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European Journal of Endocrinology, ISSN 0804-4643, 06/2017, Volume 176, Issue 6, pp. 685 - 693
Journal Article
Kidney International, ISSN 0085-2538, 07/2016, Volume 90, Issue 1, pp. 77 - 89
Bone loss and increased fractures are common complications in chronic kidney disease. Because Wnt pathway activation is essential for normal bone... 
uremia | phosphate | chronic kidney disease | bone | hyperparathyroidism | FGF23 | SCLEROSTIN | VASCULAR CALCIFICATION | MINERAL DENSITY | MASS | GENE-EXPRESSION | UROLOGY & NEPHROLOGY | PARATHYROID-HORMONE | RENAL OSTEODYSTROPHY | POSTMENOPAUSAL WOMEN | Rats, Wistar | Decalcification, Pathologic - etiology | Humans | Glucuronidase - metabolism | Male | Uremia - metabolism | Parathyroid Hormone - blood | Renal Insufficiency, Chronic - complications | Wnt Proteins - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Tibia - pathology | Renal Insufficiency, Chronic - metabolism | Calcium - blood | Fibroblast Growth Factors - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | beta Catenin - blood | Membrane Proteins - metabolism | Tibia - metabolism | Disease Models, Animal | Biomarkers - metabolism | Osteocalcin - metabolism | Osteoblasts - drug effects | Rats | Phosphorus, Dietary - adverse effects | Fibroblast Growth Factors - pharmacology | Biomarkers - blood | Uremia - complications | Animals | Wnt Signaling Pathway - drug effects | Cathepsin K - metabolism | Calcification, Physiologic | Phosphorus - blood | Phosphorus - metabolism | Cell Line, Tumor | Glucuronidase - pharmacology | Porosity | Osteoblasts - metabolism | Wnt Proteins - antagonists & inhibitors | Decalcification, Pathologic - metabolism
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 06/2017, Volume 102, Issue 6, pp. 1807 - 1815
Context: In type 2 diabetes mellitus, fracture risk is increased despite preserved areal bone mineral density. Although this apparent paradox may in part be... 
SERUM-INSULIN | OBESE CHILDREN | HOMEOSTASIS MODEL ASSESSMENT | SKELETAL-MUSCLE MASS | HEALTHY MALE SIBLINGS | ENDOCRINOLOGY & METABOLISM | PHYSICAL-ACTIVITY | POSTMENOPAUSAL WOMEN | TYPE-2 DIABETES-MELLITUS | FRACTURE RISK | BETA-CELL FUNCTION | Body Composition | Radius - pathology | Humans | Leptin - metabolism | Middle Aged | Male | Tomography, X-Ray Computed | Cortical Bone - pathology | Tibia - pathology | Tandem Mass Spectrometry | Testosterone - metabolism | Peptides - metabolism | Parathyroid Hormone - metabolism | Chromatography, Liquid | Adiponectin - metabolism | Adult | Muscle Strength | Estradiol - metabolism | Peptide Fragments - metabolism | Bone Density | Collagen Type I - metabolism | Osteocalcin - metabolism | Insulin Resistance | Tibia - diagnostic imaging | Organ Size | Sex Hormone-Binding Globulin - metabolism | Procollagen - metabolism | Absorptiometry, Photon | Insulin - metabolism | Muscle, Skeletal - diagnostic imaging | Vitamin D - analogs & derivatives | Vitamin D - metabolism | Muscle, Skeletal - pathology | Blood Glucose - metabolism | Cortical Bone - diagnostic imaging | Insulin-Like Growth Factor I - metabolism | Radius - diagnostic imaging | Serum Albumin - metabolism | Radius | Sex | Homeostasis | Population studies | Bone (trabecular) | Body composition | Adiponectin | Computed tomography | Body composition (biology) | Bone (cortical) | Age | Bone composition | Dual energy X-ray absorptiometry | Diabetes mellitus | Health risks | Muscles | Excavation | Bone turnover | Insulin | Geometry | Bone mass | Leptin | Body size | Men | Insulin resistance | Bone mineral density
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 04/2011, Volume 26, Issue 4, pp. 777 - 791
Mesenchymal stem cell commitment to an osteoprogenitor lineage requires the activity of Runx2, a molecule implicated in the etiopathology of multiple... 
CLEIDOCRANIAL DYSPLASIA | CRANIOFACIAL DEVELOPMENT | TRANSGENIC ANIMAL | NELL‐1 | RUNX2 | NELL-1 | craniofacial development | transgenic animal | cleidocranial dysplasia | CELLS | OSTEOBLAST DIFFERENTIATION | BONE MORPHOGENETIC PROTEIN-2 | CRANIOSYNOSTOSIS-ASSOCIATED GENE | CBFA1-DEFICIENT MICE | BIOLOGY | ENDOCRINOLOGY & METABOLISM | CBFA1 | TRANSCRIPTION FACTOR | EXPRESSION | RNA, Small Interfering - genetics | Osteogenesis - physiology | Core Binding Factor Alpha 1 Subunit - deficiency | Gene Expression - genetics | Alkaline Phosphatase - metabolism | Osteocalcin - genetics | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Glycoproteins - pharmacology | Core Binding Factor Alpha 1 Subunit - metabolism | Skull - pathology | Skull - abnormalities | Craniofacial Abnormalities - pathology | p38 Mitogen-Activated Protein Kinases - metabolism | Cleidocranial Dysplasia - metabolism | Phosphorylation - drug effects | Craniofacial Abnormalities - genetics | Animals, Newborn | Glycoproteins - genetics | Skull - metabolism | Alkaline Phosphatase - genetics | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Calcification, Physiologic - drug effects | Femur - metabolism | Calcification, Physiologic - physiology | Mice, Transgenic | Mice, Knockout | Cleidocranial Dysplasia - genetics | Skull - cytology | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Sp7 Transcription Factor | Dura Mater - metabolism | Mice | Calcium-Binding Proteins - pharmacology | Calcium-Binding Proteins - genetics | Osteopontin - genetics | Glycoproteins - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Osteopontin - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Tibia - metabolism | Osteoblasts - cytology | Cell Differentiation - physiology | Craniofacial Abnormalities - metabolism | Calcium-Binding Proteins - metabolism | Haploinsufficiency - genetics | Osteocalcin - metabolism | Tissue Culture Techniques | Osteogenesis - drug effects | Cells, Cultured | Transcription Factors - genetics | Mice, Inbred Strains | Cleidocranial Dysplasia - pathology | Skull - embryology | Animals | Heterozygote | Protein Kinase Inhibitors - pharmacology | Osteoblasts - metabolism | Core Binding Factor Alpha 1 Subunit - genetics | Original
Journal Article
BMC Musculoskeletal Disorders, ISSN 1471-2474, 2011, Volume 12, Issue 1, pp. 197 - 197
Background: Osteoarthritis (OA) is a debilitating, progressive joint disease. Methods: Similar to the disease progression in humans, sequential events of early... 
bone remodeling | subchondral bone | cartilage degeneration | osteoarthritis | INVERSE RELATIONSHIP | RHEUMATOLOGY | OSTEOPHYTE FORMATION | CANCELLOUS BONE | IIA PROCOLLAGEN | ANIMAL-MODELS | COLLAGEN METABOLISM | TRANSGENIC MOUSE MODEL | ORTHOPEDICS | KNEE OSTEOARTHRITIS | UP-REGULATION | EXPRESSION | Anterior Cruciate Ligament - surgery | Synovial Membrane - pathology | Male | Bone Remodeling - physiology | Gene Expression Profiling | RNA, Messenger - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Tibia - pathology | Osteophyte - pathology | Procollagen N-Endopeptidase - metabolism | Menisci, Tibial - surgery | Tibia - metabolism | Chondrocytes - metabolism | Disease Models, Animal | Fibroblasts - metabolism | Biomarkers - metabolism | Chondrocytes - pathology | Gene Expression | Menisci, Tibial - metabolism | Matrix Metalloproteinase 13 - genetics | Osteophyte - metabolism | Procollagen N-Endopeptidase - genetics | Rats | Transcription Factors - genetics | Fibroblasts - pathology | Rats, Sprague-Dawley | Synovial Membrane - metabolism | Disease Progression | ADAMTS4 Protein | Matrix Metalloproteinase 13 - metabolism | Anterior Cruciate Ligament - metabolism | Transcription Factors - metabolism | ADAM Proteins - metabolism | Animals | Osteoarthritis, Knee - pathology | Osteoarthritis, Knee - genetics | ADAM Proteins - genetics | Core Binding Factor Alpha 1 Subunit - genetics | Osteoarthritis, Knee - metabolism | Messenger RNA | Genomics | Physiological aspects | Development and progression | Research | Gene expression | Osteoarthritis | Knee | Cartilage | Sports injuries | Bone density | Rodents
Journal Article
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 01/2016, Volume 31, Issue 1, pp. 98 - 115
ABSTRACT Metabolic and bone effects were investigated in growing (G, n = 45) and mature (M, n = 45) rats fed a high‐fat/high‐sucrose diet (HFS) isocaloric to... 
BONE STRENGTH | OBESITY | CALCIUM‐PHOSPHATE METABOLISM | HISTOMORPHOMETRY | DIABETES | CALCIUM-PHOSPHATE METABOLISM | VITAMIN-D | DIET-INDUCED OBESITY | MECHANICAL-PROPERTIES | PARATHYROID-HORMONE LEVELS | VOLUNTARY WEIGHT-LOSS | GROWTH-FACTOR-BETA | MINERAL DENSITY | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | ESSENTIAL-HYPERTENSION | POSTMENOPAUSAL WOMEN | Rats, Wistar | Hypocalcemia - metabolism | Transforming Growth Factor beta1 - metabolism | Spine - metabolism | Sucrose - pharmacology | Male | Hypocalcemia - pathology | Tibia - pathology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Matrix Metalloproteinase 9 - metabolism | Hypocalcemia - chemically induced | Dietary Fats - pharmacology | Matrix Metalloproteinase 9 - genetics | Bone Marrow - metabolism | Tibia - metabolism | Spine - pathology | Dietary Fats - adverse effects | Obesity - chemically induced | Matrix Metalloproteinase 2 - metabolism | Rats | Sucrose - adverse effects | Transforming Growth Factor beta1 - genetics | Aging - pathology | Obesity - metabolism | Obesity - pathology | Matrix Metalloproteinase 2 - genetics | Tissue Inhibitor of Metalloproteinase-1 - genetics | Animals | Bone Marrow - pathology | Hypocalcemia - genetics | Aging - metabolism | Obesity | Osteoporosis | Leptin | Physiological aspects | Transforming growth factors | Sugars | Prediabetic state
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e59402
Tobacco smoking is an important risk factor for the development of several cancers, osteoporosis, and inflammatory diseases such as periodontitis. Nicotine is... 
MATRIX METALLOPROTEINASES | CELLS | CIGARETTE-SMOKING | GINGIVAL FIBROBLASTS | RISK-FACTORS | CARBONIC-ANHYDRASE-II | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | PERIODONTAL-DISEASE | NF-KAPPA-B | SUBUNIT | Vacuolar Proton-Translocating ATPases - genetics | Gene Expression - drug effects | Monocytes - cytology | Actins - metabolism | Tartrate-Resistant Acid Phosphatase | Male | Monocytes - metabolism | Osteoclasts - cytology | Vacuolar Proton-Translocating ATPases - metabolism | Actins - genetics | Nicotine - pharmacology | Acid Phosphatase - metabolism | Matrix Metalloproteinase 9 - metabolism | Bone Resorption - genetics | Isoenzymes - metabolism | Matrix Metalloproteinase 9 - genetics | Bone Resorption - metabolism | Bone Resorption - chemically induced | Tibia - metabolism | Cathepsin K - antagonists & inhibitors | Cell Line | Receptors, Nicotinic - metabolism | Bone Marrow Cells - cytology | Femur - metabolism | Isoenzymes - genetics | Mice, Inbred C57BL | Femur - drug effects | Tibia - drug effects | Osteoclasts - metabolism | Tibia - cytology | Cathepsin K - genetics | Vacuolar Proton-Translocating ATPases - antagonists & inhibitors | Actins - antagonists & inhibitors | Animals | Cathepsin K - metabolism | Bone Resorption - pathology | Femur - cytology | Mice | Cell Nucleus - drug effects | Receptors, Nicotinic - genetics | Acid Phosphatase - genetics | Bone Marrow Cells - metabolism | Osteoclasts - drug effects | Bungarotoxins - pharmacology | Osteoprogenitor cells | Cell culture | Health sciences | Tobacco smoking | Biochemistry | Kinases | Osteoblasts | Nuclei | Inflammatory diseases | Risk factors | Bone resorption | Osteoporosis | Receptors | Bungarotoxin | Actin | Acid resistance | Bone marrow | Extracellular matrix | Biocompatibility | TRANCE protein | University graduates | H+-transporting ATPase | Cathepsin K | Cytokines | Health risks | Dentistry | Gelatinase B | Osteoclastogenesis | Tobacco | Dental research | Osteoblastogenesis | Periodontitis | Oral hygiene | Morphology | Osteoclasts | Acid phosphatase (tartrate-resistant) | Nuclei (cytology) | Differentiation | Acid phosphatase | Nicotine | Smoking
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 11/2015, Volume 30, Issue 11, pp. 1959 - 1968
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 02/2003, Volume 18, Issue 2, pp. 360 - 369
Low‐intensity pulsed ultrasound, a form of mechanical energy transmitted as high‐frequency acoustical pressure waves, provides noninvasive therapeutic... 
therapeutic ultrasound | osteoblast | osteocyte | PI3K | insulin‐like growth factor I | Osteoblast | Therapeutic ultrasound | Insulin-like growth factor I | Osteocyte | SIGNAL-TRANSDUCTION PATHWAYS | insulin-like growth factor I | FLUID SHEAR-STRESS | NITRIC-OXIDE SYNTHASE | MECHANICAL RESPONSES | MESSENGER-RNA | BONE-FORMATION | STROMAL CELL CLONE | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | CYCLOOXYGENASE-2 EXPRESSION | UP-REGULATION | Osteocytes - diagnostic imaging | Bone and Bones - pathology | Up-Regulation | Calcium - metabolism | Cyclooxygenase 2 | Phosphatidylinositol 3-Kinases - metabolism | Dexamethasone - pharmacology | Time Factors | Isoenzymes - metabolism | Bone and Bones - metabolism | Ultrasonography | Cell Differentiation | Tibia - metabolism | p38 Mitogen-Activated Protein Kinases | Animals, Newborn | Cyclic AMP-Dependent Protein Kinases - metabolism | Skull - metabolism | Antineoplastic Agents, Hormonal - pharmacology | Osteocytes - metabolism | Signal Transduction | Bone Marrow Cells - cytology | Osteocalcin - metabolism | Proto-Oncogene Proteins c-fos - metabolism | Rats | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Ultrasonics | Dinoprostone - metabolism | Cell Lineage | Osteoblasts - pathology | Animals | Osteoblasts - diagnostic imaging | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Prostaglandin-Endoperoxide Synthases - metabolism | Osteocytes - pathology | Mice | Osteoblasts - metabolism | Bone Marrow Cells - metabolism | Insulin-Like Growth Factor I - metabolism | Mitogen-Activated Protein Kinases - metabolism
Journal Article