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1. Full Text A large-scale candidate gene association study of age at menarche and age at natural menopause
by He, Chunyan and Kraft, Peter and Chasman, Daniel I and Buring, Julie E and Chen, Constance and Hankinson, Susan E and Paré, Guillaume and Chanock, Stephen and Ridker, Paul M and Hunter, David J
Human genetics, ISSN 0340-6717, 11/2010, Volume 128, Issue 5, pp. 515 - 527
Human Genetics | Gene Function | Molecular Medicine | Biomedicine | Metabolic Diseases | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Fundamental and applied biological sciences. Psychology | Classical genetics, quantitative genetics, hybrids | Human | Biological and medical sciences | Genetics of eukaryotes. Biological and molecular evolution | Puberty, Delayed - genetics | Women's Health | Age Factors | Humans | Menopause - genetics | Middle Aged | Obesity - genetics | Puberty, Precocious - genetics | Gonadal Steroid Hormones - genetics | Nurses | Female | Tobacco Use Disorder - genetics | Child | Somatomedins - metabolism | Thrombophilia - genetics | Genetic Predisposition to Disease | Gonadal Steroid Hormones - metabolism | Genetic Association Studies | Signal Transduction | Menarche - genetics | Genotype | Linear Models | Smoking - genetics | Menarche - metabolism | Obesity - metabolism | Phenotype | Menopause - metabolism | Transforming Growth Factor beta - genetics | Somatomedins - genetics | Adolescent | Age of Onset | Polycystic Ovary Syndrome - genetics | Primary Ovarian Insufficiency - genetics | Polymorphism, Single Nucleotide | Thrombophilia - metabolism | Transforming Growth Factor beta - metabolism | Women | Physiological aspects | Health aspects | Genes | Menopause | Genomics | Index Medicus | age at menarche | biologically plausible pathways | candidate genes | age at natural menopause
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2. Full Text Role of hemostatic gene polymorphisms in venous and arterial thrombotic disease
by Lane, David A and Grant, Peter J
Blood, ISSN 0006-4971, 03/2000, Volume 95, Issue 5, pp. 1517 - 1532
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Biological and medical sciences | Medical sciences | Platelet diseases and coagulopathies | Hyperhomocysteinemia - epidemiology | Sequence Deletion | Humans | Thromboembolism - epidemiology | Arterial Occlusive Diseases - genetics | Thrombophilia - epidemiology | Tissue Plasminogen Activator - genetics | Factor XIII - genetics | Plasminogen Activator Inhibitor 1 - genetics | 3' Untranslated Regions | Thromboembolism - genetics | Thrombophilia - genetics | Factor VII - genetics | Genetic Predisposition to Disease | Risk Factors | Arterial Occlusive Diseases - epidemiology | Hemostasis - genetics | Platelet Membrane Glycoproteins - genetics | Blood Coagulation Factors - genetics | Fibrinogen - genetics | Polymorphism, Genetic | Point Mutation | Fibrinolysis - genetics | Venous Thrombosis - epidemiology | Hyperhomocysteinemia - genetics | Prothrombin - genetics | Factor V - genetics | Venous Thrombosis - genetics | Receptors, Cell Surface - genetics | Index Medicus | Abridged Index Medicus
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3. Full Text Genetics of venous thrombosis
by ROSENDAAL, F. R and REITSMA, P. H
Journal of thrombosis and haemostasis, ISSN 1538-7933, 07/2009, Volume 7, Issue 1, pp. 301 - 304
epidemiology | thrombophilia | risk factors | genetics | venous thrombosis | Thrombophilia | Genetics | Venous thrombosis | Epidemiology | Risk factors | Genetic Predisposition to Disease | Blood Coagulation Factors - genetics | Venous Thrombosis - genetics | Humans | Risk Factors | Pulmonary embolism | Fibrin | Protein C | Genetic aspects | Thrombophlebitis | Blood coagulation factors | Index Medicus
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4. Full Text ACMG practice guideline: Lack of evidence for MTHFR polymorphism testing
by Hickey, Scott E and Curry, Cynthia J and Toriello, Helga V
Genetics in medicine, ISSN 1098-3600, 02/2013, Volume 15, Issue 2, pp. 153 - 156
homocysteine | thrombophilia | thrombosis | folic acid | thermolabile variant | MTHFR polymorphism | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Thrombophilia - genetics | Genetic Predisposition to Disease - genetics | Genetics, Medical - methods | Genetic Testing - standards | Genetics, Medical - standards | United States | Humans | Risk Factors | Genomics - standards | Coronary Disease - diagnosis | Polymorphism, Genetic | Thrombophilia - diagnosis | Venous Thromboembolism - genetics | Coronary Disease - genetics | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | Genomics - organization & administration | Genetics, Medical - organization & administration | Hyperhomocysteinemia - diagnosis | Hyperhomocysteinemia - genetics | Genomics - methods | Practice Guidelines as Topic | Venous Thromboembolism - diagnosis | Index Medicus | Abortion
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5. Full Text Multiple thrombophilic single nucleotide polymorphisms lack a significant effect on outcomes in fresh IVF cycles: an analysis of 1717 patients
by Patounakis, George and Patounakis, George and Bergh, Eric and Bergh, Eric and Forman, Eric J and Forman, Eric J and Tao, Xin and Tao, Xin and Lonczak, Agnieszka and Lonczak, Agnieszka and Franasiak, Jason M and Franasiak, Jason M and Treff, Nathan and Treff, Nathan and Scott Jr, Richard T and Scott Jr, Richard T
Journal of assisted reproduction and genetics, ISSN 1058-0468, 1/2016, Volume 33, Issue 1, pp. 67 - 73
Human Genetics | Thrombophilia | Pregnancy | Medicine & Public Health | SNP | Gynecology | Infertility | IVF outcomes | Reproductive Medicine | Genetics & Heredity | Life Sciences & Biomedicine | Obstetrics & Gynecology | Reproductive Biology | Science & Technology | Thrombophilia - genetics | Genetic Predisposition to Disease | Thrombophilia - pathology | Infertility - pathology | Humans | Embryo Implantation - genetics | Fibrinogen - genetics | Infertility - genetics | Factor XIII - genetics | Fertilization in Vitro | Antigens, Human Platelet - genetics | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | Adult | Female | Prothrombin - genetics | Factor V - genetics | Polymorphism, Single Nucleotide | Mutation | Plasminogen Activator Inhibitor 1 - genetics | Pregnancy Outcome - genetics | Fertilization in vitro | Women | Fibrin | Medical colleges | Analysis | Fibrinogen | Resveratrol | Thrombin | Genetic aspects | Single nucleotide polymorphisms | Chromosomes | Health aspects | Index Medicus | Genetics
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6. Full Text The interaction effect of angiogenesis and endothelial dysfunction-related gene variants increases the susceptibility of recurrent pregnancy loss
by Trifonova, E.A and Swarovskaya, M.G and Ganzha, O.A and Voronkova, O.V and Gabidulina, T.V and Stepanov, V.A
Journal of assisted reproduction and genetics, ISSN 1058-0468, 2019, Volume 36, Issue 4, pp. 717 - 726
Recurrent miscarriage | Endothelial dysfunction | Gene–gene interactions | Single-nucleotide polymorphism | Genetics & Heredity | Life Sciences & Biomedicine | Obstetrics & Gynecology | Reproductive Biology | Science & Technology | Thrombophilia - genetics | Genetic Predisposition to Disease | Abortion, Spontaneous - genetics | Genetic Association Studies | Thrombophilia - physiopathology | Humans | Risk Factors | Abortion, Spontaneous - physiopathology | Genotype | Epistasis, Genetic - genetics | Vascular Endothelial Growth Factor A - genetics | Nitric Oxide Synthase Type III - genetics | Pregnancy | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | Polymorphism, Restriction Fragment Length - genetics | Alleles | Polymorphism, Single Nucleotide - genetics | Adult | Female | Women | Medical research | Endothelial growth factors | Genes | Development and progression | Disease susceptibility | Endothelium | Anopheles | Pregnant women | Analysis | Medicine, Experimental | Genetic research | Genetic aspects | Single nucleotide polymorphisms | Health aspects | Pregnancy complications | Epistasis | Methylenetetrahydrofolate reductase | Abortion | Gene polymorphism | Polymerase chain reaction | Angiogenesis | Restriction fragment length polymorphism | Genotyping | Etiology | NOS3 protein | Miscarriage | Vascular endothelial growth factor | Index Medicus | Genetics
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7. Full Text Deficiencies of antithrombin, protein C and protein S – Practical experience in genetic analysis of a large patient cohort
by M. Caspers and A. Pavlova and J. Driesen and U. Harbrecht and R. Klamroth and J. Kadar and R. Fischer and B. Kemkes-Matthes and J. Oldenburg
Thrombosis and haemostasis, ISSN 0340-6245, 08/2012, Volume 108, Issue 2, pp. 247 - 257
thrombophilia | Protein C | genetic analysis | protein S | Antithrombin | Blood Coagulation, Fibrinolysis and Cellular Haemostasis | Thrombophilia | Protein S | Genetic analysis | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Fundamental and applied biological sciences. Psychology | Hematologic and hematopoietic diseases | Biological and medical sciences | Blood coagulation. Blood cells | Molecular and cellular biology | Medical sciences | Platelet diseases and coagulopathies | Thrombophilia - genetics | Prevalence | Protein S - genetics | Humans | Genotype | Male | Antithrombin III Deficiency - genetics | Mutation, Missense | Antithrombin III - genetics | Protein S Deficiency - genetics | DNA Mutational Analysis | Female | Models, Genetic | Mutation | Protein C Deficiency - genetics | Germany | Protein C - genetics | Cohort Studies
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