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Immunity, ISSN 1074-7613, 2009, Volume 31, Issue 6, pp. 873 - 884
Journal Article
Immunity, ISSN 1074-7613, 11/2014, Volume 41, Issue 5, pp. 762 - 775
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 09/2009, Volume 86, Issue 3, pp. 655 - 662
A prototypic mechanism for enhancement of inflammation by an endogenous molecule is described. The nuclear protein HMGB1 has previously been demonstrated to... 
innate cell‐mediated immunity | cell activation | Innate cell-mediated immunity | Cell activation | CELLS | ACTIVATION | RECEPTOR | IMMUNOLOGY | innate cell-mediated immunity | HUMAN MONOCYTES | CELL BIOLOGY | GROUP CHROMOSOMAL-PROTEINS | AMPHOTERIN | DNA | HIGH-MOBILITY-GROUP-BOX-1 | MOBILITY GROUP BOX-1 | HEMATOLOGY | BINDING | Coculture Techniques | Cytokines - analysis | Humans | Synovial Membrane - immunology | HMGB1 Protein - immunology | HMGB1 Protein - isolation & purification | Lipopolysaccharides - immunology | HMGB1 Protein - genetics | Recombinant Proteins - isolation & purification | Toll-Like Receptor 9 - immunology | Signal Transduction - immunology | HMGB1 Protein - pharmacology | Leukocytes, Mononuclear - immunology | Fibroblasts - metabolism | Inflammation Mediators - immunology | Cell Survival - drug effects | Biotinylation | Leukocytes, Mononuclear - drug effects | Oligodeoxyribonucleotides - immunology | Cells, Cultured | Interleukin-1beta - immunology | Rats | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Recombinant Proteins - pharmacology | Synovial Membrane - metabolism | Arthritis, Rheumatoid - pathology | Animals | Recombinant Proteins - immunology | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Interleukin-6 - biosynthesis | Toll-Like Receptor 2 - immunology | Fibroblasts - immunology | Ligands | Toll-Like Receptor 1 - immunology
Journal Article
The Journal of Immunology, ISSN 0022-1767, 04/2009, Volume 182, Issue 7, pp. 4289 - 4295
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2007, Volume 282, Issue 36, pp. 26014 - 26025
Mycobacterium tuberculosis lipomannans (LMs) modulate the host innate immune response. The total fraction of Mycobacterium bovis BCG LM was shown both to... 
DENDRITIC CELL-FUNCTION | TUMOR-NECROSIS-FACTOR | BACILLUS-CALMETTE-GUERIN | C-TYPE LECTIN | SURFACTANT PROTEIN-A | BOVIS BCG | NITRIC-OXIDE SYNTHASE | DC-SIGN | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUBERCULOSIS INFECTION | MYO-INOSITOL ANCHOR | Mannose-Binding Lectins - metabolism | Mycobacterium bovis - immunology | Toll-Like Receptor 2 - agonists | Membrane Glycoproteins - metabolism | Humans | Lectins, C-Type - immunology | Mycobacterium tuberculosis - immunology | Lipopolysaccharides - metabolism | Cell Adhesion Molecules - immunology | Lipopolysaccharides - immunology | Lectins, C-Type - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Macrophages - secretion | Toll-Like Receptor 1 - agonists | Myeloid Differentiation Factor 88 - immunology | Tumor Necrosis Factor-alpha - immunology | Toll-Like Receptor 4 - agonists | Interleukin-12 Subunit p40 - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Acylation | Cell Line | Nitric Oxide - immunology | Immunity, Innate - drug effects | Macrophage Activation - immunology | Receptors, Interleukin-1 - immunology | Tumor Necrosis Factor-alpha - secretion | Receptors, Cell Surface - metabolism | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Cell Adhesion Molecules - metabolism | Receptors, Cell Surface - immunology | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Nitric Oxide - secretion | Animals | Mannose-Binding Lectins - immunology | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Toll-Like Receptor 1 - immunology | Macrophage Activation - drug effects | Myeloid Differentiation Factor 88 - metabolism | Toll-Like Receptor 1 | Toll-Like Receptor 2 | Toll-Like Receptor 4 | Cell Adhesion Molecules | Receptors, Cell Surface | Macrophages | Lipopolysaccharides | Life Sciences | Immunology | Membrane Glycoproteins | Mannose-Binding Lectins | Mycobacterium bovis | Signal Transduction | Lectins, C-Type | Inflammation | Myeloid Differentiation Factor 88 | Nitric Oxide | Tumor Necrosis Factor-alpha | Immunity, Natural | Macrophage Activation | Mycobacterium tuberculosis | Receptors, Interleukin-1 | Interleukin-12 Subunit p40
Journal Article
Journal Article
Clinical Immunology, ISSN 1521-6616, 2008, Volume 128, Issue 3, pp. 400 - 408
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2012, Volume 287, Issue 16, pp. 13170 - 13181
Bacterial lipoproteins/lipopeptides inducing host innate immune responses are sensed by mammalian Toll-like receptor 2 (TLR2). These bacterial lipoproteins are... 
OUTER-MEMBRANE | TOLL-LIKE-RECEPTOR-1 | GENOME SEQUENCE | IHEYA RIDGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | STAPHYLOCOCCUS-AUREUS | KAPPA-B | APOLIPOPROTEIN N-ACYLTRANSFERASE | BACILLUS | CUTTING EDGE | Geobacillus - immunology | Toll-Like Receptor 2 - genetics | Bacillus cereus - immunology | Toll-Like Receptor 6 - immunology | Toll-Like Receptor 6 - genetics | Streptococcus sanguis - metabolism | Bacillus subtilis - immunology | Gram-Positive Bacteria - immunology | Geobacillus - metabolism | Pneumonia, Mycoplasma - metabolism | Enterococcus faecalis - immunology | Toll-Like Receptor 6 - metabolism | Lactobacillus - immunology | Pneumonia, Mycoplasma - immunology | Mice, Inbred C57BL | Toll-Like Receptor 1 - metabolism | Bacillus cereus - metabolism | Macrophages, Peritoneal - immunology | Toll-Like Receptor 2 - metabolism | Lactobacillus - metabolism | Gram-Positive Bacteria - metabolism | Toll-Like Receptor 1 - genetics | Animals | Enterococcus faecalis - metabolism | Macrophages, Peritoneal - microbiology | Toll-Like Receptor 2 - immunology | Mice | Toll-Like Receptor 1 - immunology | Streptococcus sanguis - immunology | Bacillus subtilis - metabolism | Host-Pathogen Interactions | Toll-like Receptors (TLR) | Immunology | Low-GC Content | Innate Immunity | Mass Spectrometry (MS) | Lipoprotein | Gram-positive Bacteria
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 04/2013, Volume 133, Issue 4, pp. 988 - 998
Atopic dermatitis (AD) is characterized by epidermal tight junction (TJ) defects and a propensity for skin infections. is sensed by many pattern recognition... 
KAPPA-B ACTIVATION | CYTOKINE PRODUCTION | MESSENGER-RNA | CELL WALL COMPONENTS | CUTANEOUS PERMEABILITY BARRIER | STRATUM-CORNEUM | GENE-EXPRESSION | INNATE IMMUNE-SYSTEM | STAPHYLOCOCCUS-AUREUS | UP-REGULATION | DERMATOLOGY | Toll-Like Receptor 2 - agonists | Toll-Like Receptor 2 - genetics | Humans | Male | RNA, Messenger - metabolism | Dermatitis, Atopic - immunology | Female | Tight Junctions - metabolism | Epidermis - metabolism | Epidermis - pathology | Transcutaneous Electric Nerve Stimulation | Mice, Inbred C57BL | Toll-Like Receptor 1 - metabolism | Antibodies, Neutralizing - pharmacology | Dermatitis, Atopic - pathology | Keratinocytes - cytology | Permeability | Toll-Like Receptor 2 - metabolism | Keratinocytes - immunology | Mice, Knockout | Toll-Like Receptor 1 - genetics | Bacterial Proteins - pharmacology | Animals | Epidermis - immunology | Foreskin - cytology | Tight Junctions - immunology | Keratinocytes - metabolism | Mice | Wound Healing - physiology | Toll-Like Receptor 1 - immunology | Peptidoglycan - pharmacology | Dermatitis, Atopic - metabolism | Tight Junctions - pathology | Immune response | Wound healing | Tight junctions | Keratinocytes | Epidermis | peptidoglycans | Pattern recognition | Zonula occludens-1 protein | Infection | Water loss | Atopic dermatitis | TLR2 protein | Toll-like receptors | Skin
Journal Article
Cellular Immunology, ISSN 0008-8749, 2009, Volume 258, Issue 1, pp. 29 - 37
Journal Article
International Journal of Oncology, ISSN 1019-6439, 01/2017, Volume 50, Issue 1, pp. 41 - 48
Blockade of the programmed-death 1 receptor (PD-1)/programmed-death ligand (PD-L1) pathway efficiently reduces tumour growth and improves survival. Durable... 
PD-L1 | Epithelial-mesenchymal transition | Tumour microenvironment | RECEPTOR | TUMOR-CELLS | POTENTIAL MECHANISM | LUNG-CANCER | ANTI-PD-L1 ANTIBODY | CANCER-PATIENTS | ONCOLOGY | epithelial-mesenchymal transition | tumour microenvironment | B7-H1 | oral cancer | T-CELLS | CLINICAL-SIGNIFICANCE | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Dendritic Cells - immunology | Humans | Gene Expression Regulation, Neoplastic | Dendritic Cells - pathology | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Tumor Microenvironment - genetics | Immunotherapy | Mouth Neoplasms - genetics | Macrophages - immunology | Macrophages - pathology | Antigens, Neoplasm - immunology | Neoplasm Invasiveness - immunology | Carcinoma, Squamous Cell - therapy | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Carcinoma, Squamous Cell - immunology | Mouth Neoplasms - therapy | Mouth Neoplasms - immunology | Tumor Microenvironment - immunology | B7-H1 Antigen - antagonists & inhibitors | Epithelial-Mesenchymal Transition - immunology | Mouth Neoplasms - pathology | Programmed Cell Death 1 Receptor - immunology | CD8-Positive T-Lymphocytes - immunology | Neoplasm Invasiveness - genetics | Programmed Cell Death 1 Receptor - genetics
Journal Article