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PLoS Medicine, ISSN 1549-1277, 2006, Volume 3, Issue 7, pp. 1140 - 1151
The JAK2V617F allele has recently been identified in patients with polycythemia vera (PV), essential thrombocytosis (ET), and myelofibrosis with myeloid... 
Recombinant Fusion Proteins - adverse effects | Humans | Myeloproliferative Disorders - pathology | Mutation, Missense | Primary Myelofibrosis - pathology | Receptors, Cytokine - physiology | Janus Kinases - physiology | Bone Marrow Transplantation | Hematopoiesis - physiology | Myeloid Cells - drug effects | Transcription, Genetic | Colony-Forming Units Assay | Phosphorylation - drug effects | mRNA Cleavage and Polyadenylation Factors - genetics | Hematologic Neoplasms - physiopathology | Disease Models, Animal | Cells, Cultured - drug effects | Bone Marrow Cells - pathology | Hematopoiesis - genetics | Signal Transduction - genetics | Thrombocytosis - etiology | Sequence Analysis, DNA | Cell Division - drug effects | mRNA Cleavage and Polyadenylation Factors - physiology | Receptor, Platelet-Derived Growth Factor alpha - physiology | Hematologic Neoplasms - drug therapy | Mice | Mice, Inbred BALB C | Myeloid Cells - pathology | Protein Processing, Post-Translational - genetics | Myeloproliferative Disorders - genetics | Oncogene Proteins, Fusion - physiology | STAT Transcription Factors - physiology | Protein Processing, Post-Translational - drug effects | Cells, Cultured - pathology | Bone Marrow Cells - drug effects | Spleen - pathology | Thrombocytosis - pathology | Gene Expression Regulation | Janus Kinases - antagonists & inhibitors | Point Mutation | Megakaryocytes - pathology | Primary Myelofibrosis - genetics | Animals | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Receptor, Platelet-Derived Growth Factor alpha - genetics | Myeloproliferative Disorders - etiology | Signal Transduction - physiology | Protein Kinase Inhibitors - pharmacology | Genetic Vectors | Megakaryocytes - drug effects | Cytokines - pharmacology | Amino Acid Substitution | Thrombocytosis - genetics | Genetic aspects | Myelofibrosis | Research | Hematology | Gene Therapy | Genomics | Genetics | Oncology | Cancer Biology | Hematology (including Blood Transfusion)
Journal Article
Journal of Immunology, ISSN 0022-1767, 01/2015, Volume 194, Issue 2, pp. 750 - 760
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e77819
Exome sequencing of primary tumors identifies complex somatic mutation patterns. Assignment of relevance of individual somatic mutations is difficult and poses... 
POLYCYTHEMIA-VERA | GAIN-OF-FUNCTION | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | TYROSINE KINASE JAK2 | ACQUIRED UNIPARENTAL DISOMY | MYELOPROLIFERATIVE NEOPLASMS | GENOMIC ABERRATIONS | TANDEM DUPLICATION | DNA-SEQUENCING DATA | ESSENTIAL THROMBOCYTHEMIA | Proto-Oncogene Proteins c-cbl - genetics | Histone-Lysine N-Methyltransferase | Humans | Polycythemia Vera - genetics | Proto-Oncogene Proteins - genetics | Chromosomes, Human, Pair 11 - genetics | DNA-Binding Proteins - genetics | Exome - genetics | Primary Myelofibrosis - genetics | Myeloid-Lymphoid Leukemia Protein - genetics | Adaptor Proteins, Signal Transducing - genetics | Chromosome Aberrations | Polymerase Chain Reaction | LIM Domain Proteins - genetics | Myelodysplastic Syndromes - genetics | Leukemia, Myeloid, Acute - genetics | Thrombocytosis - genetics | Genetic research | Genetic aspects | Gene mutations | Analysis | Genes | Cancer | Target recognition | Disease | Leukemia | Oncology | Genomes | Single-nucleotide polymorphism | Gene deletion | DNA repair | Defects | Proteins | Chromosome 11 | Lesions | Chromosomes | Bioinformatics | Deoxyribonucleic acid--DNA | Chronic illnesses | Hematology | Internal medicine | Myeloid leukemia | Medicine | Hospitals | DNA microarrays | Point mutation | Cytogenetics | Tumor suppressor genes | Genetic engineering | Mutation | Aberration | Acute myeloid leukemia | Chromosome aberrations | Tumors | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, p. e57915
Obesity and type 2 diabetes are associated with increased production of Galectin-3 (Gal-3), a protein that modulates inflammation and clearance of glucose... 
INDUCED GLOMERULAR INJURY | RECEPTOR-MEDIATED MECHANISMS | ACTIVATION | GALECTIN-3/AGE-RECEPTOR-3 KNOCKOUT MICE | LIPOXIDATION | MULTIDISCIPLINARY SCIENCES | FATTY LIVER | AGE-RECEPTOR | DIET-INDUCED OBESITY | INSULIN SENSITIVITY | EXPRESSION | Galectin 3 - metabolism | Glucose Tolerance Test - methods | Fibroblast Growth Factors - genetics | Monocytes - metabolism | PPAR gamma - metabolism | Obesity - genetics | Galectin 3 - deficiency | Fibroblast Growth Factors - metabolism | Inflammation - metabolism | Adiponectin - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Adiponectin - metabolism | Insulin - genetics | PPAR gamma - genetics | Liver - metabolism | Glucose - genetics | Lipase - metabolism | Transcription Factors - genetics | Galectin 3 - genetics | Adiposity - genetics | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Diet | Insulin Resistance - genetics | Glucose - metabolism | Inflammation - genetics | Mice, Obese | Adiposity - physiology | Mice | Lipase - genetics | Type 2 diabetes | Obesity | Glucose metabolism | Blood sugar monitoring | Liver | Physiological aspects | Inflammation | Glucose | Glucose tolerance tests | Insulin | Statistics | Dextrose | Fibroblast growth factor | Oxidative stress | Adipose tissue | Microflora | Adipocytes | Galectin-3 | Adiponectin | Proteins | Microbiota | Hyperglycemia | Rodents | Thrombocytosis | Age | Adducts | Heart failure | Medical research | Fasting | Cytokines | Anemia | Diabetes mellitus | Metabolism | Glucose tolerance | Nutrition research | Pathology | Monocytes | Acute phase substances | Antibiotics | Neutrophilia | Diabetes | Kinesiology
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2015, Volume 212, Issue 12, pp. 2133 - 2146
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Journal Article
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