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Nature Immunology, ISSN 1529-2908, 04/2008, Volume 9, Issue 4, pp. 361 - 368
Journal Article
Nature medicine, ISSN 1078-8956, 2010, Volume 16, Issue 4, pp. 413 - 419
Journal Article
Journal Article
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2012, Volume 122, Issue 10, pp. 3476 - 3489
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 07/2010, Volume 114, Issue 1, pp. 13 - 27
P>The responses of the brain to infection, ischemia and trauma share remarkable similarities. These and other conditions of the CNS coordinate an innate immune... 
MD-2 | Neuroinflammation | Sepsis | Glia | Endogenous ligand | Toll-like receptor | INFLAMMATORY RESPONSES | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | endogenous ligand | DORSAL-VENTRAL POLARITY | EXPERIMENTAL STROKE | glia | NEUROSCIENCES | HEAT-SHOCK PROTEINS | AMYLOID BETA-PEPTIDE | neuroinflammation | LIPID-RAFTS | SIGNAL-TRANSDUCTION INHIBITOR | sepsis | GENE-PRODUCT | INNATE IMMUNITY | Neoplasms - metabolism | Humans | Brain Ischemia - metabolism | Pain - immunology | Brain Ischemia - immunology | Pain - metabolism | Central Nervous System Diseases - metabolism | Synaptic Transmission | Sepsis - drug therapy | Inflammation - metabolism | Pain - drug therapy | Sepsis - metabolism | Toll-Like Receptor 4 - agonists | Toll-Like Receptor 4 - antagonists & inhibitors | Alzheimer Disease - immunology | Stroke - immunology | Sepsis - immunology | Signal Transduction | Neuroglia - physiology | Alzheimer Disease - drug therapy | Central Nervous System Diseases - immunology | Inflammation - immunology | Stroke - drug therapy | Immunity, Innate | Neoplasms - drug therapy | Toll-Like Receptor 4 - physiology | Stroke - metabolism | Animals | Neoplasms - immunology | Brain Ischemia - drug therapy | Alzheimer Disease - metabolism | Central Nervous System Diseases - drug therapy | Neuroimmunomodulation | Neurosciences | Pathology | Brain | Ischemia | Neurons | Trauma
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1279 - 1285
Toll-like receptor 4 (TLR4) has a key role in innate immunity by activating an inflammatory signaling pathway. Free fatty acids (FFAs) stimulate adipose tissue... 
PATHWAYS | MEDICINE, RESEARCH & EXPERIMENTAL | ADIPOCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR-KAPPA-B | SATURATED FATTY-ACIDS | DIET-INDUCED OBESITY | CELL BIOLOGY | TOLL-LIKE RECEPTOR-4 | CHRONIC INFLAMMATION | EXPRESSION | ADIPOSE-TISSUE | INNATE IMMUNITY | Inflammation - chemically induced | Humans | Molecular Sequence Data | NF-kappa B - metabolism | Adipocytes - drug effects | Fatty Acids, Nonesterified - pharmacology | Adipocytes - physiology | Obesity - blood | Insulin Resistance - physiology | Base Sequence | alpha-2-HS-Glycoprotein - antagonists & inhibitors | Toll-Like Receptor 4 - antagonists & inhibitors | Binding Sites | Cytokines - genetics | Toll-Like Receptor 4 - analysis | Blood Glucose - analysis | Obesity - physiopathology | Toll-Like Receptor 4 - chemistry | 3T3-L1 Cells | alpha-2-HS-Glycoprotein - physiology | Mice, Knockout | Gene Expression Regulation - drug effects | Toll-Like Receptor 4 - physiology | Receptors, Leptin - deficiency | Diabetes Mellitus, Type 2 - blood | alpha-2-HS-Glycoprotein - toxicity | Animals | Diabetes Mellitus, Type 2 - physiopathology | alpha-2-HS-Glycoprotein - chemistry | Signal Transduction - drug effects | Galactosides - metabolism | Ligands | Mice, Obese | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | Chronic Disease | Dietary Fats - toxicity | Cytokines - biosynthesis | Adipocytes - chemistry | Inflammation - physiopathology | Insulin resistance | Alpha fetoproteins | Care and treatment | Diagnosis | Health aspects | Ligands (Biochemistry) | Signal transduction | Lipids | Cytokines | Gene expression | Immune system
Journal Article