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Nature cell biology, ISSN 1476-4679, 2011, Volume 13, Issue 10, pp. 1202 - 1213
Angiogenesis, the growth of new blood vessels, involves specification of endothelial cells to tip cells and stalk cells, which is controlled by Notch signalling, whereas vascular endothelial growth factor receptor (VEGFR... 
LYMPHATIC ENDOTHELIUM | KINASE-ACTIVITY | VASCULAR NETWORK FORMATION | ENDOTHELIAL-GROWTH-FACTOR | FACTOR-C | MOUSE EMBRYOS | EXTRACELLULAR-MATRIX | MONOCLONAL-ANTIBODY | TUMOR ANGIOGENESIS | FACTOR RECEPTOR-3 | CELL BIOLOGY | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Receptors, Notch - antagonists & inhibitors | Transfection | RNA Interference | Time Factors | Forkhead Transcription Factors - metabolism | Vascular Endothelial Growth Factor D - genetics | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor Receptor-3 - deficiency | Transduction, Genetic | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Rhombencephalon - blood supply | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Forkhead Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Mice, Knockout | Antibodies - pharmacology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Vascular Endothelial Growth Factor B - genetics | Mice | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Physiological aspects | Blood vessels | Cellular signal transduction | Growth factor receptors | Research | Vascular endothelial growth factor
Journal Article
SAE technical paper series, Volume 972670.
This paper describes the application of human factors research tools to the design of Intelligent Transportation Systems... 
Human factors | Reaction and response times | Design Engineering and Styling | Intelligent transportation systems
eJournal
SAE technical paper series, Volume 1999-01-0815
This paper summarizes the activities and results of a preliminary human factors review for the Intelligent Vehicle Initiative (IVI) Program... 
Human factors | Reaction and response times | Intelligent transportation systems | Public transportation systems
eJournal
Journal Article
SAE technical paper series, Volume 951959.
.... A new reconfigurable research tool is being developed to perform human factors experiments in the areas of vehicle display type, placement and symbology... 
Human factors | Reaction and response times | Data acquisition and handling | Design Engineering and Styling
eJournal
SAE technical paper series, Volume 2005-01-0434.
The present paper describes two studies performed with the aim of developing a human factors evaluation technique for the total vehicle based upon users' subjective experience while interacting... 
Human factors | Reaction and response times | Displays | Design Engineering and Styling
eJournal
SAE technical paper series, Volume 951482.
The U.S. Army Research Laboratory's (ARL) Human Research andamp; Engineering Directorate (HRED) is currently pursuing various research and development projects... 
Human factors | Reaction and response times | Driver Assistance systems | Human machine interface (HMI)
eJournal
SAE technical paper series, Volume 2001-01-0358.
The future of automotive vehicle interiors is becoming more complex each year. Extensive consumer research demonstrates that consumers want new convenience... 
Human factors | Reaction and response times
eJournal
SAE technical paper series, Volume 2005-01-0426.
Video recordings of real life traffic crashes and near crashes were analysed for driver response choice. These responses were compared to problem solving... 
Vehicle drivers | Accident reconstruction | Human factors | Reaction and response times | Crash research | Collision avoidance systems
eJournal
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2011, Volume 108, Issue 17, pp. 6889 - 6892
.... In contrast legal realists argue that the rational application of legal reasons does not sufficiently explain the decisions of judges and that psychological, political, and social factors influence judicial rulings... 
Prisons | Judicial rulings | Criminal punishment | Ethnicity | Parole boards | Breakfasts | Parole | Judges | Attorneys | Criminal offenses | Expert decisionmaking | Decisionmaking | Ego depletion | Mental depletion | Legal realism | RESOURCES | SELF-CONTROL | ego depletion | MULTIDISCIPLINARY SCIENCES | decisionmaking | mental depletion | expert decisionmaking | legal realism | Mental Fatigue | Choice Behavior | Time Factors | Humans | Feeding Behavior | Female | Male | Cognition | Jurisprudence | Workload | Judicial opinions | Research | Social Sciences
Journal Article
Gene, ISSN 0378-1119, 01/2017, Volume 599, pp. 36 - 52
.... E2F1 is the transcription factor most closely associated with genes down-regulated by ageing and is down-regulated at the protein level in osteocytes... 
Bone | Proliferation | Mechanical loading | Ageing | OSTEOBLAST | SCLEROSTIN ANTIBODY | RNA | CYTOSCAPE | RECEPTOR | CANCELLOUS BONE | PATHWAY | GENETICS & HEREDITY | GENE-EXPRESSION | MICE | ADAPTIVE RESPONSE | Adaptation, Physiological | Cell Cycle - genetics | Cell Proliferation - genetics | Osteocytes - metabolism | Humans | Mice, Inbred C57BL | Transcriptome | Carbohydrate Metabolism - genetics | Signal Transduction - genetics | Gene Regulatory Networks | Tibia - pathology | Aging - pathology | Weight-Bearing - physiology | Extracellular Matrix - genetics | Animals | Aging - genetics | Aging - physiology | Female | Osteocytes - pathology | Mice | E2F1 Transcription Factor - genetics | Energy Metabolism - genetics | Tibia - physiopathology | DNA microarrays | Analysis | Cell cycle | Physiological aspects | Carbohydrate metabolism | Genetic transcription | DNA binding proteins | Transforming growth factors | NADH, Nicotinamide adenine dinucleotide | EGF, epidermal growth factor | Sp7, Sp7 transcription factor 7 | Ezh2, enhancer of zeste 2 polycomb repressive complex 2 subunit | Gtse, G2 and S-phase expressed 1 | Cdk6, cyclin dependent kinase 6 | Cdca3, cell division cycle associated 3 | RNA, Ribonucleic acid | BMD, bone mineral density | Research Paper | E2F1, E2F transcription factor 1 | CO2, Carbon dioxide | BMP, bone morphogenetic protein | Ttn, titin | DAVID, Database for Annotation, Visualization and Integrated Discovery | Bcl212, Bcl-2-like protein 2 | Itgb1, integrin subunit beta 1 | Bcl11a, B-cell CLL | Sox9, SRY-box 9 | Gnas, GNAS complex locus | Grb10, growth factor receptor bound protein 10 | ATP, Adenosine triphosphate | EGR2, early growth response 2 | Wnt16, Wnt family member 16 | Vcam1, vascular cell adhesion molecule 1 | Mark3, microtubule affinity regulating kinase 3 | Fzd7, frizzled class receptor 7 | b2MG, Beta-2-microglobulin | KEGG, Kyoto Encyclopaedia of Genes and Genomes | Phex, phosphate regulating endopeptidase homolog, X-linked | RarB, retinoic acid receptor beta | SPEED, Signalling Pathway Enrichment using Experimental Datasets | BiNGO, A Biological Network Gene Ontology tool | PASTAA, Predicted Association of Transcription factors from Annotated Affinities | lymphoma 11A | TGFb, transforming growth factor beta 1 | Tcf15, transcription factor 15 | Igf1, insulin like growth factor 1 | Dkk1, dickkopf WNT signalling pathway inhibitor 1 | Des, desmin | MAPK, mitogen-activated protein kinase | Dab2IP, disabled 2 interacting protein | Mmp25, matrix metallopeptidase 25 | Actn2, actinin alpha 2 | qRT-PCR, quantitative real time polymerase chain reaction | Mef2c, myocyte enhancer factor 2C | Mki67, marker of proliferation Ki-67 | Pitx2, paired-like homeodomain transcription factor 2 | MCM, Minichromosome maintenance protein complex | Cdc20, cell division cycle 20 | MEPE, matrix extracellular phosphoglycoprotein | QTC, Quality Threshold Clustering | TNFa, tumor necrosis factor a | Fgf7, fibroblast growth factor 7 | Igf2, insulin like growth factor 2 | Ccna2, cyclin A2 | Fgfr1, fibroblast growth factor receptor 1 | Pkia, protein kinase (cAMP-dependent, catalytic) inhibitor alpha | Ccnd3, cyclin D3 | Myf6, myogenic factor 6 | Sost, sclerostin | Sox6, SRY-box 6 | Atf3, activating transcription factor 3 | MMP13, matrix metallopeptidase 13 | AMPK, adenosine monophosphate-activated protein kinase | Mapt, microtubule associated protein tau | Scyl1, SCY1 like pseudokinase 1 | Insig2, insulin induced gene 2 | PBS, Phosphate buffered saline | Kcnma1, potassium calcium-activated channel subfamily M alpha 1 | FDR, false discovery rate | VegfA, vascular endothelial growth factor A | Nusap1, nucleolar and spindle associated protein 1 | ECM, Extracellular matrix | PdgfA, platelet derived growth factor, alpha
Journal Article
Journal of Thrombosis and Haemostasis, ISSN 1538-7933, 05/2017, Volume 15, Issue 5, pp. 1009 - 1019
Journal Article
Biochemical journal, ISSN 1470-8728, 2013, Volume 451, Issue 3, pp. 427 - 437
The compound BAY 11-7082 inhibits IκBα [inhibitor of NF-κB (nuclear factor κB)α] phosphorylation in cells and has been used to implicate the canonical IKKs (IκB kinases) and NF-κB in >350 publications... 
Linear ubiquitin assembly complex (LUBAC) | Myeloid differentiation factor 88 (MyD88) | Nuclear factor κB (NF-κB) | Lymphoma | Ubiquitin conjugating 13 (Ubc13) | Proteasome | nuclear factor kappa B (NF-kappa B) | proteasome | ACTIVATION | CELL LYMPHOMA-CELLS | LINEAR POLYUBIQUITIN CHAINS | linear ubiquitin assembly complex (LUBAC) | BIOCHEMISTRY & MOLECULAR BIOLOGY | lymphoma | KINASE | IKK | myeloid differentiation factor 88 (MyD88) | CROSS-TALK | IN-VIVO | CONJUGATING ENZYMES | NF-KAPPA-B | NEMO | ubiquitin conjugating 13 (Ubc13) | Nitriles - pharmacology | Humans | Receptors, Interleukin-1 - genetics | Ubiquitin - metabolism | Molecular Sequence Data | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | I-kappa B Proteins - metabolism | Sulfones - pharmacology | Proteasome Endopeptidase Complex - drug effects | Ubiquitination - drug effects | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Myeloid Differentiation Factor 88 - antagonists & inhibitors | Amino Acid Sequence | NF-KappaB Inhibitor alpha | Ubiquitin - antagonists & inhibitors | NF-kappa B p50 Subunit - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Myeloid Differentiation Factor 88 - genetics | Ubiquitin-Conjugating Enzymes - genetics | Macrophages - cytology | Interleukin-1 - pharmacology | Receptors, Interleukin-1 - metabolism | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Ubiquitin-Conjugating Enzymes - metabolism | Lipopolysaccharides - pharmacology | Cell Line, Tumor | Macrophages - drug effects | Mice | Proteasome Endopeptidase Complex - metabolism | Ubiquitin-Conjugating Enzymes - antagonists & inhibitors | Myeloid Differentiation Factor 88 - metabolism | TRAF, tumour-necrosis-factor-receptor-associated factor | HRMS, high-resolution mass spectra | K48-pUb, Lys48-linked polyubiquitin | UBE, ubiquitin-activating enzyme | IKK, IκB kinase | NEMO, NF-κB essential modifier | HEK, human embryonic kidney | TBK1, tumour-necrosis-factor-receptor-associated factor-associated NF-κB activator-binding kinase 1 | JNK, c-Jun N-terminal kinase | HOIP, haem-oxidized IRP2 ligase-1-interacting protein | LUBAC, linear ubiquitin assembly complex | ERK, extracellular-signal-regulated kinase | HTLV-1, human T-cell lymphotropic virus 1 | K63-pUb, Lys63-linked polyubiquitin | IRAK, IL-receptor-associated kinase | pUb, polyubiquitin | IκB, inhibitor of NF-κB | MALDI–TOF, matrix-assisted laser-desorption ionization–time-of-flight | GAPDH, glyceraldehyde-3-phosphate dehydrogenase | MyD88, myeloid differentiation factor 88 | DLBCL, diffuse large B-cell lymphoma | GFP, green fluorescent protein | NF-κB, nuclear factor κB | MS, tandem MS | Ubc, ubiquitin conjugating | TAK1, transforming growth factor β-activated kinase 1 | DAPI, 4′,6-diamidino-2-phenylindole | IL-1R, IL-1 receptor | DMEM, Dulbecco’s modified Eagle’s medium | HIF1α, hypoxia-inducible factor 1α | IL, interleukin | NEDD8, neural-precursor-cell-expressed developmentally down-regulated 8 | RBR, RING-between-RING, TAB, TAK1-binding protein | LPS, lipopolysaccharide | nuclear factor κB (NF-κB) | MAPK, mitogen-activated protein kinase | PAMP, pathogen-associated molecular pattern
Journal Article