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PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e57243
Accumulating evidence suggests that alterations in immune function may be important in the etiology of papillary thyroid cancer (PTC). To identify genetic... 
UNITED-STATES | LINKAGE DISEQUILIBRIUM | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | PROTEIN-C INHIBITOR | US RADIOLOGIC TECHNOLOGISTS | FAMILY-HISTORY | AUTOIMMUNE-DISEASE | CARCINOMA | ASSOCIATION | RADIATION | Genetic Predisposition to Disease | Humans | Middle Aged | Risk Factors | Male | Protein C Inhibitor - genetics | Genetic Loci | Thyroid Cancer, Papillary | Autoimmune Diseases - genetics | Thyroid Neoplasms - genetics | Carcinoma, Papillary | Adult | Blood Coagulation - genetics | Carcinoma - genetics | Female | Aged | Polymorphism, Single Nucleotide | Carcinoma - metabolism | Carcinoma - pathology | Autoimmune Diseases - metabolism | Protein C Inhibitor - metabolism | Thyroid Neoplasms - metabolism | Thyroid Neoplasms - pathology | Genetic markers | Ionizing radiation | Thyroid cancer | Genes | Development and progression | Disease susceptibility | Genetic aspects | Single nucleotide polymorphisms | Arachidonic acid | Health aspects | Risk factors | Cancer | Papillary thyroid cancer | Leukocyte migration | Genomics | Coagulation | Risk | Single-nucleotide polymorphism | Epidemiology | Confidence intervals | Signal transduction | Regression models | Etiology | Extravasation | Thyroid | Thyroiditis | NF-κB protein | Immune response | Statistical analysis | Susceptibility | Health risks | Antimicrobial agents | Protein C | Regression analysis | Metabolism | Biological activity | White blood cells | Signaling | Tumor necrosis factor | Health risk assessment
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