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1. Full Text Extracorporeal photochemotherapy for the treatment of steroid-resistant chronic GVHD
by Couriel, Daniel R and Hosing, Chitra and Saliba, Rima and Shpall, Elizabeth J and Anderlini, Paolo and Rhodes, Beverly and Smith, Veronica and Khouri, Issa and Giralt, Sergio and De Lima, Marcos and Hsu, Yvonne and Ghosh, Shubhra and Neumann, Joyce and Andersson, Borje and Qazilbash, Muzzafar and Hymes, Sharon and Kim, Stella and Champlin, Richard and Donato, Michele
Blood, ISSN 0006-4971, 04/2006, Volume 107, Issue 8, pp. 3074 - 3080
Chronic graft-versus-host disease (GVHD) is a major limitation of successful allogeneic hematopoietic stem cell transplantation (HSCT). Extracorporeal... 
PREDICTORS | PEDIATRIC-PATIENTS | T-CELL LYMPHOMA | PHOTOPHERESIS THERAPY | MECHANISMS | PARAMETERS | COMBINATION | HEMATOLOGY | VERSUS-HOST-DISEASE | TRANSPLANTATION | LYMPHOCYTES | Anemia, Sickle Cell - pathology | Graft vs Host Disease - therapy | Eye - pathology | Liver - pathology | Immunosuppression - methods | Humans | Middle Aged | Child, Preschool | Male | Hematopoietic Stem Cell Transplantation - mortality | Anemia, Aplastic - therapy | Transplantation, Homologous | Breast Neoplasms - therapy | Immunosuppression - mortality | Anemia, Aplastic - mortality | Graft vs Host Disease - mortality | Immunosuppression - adverse effects | Photopheresis - mortality | Adult | Female | Photopheresis - methods | Retrospective Studies | Steroids - therapeutic use | Mouth Mucosa - pathology | Anemia, Aplastic - complications | Child | Graft vs Host Disease - etiology | Skin - pathology | Anemia, Aplastic - pathology | Steroids - adverse effects | Anemia, Sickle Cell - complications | Treatment Outcome | Drug Resistance - drug effects | Thrombocytopenia - pathology | Lymphoproliferative Disorders - pathology | Graft vs Host Disease - pathology | Anemia, Sickle Cell - therapy | Breast Neoplasms - complications | Disease-Free Survival | Lymphoproliferative Disorders - mortality | Anemia, Sickle Cell - mortality | Breast Neoplasms - pathology | Adolescent | Breast Neoplasms - mortality | Aged | Hematopoietic Stem Cell Transplantation - methods | Chronic Disease
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2. Full Text Thrombocytopenic coagulopathy (Kasabach-Merritt Phenomenon) is associated with Kaposiform hemangioendothelioma and not with common infantile hemangioma
by Sarkar, Molly and Mulliken, John B and Kozakewich, Harry P. W and Robertson, Richard L and Burrows, Patricia E
Plastic and Reconstructive Surgery, ISSN 0032-1052, 1997, Volume 100, Issue 6, pp. 1377 - 1386
Children with a large vascular tumor and associated Kasabach-Merritt coagulopathy respond inconsistently to therapy and have a high mortality rate. For this... 
TRANEXAMIC ACID | SURGERY | EPSILON-AMINOCAPROIC ACID | PREDNISONE | THERAPY | SPINDLE-CELL HEMANGIOENDOTHELIOMA | VASCULAR MALFORMATIONS | FIBROBLAST GROWTH-FACTOR | ALPHA-INTERFERON | INFANCY | GIANT HEMANGIOMA | Thoracic Neoplasms - therapy | Hemangioendothelioma - diagnosis | Blood Vessels - pathology | Humans | Retroperitoneal Neoplasms - therapy | Infant | Male | Antineoplastic Agents - therapeutic use | Recombinant Proteins | Ecchymosis - pathology | Basement Membrane - pathology | Retroperitoneal Neoplasms - diagnosis | Thrombocytopenia - complications | Hemangioendothelioma - pathology | Skin Neoplasms - diagnosis | Female | Retrospective Studies | Retroperitoneal Neoplasms - pathology | Infant, Newborn | Skin Neoplasms - pathology | Hemangioendothelioma - therapy | Skin Neoplasms - therapy | Adipose Tissue - pathology | Head and Neck Neoplasms - therapy | Interferon-alpha - therapeutic use | Thoracic Neoplasms - pathology | Retroperitoneal Neoplasms - complications | Hemangioma - diagnosis | Microscopy, Electron | Thrombocytopenia - pathology | Syndrome | Head and Neck Neoplasms - pathology | Skin Neoplasms - complications | Lymphoid Tissue - pathology | Magnetic Resonance Imaging | Purpura, Thrombocytopenic - pathology | Thoracic Neoplasms - diagnosis | Thoracic Neoplasms - complications | Interferon alpha-2 | Endothelium, Vascular - pathology | Head and Neck Neoplasms - complications | Head and Neck Neoplasms - diagnosis | Thrombocytopenia - diagnosis | Hemosiderin - analysis | Edema - pathology | Hemangioendothelioma - complications
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3. Full Text Hairy cell leukemia: Uncommon clinical features, unusual sites of involvement and some rare associations
by Tadmor, Tamar and Polliack, Aaron
Best Practice & Research: Clinical Haematology, ISSN 1521-6926, 2015, Volume 28, Issue 4, pp. 193 - 199
Abstract Unusual clinical manifestations and associations with auto-immunity or other systemic disorders are uncommon clinical features of hairy cell leukemia... 
Hematology, Oncology and Palliative Medicine | Rare manifestations | Clinical features | Hairy cell leukemia (HCL) | DIAGNOSIS | LYMPHADENOPATHY | COMPLICATIONS | HEMATOLOGY | Bone and Bones - pathology | Liver - pathology | Lymph Nodes - pathology | Humans | Male | Behcet Syndrome - pathology | Splenomegaly - pathology | Antiphospholipid Syndrome - complications | Thrombocytopenia - complications | Splenomegaly - complications | Splenomegaly - diagnosis | Liver - immunology | Anemia, Hemolytic, Autoimmune - pathology | Lymphatic Diseases - diagnosis | Anemia, Hemolytic, Autoimmune - diagnosis | Female | B-Lymphocytes - pathology | Leukemia, Hairy Cell - pathology | Skin - pathology | Skin - immunology | Leukemia, Hairy Cell - immunology | Antiphospholipid Syndrome - diagnosis | Lymphatic Diseases - immunology | Splenomegaly - immunology | Thrombocytopenia - immunology | Behcet Syndrome - complications | Lymphatic Diseases - complications | Anemia, Hemolytic, Autoimmune - complications | Lymph Nodes - immunology | Thrombocytopenia - pathology | Leukemia, Hairy Cell - diagnosis | Antiphospholipid Syndrome - immunology | B-Lymphocytes - drug effects | Leukemia, Hairy Cell - complications | Antiphospholipid Syndrome - pathology | Anemia, Hemolytic, Autoimmune - immunology | Sex Factors | Lymphatic Diseases - pathology | Thrombocytopenia - diagnosis | Behcet Syndrome - diagnosis | Bone and Bones - immunology | Behcet Syndrome - immunology | Leukemia, Hairy cell
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4. Full Text Inflammation and oxidative stress in viper bite: An insight within and beyond
by Sunitha, K and Hemshekhar, M and Thushara, R.M and Santhosh, M. Sebastin and Sundaram, M. Shanmuga and Kemparaju, K and Girish, K.S
Toxicon, ISSN 0041-0101, 05/2015, Volume 98, pp. 89 - 97
Though systemic and local manifestations of snakebite are considered serious, the relevance of oxidative stress in viper bite pathology is largely denied.... 
Hyaluronidase | Oxidative stress | Damage associated molecular patterns (DAMPs) | SVMPs | Secondary complications | Viper venom | Damage associated molecular patterns | DAMPs | ACUTE KIDNEY INJURY | AMINOPEPTIDASE ACTIVITIES | SRI-LANKA | RENAL-FUNCTION | DABOIA-RUSSELII | SNAKE-VENOM HYALURONIDASE | PHOSPHOLIPASES A | OVERLOOKED ISSUES | LOCAL TISSUE-DAMAGE | PHARMACOLOGY & PHARMACY | TOXICOLOGY | Damage associated molecular patterns (DAMPS) | DURISSUS-TERRIFICUS ENVENOMATION | Inflammation - pathology | Urogenital Abnormalities - etiology | Kidney - pathology | Humans | Homeostasis | Hemorrhage - prevention & control | Antivenins - therapeutic use | Necrosis - pathology | Snake Bites - pathology | Inflammation - drug therapy | Urogenital Abnormalities - prevention & control | Hemorrhage - etiology | Hypopituitarism - pathology | Hypopituitarism - prevention & control | Snake Bites - complications | Disease Models, Animal | Snake Bites - drug therapy | Thrombocytopenia - etiology | Hypopituitarism - etiology | Thrombocytopenia - pathology | Inflammation - etiology | Necrosis - prevention & control | Viper Venoms - toxicity | Antioxidants - therapeutic use | Urogenital Abnormalities - pathology | Animals | Kidney - abnormalities | Necrosis - etiology | Thrombocytopenia - prevention & control | Oxidative Stress - drug effects | Hemorrhage - pathology | Poisonous snakes | Antioxidants | Enzymes | Inflammation | Index Medicus | Stresses | Pathology | Turbulence | Fluid dynamics | Abnormalities | Disturbances | Exploration | Dampness
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5. Full Text Nontraditional Applications in Clinical Pathology
by Jordan, Holly L and Register, Thomas C and Tripathi, Niraj K and Bolliger, Anne Provencher and Everds, Nancy and Zelmanovic, David and Poitout, Florence and Bounous, Denise I and Wescott, Debra and Ramaiah, Shashi K
Toxicologic Pathology, ISSN 0192-6233, 10/2014, Volume 42, Issue 7, pp. 1058 - 1068
Most published reviews of preclinical toxicological clinical pathology focus on the fundamental aspects of hematology, clinical chemistry, coagulation, and... 
guinea pig | minipig | hamster | Advia | hematology | bone biomarkers | Wistar rat | rabbit | bone markers | clinical pathology | juvenile | transgenic | pregnant | Advia((R)) | PLATELET-FUNCTION | MARKERS | PATHOLOGY | CHEMISTRY | CYNOMOLGUS MONKEYS | TOXICOLOGY | SERUM BIOCHEMICAL VALUES | PREGNANCY | BIOMARKERS | THROMBOCYTOPENIA | MONKEYS MACACA-FASCICULARIS | BEAGLES | Rabbits | Cricetinae | Guinea Pigs | Humans | Rats | Biomarkers - blood | Pathology, Clinical - methods | Animals | Bone and Bones - metabolism | Dogs | Endpoint Determination | Primates | Mice | Drug Evaluation, Preclinical | Disease Models, Animal | Index Medicus
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6. Full Text Only severe thrombocytopenia results in bleeding and defective thrombus formation in mice
by Morowski, Martina and Voegtle, Timo and Kraft, Peter and Kleinschnitz, Christoph and Stoll, Guido and Nieswandt, Bernhard
Blood, ISSN 0006-4971, 06/2013, Volume 121, Issue 24, pp. 4938 - 4947
Platelets are essential mediators of hemostasis and thrombosis. Platelet counts (PCs) in humans average 250 platelets/nL, but it is not entirely clear how... 
PROTECTS MICE | DRUG-INDUCED THROMBOCYTOPENIA | GLYCOPROTEIN-VI | PRIMARY IMMUNE THROMBOCYTOPENIA | PLATELET COUNTS | PURPURA | CHRONIC HEPATITIS-C | ARTERIAL THROMBOSIS | HEMATOLOGY | ISCHEMIC-STROKE | DEFICIENCY | Blood Platelets - pathology | Carotid Arteries - metabolism | Hemorrhage - blood | Aorta - metabolism | Thrombocytopenia - pathology | Thrombocytopenia - blood | Aorta - pathology | Stroke - blood | Stroke - pathology | Thrombosis - pathology | Animals | Platelet Count | Blood Platelets - metabolism | Carotid Arteries - pathology | Mice | Thrombosis - blood | Hemorrhage - pathology | Disease Models, Animal
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7. Full Text Delineation of EFTUD2 Haploinsufficiency‐Related Phenotypes Through a Series of 36 Patients
by Lehalle, Daphné and Gordon, Christopher T and Oufadem, Myriam and Goudefroye, Géraldine and Boutaud, Lucile and Alessandri, Jean‐Luc and Baena, Neus and Baujat, Geneviève and Baumann, Clarisse and Boute‐Benejean, Odile and Caumes, Roseline and Decaestecker, Charles and Gaillard, Dominique and Goldenberg, Alice and Gonzales, Marie and Holder‐Espinasse, Muriel and Jacquemont, Marie‐Line and Lacombe, Didier and Manouvrier‐Hanu, Sylvie and Marlin, Sandrine and Mathieu‐Dramard, Michèle and Morin, Gilles and Pasquier, Laurent and Petit, Florence and Rio, Marlène and Smigiel, Robert and Thauvin‐Robinet, Christel and Vasiljevic, Alexandre and Verloes, Alain and Malan, Valérie and Munnich, Arnold and Pontual, Loïc and Vekemans, Michel and Lyonnet, Stanislas and Attié‐Bitach, Tania and Amiel, Jeanne
Human Mutation, ISSN 1059-7794, 04/2014, Volume 35, Issue 4, pp. 478 - 485
ABSTRACT Mandibulofacial dysostosis, Guion‐Almeida type (MFDGA) is a recently delineated multiple congenital anomalies/mental retardation syndrome... 
EFTUD2 | microcephaly | mandibulofacial dysostosis | spliceosome | Spliceosome | Microcephaly | Mandibulofacial dysostosis | ESOPHAGEAL ATRESIA | COMPLEX | MENTAL-RETARDATION | ANOMALIES | GENETICS & HEREDITY | OTO-FACIAL SYNDROME | MUTATIONS | SPECTRUM | Abnormalities, Multiple - pathology | Peptide Elongation Factors - metabolism | Microcephaly - genetics | Ear, External - pathology | Humans | Mandibulofacial Dysostosis - pathology | Child, Preschool | Ribonucleoprotein, U5 Small Nuclear - metabolism | Infant | Male | Anus, Imperforate - pathology | Ribonucleoprotein, U5 Small Nuclear - genetics | Intellectual Disability - genetics | Thrombocytopenia - genetics | Mandibulofacial Dysostosis - genetics | Hearing Loss, Bilateral - pathology | Microcephaly - pathology | Female | Child | Peptide Elongation Factors - genetics | Abnormalities, Multiple - genetics | Diagnosis, Differential | Ophthalmoplegia - pathology | Prenatal Diagnosis | Intellectual Disability - pathology | Hearing Loss, Bilateral - genetics | Thrombocytopenia - pathology | Haploinsufficiency | Pregnancy | Phenotype | Hand Deformities, Congenital - genetics | Anus, Imperforate - genetics | Ophthalmoplegia - genetics | Mutation | Hand Deformities, Congenital - pathology | Genetic aspects | Genetic disorders | Birth defects
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8. Full Text Lethal Crimean-Congo Hemorrhagic Fever Virus Infection in Interferon α/β Receptor Knockout Mice Is Associated With High Viral Loads, Proinflammatory Responses, and Coagulopathy
by Marko Zivcec and David Safronetz and Dana Scott and Shelly Robertson and Hideki Ebihara and Heinz Feldmann
The Journal of Infectious Diseases, ISSN 0022-1899, 6/2013, Volume 207, Issue 12, pp. 1909 - 1921
Crimean-Congo hemorrhagic fever (CCHF) is a widely distributed viral hemorrhagic fever characterized by rapid onset of flu-like symptoms often followed by... 
Pathology | Disease models | Interleukins | Tissue samples | Crimean Congo hemorrhagic fever virus | Viral diseases | Viruses | Infections | Platelets | Dosage | pathology | coagulopathy | proinflammatory response | thrombocytopenia | CCHFV | interferon α/β receptor knockout mice | CELLS | INFECTIOUS DISEASES | PREDICTOR | MICROBIOLOGY | MODEL | IMMUNOLOGY | PATHOGENESIS | TRANSMISSION | REPLICATION | DISSEMINATED INTRAVASCULAR COAGULATION | beta receptor knockout mice | CCHF VIRUS | AFRICAN | interferon alpha | Liver - virology | Spleen - virology | Liver - pathology | Viremia | Hemorrhagic Fever, Crimean - complications | Humans | Liver Failure, Acute - virology | Viral Load | Blood Coagulation Disorders - virology | Hemorrhagic Fever, Crimean - immunology | Receptor, Interferon alpha-beta - genetics | Spleen - pathology | Disease Models, Animal | Hemorrhagic Fever Virus, Crimean-Congo - physiology | Cytokines - metabolism | Lymphoid Tissue - virology | Mice, Inbred C57BL | Thrombocytopenia - immunology | Hemorrhagic Fever Virus, Crimean-Congo - immunology | Thrombocytopenia - pathology | Thrombocytopenia - virology | Disease Progression | Mice, Knockout | Lymphoid Tissue - pathology | Animals | Hemorrhagic Fever, Crimean - pathology | Blood Coagulation Disorders - pathology | Receptor, Interferon alpha-beta - immunology | Blood Coagulation Disorders - immunology | Liver Failure, Acute - immunology | Liver Failure, Acute - pathology | Chemokines - metabolism | Mice | Major and Brief Reports | β receptor knockout mice | interferon α
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9. Full Text Phase I/II study of the hypoxia-activated prodrug PR104 in refractory/relapsed acute myeloid leukemia and acute lymphoblastic leukemia
by Konopleva, Marina and Thall, Peter F and Yi, Cecilia Arana and Borthakur, Gautam and Coveler, Andrew and Bueso-Ramos, Carlos and Benito, Juliana and Konoplev, Sergej and Gu, Yongchuan and Ravandi, Farhad and Jabbour, Elias and Faderl, Stefan and Thomas, Deborah and Cortes, Jorge and Kadia, Tapan and Kornblau, Steven and Daver, Naval and Pemmaraju, Naveen and Nguyen, Hoang Q and Feliu, Jennie and Lu, Hongbo and Wei, Caimiao and Wilson, William R and Melink, Teresa J and Gutheil, John C and Andreeff, Michael and Estey, Elihu H and Kantarjian, Hagop
Haematologica, ISSN 0390-6078, 07/2015, Volume 100, Issue 7, pp. 927 - 934
We previously demonstrated vast expansion of hypoxic areas in the leukemic microenvironment and provided a rationale for using hypoxia-activated prodrugs.... 
STEM-CELLS | INHIBITION | PR-104A | ACUTE MYELOGENOUS LEUKEMIA | BONE-MARROW | I TRIAL | KETO REDUCTASE 1C3 | HIF-1-ALPHA | HEMATOLOGY | CANCER-THERAPY | CHEMOTHERAPY | Recurrence | Humans | Middle Aged | Male | Antineoplastic Agents, Alkylating - administration & dosage | Carbonic Anhydrases - metabolism | Thrombocytopenia - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Anemia - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Bone Marrow - metabolism | Enterocolitis - genetics | Antigens, Neoplasm - metabolism | Bone Marrow - drug effects | Biomarkers - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - complications | Gene Expression | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Leukemia, Myeloid, Acute - pathology | Anemia - chemically induced | Hypoxia - complications | Thrombocytopenia - chemically induced | Thrombocytopenia - pathology | Nitrogen Mustard Compounds - adverse effects | Remission Induction | Hypoxia - pathology | Antineoplastic Agents, Alkylating - adverse effects | Enterocolitis - pathology | Prodrugs - administration & dosage | Hypoxia - drug therapy | Neutropenia - metabolism | Nitrogen Mustard Compounds - administration & dosage | Carbonic Anhydrases - genetics | Anemia - pathology | Leukemia, Myeloid, Acute - complications | Prodrugs - metabolism | Leukemia, Myeloid, Acute - drug therapy | Neutropenia - pathology | Adult | Female | Neutropenia - chemically induced | Neutropenia - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Carbonic Anhydrase IX | Enterocolitis - chemically induced | Antigens, Neoplasm - genetics | Nitroimidazoles - pharmacology | Thrombocytopenia - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Anemia - metabolism | Prodrugs - adverse effects | Hypoxia - genetics | Nitrogen Mustard Compounds - metabolism | Bone Marrow - pathology | Aged | Enterocolitis - metabolism | Antineoplastic Agents, Alkylating - metabolism | Leukemia, Myeloid, Acute - genetics
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10. Full Text Episodic angioedema with eosinophilia (Gleich syndrome) is a multilineage cell cycling disorder
by Khoury, Paneez and Herold, Acqueline and Alpaugh, Alexandra and Dinerman, Ellen and Holland-Thomas, Nicole and Stoddard, Jennifer and Gurprasad, Shakuntala and Maric, Irina and Simakova, Olga and Schwartz, Lawrence B and Fong, Juelia and Richard Lee, Chyi-Chia and Xi, Liqiang and Wang, Zengfeng and Raffeld, Mark and Klion, Amy D
Haematologica, ISSN 0390-6078, 2015, Volume 100, Issue 3, pp. 300 - 307
Episodic angioedema with eosinophilia (Gleich syndrome) is a rare disorder characterized by episodes of angioedema and eosinophilia that occur at monthly... 
ACTIVATION | ELASTASE | SEVERE CONGENITAL NEUTROPENIA | HYPEREOSINOPHILIA | THROMBOCYTOPENIA | HISTAMINE-RELEASE | PERIPHERAL-BLOOD | PROLIFERATION | MUTATIONS | HEMATOLOGY | LYMPHOCYTES | Antigens, CD - immunology | Angioedema - complications | Bone Marrow - immunology | Humans | Middle Aged | Male | Angioedema - immunology | Eosinophils - immunology | Eosinophilia - pathology | Adult | Female | Eosinophils - pathology | T-Lymphocytes - pathology | Eosinophilia - diagnosis | Skin - pathology | Cytokines - immunology | Neutrophils - pathology | Skin - immunology | Mast Cells - immunology | Gene Expression | Angioedema - pathology | Neutrophils - immunology | Periodicity | Immunophenotyping | Syndrome | Eosinophilia - immunology | Bone Marrow - pathology | Mast Cells - pathology | Angioedema - diagnosis | T-Lymphocytes - immunology | Eosinophilia - complications | Cell Lineage - immunology
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11. Full Text Lenalidomide therapy in myelofibrosis with myeloid metaplasia
by Tefferi, Ayalew and Cortes, Jorge and Verstovsek, Srdan and Mesa, Ruben A and Thomas, Deborah and Lasho, Terra L and Hogan, William J and Litzow, Mark R and Allred, Jacob B and Jones, Dan and Byrne, Catriona and Zeldis, Jerome B and Ketterling, Rhett P and McClure, Rebecca F and Giles, Francis and Kantarjian, Hagop M
Blood, ISSN 0006-4971, 08/2006, Volume 108, Issue 4, pp. 1158 - 1164
We present results of 2 similarly designed but separate phase 2 studies involving single-agent lenalidomide (CC-5013, Revlimid) in a total of 68 patients with... 
MULTIPLE-MYELOMA | GROWTH-FACTOR-BETA | LOW-DOSE THALIDOMIDE | CC-5013 | MYELOPROLIFERATIVE-DISORDERS | ANGIOGENESIS | ANALOGS | MUTATION | CELL TRANSPLANTATION | MYELODYSPLASTIC-SYNDROMES | HEMATOLOGY | Humans | Male | Primary Myelofibrosis - pathology | Neovascularization, Pathologic - complications | Primary Myelofibrosis - blood | Thrombocytopenia - genetics | Thalidomide - analogs & derivatives | Anemia - genetics | Anemia - drug therapy | Anemia, Myelophthisic - genetics | Time Factors | Splenomegaly - blood | Neutropenia - blood | Thalidomide - adverse effects | Primary Myelofibrosis - complications | Splenomegaly - genetics | Administration, Oral | Anemia, Myelophthisic - pathology | Thrombocytopenia - chemically induced | Thalidomide - administration & dosage | Thrombocytopenia - pathology | Remission Induction | Neovascularization, Pathologic - drug therapy | Anemia, Myelophthisic - complications | Sequence Deletion | Splenomegaly - drug therapy | Anemia - blood | Anemia - pathology | Splenomegaly - pathology | Anemia, Myelophthisic - blood | Neovascularization, Pathologic - blood | Neovascularization, Pathologic - pathology | Janus Kinase 2 | Splenomegaly - complications | Protein-Tyrosine Kinases - genetics | Thrombocytopenia - drug therapy | Neutropenia - pathology | Adult | Female | Neutropenia - chemically induced | Neutropenia - genetics | Primary Myelofibrosis - drug therapy | Proto-Oncogene Proteins - genetics | Thrombocytopenia - blood | Anemia, Myelophthisic - drug therapy | Point Mutation | Anemia - complications | Primary Myelofibrosis - genetics | Platelet Count | Neovascularization, Pathologic - genetics | Aged | Hemoglobins - analysis
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12. Full Text Chronic murine typhoid fever is a natural model of secondary hemophagocytic lymphohistiocytosis
by Brown, Diane E and McCoy, Melissa W and Pilonieta, M. Carolina and Nix, Rebecca N and Detweiler, Corrella S
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, p. e9441
Hemophagocytic lymphohistiocytosis (HLH) is a hyper-inflammatory clinical syndrome associated with neoplastic disorders especially lymphoma, autoimmune... 
DIAGNOSIS | MUTANT MICE | MACROPHAGES | MALIGNANT HISTIOCYTOSIS | IN-VIVO | GENES | BIOLOGY | SALMONELLA-TYPHIMURIUM | INFECTION | IRON-DEFICIENCY | SPECTRUM | Lymphohistiocytosis, Hemophagocytic - etiology | Inflammation - pathology | Liver - pathology | Lymphohistiocytosis, Hemophagocytic - pathology | Typhoid Fever - complications | Humans | Bone Marrow Diseases - pathology | Splenomegaly - pathology | Mice, Inbred Strains | Thrombocytopenia - pathology | Salmonella Infections, Animal - complications | Host-Pathogen Interactions | Animals | Typhoid Fever - microbiology | Ferritins - blood | Salmonella typhi - physiology | Salmonella Infections, Animal - microbiology | Female | Mice | Chronic Disease | Fever - pathology | Disease Models, Animal | Lymphohistiocytosis, Hemophagocytic - blood | Anemia | Analysis | Typhoid fever | Lymphomas | Macrophages | Health aspects | Diseases | Water-borne diseases | Salmonella | Splenomegaly | Disease | Laboratories | Pathogenesis | Disorders | Chronic infection | Viruses | Iron | Infections | Veterinary medicine | Fungi | Lymphocytes | Rodents | Bone marrow | Bacteria | Spleen | Thrombocytopenia | Protozoa | Lymphatic system | Cytokines | Hematology | Developmental biology | Body temperature | Inflammation | Typhoid | Histiocytosis | Lymphoma | Fever | Pathology | Lipoproteins | Infectious diseases | Mutation | Lymphocytosis | Metabolic disorders | Viral infections
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13. Full Text Alveolar Septal Deposition of Immunoglobulin and Complement Parallels Pulmonary Hemorrhage in a Guinea Pig Model of Severe Pulmonary Leptospirosis
by Nally, Jarlath E and Chantranuwat, Chavit and Wu, Xiao-Yang and Fishbein, Michael C and Pereira, Martha M and da Silva, João José Pereira and Blanco, David R and Lovett, Michael A
The American Journal of Pathology, ISSN 0002-9440, 2004, Volume 164, Issue 3, pp. 1115 - 1127
Human patients suffering from leptospirosis present with a diverse array of clinical manifestations, including the more severe and often fatal pulmonary form... 
ANTIGENIC RELATIONSHIP | EQUINE CORNEA | PATHOGENESIS | LUNG | THROMBOCYTOPENIA | INFECTION | ELECTRON MICROSCOPY | INTERROGANS | PATHOLOGY | FEATURES | ICTEROHAEMORRHAGIAE | Immunohistochemistry | Liver - pathology | Spleen - immunology | Immunoglobulins - immunology | Kidney - pathology | Lung Diseases - microbiology | Kidney - immunology | Liver - microbiology | Complement System Proteins - immunology | Male | Intestines - immunology | Spleen - microbiology | Complement System Proteins - metabolism | Immunoglobulins - metabolism | Liver - immunology | Hemorrhage - etiology | Spleen - pathology | Leptospirosis - immunology | Disease Models, Animal | Guinea Pigs | Intestines - pathology | Pulmonary Alveoli - pathology | Autoimmune Diseases - complications | Microscopy, Electron | Pulmonary Alveoli - ultrastructure | Lung Diseases - immunology | Kidney - microbiology | Animals | Intestines - microbiology | Leptospirosis - pathology | Leptospira - immunology | Lung Diseases - pathology | Hemorrhage - pathology | Pulmonary Alveoli - immunology | Animal Model
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