothelium | Medical colleges | Inositol | Actin | Nitric oxide | Permeability | Muscle proteins | Index Medicus | cerebral microvasculature | calcium signaling
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 879 - 15
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 06/2007, Volume 18, Issue 6, pp. 2002 - 2012
Reactive oxygen species (ROS) have been implicated in both cell signaling and pathology. A major source of ROS in endothelial cells is NADPH oxidase, which... 
Index Medicus
Journal Article