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Nature Medicine, ISSN 1078-8956, 02/2017, Volume 23, Issue 2, pp. 174 - 184
Low-grade, chronic inflammation has been associated with many diseases of aging, but the mechanisms responsible for producing this inflammation remain unclear.... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | INTERLEUKIN-18 LEVELS | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | CARDIOVASCULAR-DISEASE | RECEPTOR | ESSENTIAL-HYPERTENSION | PROTEINS | T-CELLS | AGE | CELL BIOLOGY | Toll-Like Receptor 5 - genetics | Humans | Middle Aged | Oxidative Stress - immunology | Transcriptome | Immunoblotting | Male | Purinergic P1 Receptor Antagonists - pharmacology | Young Adult | Toll-Like Receptor 8 - immunology | Aging - immunology | Toll-Like Receptor 6 - immunology | Aging - genetics | Calcium-Binding Proteins - immunology | Interleukin-1beta - metabolism | Aged, 80 and over | Caffeine - pharmacology | Hypertension - genetics | Intracellular Signaling Peptides and Proteins - genetics | Interleukin 1 Receptor Antagonist Protein - immunology | Carotid Intima-Media Thickness | Mortality | Neutrophils - drug effects | Hypertension - immunology | Nucleotides - metabolism | Toll-Like Receptor 5 - immunology | Monocytes - drug effects | Regression Analysis | Phenotype | Mice | Calcium-Binding Proteins - genetics | Metabolomics | Pulse Wave Analysis | Neutrophil Activation - drug effects | Cytidine - pharmacology | Interleukin 1 Receptor Antagonist Protein - genetics | Intracellular Signaling Peptides and Proteins - immunology | Cytidine - analogs & derivatives | CARD Signaling Adaptor Proteins - genetics | Toll-Like Receptor 6 - genetics | Adult | Female | Blood Pressure - drug effects | Blood Platelets - drug effects | Macrophages - immunology | Cytokines - immunology | CARD Signaling Adaptor Proteins - immunology | Cell Line | Vascular Stiffness - genetics | Cytokines - metabolism | Oxidative Stress - genetics | Interleukin-1beta - immunology | Adenine - pharmacology | Inflammation - immunology | Inflammasomes - genetics | Vascular Stiffness - immunology | Animals | Inflammasomes - immunology | Toll-Like Receptor 8 - genetics | Inflammation - genetics | Aged | Platelet Activation - drug effects | Genetic aspects | Immune response | Gene expression | Health aspects | Aging | Inflammation | Immune system | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, pp. e15041 - e15041
Newborns and young infants suffer increased infectious morbidity and mortality as compared to older children and adults. Morbidity and mortality due to... 
VACCINE DEVELOPMENT | INFECTIONS | HUMAN NEWBORN | MIFLOWCYT | BIOLOGY | INNATE IMMUNE-RESPONSE | FLOW-CYTOMETRY | BIRTH | INFANT | CHILDHOOD | AGE | Leukocytes, Mononuclear - metabolism | Toll-Like Receptor 2 - agonists | Age Factors | Lipopeptides - pharmacology | Dendritic Cells - immunology | Humans | Middle Aged | Child, Preschool | Infant | Toll-Like Receptor 9 - agonists | Toll-Like Receptor 9 - immunology | Young Adult | Toll-Like Receptor 8 - immunology | Leukocytes, Mononuclear - immunology | Toll-Like Receptor 7 - immunology | Toll-Like Receptor 4 - agonists | Adult | Dendritic Cells - drug effects | Dendritic Cells - metabolism | Infant, Newborn | Cytokines - immunology | Toll-Like Receptor 8 - agonists | Toll-Like Receptor 7 - agonists | Antigen-Presenting Cells - metabolism | Leukocytes, Mononuclear - drug effects | Cytokines - metabolism | Antigen-Presenting Cells - drug effects | Toll-Like Receptors - immunology | Toll-Like Receptor 4 - immunology | Antigen-Presenting Cells - immunology | Toll-Like Receptors - agonists | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Oligodeoxyribonucleotides - pharmacology | Cohort Studies | Infants (Newborn) | Cytokines | Mortality | Vaccines | B cells | Comparative analysis | Health aspects | Neonates | Flow cytometry | Pediatrics | Helper cells | Leukocytes (mononuclear) | Stimulation | Interleukin 23 | Infants | Lymphocytes T | Infections | Ontogeny | Birth | Immunity | Interleukin 6 | Proteins | Immunology | Newborn babies | Toll-like receptors | Children | Age | Immune system | Medical research | Antigens | Immune response | Fetuses | Interleukin 12 | T cell receptors | Tumor necrosis factor-α | Morbidity | Childbirth & labor | Immune systems | Herpes viruses | γ-Interferon | Interleukin 10 | Interferon | Adults | Human behavior | Index Medicus
Journal Article
IMMUNITY, ISSN 1074-7613, 11/2005, Volume 23, Issue 5, pp. 465 - 478
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 07/2017, Volume 214, Issue 7, pp. 2103 - 2119
Neutrophils play a crucial role in host defense. However, neutrophil activation is also linked to autoimmune diseases such as systemic lupus erythematosus... 
PLASMACYTOID DENDRITIC CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | NADPH OXIDASE | SYSTEMIC-LUPUS-ERYTHEMATOSUS | PROPROTEIN CONVERTASES | C1Q DEFICIENCY | INFLAMMATION | FC-GAMMA-RIIIB | TOLL-LIKE RECEPTORS | ALPHA | IMMUNOLOGY | EXTRACELLULAR TRAPS | Extracellular Traps - metabolism | Dendritic Cells - immunology | Humans | Complement C5a - metabolism | Lupus Erythematosus, Systemic - metabolism | Monocytes - metabolism | Monocytes - immunology | Complement Activation - immunology | Receptors, IgG - metabolism | Toll-Like Receptor 8 - immunology | Extracellular Traps - immunology | Flow Cytometry | Toll-Like Receptor 7 - immunology | Lupus Erythematosus, Systemic - immunology | Neutrophils - metabolism | Dendritic Cells - metabolism | Toll-Like Receptor 7 - metabolism | Cells, Cultured | Neutrophils - immunology | Phagocytosis - immunology | Receptors, IgG - immunology | Blotting, Western | Complement C5a - immunology | Antigen-Antibody Complex - immunology | Toll-Like Receptor 8 - metabolism | Shedding | Complement component C5a | Immune clearance | Activation | Proteins | Cell activation | Systemic lupus erythematosus | Proteolysis | Toll-like receptors | Furin | Cleavage | Antigen-antibody complexes | TLR7 protein | Dendritic cells | Neutrophils | Leukocytes (neutrophilic) | Inflammation | Nucleic acids | Immune systems | Signaling | Monocytes | Chronic conditions | Complement activation | Information processing | Autoimmune diseases | Phagocytosis | Index Medicus | s | 311 | 300
Journal Article
Journal of Virology, ISSN 0022-538X, 03/2014, Volume 88, Issue 6, pp. 3329 - 3339
Journal Article
European Journal of Immunology, ISSN 0014-2980, 05/2014, Volume 44, Issue 5, pp. 1503 - 1516
Journal Article
Nature immunology, ISSN 1529-2908, 2010, Volume 11, Issue 5, pp. 419 - U81
Pattern-recognition receptors (PRRs) elicit antiviral immune responses to human immunodeficiency virus type 1 (HIV-1). Here we show that HIV-1 required... 
MYCOBACTERIUM-TUBERCULOSIS | TUMOR-NECROSIS-FACTOR | ACTIVATION | LONG TERMINAL REPEAT | IMMUNE-RESPONSES | IMMUNOLOGY | HUMAN-IMMUNODEFICIENCY-VIRUS | NF-KAPPA-B | MEDIATED INTERNALIZATION | T-CELLS | LANGERHANS CELLS | HIV Envelope Protein gp120 - genetics | HIV-1 - pathogenicity | Phosphorylation | Protein Binding - genetics | Transcriptional Activation - genetics | Virus Replication - genetics | Dendritic Cells - immunology | Humans | Lectins, C-Type - immunology | Dendritic Cells - pathology | NF-kappa B - metabolism | Cell Adhesion Molecules - immunology | Positive Transcriptional Elongation Factor B - metabolism | RNA Polymerase II - metabolism | Transcriptional Activation - immunology | HIV Envelope Protein gp120 - metabolism | HIV Infections - immunology | Lectins, C-Type - metabolism | Toll-Like Receptor 8 - immunology | HIV-1 - physiology | Second Messenger Systems - genetics | Protein Engineering | Dendritic Cells - virology | Dendritic Cells - metabolism | Virus Replication - drug effects | HIV Infections - genetics | HIV Infections - virology | Cells, Cultured | Receptors, Cell Surface - metabolism | Virus Replication - immunology | Immunity, Innate | Cell Adhesion Molecules - metabolism | Receptors, Cell Surface - immunology | Second Messenger Systems - immunology | Proto-Oncogene Proteins c-raf - metabolism | NF-kappa B - genetics | Toll-Like Receptor 8 - metabolism | HIV Infections - metabolism | Sequence Deletion - genetics | Prevention | Transcription factors | Immune response | Dendritic cells | Physiological aspects | Causes of | Genetic aspects | Research | HIV (Viruses) | Health aspects | HIV infection | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2015, Volume 195, Issue 3, pp. 1100 - 1111
Journal Article