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Molecular Immunology, ISSN 0161-5890, 02/2018, Volume 94, pp. 200 - 208
Recent studies have demonstrated the important role of toll-like receptor 9 (TLR9) signalling in autoimmune diseases, but its role in myasthenia gravis (MG)... 
Germinal centre | Follicular helper T cells | Experimental autoimmune myasthenia gravis | Toll-like receptor 9 | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUBSETS | IMMUNOLOGY | HELPER T-CELLS | MULTIPLE-SCLEROSIS | PATHWAY | TLR9 | ANTIBODY-RESPONSES | INNATE IMMUNITY | Oligodeoxyribonucleotides, Antisense - pharmacology | Spleen - immunology | Myasthenia Gravis, Autoimmune, Experimental - immunology | Dendritic Cells - immunology | Myasthenia Gravis, Autoimmune, Experimental - genetics | Rats, Inbred Lew | Toll-Like Receptor 9 - genetics | Germinal Center - drug effects | Spleen - drug effects | T-Lymphocytes, Helper-Inducer - drug effects | T-Lymphocytes, Helper-Inducer - immunology | Dendritic Cells - drug effects | Female | Myasthenia Gravis, Autoimmune, Experimental - drug therapy | Toll-Like Receptor 9 - antagonists & inhibitors | Myasthenia Gravis, Autoimmune, Experimental - pathology | Immunity, Humoral - genetics | Oligodeoxyribonucleotides, Antisense - therapeutic use | Rats | Germinal Center - pathology | Down-Regulation - drug effects | Down-Regulation - genetics | B-Lymphocytes - drug effects | Animals | B-Lymphocytes - immunology | Immunity, Humoral - drug effects | Cell Proliferation - drug effects | Lupus | Viral antibodies | Dendritic cells | Antibodies | B cells | T cells | Autoimmune diseases | Index Medicus
Journal Article
ACS MEDICINAL CHEMISTRY LETTERS, ISSN 1948-5875, 11/2014, Volume 5, Issue 11, pp. 1235 - 1239
Toll-like receptors (TLRs) play important roles in the innate immune system. In fact, recognition of endogenous immune complexes containing self-nucleic acids... 
POTENT | CHEMISTRY, MEDICINAL | LLE | TLR9 | dihydropyrrolo[2,3-d]pyrimidine | MODEL | Toll-like receptor 9 | autoimmune disease | sepsis | PATHOGEN RECOGNITION
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 8723 - 17
Spinal cord (SC) trauma elicits pathological changes at the primary lesion and in regions distant from the injury epicenter. Therapeutic agents that target... 
PATHWAYS | NEUROPATHIC PAIN | ACTIVATION | GLT1 | MICROGLIA | MULTIDISCIPLINARY SCIENCES | MECHANISMS | RAT-BRAIN | INFLAMMATORY RESPONSE | NF-KAPPA-B | ASTROCYTES | Spinal cord | Cytokines | Oligonucleotides | Amino acids | Thorax | Spinal cord injury | Trauma | TLR9 protein | Microglia | γ-Aminobutyric acid | Rodents | Toll-like receptors | Glutamic acid transporter | Growth factors | Sodium-glutamic acid cotransporter | Dorsal horn
Journal Article
Journal Article
ACS Medicinal Chemistry Letters, ISSN 1948-5875, 11/2014, Volume 5, Issue 11, pp. 1235 - 1239
Toll-like receptors (TLRs) play important roles in the innate immune system. In fact, recognition of endogenous immune complexes containing self-nucleic acids... 
LLE | Toll-like receptor 9 | autoimmune disease | dihydropyrrolo[2,3- d ]pyrimidine | sepsis
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 11/2014, Volume 31, Issue 21, pp. 18 - 1806
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2011, Volume 186, Issue 8, pp. 4794 - 4804
Endosomal TLRs play an important role in innate immune response as well as in autoimmune processes. In the therapy of systemic lupus erythematosus,... 
IMMUNOSTIMULATORY CPG-OLIGODEOXYNUCLEOTIDES | CELLS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | ACTIVATION | DNA | COMPLEXES | TOLL-LIKE RECEPTORS | H+-ATPASE | IFN-ALPHA INDUCER | IMMUNOLOGY | NUCLEIC-ACIDS | Humans | Toll-Like Receptor 9 - genetics | Toll-Like Receptor 3 - antagonists & inhibitors | Endosomes - metabolism | Quinolines - pharmacology | Chloroquine - pharmacology | Quinacrine - pharmacology | Transfection | Hydrogen-Ion Concentration - drug effects | HEK293 Cells | Toll-Like Receptors - antagonists & inhibitors | Toll-Like Receptor 3 - genetics | Culture Media - chemistry | Imidazoles - metabolism | Toll-Like Receptor 8 - agonists | Binding, Competitive | Toll-Like Receptor 7 - agonists | Toll-Like Receptor 7 - genetics | Cell Line | Antimalarials - metabolism | Toll-Like Receptor 7 - metabolism | Toll-Like Receptor 9 - antagonists & inhibitors | Cytokines - metabolism | Cells, Cultured | Imidazoles - pharmacology | Nucleic Acids - metabolism | Toll-Like Receptor 3 - metabolism | Toll-Like Receptors - agonists | Antimalarials - pharmacology | Quinolines - metabolism | Animals | Toll-Like Receptors - genetics | Culture Media - pharmacology | Toll-Like Receptor 8 - genetics | Endosomes - chemistry | Toll-Like Receptor 9 - metabolism | Toll-Like Receptor 8 - metabolism | Serum - chemistry | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, pp. e84634 - e84634
Psoriasis is a complex inflammatory disease resulting from the activation of T helper (Th) 1 and Th17 cells. Recent evidence suggests that abnormal activation... 
SIGNALING PATHWAYS | IN-VITRO | MULTIDISCIPLINARY SCIENCES | KERATINOCYTES | EPIDERMAL HYPERPLASIA | ANTIMICROBIAL PEPTIDES | PLASMACYTOID DENDRITIC CELL | EXPRESSION | T-CELLS | PSORIASIS-VULGARIS | INNATE IMMUNITY | RNA, Messenger - immunology | Interleukin-23 - toxicity | Toll-Like Receptor 7 - antagonists & inhibitors | Toll-Like Receptor 9 - antagonists & inhibitors | Dermatitis - immunology | Gene Expression Regulation - immunology | Dermatitis - prevention & control | Gene Expression Regulation - drug effects | Toll-Like Receptor 9 - immunology | Dermatitis - pathology | Signal Transduction - immunology | Toll-Like Receptor 8 - immunology | Animals | Membrane Glycoproteins - antagonists & inhibitors | Toll-Like Receptor 8 - antagonists & inhibitors | Signal Transduction - drug effects | Toll-Like Receptor 7 - immunology | Th17 Cells - immunology | Female | Mice | Membrane Glycoproteins - immunology | Th17 Cells - pathology | Keratin | Psoriasis | Genes | Skin | Inflammation | B cells | Comparative analysis | Gene expression | Dermatitis | Laboratories | Pathogenesis | Hyperplasia | Helper cells | Interleukin 23 | Activation | Antagonists | Lymphocytes T | Kinases | Proteins | Genotype & phenotype | Receptors | Psoriasis vulgaris | Pathways | Lymphocytes | Rodents | Modulation | Toll-like receptors | Skin diseases | Lesions | Growth factors | Antigens | Cytokines | Dendritic cells | Dermatology | Injection | Interleukin 17 | Immune systems | Signaling | Aberration | Pharmaceuticals | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 05/2012, Volume 484, Issue 7397, pp. 251 - 255
Journal Article
Journal Article