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Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 11, pp. 2563 - 2570
Summary BRAF mutation is seen in a variety of human neoplasms including cutaneous malignant melanoma, papillary thyroid carcinoma, colorectal carcinoma, non... 
Pathology | Immunohistochemistry | Anti–B-Raf | BRAF V600E | VE1 | Glioma | Melanoma | Thyroid carcinoma | Anti-B-Raf | PROTEIN | IMPROVED SURVIVAL | PATHOLOGY | CANCER | TUMORS | THERAPY | MEK INHIBITION | GENE | MUTANT BRAF | Neoplasms - metabolism | Gastrointestinal Neoplasms - genetics | Humans | Lung Neoplasms - metabolism | Ovarian Neoplasms - pathology | Lung Neoplasms - pathology | Mutation, Missense | Breast Neoplasms - metabolism | Glioma - metabolism | Glioma - genetics | Ovarian Neoplasms - genetics | Gastrointestinal Neoplasms - pathology | Neoplasms - genetics | DNA Mutational Analysis | Melanoma - genetics | Glioma - pathology | Sensitivity and Specificity | Biomarkers, Tumor - metabolism | Female | Ovarian Neoplasms - metabolism | Carcinoma - pathology | Antibodies, Monoclonal - immunology | Proto-Oncogene Proteins B-raf - metabolism | Gastrointestinal Neoplasms - metabolism | Lung Neoplasms - genetics | Melanoma - metabolism | Mutant Proteins - genetics | Mutant Proteins - metabolism | Melanoma - pathology | Thyroid Neoplasms - genetics | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Proto-Oncogene Proteins B-raf - genetics | Carcinoma - genetics | Biomarkers, Tumor - genetics | Carcinoma - metabolism | Neoplasms - pathology | Amino Acid Substitution | Thyroid Neoplasms - metabolism | Thyroid Neoplasms - pathology | Carcinoma | Gene mutations | Gliomas | Thyroid diseases | Monoclonal antibodies | Genetic aspects | Comparative analysis | Lung cancer, Non-small cell | Cancer | Breast cancer | Mutation | Tumors | Skin cancer
Journal Article
Journal Article
Journal Article
Cancer Science, ISSN 1347-9032, 07/2019, Volume 110, Issue 7, pp. 2180 - 2188
Journal Article
Genes and Development, ISSN 0890-9369, 06/2006, Volume 20, Issue 12, pp. 1569 - 1574
The tumor suppressor PTEN is frequently inactivated in human cancers. A major downstream effector of PTEN is Akt, which is hyperactivated via PTEN... 
Intestinal polyps | PIN | Endometrium carcinoma | Thyroid and adrenal tumors | Cancer therapy | AKT/PKB | GENETICALLY-ENGINEERED MICE | PTEN | DEVELOPMENTAL BIOLOGY | PHOSPHOINOSITIDE 3-KINASE | CANCER | CELL BIOLOGY | cancer therapy | PATHWAY | thyroid and adrenal tumors | NEOPLASIA | RELEVANCE | intestinal polyps | MUTATION | GENETICS & HEREDITY | endometrium carcinoma | Prostatic Intraepithelial Neoplasia - pathology | Thyroid Neoplasms - enzymology | Proto-Oncogene Proteins c-akt - deficiency | Male | Intestinal Polyps - enzymology | Proto-Oncogene Proteins c-akt - genetics | Adrenal Gland Neoplasms - enzymology | Prostatic Neoplasms - genetics | Endometrial Neoplasms - genetics | Neoplasms - genetics | Female | Pseudolymphoma - genetics | Pseudolymphoma - enzymology | Adrenal Gland Neoplasms - pathology | PTEN Phosphohydrolase - genetics | Prostatic Neoplasms - pathology | Endometrial Neoplasms - enzymology | Neoplasms - enzymology | PTEN Phosphohydrolase - metabolism | Pseudolymphoma - pathology | Thyroid Neoplasms - genetics | Animals | Prostatic Intraepithelial Neoplasia - enzymology | Intestinal Polyps - pathology | Prostatic Neoplasms - enzymology | Endometrial Neoplasms - pathology | Heterozygote | Mice | Prostatic Intraepithelial Neoplasia - genetics | Neoplasms - pathology | Adrenal Gland Neoplasms - genetics | Thyroid Neoplasms - pathology | Research Communication
Journal Article
Scientific reports, ISSN 2045-2322, 2014, Volume 4, Issue 1, p. 6088
Some long noncoding RNAs (lncRNAs) play important roles in the regulation of gene expression by acting as competing endogenous RNAs (ceRNAs). However, the... 
MESSENGER-RNAS | DATABASE | GENE | ANNOTATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MICRORNA TARGETS | CERNA HYPOTHESIS | NETWORKS | IDENTIFICATION | EXPRESSION | Oligonucleotide Array Sequence Analysis | Carcinoma, Squamous Cell - genetics | Humans | Male | Gene Expression Profiling | Leukemia, Lymphocytic, Chronic, B-Cell - genetics | Ovarian Neoplasms - genetics | Retinoblastoma Protein - biosynthesis | Squamous Cell Carcinoma of Head and Neck | Prostatic Neoplasms - genetics | RNA Interference | Cell Transformation, Neoplastic - genetics | Female | Gene Expression Regulation, Neoplastic - genetics | Stomach Neoplasms - genetics | Pituitary Neoplasms - genetics | RNA, Long Noncoding - genetics | Thyroid Cancer, Papillary | Thyroid Neoplasms - genetics | Algorithms | Carcinoma, Papillary | Retinoblastoma Protein - genetics | Cell Line, Tumor | Carcinoma - genetics | Head and Neck Neoplasms - genetics | MicroRNAs - genetics | RNA, Small Interfering | Post-transcription | Transcription | Gene regulation | Regulatory sequences | Stomach cancer | Ovarian cancer | miRNA | Gastric cancer | Thyroid | Squamous cell carcinoma | Chronic lymphatic leukemia | Pituitary (anterior) | Runx1 protein | siRNA | Lymphatic leukemia | Gene expression | DNA microarrays | Pituitary | Interleukin 10 | Head and neck cancer | Prostate | Prostate cancer | PTEN protein | Papillary thyroid carcinoma | Tumors
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 379, Issue 1, pp. 94 - 99
Highlights • First population-based study on risk of specific SPCs after malignant mesothelioma. • Using the approach of bidirectional analyses of second... 
Hematology, Oncology and Palliative Medicine | Second primary cancers | Malignant mesothelioma | Peritoneal mesothelioma | Cancer registry | Pleural mesothelioma | Etiology | SWEDEN | FAMILY-HISTORY | GENETIC SUSCEPTIBILITY | MELANOMA | DATABASE | ONCOLOGY | BAP1 MUTATIONS PREDISPOSE | SWEDISH | ASBESTOS | Kidney Neoplasms - genetics | Pleural Neoplasms - genetics | Kidney Neoplasms - epidemiology | Mesothelioma - pathology | Peritoneal Neoplasms - epidemiology | Humans | Middle Aged | Lung Neoplasms - pathology | Male | Young Adult | Time Factors | Tumor Suppressor Proteins - genetics | Aged, 80 and over | Germ-Line Mutation | Adult | Female | Registries | Neoplasms, Second Primary - genetics | Pleural Neoplasms - pathology | Lung Neoplasms - genetics | Peritoneal Neoplasms - pathology | Pleural Neoplasms - epidemiology | Genetic Predisposition to Disease | Risk Assessment | Risk Factors | Mesothelioma - genetics | Sweden - epidemiology | Ubiquitin Thiolesterase - genetics | Neoplasms, Second Primary - epidemiology | Phenotype | Lung Neoplasms - epidemiology | Peritoneal Neoplasms - genetics | Mesothelioma - epidemiology | Environment | Kidney Neoplasms - pathology | Aged | Neoplasms, Second Primary - pathology | Mesothelioma | Health aspects | Risk factors | Cancer | Clinical Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin | Cancer and Oncology | Cancer och onkologi
Journal Article
Seminars in Oncology, ISSN 0093-7754, 12/2013, Volume 40, Issue 6, pp. 676 - 689
Eighteen percent of incident malignancies in the United States are a second (or subsequent) cancer. Second primary neoplasms... 
LONG-TERM SURVIVORS | PRIMARY THYROID-CANCER | LI-FRAUMENI-SYNDROME | ONCOLOGY | EXTENDED FOLLOW-UP | ACUTE LYMPHOBLASTIC-LEUKEMIA | SQUAMOUS-CELL CARCINOMA | NON-HODGKINS-LYMPHOMA | UPPER AERODIGESTIVE TRACT | CORONARY-HEART-DISEASE | CHILDHOOD-CANCER SURVIVOR | Smoking - adverse effects | United States | Humans | Neoplasms, Radiation-Induced - etiology | Thyroid Neoplasms - epidemiology | Breast Neoplasms - etiology | Male | Radiotherapy - adverse effects | Gene-Environment Interaction | Neoplastic Syndromes, Hereditary | Hodgkin Disease - radiotherapy | Colorectal Neoplasms - etiology | Squamous Cell Carcinoma of Head and Neck | Skin Neoplasms - prevention & control | Head and Neck Neoplasms - epidemiology | Female | Neoplasms, Radiation-Induced - prevention & control | Stomach Neoplasms - epidemiology | Stomach Neoplasms - etiology | Thyroid Neoplasms - prevention & control | Breast Neoplasms - epidemiology | Colorectal Neoplasms - epidemiology | Genetic Predisposition to Disease | Head and Neck Neoplasms - prevention & control | Risk Assessment | Risk Reduction Behavior | Carcinoma, Squamous Cell - epidemiology | Risk Factors | Thyroid Neoplasms - etiology | Colorectal Neoplasms - prevention & control | Carcinoma, Squamous Cell - prevention & control | Skin Neoplasms - epidemiology | Breast Neoplasms - prevention & control | Neoplasms, Second Primary - epidemiology | Neoplasms, Second Primary - etiology | Neoplasms, Second Primary - prevention & control | Carcinoma, Squamous Cell - etiology | Head and Neck Neoplasms - etiology | Stomach Neoplasms - prevention & control | Skin Neoplasms - etiology | Antineoplastic Agents, Alkylating - adverse effects | Neoplasms, Radiation-Induced - epidemiology
Journal Article
The journal of clinical endocrinology and metabolism, ISSN 1945-7197, 2017, Volume 102, Issue 6, pp. 1898 - 1907
Journal Article
Cancer, ISSN 0008-543X, 2015, Volume 121, Issue 5, pp. 648 - 663
Subsequent malignant neoplasms (SMNs) are associated with significant morbidity and are a major cause of premature mortality among cancer survivors... 
gene‐environment interactions | therapeutic exposures | genetic susceptibility | cancer survivors | second cancers | Cancer survivors | Gene-environment interactions | Therapeutic exposures | Genetic susceptibility | Second cancers | CONTRALATERAL BREAST-CANCER | PRIMARY THYROID-CANCER | LONG-TERM SURVIVORS | DNA-DAMAGE RESPONSE | ACUTE MYELOID-LEUKEMIA | MYELODYSPLASTIC SYNDROME | 2ND PRIMARY NEOPLASMS | CHILDHOOD-CANCER | ONCOLOGY | gene-environment interactions | SQUAMOUS-CELL CARCINOMA | GENOME-WIDE ASSOCIATION | DNA Damage - drug effects | Genetic Predisposition to Disease | Humans | Neoplasm Recurrence, Local - therapy | Neoplasms - mortality | Neoplasms, Second Primary - mortality | Antineoplastic Agents - therapeutic use | Radiotherapy - adverse effects | DNA Repair - genetics | Neoplasm Recurrence, Local - mortality | Genetic Variation | Neoplasms, Radiation-Induced - mortality | Neoplasms, Radiation-Induced - therapy | Neoplasms - therapy | Neoplasms, Radiation-Induced - genetics | Neoplasms - genetics | Antineoplastic Agents - adverse effects | Neoplasms, Second Primary - therapy | Neoplasm Recurrence, Local - genetics | Neoplasms, Second Primary - genetics | DNA Damage - radiation effects | Neuroleptic malignant syndrome | Usage | Genetic variation | Therapeutics | Research | Gene expression | Health aspects | Risk factors | Homeopathy | Materia medica and therapeutics
Journal Article