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The Lancet. Haematology, ISSN 2352-3026, 05/2016, Volume 3, Issue 5, pp. e217 - e227
Summary Background Standard treatment for patients with primary CNS lymphoma remains to be defined. Active therapies are often associated with increased risk... 
Hematology, Oncology and Palliative Medicine | Life Sciences & Biomedicine | Hematology | Science & Technology | Follow-Up Studies | Cytarabine - therapeutic use | Humans | Lymphoma, B-Cell - therapy | Middle Aged | Male | Comparative Effectiveness Research | Hyperglycemia - chemically induced | Radiotherapy, Adjuvant - adverse effects | Hematopoietic Stem Cell Transplantation - adverse effects | Lymphoma, B-Cell - diagnostic imaging | Transplantation Conditioning - methods | Rituximab - adverse effects | Induction Chemotherapy - methods | Anemia - chemically induced | Central Nervous System Neoplasms - therapy | Thrombocytopenia - chemically induced | Mucositis - chemically induced | Magnetic Resonance Imaging | Methotrexate - adverse effects | Dexamethasone - therapeutic use | Radiotherapy, Adjuvant - methods | Transplantation, Autologous - adverse effects | Heart Injuries - chemically induced | Italy | Hematopoietic Stem Cell Transplantation - methods | Central Nervous System Neoplasms - mortality | Germany | Thiotepa - adverse effects | Thiotepa - therapeutic use | Thrombosis - chemically induced | Infections - chemically induced | Intraocular Lymphoma - therapy | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Cytarabine - adverse effects | Methotrexate - therapeutic use | Rituximab - therapeutic use | Stroke - chemically induced | Optic Nerve Neoplasms - diagnostic imaging | Poisoning - epidemiology | Female | Remission Induction - methods | Transplantation Conditioning - adverse effects | Acute Kidney Injury - chemically induced | Neutropenia - chemically induced | Optic Nerve Neoplasms - therapy | Death, Sudden - epidemiology | Lymphoma, B-Cell - mortality | Intraocular Lymphoma - diagnostic imaging | Kaplan-Meier Estimate | Chemical and Drug Induced Liver Injury - epidemiology | Treatment Outcome | United Kingdom | Combined Modality Therapy - adverse effects | Switzerland | Induction Chemotherapy - adverse effects | Disease-Free Survival | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Central Nervous System Neoplasms - diagnostic imaging | Denmark | Combined Modality Therapy - methods | Neurotoxicity Syndromes - epidemiology | Gastrointestinal Diseases - chemically induced | Index Medicus
Journal Article
JNCI : Journal of the National Cancer Institute, ISSN 1460-2105, 09/2011, Volume 103, Issue 17, pp. 1299 - 1309
Journal Article
The New England journal of medicine, ISSN 1533-4406, 01/2017, Volume 376, Issue 2, pp. 136 - 148
Journal Article
The New England journal of medicine, ISSN 1533-4406, 09/2016, Volume 375, Issue 12, pp. 1131 - 1141
Journal Article
International journal of clinical oncology, ISSN 1341-9625, 2/2016, Volume 21, Issue 1, pp. 110 - 117
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 05/2009, Volume 101, Issue 5, pp. 860 - 866
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2013, Volume 59, Issue 2, pp. 318 - 326
.... To date no relevant experimental models of oxaliplatin induced SOS have been described. The aim of this project was to establish a rodent model which could be... 
Gastroenterology and Hepatology | Sinusoidal obstruction syndrome | Oxaliplatin | Colorectal liver metastases | Chemotherapy induced liver injury | Keywords Sinusoidal obstruction syndrome Oxaliplatin Colorectal liver metastases Chemotherapy induced liver injury | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Thrombosis - chemically induced | Oxidative Stress | Humans | Fluorouracil - toxicity | Liver Cirrhosis - chemically induced | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Colorectal Neoplasms - drug therapy | Inflammation Mediators - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Hepatic Veno-Occlusive Disease - pathology | Liver Neoplasms - secondary | Cytokines - genetics | Disease Models, Animal | Cytokines - metabolism | Mice, Inbred C57BL | Liver Neoplasms - drug therapy | Hepatic Veno-Occlusive Disease - metabolism | Liver Neoplasms - surgery | Neovascularization, Pathologic - chemically induced | Antineoplastic Combined Chemotherapy Protocols - toxicity | Hepatic Veno-Occlusive Disease - chemically induced | Organoplatinum Compounds - toxicity | Serpin E2 - genetics | Animals | Cell Cycle | Antioxidants - administration & dosage | Leucovorin - toxicity | Mice | Serpin E2 - metabolism | Chemotherapy | Analysis | Cancer | Index Medicus | MCP1, monocyte chemotactic protein-1 | CRLM, colorectal liver metastases | NRF2, nuclear factor (erythroid-derived 2)-like 2 | HPF, high powered field | PAI-1, plasminogen activatior inhibitor 1 | ALP, alkaline phosphatase | CXCL1 | IL-6, interleukin 6 | NAC, N-acetylcysteine | PCNA, proliferating cell nuclear antigen | C, vascular endothelial growth factor A | PAR 1 | TXN1, thioredoxin 1 | 2, protease activated receptor 1 | 2, vascular endothelial growth factor receptor | NQO1, NAD(P)H dehydrogenase 1 | ALT, alanine aminotransferase | BHA, butylated hydroxyanisole | SOS, sinusoidal obstruction syndrome | STAT3, signal transducer and activator of transcription 3 | H&E, haematoxylin and eosin | VEGF-A | vWF, von Willebrand factor | 2, chemokine (C-X-C motif) ligand 1 | GAPDH, gyceraldehyde 3-phosphate dehydrogenase | γH2AX, phosphorylated form of the H2A histone family, member X | i.p., intraperitoneal | VEGFR-1 | AST, aspartate aminotransferase
Journal Article