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Nature immunology, ISSN 1529-2916, 2010, Volume 11, Issue 5, pp. 411 - 418
Sensors of pathogens, such as Toll-like receptors (TLRs), detect microbes to activate transcriptional programs that orchestrate adaptive responses to specific insults... 
UNFOLDED PROTEIN RESPONSE | PATHWAY | ER STRESS | HOST-DEFENSE | ENDOPLASMIC-RETICULUM STRESS | FRANCISELLA-TULARENSIS INFECTION | IMMUNOLOGY | LIVE VACCINE STRAIN | INFLAMMATORY RESPONSE | NEGATIVE REGULATOR | SYSTEMS BIOLOGY | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2009, Volume 284, Issue 36, pp. 24035 - 24048
..., macrophages need double stimulation with ligands to both Toll-like receptors (TLRs) for IL-1β... 
GENE | LEUCINE-RICH PROTEOGLYCANS | CASPASE-1 | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE IMMUNE-RESPONSE | HYALURONAN | IL-1-BETA | SECRETION | ATP | NALP3 INFLAMMASOME | Toll-Like Receptor 2 - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Immunity, Innate - genetics | Extracellular Matrix - metabolism | Caspase 1 - metabolism | Male | Ureteral Obstruction - metabolism | Interleukin-1beta - genetics | Inflammation - metabolism | Kidney - metabolism | Carrier Proteins - immunology | Signal Transduction | Proteoglycans - immunology | Extracellular Matrix Proteins - genetics | Proteoglycans - metabolism | Toll-Like Receptor 4 - genetics | Biglycan | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Ureteral Obstruction - immunology | Macrophages - metabolism | Receptors, Purinergic P2 - metabolism | Toll-Like Receptor 2 - immunology | Mice | Proteoglycans - genetics | Extracellular Matrix - immunology | Kidney - immunology | Caspase 1 - immunology | Extracellular Matrix - genetics | Receptors, Purinergic P2 - genetics | Ureteral Obstruction - genetics | Interleukin-1beta - biosynthesis | Lung - metabolism | Receptors, Purinergic P2 - immunology | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Carrier Proteins - biosynthesis | Interleukin-1beta - immunology | Inflammation - immunology | Protein Structure, Tertiary - genetics | Receptors, Purinergic P2X7 | Carrier Proteins - genetics | Immunity, Innate - immunology | Animals | Caspase 1 - genetics | Extracellular Matrix Proteins - immunology | Receptors, Purinergic P2X4 | Inflammation - genetics | Lung - immunology | Animal models | Reactive oxygen species | Oligomerization | Proteoglycans | Transcription | Lung | Inflammation | Macrophages | Kidney | Stress | Caspase-1 | Purine P2X receptors | Signal transduction | Cell activation | TLR2 protein | Interleukin 1 | Toll-like receptors | Extracellular matrix | Sepsis | Cooperativity | Injuries | Glycobiology and Extracellular Matrices
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 09/2008, Volume 283, Issue 36, pp. 24748 - 24759
...), have been shown to inhibit signaling induced by bacterial lipopeptide or lipopolysaccharide (LPS), yet the mechanisms responsible for the inhibition of Toll-like receptor... 
ACTIVATION | COMPLEX | ESCHERICHIA-COLI LIPOPOLYSACCHARIDE | INDUCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | LIPID RAFTS | LOW-DENSITY-LIPOPROTEIN | MD-2 | HEME OXYGENASE-1 | MONOCYTE CHEMOTACTIC PROTEIN-1 | OXIDATION-PRODUCTS | Phosphatidylcholines - immunology | Reactive Oxygen Species - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lymphocyte Antigen 96 - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | Macrophages, Peritoneal - cytology | Flagellin - pharmacology | Lymphocyte Antigen 96 - agonists | Oxidation-Reduction - drug effects | Tumor Necrosis Factor-alpha - immunology | Toll-Like Receptor 4 - agonists | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Carrier Proteins - immunology | Guanosine - analogs & derivatives | Poly I-C - pharmacology | Macrophages, Peritoneal - immunology | Toll-Like Receptor 4 - genetics | Lipopolysaccharide Receptors - immunology | Toll-Like Receptor 4 - immunology | p38 Mitogen-Activated Protein Kinases - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | I-kappa B Kinase - immunology | Mice | Mice, Inbred BALB C | Macrophages, Peritoneal - metabolism | Oligodeoxyribonucleotides - pharmacology | Immunologic Factors - pharmacology | Lymphocyte Antigen 96 - immunology | Tumor Necrosis Factor-alpha - metabolism | Toll-Like Receptor 2 - agonists | Acute-Phase Proteins - genetics | Lipopolysaccharide Receptors - metabolism | Lymphocyte Antigen 96 - genetics | Carrier Proteins - agonists | I-kappa B Kinase - metabolism | Reactive Oxygen Species - immunology | Female | Membrane Glycoproteins - immunology | Cell Line | Acute-Phase Proteins - metabolism | Phosphatidylcholines - pharmacology | Membrane Glycoproteins - agonists | p38 Mitogen-Activated Protein Kinases - genetics | I-kappa B Kinase - genetics | Membrane Glycoproteins - genetics | Carrier Proteins - genetics | Phosphatidylcholines - genetics | Animals | Carrier Proteins - metabolism | RNA - pharmacology | Guanosine - pharmacology | Lipopolysaccharide Receptors - genetics | Signal Transduction - physiology | Acute-Phase Proteins - agonists | Acute-Phase Proteins - immunology | Lipids and Lipoproteins | Metabolism, Regulation, and Signaling
Journal Article
Autoimmunity, ISSN 1607-842X, 2014, Volume 48, Issue 1, pp. 1 - 12
.... It requires IgH germline IH-CH transcription and expression of AID, both of which are induced by engagement of CD40 or dual engagement of a Toll-like receptor (TLR... 
lipopolysaccharides | Antibody | T-independent antibody response | class switch DNA recombination | B cells | CpG | B cell receptor | Toll-like receptor | Class switch DNA recombination | Lipopolysaccharides | CROSS-REGULATION | ACTIVATION | RECOGNITION | INNATE | MICRORNAS | IMMUNOLOGY | AUTOIMMUNE | TOLL-LIKE RECEPTORS | AID | NF-KAPPA-B | EXPRESSION | Toll-Like Receptor 2 - genetics | Calcium - metabolism | Lipopeptides - pharmacology | Toll-Like Receptor 9 - genetics | Calcium - immunology | Immunoglobulin G - classification | Toll-Like Receptor 9 - immunology | Immunoglobulin G - biosynthesis | Recombination, Genetic | Toll-Like Receptor 7 - immunology | CD40 Antigens - genetics | Cell Differentiation | Immunoglobulin Class Switching - drug effects | Membrane Glycoproteins - immunology | Toll-Like Receptor 7 - genetics | B-Lymphocytes - cytology | Signal Transduction | Gene Expression Regulation - immunology | Mice, Inbred C57BL | Toll-Like Receptor 4 - genetics | Imidazoles - pharmacology | Toll-Like Receptor 4 - immunology | Receptors, Antigen, B-Cell - immunology | Membrane Glycoproteins - genetics | Receptor Cross-Talk - immunology | Toll-Like Receptor 1 - genetics | B-Lymphocytes - drug effects | Animals | B-Lymphocytes - immunology | CD40 Ligand - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Receptors, Antigen, B-Cell - genetics | Mice | Toll-Like Receptor 1 - immunology | CD40 Antigens - immunology | Cytokines - biosynthesis | antibody, AID | cytokine | immunoglobulin | B cell receptor (BCR) | lipopolysaccharides (LPS) | germinal center | Toll-like Receptor (TLR) | class switch DNA recombination (CSR)
Journal Article
BMC gastroenterology, ISSN 1471-230X, 2011, Volume 11, Issue 1, pp. 138 - 138
Background: Dysregulation of innate immune response by Toll-Like Receptors (TLRs) is a key feature in Ulcerative Colitis... 
MESSENGER-RNA EXPRESSION | POUCH MUCOSA | GASTROENTEROLOGY & HEPATOLOGY | TLR4 | INTESTINAL EPITHELIAL-CELLS | COLONIC-MUCOSA | BACTERIAL-DNA | BOWEL-DISEASE | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Toll-Like Receptor 2 - genetics | Toll-Like Receptor 5 - genetics | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Toll-Like Receptor 9 - genetics | Male | Gene Expression Profiling | RNA, Messenger - metabolism | Young Adult | Toll-Like Receptor 6 - genetics | Statistics, Nonparametric | Adult | Female | Toll-Like Receptor 3 - genetics | Toll-Like Receptors - metabolism | Interleukin-6 - metabolism | Severity of Illness Index | Toll-Like Receptor 6 - metabolism | Toll-Like Receptor 7 - genetics | Toll-Like Receptor 5 - metabolism | Toll-Like Receptor 7 - metabolism | Interleukin-6 - genetics | Colitis, Ulcerative - metabolism | Toll-Like Receptor 1 - metabolism | Toll-Like Receptor 4 - genetics | Colitis, Ulcerative - pathology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Toll-Like Receptor 1 - genetics | Biopsy | Toll-Like Receptors - genetics | Adolescent | Toll-Like Receptor 8 - genetics | Aged | Toll-Like Receptor 9 - metabolism | Toll-Like Receptor 8 - metabolism | Measurement | Toll-like receptors | Messenger RNA | Research | Patient outcomes | Ulcerative colitis | Inflammatory bowel disease | Studies | Proteins | Nutrition research | Gene expression | Manuscripts
Journal Article
The Journal of immunology (1950), ISSN 0022-1767, 11/2005, Volume 175, Issue 10, pp. 6723 - 6732
Antiviral immunity requires early and late mechanisms in which IFN-alpha and IL-12 play major roles. However, the initial events leading to their production... 
PLASMACYTOID DENDRITIC CELLS | NATURAL-KILLER-CELLS | DOUBLE-STRANDED-RNA | ANTIVIRAL DEFENSE | INTERFERON-GAMMA | NK CELLS | TOLL-LIKE RECEPTOR-9 | MOUSE-CYTOMEGALOVIRUS | IMMUNOLOGY | VIRAL-INFECTION | ACTIVATION RECEPTOR | Liver - pathology | Toll-Like Receptor 2 - genetics | Dendritic Cells - immunology | Toll-Like Receptor 9 - genetics | Dendritic Cells - pathology | Cytomegalovirus Infections - immunology | Toll-Like Receptor 9 - immunology | Toll-Like Receptor 2 - deficiency | Liver - immunology | Toll-Like Receptor 4 - deficiency | Toll-Like Receptor 3 - deficiency | Base Sequence | Adaptor Proteins, Signal Transducing - immunology | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Female | Cell Differentiation | Cytomegalovirus Infections - pathology | Toll-Like Receptor 3 - genetics | Receptors, Immunologic - immunology | Immunoglobulin Class Switching | Interleukin-12 - biosynthesis | Antibodies, Viral - biosynthesis | Interferon-alpha - biosynthesis | Mice, Inbred C57BL | Toll-Like Receptor 9 - deficiency | Receptors, Immunologic - deficiency | Toll-Like Receptor 4 - genetics | Muromegalovirus - pathogenicity | Toll-Like Receptor 3 - immunology | Toll-Like Receptor 4 - immunology | Myeloid Differentiation Factor 88 | Mice, Knockout | Muromegalovirus - immunology | Dendritic Cells - classification | Animals | Adaptor Proteins, Signal Transducing - deficiency | Adaptor Proteins, Signal Transducing - genetics | Toll-Like Receptor 2 - immunology | Antigens, Differentiation - genetics | Mice | Muromegalovirus - genetics | DNA, Viral - genetics | Receptors, Immunologic - genetics | Interferon-gamma - biosynthesis | Antigens, Differentiation - immunology | Cytotoxicity, Immunologic | Life Sciences | Immunology
Journal Article
Circulation research, ISSN 1524-4571, 2013, Volume 112, Issue 1, pp. 103 - 112
.... Such conditions are associated with an appearance of altered-self ligands in circulation that can be recognized by Toll-like receptors (TLRs... 
carboxy(alkylpyrrole) protein adducts | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | HUMAN PLASMA | MACROPHAGES | DEPENDENT PROTEIN-KINASE | MACULAR DEGENERATION | Toll-like receptor 9 | platelets | LOW-DENSITY LIPOPROTEINS | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | OXIDIZED PHOSPHOLIPIDS | CD36 | HEMATOLOGY | oxidative stress | B SCAVENGER RECEPTORS | Toll-Like Receptor 9 - blood | CD36 Antigens - deficiency | Phosphorylation | Blood Platelets - immunology | CD36 Antigens - genetics | Toll-Like Receptor 2 - genetics | Oxidative Stress | Humans | Toll-Like Receptor 9 - genetics | Toll-Like Receptor 2 - deficiency | Transfection | src-Family Kinases - blood | Time Factors | Toll-Like Receptor 6 - genetics | Phosphatidylinositol 3-Kinase - blood | Thrombosis - blood | Genes, Reporter | Disease Models, Animal | Cell Line | Scavenger Receptors, Class B - deficiency | Signal Transduction | Mice, Inbred C57BL | Toll-Like Receptor 9 - deficiency | Myeloid Differentiation Factor 88 - genetics | Thrombosis - immunology | Scavenger Receptors, Class B - genetics | Immunity, Innate | Toll-Like Receptor 6 - deficiency | Mice, Knockout | Platelet Aggregation | Animals | Myeloid Differentiation Factor 88 - deficiency | Blood Platelets - metabolism | Platelet Activation | Proto-Oncogene Proteins c-akt - blood | Ligands | Thrombosis - genetics | Interleukin-1 Receptor-Associated Kinases - blood | Mice | Serum Albumin - metabolism
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2005, Volume 175, Issue 10, pp. 6303 - 6310
Induction of tissue-specific experimental autoimmune diseases involves an obligatory adjuvant effect to trigger an innate response of a type that will drive a... 
MYCOBACTERIUM-TUBERCULOSIS | MYD88-DEFICIENT MICE | ENCEPHALITOGENIC T-CELLS | DENDRITIC CELLS | TOLL-LIKE RECEPTORS | IL-18-DEFICIENT MICE | DEFICIENT MICE | IMMUNOLOGY | ADAPTER PROTEIN | CUTTING EDGE | INNATE IMMUNITY | Uveitis - etiology | Interleukin-18 - immunology | Toll-Like Receptor 2 - genetics | Receptors, Interleukin-1 - genetics | Toll-Like Receptor 9 - genetics | Male | Th2 Cells - immunology | Autoimmune Diseases - etiology | Th1 Cells - immunology | Toll-Like Receptor 9 - immunology | Toll-Like Receptor 2 - deficiency | Toll-Like Receptor 4 - deficiency | Receptors, Interleukin-1 - deficiency | Adaptor Proteins, Signal Transducing - immunology | Female | Receptors, Immunologic - immunology | Autoimmune Diseases - pathology | Signal Transduction | Receptors, Interleukin-1 - immunology | Mice, Inbred C57BL | Toll-Like Receptor 9 - deficiency | Autoimmune Diseases - immunology | Receptors, Immunologic - deficiency | Interleukin-18 - deficiency | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | Myeloid Differentiation Factor 88 | Mice, Knockout | Animals | Uveitis - immunology | Adaptor Proteins, Signal Transducing - deficiency | Adaptor Proteins, Signal Transducing - genetics | Toll-Like Receptor 2 - immunology | Antigens, Differentiation - genetics | Mice | Receptors, Interleukin-1 Type I | Receptors, Immunologic - genetics | Interleukin-18 - genetics | Uveitis - pathology | Antigens, Differentiation - immunology
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2008, Volume 121, Issue 4, pp. 1013 - 1019
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2013, Volume 39, Issue 2, pp. 372 - 385
...), highly adaptive lactobacilli are expanded and produce an aryl hydrocarbon receptor (AhR) ligand—indole-3-aldehyde—that contributes to AhR-dependent Il22... 
INTESTINAL INFLAMMATION | LACTOBACILLUS-PLANTARUM | CANDIDA-ALBICANS | GASTROINTESTINAL-TRACT | GUT | REGULATORY T-CELLS | MICE | COLONIZATION | JOHNSONII STRAIN NCC533 | INNATE LYMPHOID-CELLS | IMMUNOLOGY | Indoleamine-Pyrrole 2,3,-Dioxygenase - genetics | Lactobacillus reuteri - metabolism | Receptors, Aryl Hydrocarbon - deficiency | Toll-Like Receptor 2 - genetics | Tryptophan - chemistry | Indoleamine-Pyrrole 2,3,-Dioxygenase - deficiency | Lactobacillus reuteri - immunology | Tryptophan - metabolism | Interleukins - metabolism | Gastrointestinal Tract - immunology | Toll-Like Receptor 2 - deficiency | Indoles - metabolism | Receptors, Aryl Hydrocarbon - metabolism | Female | Candidiasis - immunology | Basic Helix-Loop-Helix Transcription Factors - deficiency | Basic Helix-Loop-Helix Transcription Factors - genetics | Mice, Inbred C57BL | Myeloid Differentiation Factor 88 - genetics | Receptors, Aryl Hydrocarbon - genetics | Gastrointestinal Tract - microbiology | Interleukin-17 - genetics | Interleukin-17 - deficiency | Candida albicans - immunology | Mice, SCID | Gastrointestinal Tract - metabolism | Mice, Inbred C3H | Mice, Knockout | Probiotics | Animals | Myeloid Differentiation Factor 88 - deficiency | Energy Metabolism | Lactobacillus reuteri - growth & development | Mice | Mice, Inbred BALB C | Metagenome | Tryptophan | Metabolites | Interleukins | Aldehydes | Medical research | Stomach | Colleges & universities | Homeostasis | Ligands | Software | Infections | Metabolism | Experiments
Journal Article