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Journal Article
BIOORGANIC & MEDICINAL CHEMISTRY LETTERS, ISSN 0960-894X, 06/2013, Volume 23, Issue 11, pp. 3239 - 3243
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2012, Volume 32, Issue 9, pp. 2185 - 2198
OBJECTIVE—Blood-sucking arthropods’ salivary glands contain a remarkable diversity of antihemostatics. The aim of the present study was to identify the unique... 
hematophagy | thrombosis | leishmaniasis | microcirculation | vector biology | BLOOD-COAGULATION | COAGULATION-FACTOR-XA | PLATELET-AGGREGATION | ANCYLOSTOMA-CANINUM | IXODES-SCAPULARIS | ANTICOAGULANT PEPTIDE | BREAST-CANCER | HEMATOPHAGOUS ANIMALS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ARTHROPOD SALIVA | TISSUE FACTOR | Surface Plasmon Resonance | Molecular Weight | Factor Xa Inhibitors | Humans | Fibrinolytic Agents - isolation & purification | Thromboplastin - antagonists & inhibitors | Inflammation - blood | Inflammation - metabolism | Time Factors | Anti-Inflammatory Agents - isolation & purification | Insect Proteins - isolation & purification | Disease Models, Animal | Amino Acid Sequence | Recombinant Proteins - antagonists & inhibitors | Anti-Inflammatory Agents - pharmacology | Rats | Factor Xa - metabolism | Salivary Glands - chemistry | Thrombosis - metabolism | Partial Thromboplastin Time | Insect Proteins - chemistry | Cell Line, Tumor | Calorimetry | Inflammation - prevention & control | Mice | Fibrinolytic Agents - chemistry | Thrombosis - chemically induced | Prothrombin Time | Receptor, PAR-2 - metabolism | Molecular Sequence Data | Dose-Response Relationship, Drug | Receptor, PAR-2 - antagonists & inhibitors | Chlorides | Cloning, Molecular | HEK293 Cells | Female | Thrombosis - blood | Thrombosis - prevention & control | Recombinant Proteins - metabolism | Insect Proteins - pharmacology | Mice, Inbred C57BL | Psychodidae - chemistry | Ferric Compounds | Fibrinolytic Agents - pharmacology | Animals | Anti-Inflammatory Agents - chemistry | Blood Coagulation - drug effects | Protein Binding | Thromboplastin - metabolism | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology: Journal of the American Heart Association, ISSN 1079-5642, 04/2000, Volume 20, Issue 4, pp. 1162 - 1167
Journal Article
JOURNAL OF BIOLOGICAL CHEMISTRY, ISSN 0021-9258, 08/2017, Volume 292, Issue 33, pp. 13688 - 13701
Cell migration and invasion are very characteristic features of cancer cells that promote metastasis, which is one of the most common causes of mortality among... 
EPITHELIAL-MESENCHYMAL TRANSITION | MIGRATION | CELLS | CYCLIN D1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEINS | EXPRESSION | BINDING | TISSUE FACTOR | INSIGHTS | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Humans | Receptor, PAR-2 - metabolism | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Breast - metabolism | Proto-Oncogene Proteins c-akt - genetics | Thromboplastin - agonists | Breast Neoplasms - metabolism | Breast Neoplasms - enzymology | Factor VIIIa - metabolism | Receptor, PAR-2 - antagonists & inhibitors | RNA Interference | Neoplasm Invasiveness - pathology | Female | Glycogen Synthase Kinase 3 beta - chemistry | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Breast - pathology | Phosphatidylinositol 3-Kinase - metabolism | Receptor, PAR-2 - genetics | Recombinant Proteins - metabolism | Breast - cytology | beta Catenin - agonists | Enzyme Inhibitors - pharmacology | Recombinant Proteins - chemistry | Glycogen Synthase Kinase 3 beta - antagonists & inhibitors | Factor VIIIa - genetics | Glycogen Synthase Kinase 3 beta - metabolism | beta Catenin - metabolism | beta Catenin - genetics | Phosphatidylinositol 3-Kinase - chemistry | Cell Movement - drug effects | Neoplasm Proteins - agonists | Signal Transduction - drug effects | Breast Neoplasms - pathology | beta Catenin - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - agonists | Thromboplastin - genetics | Receptor, PAR-2 - agonists | Cell Line, Tumor | Genes, Reporter - drug effects | Thromboplastin - metabolism | Oligopeptides - pharmacology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Index Medicus | coagulation factor | β-catenin | breast cancer | signaling | Signal Transduction | tissue factor
Journal Article
Journal Article
2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC), ISSN 1094-687X, 07/2013, Volume 2013, pp. 4118 - 4120
Conference Proceeding
Blood, ISSN 0006-4971, 01/2015, Volume 125, Issue 4, pp. 710 - 719
The precise mechanism for reduced thrombosis in prekallikrein null mice (Klkb1(-/-)) is unknown. Klkb1(-/-) mice have delayed carotid artery occlusion times on... 
PLASMA KALLIKREIN | MOLECULAR-WEIGHT KININOGEN | ENDOTHELIAL-CELLS | IN-VIVO | NITRIC-OXIDE | FACTOR EXPRESSION | VASCULAR-PERMEABILITY | DEFICIENT MICE | CONTACT ACTIVATION | HEMATOLOGY | FACTOR-XII | Epoprostenol - genetics | Receptor, Bradykinin B2 - genetics | Receptors, G-Protein-Coupled - metabolism | Epoprostenol - biosynthesis | Peptide Fragments - pharmacology | Carotid Artery Thrombosis - metabolism | Pyrones - pharmacology | Sirtuin 1 - genetics | Thromboplastin - antagonists & inhibitors | Nerve Tissue Proteins - biosynthesis | Synaptotagmins - biosynthesis | Carotid Artery Thrombosis - chemically induced | Prekallikrein | Thromboplastin - biosynthesis | Proto-Oncogene Proteins - metabolism | Kruppel-Like Transcription Factors - biosynthesis | Proto-Oncogene Proteins - antagonists & inhibitors | Angiotensin II - pharmacology | Receptor, Bradykinin B2 - biosynthesis | Sirtuin 1 - biosynthesis | Proto-Oncogene Proteins - genetics | Imidazoles - pharmacology | Angiotensin II - analogs & derivatives | Sulfonamides - pharmacology | Nerve Tissue Proteins - genetics | Sirtuin 1 - antagonists & inhibitors | Mice, Knockout | Naphthalenes - pharmacology | Animals | Partial Thromboplastin Time | Thromboplastin - genetics | Receptors, G-Protein-Coupled - antagonists & inhibitors | Mice | Receptors, G-Protein-Coupled - genetics | Synaptotagmins - genetics | Carotid Artery Thrombosis - genetics | Carotid Artery Thrombosis - pathology | Kruppel-Like Transcription Factors - antagonists & inhibitors | Kruppel-Like Transcription Factors - genetics | RNA, Messenger | Index Medicus | Abridged Index Medicus | Thrombosis and Hemostasis
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 12/2018, Volume 118, Issue 12, pp. 2145 - 2151
Abstract Background  The stroke severity or functional outcomes could differ because the efficacy of non-vitamin K antagonist oral anticoagulants (NOACs) could... 
Stroke, Systemic or Venous Thromboembolism | atrial fibrillation | stroke | outcome | NOAC | ASIAN PATIENTS | PREVENTION | PARTIAL THROMBOPLASTIN TIME | ATRIAL-FIBRILLATION | ANTITHROMBOTIC TREATMENT | DABIGATRAN | WARFARIN | ACUTE ISCHEMIC-STROKE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY
Journal Article
JOURNAL OF THROMBOSIS AND HAEMOSTASIS, ISSN 1538-7933, 03/2007, Volume 5, Issue 3, pp. 571 - 576
Background: Blockade of the thrombin receptors protease-activated receptor (PAR)1 and PAR4 with pepducins, cell-penetrating lipopeptides based on the third... 
protease-activated receptors | thrombin | cell penetrating antagonists | ANALOGS | BIOLOGY | PERIPHERAL VASCULAR DISEASE | MODEL | HEMATOLOGY | platelets | PROBES | Lipoproteins - therapeutic use | Receptors, Proteinase-Activated - metabolism | Humans | Carotid Artery, Common - surgery | Cell Membrane Permeability | Fibrinolytic Agents - metabolism | Male | Microscopy, Video | Receptors, Thrombin - antagonists & inhibitors | Dose-Response Relationship, Drug | Flow Cytometry | Time Factors | Fibrinolytic Agents - therapeutic use | Lipoproteins - metabolism | Fluorescence Resonance Energy Transfer | Platelet Aggregation Inhibitors - pharmacology | Cell Membrane - metabolism | Platelet Aggregation - drug effects | Platelet Aggregation Inhibitors - metabolism | Thrombosis - blood | Thrombosis - prevention & control | Blood Platelets - drug effects | Platelet Aggregation Inhibitors - therapeutic use | Disease Models, Animal | Receptor, PAR-1 - antagonists & inhibitors | Anticoagulants - metabolism | Anticoagulants - therapeutic use | Receptors, Proteinase-Activated - antagonists & inhibitors | Fibrinolytic Agents - pharmacology | Thrombosis - metabolism | Animals | Blood Platelets - metabolism | Lipoproteins - pharmacology | Anticoagulants - pharmacology | Mice | Thrombin - metabolism | Thromboplastin - metabolism | In Vitro Techniques | Index Medicus
Journal Article