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Clinical science (London, England : 1979), ISSN 0143-5221, 2015, Volume 129, Issue 2, pp. 187 - 198
Myocardial ischaemia-reperfusion (MIR) triggers a sterile inflammatory response important for myocardial healing, but which may also contribute to adverse... 
Myocardial infarction | Toll-like receptors | Animal model | Oxidative stress | Innate immunity | Inflammation | CATALASE | MEDICINE, RESEARCH & EXPERIMENTAL | ISCHEMIA/REPERFUSION INJURY | ACTIVATED PROTEIN-KINASE | toll-like receptors | PEROXYNITRITE | myocardial infarction | IDENTIFICATION | HEART | INFARCTION | innate immunity | inflammation | IN-VIVO | MICE | animal model | oxidative stress | Myocardial Infarction - genetics | Phosphorylation | Myocardium - immunology | Oxidative Stress | Ventricular Function, Left | Toll-Like Receptor 5 - genetics | Male | Myocardial Infarction - immunology | Inflammation - metabolism | Myocardial Reperfusion Injury - pathology | Myocardial Infarction - pathology | Myocardium - metabolism | Inflammation Mediators - metabolism | Ventricular Dysfunction, Left - pathology | Myocardial Infarction - physiopathology | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Myocardial Reperfusion Injury - genetics | Disease Models, Animal | Myocardial Reperfusion Injury - immunology | Mice, Inbred C57BL | Genotype | Myocardium - pathology | Inflammation - immunology | Myocardial Infarction - metabolism | Toll-Like Receptor 5 - deficiency | Immunity, Innate | Ventricular Dysfunction, Left - immunology | Myocardial Reperfusion Injury - physiopathology | Ventricular Dysfunction, Left - physiopathology | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Myocardial Reperfusion Injury - metabolism | Phenotype | Animals | Inflammation - genetics
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 11/2015, Volume 1852, Issue 11, pp. 2456 - 2466
Vascular dysfunction, characterized by the endothelial-to-mesenchymal transition (EndMT), contributes to the development of cardiac fibrosis induced by... 
Toll-like receptor 5 | Cardiac fibrosis | Endothelial-to-mesenchymal transition | Endothelium | HYPERTROPHY | BIOPHYSICS | BIOCHEMISTRY & MOLECULAR BIOLOGY | Inflammation | Analysis | Fibrosis | Stem cells
Journal Article
Science, ISSN 0036-8075, 4/2010, Volume 328, Issue 5975, pp. 228 - 231
Journal Article
Journal Article
Cell Host & Microbe, ISSN 1931-3128, 08/2012, Volume 12, Issue 2, pp. 139 - 152
Journal Article
Cell Metabolism, ISSN 1550-4131, 12/2015, Volume 22, Issue 6, pp. 983 - 996
Journal Article
Journal of Immunology, ISSN 0022-1767, 02/2014, Volume 192, Issue 4, pp. 1587 - 1596
Journal Article
Scientific reports, ISSN 2045-2322, 2017, Volume 7, Issue 1, p. 42688
Toll-like receptors (TLR) provide a critical link between innate and adaptive immunity, both important players in atherosclerosis. Since evidence for the role... 
IMMUNE-RESPONSE | GENE | INFLAMMATION | CHEMOKINES | MULTIDISCIPLINARY SCIENCES | DISEASE | TOLL-LIKE RECEPTORS | BACTERIAL FLAGELLIN | MICE | EXPRESSION | INNATE IMMUNITY | T-Lymphocyte Subsets - immunology | Plaque, Atherosclerotic - genetics | T-Lymphocyte Subsets - cytology | Monocytes - metabolism | Monocytes - immunology | Cell Movement - genetics | Cell Movement - immunology | Leukocytes - immunology | Atherosclerosis - etiology | Cell Differentiation - genetics | Inflammation Mediators - metabolism | Leukocyte Count | Plaque, Atherosclerotic - pathology | Macrophages - immunology | Disease Models, Animal | Atherosclerosis - pathology | Cytokines - metabolism | Plaque, Atherosclerotic - metabolism | Plaque, Atherosclerotic - immunology | Toll-Like Receptor 5 - deficiency | Atherosclerosis - metabolism | Mice, Knockout | Atherosclerosis - blood | Cell Differentiation - immunology | Macrophages - metabolism | Animals | T-Lymphocyte Subsets - metabolism | Mice | Leukocytes - metabolism | Spleen | TLR5 protein | Flagellin | T cell receptors | Lymphocytes T | Adaptive immunity | Macrophages | Hemopoiesis | CD4 antigen | T-cell receptor | Interleukin 6 | Cell activation | Arteriosclerosis | Rodents | Atherosclerosis | Toll-like receptors | Lipoprotein (low density) receptors | Plasma levels | Receptor mechanisms | Chimeras | Monocyte chemoattractant protein 1
Journal Article
Gut, ISSN 0017-5749, 07/2014, Volume 63, Issue 7, pp. 1069 - 1080
Background Inflammatory bowel disease (IBD) is driven by a seemingly aberrant immune response to the gut microbiota with disease development dictated by... 
PATHOGENESIS | INFLAMMATORY-BOWEL-DISEASE | CROHNS-DISEASE | GENE | GUT | ILEAL MUCOSA | MICE | COLONIZATION | RECEPTOR 5 | GASTROENTEROLOGY & HEPATOLOGY | GENOME-WIDE ASSOCIATION | Lipopolysaccharides - metabolism | Feces - microbiology | Microbiota | Intestinal Mucosa - immunology | Flagellin - metabolism | DNA, Bacterial - analysis | Colitis - immunology | Biomarkers - metabolism | Lipocalins - metabolism | Enzyme-Linked Immunosorbent Assay | Acute-Phase Proteins - metabolism | Bacterial Adhesion - immunology | Oncogene Proteins - metabolism | Escherichia coli - immunology | Bacterial Load | Intestinal Mucosa - microbiology | Toll-Like Receptor 5 - deficiency | Immunity, Innate | Sequence Analysis, DNA | Mice, Knockout | Animals | Colitis - microbiology | Feces - chemistry | Escherichia coli - genetics | Lipocalin-2 | Germ-Free Life | Escherichia coli Infections - immunology | Colon - microbiology | Mice | Chronic Disease | Escherichia coli Infections - complications | Microbiota (Symbiotic organisms) | Escherichia coli | Analysis | Physiological aspects | Environmental aspects | Research | Colitis | Risk factors | Inflammatory bowel disease | Antibiotics | E coli | Rodents | Bacteria | Inflammation | Gene expression | Crohns disease | colitis | gut microbial diversity | 16S rRNA gene pyrosequencing | TLR5 | adherent-invasive Escherichia coli
Journal Article