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BMC Nephrology, ISSN 1471-2369, 01/2017, Volume 18, Issue 1, pp. 35 - 35
Background: During chronic kidney disease progression, kidney-specific risk factors for cardiovascular disease come into play. The present study investigated... 
Prothrombotic state | Hemostatic disorder | Indoxyl sulfate | von Willebrand factor | Tissue factor | Tryptophan derivatives | Uremic toxin | Cardiovascular disease | Chronic kidney disease | Monocytes activation | MORTALITY | KAPPA-B ACTIVATION | OXIDATIVE STRESS | RISK-FACTORS | KYNURENINE PATHWAY | ARYL-HYDROCARBON RECEPTOR | INFLAMMATION | ENDOTHELIAL-CELLS | UROLOGY & NEPHROLOGY | CARDIORENAL SYNDROME | EXPRESSION | Prevalence | C-Reactive Protein | Oxidative Stress | Tissue Plasminogen Activator - metabolism | Humans | Middle Aged | Male | Case-Control Studies | Renal Insufficiency, Chronic - epidemiology | Renal Insufficiency, Chronic - metabolism | Plasminogen Activator Inhibitor 1 - metabolism | Hemostasis | Lipoproteins - metabolism | Cardiovascular Diseases - epidemiology | Adult | Female | Prothrombin - metabolism | von Willebrand Factor - metabolism | Peptide Fragments - metabolism | Cardiovascular Diseases - metabolism | Inflammation | Hydrogen Peroxide - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Urokinase-Type Plasminogen Activator - metabolism | Indican - metabolism | Aged | Thrombin - metabolism | Thromboplastin - metabolism | Neopterin - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Physiological aspects | Toxins | Chronic kidney failure | Cardiovascular diseases | Health aspects | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2010, Volume 120, Issue 5, pp. 1396 - 1399
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2013, Volume 123, Issue 3, pp. 1310 - 1322
Coagulation is a host defense system that limits the spread of pathogens. Coagulation proteases, such as thrombin, also activate cells by cleaving PARs. In... 
TOLL-LIKE RECEPTOR-3 | MEDICINE, RESEARCH & EXPERIMENTAL | NATURAL-KILLER-CELLS | ISCHEMIA/REPERFUSION INJURY | PROTEASE-ACTIVATED RECEPTORS | VIRUS-INFECTION | IN-VIVO | ENDOTHELIAL-CELLS | FACTOR MESSENGER-RNA | INTERFERON-BETA | TISSUE FACTOR EXPRESSION | Receptor, PAR-1 - metabolism | Humans | Male | Troponin I - blood | STAT1 Transcription Factor - metabolism | Myocardium - metabolism | HEK293 Cells | Interferon-beta - genetics | Influenza A virus - immunology | Myocarditis - immunology | Influenza A virus - physiology | Orthomyxoviridae Infections - metabolism | Coxsackievirus Infections - metabolism | Liver - metabolism | Mice, Inbred C57BL | Enterovirus - physiology | Myocarditis - blood | Immunity, Innate | STAT1 Transcription Factor - genetics | Toll-Like Receptor 3 - metabolism | Receptor, PAR-1 - genetics | Mice, Knockout | Myocarditis - virology | Animals | Interferon-beta - metabolism | Thromboplastin - genetics | Enterovirus - immunology | Mice | HeLa Cells | Thrombin - metabolism | Thromboplastin - metabolism | Fibrin - metabolism | Chemokine CXCL10 - metabolism | Coxsackievirus Infections - immunology | Orthomyxoviridae Infections - immunology | Receptor, PAR-1 - physiology | Virus diseases | Cell receptors | Immune response | Physiological aspects | Development and progression | Genetic aspects | Research | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, pp. e104200 - e104200
Elevated levels of erythrocyte-derived microparticles are present in the circulation in medical conditions affecting the red blood cells. Erythrocyte-derived... 
FACTOR XA | THROMBIN GENERATION | FACTOR-IXA | TISSUE FACTOR PATHWAY | FACTOR VA | MULTIDISCIPLINARY SCIENCES | VITAMIN-K | TFPI-ALPHA | SICKLE-CELL-DISEASE | FLOW-CYTOMETRY | FACTOR PATHWAY INHIBITOR | Thromboplastin - pharmacology | Factor Va - pharmacology | Annexin A5 - pharmacology | Humans | Cell-Derived Microparticles - chemistry | Ultracentrifugation | Erythrocytes - chemistry | Protein C - pharmacology | Protein S - metabolism | Lipoproteins - antagonists & inhibitors | Factor VIIIa - metabolism | Thrombin - biosynthesis | Cell-Derived Microparticles - drug effects | Lipoproteins - metabolism | Antigens, Surface - metabolism | Protein S - pharmacology | Cell-Derived Microparticles - metabolism | Factor VIIIa - pharmacology | Milk Proteins - metabolism | Factor V - metabolism | Phosphatidylserines - metabolism | Cells, Cultured | Phosphatidylserines - chemistry | Calcimycin - pharmacology | Factor Xa - metabolism | Antibodies - pharmacology | Blood Coagulation - drug effects | Factor Va - metabolism | Blood Coagulation Tests | Protein C - metabolism | Thromboplastin - metabolism | Monoclonal antibodies | Thrombin | Protein C | Ionophores | Protein binding | Flow cytometry | Blood coagulation | Sickle cell anemia | Laboratories | Prothrombinase | Erythrocytes | Tissue factor | Coagulation | Assaying | Blood | Microparticles | Cofactors | Proteins | Red blood cells | Blood platelets | Inhibition | Calcimycin | Sickle cell disease | Phosphatidylserine | Anticoagulants | Enzymes | Incubation | Blood cells | Activated protein C | Medicine | Cytometry | Hospitals | Blood circulation | Protein S | In vivo methods and tests | Annexin V | Index Medicus | Medicinal Chemistry | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Läkemedelskemi | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 10/2011, Volume 51, Issue 7, pp. 1390 - 1398
The acute-phase protein serum amyloid A (SAA) is elevated during inflammation and may be deposited in atheroma where it promotes atherosclerosis. We... 
Inflammation | Free radicals | Endothelial function | Nitric oxide | Acute-phase protein | C-REACTIVE PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL PROLIFERATION | OXIDIZED LDL | RECEPTOR | VASCULAR CELLS | HDL | A SAA | ENDOCRINOLOGY & METABOLISM | NITRIC-OXIDE | CORONARY-HEART-DISEASE | EXPRESSION | Endothelium, Vascular - cytology | Inflammation - pathology | Superoxide Dismutase - genetics | Rats, Wistar | Calcium - metabolism | Serum Amyloid A Protein - pharmacology | Humans | Arginase - metabolism | Lipoproteins, HDL - metabolism | Endothelium, Vascular - drug effects | Male | NF-kappa B - metabolism | Aorta - metabolism | Nitric Oxide - analysis | Lipoproteins, HDL - pharmacology | Calcium - antagonists & inhibitors | Inflammation - metabolism | Superoxides - metabolism | Lipoproteins, HDL - therapeutic use | Superoxides - antagonists & inhibitors | Superoxide Dismutase - metabolism | Atherosclerosis - pathology | Gene Expression | Serum Amyloid A Protein - metabolism | Endothelial Cells - metabolism | Aorta - drug effects | Rats | Arginase - genetics | Atherosclerosis - metabolism | Blotting, Western | Aorta - pathology | Animals | NF-kappa B - genetics | Signal Transduction - drug effects | Endothelial Cells - cytology | Endothelium, Vascular - metabolism | Thromboplastin - genetics | Thromboplastin - metabolism | Nitric Oxide - metabolism | Serum Amyloid A Protein - adverse effects | Endothelial Cells - drug effects | Atherosclerosis | Acetylcholine | Superoxide | Gene expression | Polyols | Endothelium | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2011, Volume 286, Issue 7, pp. 5756 - 5767
Journal Article
Microvascular Research, ISSN 0026-2862, 05/2018, Volume 117, pp. 37 - 43
Journal Article
Journal Article
Journal Article
Blood, ISSN 0006-4971, 01/2006, Volume 107, Issue 2, pp. 558 - 565
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2017, Volume 37, Issue 7, pp. 1323 - 1331
Journal Article