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Nature, ISSN 0028-0836, 2013, Volume 494, Issue 7436, pp. 247 - 250
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2013, Volume 15, Issue 7, pp. 807 - 817
Journal Article
Blood, ISSN 0006-4971, 01/2008, Volume 111, Issue 2, pp. 613 - 623
Platelet a-granules constitute the major rapidly releasable reservoir of thrombospondin-1 in higher animals. Although some fragments and peptides derived from... 
ACTIVATION | CELL RESPONSES | FACTOR MULTIMER SIZE | L-ARGININE | GLYCOPROTEIN-IV | MONOCLONAL-ANTIBODY | TERMINAL PEPTIDE | KINASE-I | HEMATOLOGY | INTEGRIN-ASSOCIATED PROTEIN | VON-WILLEBRAND-FACTOR | Cyclic GMP - pharmacology | Immunologic Capping - physiology | Cell Adhesion Molecules - genetics | CD36 Antigens - genetics | Peptides - genetics | Secretory Vesicles - metabolism | Fibrinolytic Agents - metabolism | Phosphoproteins - metabolism | Platelet Adhesiveness - genetics | Secretory Vesicles - genetics | Peptides - metabolism | Thrombin - pharmacology | CD36 Antigens - metabolism | Blood Platelets - cytology | Arginine - genetics | Platelet Glycoprotein GPIIb-IIIa Complex - metabolism | Thrombospondin 1 - genetics | Immunologic Capping - drug effects | Microfilament Proteins - metabolism | Platelet Aggregation - drug effects | Platelet Glycoprotein GPIIb-IIIa Complex - genetics | Microfilament Proteins - genetics | CD47 Antigen - genetics | rap1 GTP-Binding Proteins - metabolism | Shear Strength | Nitric Oxide - antagonists & inhibitors | Platelet Aggregation - physiology | CD47 Antigen - metabolism | Phosphoproteins - genetics | Cell Adhesion Molecules - metabolism | Thrombin - genetics | Mice, Knockout | Peptides - pharmacology | Platelet Adhesiveness - drug effects | Fibrinolytic Agents - pharmacology | Animals | Cyclic GMP - metabolism | Blood Platelets - metabolism | Nitric Oxide - genetics | Cyclic GMP - genetics | Thrombospondin 1 - pharmacology | Mice | Thrombin - metabolism | Thrombospondin 1 - metabolism | Nitric Oxide - metabolism | rap1 GTP-Binding Proteins - genetics | Arginine - metabolism | Cyclic GMP - antagonists & inhibitors | Index Medicus | Abridged Index Medicus | Hemostasis, Thrombosis, and Vascular Biology
Journal Article
Circulation, ISSN 0009-7322, 07/2003, Volume 108, Issue 2, pp. 198 - 204
Background-Intravitreal neovascular diseases, as in ischemic retinopathies, are a major cause of blindness. Because inflammatory mechanisms influence vitreal... 
Prostaglandins | Ischemia | Diabetes mellitus | Vasculature | vasculature | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | OXYGEN-INDUCED RETINOPATHY | diabetes mellitus | OCULAR NEOVASCULARIZATION | ischemia | RETINAL NEOVASCULARIZATION | INHIBITION | IN-VIVO | prostaglandins | PERIPHERAL VASCULAR DISEASE | PLATELET-ACTIVATING-FACTOR | GROWTH-FACTOR | PROSTAGLANDIN E-2 | Retina - drug effects | Endothelial Growth Factors - metabolism | Vascular Endothelial Growth Factors | Humans | Middle Aged | Astrocytes - pathology | Male | Astrocytes - enzymology | Receptors, Lipoprotein - metabolism | Diabetic Retinopathy - complications | Receptors, Prostaglandin E, EP4 Subtype | Retinal Vessels - pathology | Lymphokines - metabolism | Ischemia - pathology | Disease Models, Animal | Enzyme Inhibitors - pharmacology | Rats | Retinal Vessels - drug effects | Rats, Sprague-Dawley | Receptors, Prostaglandin E, EP3 Subtype | Cell Division - drug effects | Dinoprostone - metabolism | Membrane Proteins | Receptors, Scavenger | Neovascularization, Pathologic - drug therapy | Receptors, Immunologic | Mice | Thrombospondin 1 - metabolism | Vitreoretinopathy, Proliferative - enzymology | Retina - pathology | Diabetic Retinopathy - drug therapy | Vascular Endothelial Growth Factor A | Vitreoretinopathy, Proliferative - complications | Cyclooxygenase 2 | Ischemia - enzymology | Diabetic Retinopathy - pathology | Neovascularization, Pathologic - pathology | Intercellular Signaling Peptides and Proteins - metabolism | CD36 Antigens - metabolism | Ischemia - complications | Isoenzymes - metabolism | Retina - enzymology | Adult | Female | Astrocytes - drug effects | Vitreoretinopathy, Proliferative - drug therapy | Mice, Inbred C57BL | Vitreoretinopathy, Proliferative - pathology | Cells, Cultured | Receptors, Prostaglandin E - drug effects | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Animals | Diabetic Retinopathy - enzymology | Prostaglandin-Endoperoxide Synthases - metabolism | Aged | Isoenzymes - antagonists & inhibitors | Receptors, Prostaglandin E - metabolism | Index Medicus | Abridged Index Medicus | Enzyme Inhibitors | Astrocytes | Antigens, CD36 | Neurons and Cognition | Endothelial Growth Factors | Dinoprostone | Intercellular Signaling Peptides | Life Sciences | Cell Division | Diabetic Retinopathy
Journal Article
Fertility and Sterility, ISSN 0015-0282, 2015, Volume 104, Issue 6, pp. 1552 - 1560.e3
Objective To investigate the engraftment and proliferation of superparamagnetic iron oxide nanoparticles (SPIOs)-labeled human CD133+ bone marrow-derived stem... 
Internal Medicine | Obstetrics and Gynecology | Asherman syndrome | bone marrow-derived stem cells (BMDSCs) | CD133+cells | superparamagnetic iron oxide nanoparticles (SPIOs) | cells | CD133 | POPULATION | MOBILIZATION | REGENERATION | PHENOTYPE | MOUSE ENDOMETRIUM | CD133(+) cells | IDENTIFICATION | RAT MODEL | OBSTETRICS & GYNECOLOGY | REPRODUCTIVE BIOLOGY | THIN ENDOMETRIUM | DISEASE | DIFFERENTIATION | Cell Proliferation | Prospective Studies | Humans | Middle Aged | Cell Tracking - methods | Glycoproteins - metabolism | Ki-67 Antigen - metabolism | Insulin-Like Growth Factor I - genetics | Gynatresia - physiopathology | Stem Cells - metabolism | Gynatresia - genetics | Antigens, CD - metabolism | Atrophy | Stem Cell Transplantation | Peptides - metabolism | Thrombospondin 1 - genetics | Bone Marrow Transplantation | Adult | Female | Disease Models, Animal | Endometrium - metabolism | Biomarkers - metabolism | Gynatresia - surgery | Paracrine Communication | Cell Survival | Glycosaminoglycans - metabolism | Gene Expression Regulation | Graft Survival | Clinical Trials as Topic | Mice, SCID | AC133 Antigen | Collagen - metabolism | Animals | Gynatresia - metabolism | Endometrium - physiopathology | Mice, Inbred NOD | Mice | Thrombospondin 1 - metabolism | Endometrium - pathology | Gynatresia - pathology | Bone Marrow Cells - metabolism | Insulin-Like Growth Factor I - metabolism | Index Medicus
Journal Article
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2013, Volume 21, Issue 12, pp. 1913 - 1923
Summary Objective The aim of this study was to investigate the link between the hypertrophic phenotype of chondrocytes and angiogenesis in osteoarthritis (OA)... 
Rheumatology | Angiogenesis | Bone sialoprotein | Chondrocyte hypertrophy | Osteoarthritis | RHEUMATOLOGY | METALLOPROTEINASES | OSTEOCHONDRAL JUNCTION | ARTICULAR-CARTILAGE | SIALOPROTEIN | ENDOTHELIAL-CELLS | GROWTH-FACTOR | BONE | DIFFERENTIATION | ORTHOPEDICS | EXPRESSION | ALGINATE BEADS | Osteopontin - genetics | Up-Regulation | Thrombospondins - genetics | Humans | Osteopontin - metabolism | Vascular Endothelial Growth Factor A - metabolism | Angiopoietin-1 - metabolism | Vascular Endothelial Growth Factor A - genetics | Osteoarthritis - physiopathology | Intercellular Signaling Peptides and Proteins - metabolism | Cartilage, Articular - metabolism | Thrombospondin 1 - genetics | Fibroblast Growth Factor 2 - metabolism | Neovascularization, Pathologic - physiopathology | Membrane Proteins - metabolism | Chondrocytes - metabolism | Endothelial Cells - physiology | Chondrocytes - pathology | Nerve Growth Factor - metabolism | Cartilage, Articular - cytology | Matrix Metalloproteinase 2 - metabolism | Membrane Proteins - genetics | Cells, Cultured | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Osteoarthritis - metabolism | Nerve Growth Factor - genetics | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion | Osteoarthritis - genetics | Angiopoietin-1 - genetics | Matrix Metalloproteinase 2 - genetics | Fibroblast Growth Factor 2 - genetics | Integrin-Binding Sialoprotein - genetics | Neovascularization, Pathologic - genetics | Neovascularization, Pathologic - metabolism | Thrombospondin 1 - metabolism | Integrin-Binding Sialoprotein - metabolism | Thrombospondins - metabolism | Cell Movement | Hypertrophy | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 05/2012, Volume 484, Issue 7397, pp. 195 - 202
Journal Article
Cell Reports, ISSN 2211-1247, 12/2015, Volume 13, Issue 11, pp. 2456 - 2469
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2015, Volume 35, Issue 12, pp. 2626 - 2637
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, pp. e48535 - e48535
Inflammation is part of a complex biological response of vascular tissue to pathogens or damaged cells. First inflammatory cells attempt to remove the... 
GAMMA | CELLS | PHAGOCYTOSIS | ACTIVATED PROTEIN-KINASE | RECOGNITION | MULTIDISCIPLINARY SCIENCES | RECEPTOR | FACTOR-I | HIF-1-ALPHA | EXPRESSION | BOWEL-DISEASE | Crohn Disease - genetics | Intestinal Mucosa - metabolism | Humans | Middle Aged | Colitis, Ulcerative - genetics | Crohn Disease - metabolism | Male | Colitis, Ulcerative - immunology | Hypoxia-Inducible Factor 1 - metabolism | Hypoxia - metabolism | Young Adult | Inflammation - metabolism | CD36 Antigens - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Intestinal Mucosa - immunology | Thrombospondin 1 - genetics | Adult | Female | p38 Mitogen-Activated Protein Kinases - metabolism | Protein Stability | Neutrophils - metabolism | Macrophages - immunology | Cell Line | Promoter Regions, Genetic | Signal Transduction | Colitis, Ulcerative - metabolism | Neutrophils - immunology | Phagocytosis - immunology | Hypoxia - immunology | Inflammation - immunology | Crohn Disease - immunology | Macrophages - metabolism | Apoptosis - immunology | Adolescent | Inflammation - genetics | Protein Binding | Thrombospondin 1 - metabolism | Intestinal Mucosa - pathology | Proteins | Inflammation | Macrophages | Gastrointestinal diseases | Apoptosis | Cell culture | Hypoxia-inducible factor 1 | Inflammatory bowel diseases | Transcription | Gene regulation | Mucosa | Kinases | Tissues | Immunology | Transfection | Intestine | Rodents | Damage | Thrombospondin | Hypoxia-inducible factors | Pathogens | Pulmonary arteries | Neutrophils | Lamina propria | MAP kinase | Leukocytes (neutrophilic) | CD36 antigen | Gene expression | Patients | Inflammatory bowel disease | Hospitals | Nitric oxide | Vascular tissue | Hypoxia | Phagocytosis | Index Medicus
Journal Article