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Cell Communication and Signaling, ISSN 1478-811X, 2014, Volume 12, Issue 1, pp. 1 - 13
Background: B cells are important effectors and regulators of adaptive and innate immune responses, inflammation and autoimmunity, for instance in... 
B cell | B10 | Ifenprodil | Memantine | K Ca 3.1 | 3.1 | 1.3 | Nmda-receptor antagonist | K v 1.3 | IL-10 | LPS | NMDA-receptor antagonist | K(v)1.3 | ACTIVATION | POTASSIUM CHANNELS | ALZHEIMERS-DISEASE | B10 CELLS | AUTOIMMUNE ENCEPHALOMYELITIS | CELL BIOLOGY | K(Ca)3.1 | TOLL-LIKE RECEPTORS | ION CHANNELS | LYMPHOCYTE FUNCTION | EXPRESSION | T-CELLS | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Apoptosis - drug effects | Mice, Inbred C57BL | Mice, Transgenic | Interferon-gamma - metabolism | Receptors, Antigen, B-Cell - metabolism | Intermediate-Conductance Calcium-Activated Potassium Channels - metabolism | Toll-Like Receptor 4 - metabolism | Immunoglobulin M - metabolism | B-Lymphocytes - drug effects | Animals | CD40 Antigens - metabolism | Piperidines - pharmacology | Interleukin-10 - genetics | Lipopolysaccharides - pharmacology | Interleukin-10 - metabolism | Kv1.3 Potassium Channel - metabolism | Cell Proliferation - drug effects | B-Lymphocytes - metabolism | Immunoglobulin G - metabolism | Autoimmunity | Encephalitis | Methyl aspartate | Care and treatment | Immunoglobulin G | B cells | Diagnosis | Health aspects | Alzheimer's disease | Risk factors | Immune response | Mitogens | Competition | Antigens | Neurosciences | Multiple sclerosis | Statistical analysis | Experiments | Cell adhesion & migration | Cell growth | Rodents | Ligands | Alzheimers disease | Binding sites | Immune system | Apoptosis | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 140, Issue 2, pp. 697 - 708.e4
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2016, Volume 11, Issue 5, pp. e0155944 - e0155944
Duchenne muscular dystrophy (DMD) is an X-linked muscle disease caused by mutations in the dystrophin gene. The pathology of DMD manifests in patients with... 
RHEUMATOID-ARTHRITIS | CONTROLLED-TRIAL | INTERLEUKIN-1 | INHIBITION | LONG-TERM BENEFITS | REDUCES SKELETAL | MULTIDISCIPLINARY SCIENCES | SKELETAL-MUSCLE DEGENERATION | DUCHENNE MUSCULAR-DYSTROPHY | TNF-ALPHA | ANXIETY-LIKE BEHAVIOR | Interleukin 1 Receptor Antagonist Protein - administration & dosage | Muscular Dystrophy, Duchenne - drug therapy | Humans | Motor Activity - drug effects | Dose-Response Relationship, Drug | Forelimb - physiopathology | Animals | Signal Transduction - drug effects | Interleukin-1beta - metabolism | Muscular Dystrophy, Duchenne - physiopathology | Injections, Subcutaneous | Muscular Dystrophy, Duchenne - immunology | Mice, Inbred mdx | Female | Mice | Interleukin 1 Receptor Antagonist Protein - pharmacology | Forelimb - drug effects | Disease Models, Animal | Locomotion - drug effects | Disease | Interleukin | Stimulation | Arthritis | Muscular dystrophy | Lipopolysaccharides | Proteins | Signal transduction | Splenocytes | Duchenne's muscular dystrophy | Interleukin 1 | Toll-like receptors | Tumor necrosis factor-TNF | Mathematical models | Inhibition | Grip strength | Dystrophin | Age | Recombinant | Phenotypes | Parameters | Cytokines | Coordination | Interleukin 1 receptor antagonist | Medical treatment | Muscles | Blocking | Inflammation | Gene expression | Disease control | Skeletal muscle | Medicine | Pathology | Signaling | Inhibitors | Anxieties | Correlation analysis | Rheumatoid arthritis | Dystrophy | Diabetes | Mutation | Laboratory animals | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 04/2017, Volume 7, Issue 1, pp. 46165 - 46165
Activation of TLR4 by lipopolysaccharide (LPS) induces both pro-inflammatory and anti-inflammatory cytokine production in macrophages. Type 4... 
CYCLIC-AMP | INHIBITION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | SEPTIC SHOCK | LIPOPOLYSACCHARIDE | CYTOKINE | IL-1 RECEPTOR | TNF-ALPHA | HUMAN MONOCYTES | AUTOINFLAMMATORY DISEASE | Interleukin 1 Receptor Antagonist Protein - genetics | RNA Stability - drug effects | RNA, Messenger - metabolism | Interleukin 1 Receptor Antagonist Protein - blood | Interleukin-1beta - blood | Cyclic Nucleotide Phosphodiesterases, Type 4 - deficiency | Sepsis - pathology | Interleukin 1 Receptor Antagonist Protein - metabolism | Interleukin 1 Receptor Antagonist Protein - secretion | Phosphorylation - drug effects | Cyclic Nucleotide Phosphodiesterases, Type 4 - metabolism | Rolipram - pharmacology | Cyclic AMP - metabolism | Macrophages, Peritoneal - drug effects | STAT3 Transcription Factor - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Cell Survival - drug effects | Mice, Inbred C57BL | RNA, Messenger - genetics | Phosphodiesterase 4 Inhibitors - pharmacology | Up-Regulation - drug effects | Animals | Signal Transduction - drug effects | Cyclic AMP Response Element-Binding Protein - metabolism | Lipopolysaccharides - pharmacology | RAW 264.7 Cells | Mice | Sepsis - blood | Macrophages, Peritoneal - metabolism | Protein kinase A | Phosphorylation | Transcription | Interleukin 1 receptors | Macrophages | Lipopolysaccharides | Cell activation | Rodents | Interleukin 1 | Toll-like receptors | Phosphodiesterase | Enzymes | Cytokines | Interleukin 1 receptor antagonist | Secretion | Stat3 protein | Cyclic AMP | Inflammation | TLR4 protein | Cyclic AMP response element-binding protein | Rolipram | Isoforms | Sepsis | Peritoneum | mRNA stability | Index Medicus
Journal Article
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 03/2019, Volume 78, Issue Suppl 1, p. A42
Career situation of first and presenting authorPost-doctoral fellow.IntroductionThe inflammatory effects of interleukin (IL)-1 are tightly controlled by IL-1... 
Alternative splicing | Imiquimod | Exons | Cream | mRNA | Caspase-1 | Rodents | Interleukin 1 | Toll-like receptors | Enzyme-linked immunosorbent assay | Phenotypes | Psoriasis | Cytokines | Interleukin 1 receptor antagonist | Translation initiation | Keratinocytes | Caspase | Data processing | Inflammation | IL-1β | Cell death | Biopsy | Isoforms | Lysis | Ear | Skin | Topical application | Intracellular | Immunofluorescence
Journal Article
Microbiome, ISSN 2049-2618, 2017, Volume 5, Issue 1, pp. 63 - 63
BACKGROUND: Perturbation of commensal intestinal microbiota has been associated with several autoimmune diseases. Mice deficient in interleukin-1 receptor... 
Toll-like receptors | Autoimmune arthritis | Microbiota | T helper 17 cells | IL-1 receptor antagonist | RHEUMATOID-ARTHRITIS | MICROBIOLOGY | GUT MICROBIOTA | SEGMENTED FILAMENTOUS BACTERIA | MICE LACKING | CYTOKINE PRODUCTION | DESTRUCTIVE ARTHRITIS | T-CELLS | TH17 CELLS | Helicobacter - genetics | Interleukin-17 - immunology | Interleukin 1 Receptor Antagonist Protein - genetics | Genetic Variation | Hereditary Autoinflammatory Diseases - microbiology | Intestinal Mucosa - immunology | Arthritis - immunology | Prevotella - genetics | RNA, Ribosomal, 16S | Arthritis - microbiology | Interleukin 1 Receptor Antagonist Protein - deficiency | Interleukin 1 Receptor Antagonist Protein - immunology | Autoimmune Diseases - immunology | Gastrointestinal Microbiome | Hereditary Autoinflammatory Diseases - immunology | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | Interleukin 1 Receptor Antagonist Protein - physiology | Intestinal Mucosa - microbiology | Ruminococcus - genetics | Mice, Knockout | Animals | Mucous Membrane - immunology | Autoimmune Diseases - microbiology | Th17 Cells - immunology | High-Throughput Nucleotide Sequencing | Mice | Anti-Bacterial Agents - administration & dosage | Mucous Membrane - microbiology | Immune response | Psoriasis | Interleukins | Microbiota (Symbiotic organisms) | RNA | Development and progression | Arthritis | Disease susceptibility | T cells | Cluster analysis | rRNA 16S | Mucosal immunity | Encephalomyelitis | Disease | Helper cells | Lymphocytes T | Genotype & phenotype | Intestine | Interleukin 1 | Intestinal microflora | Feces | Gnotobiotics | Cytokines | Interleukin 1 receptor antagonist | Diabetes mellitus | Lamina propria | Inflammation | TLR4 protein | Multiculturalism & pluralism | Interleukin 17 | Rheumatoid arthritis | Diabetes | Tobramycin | Autoimmune diseases | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2011, Volume 286, Issue 30, pp. 26470 - 26479
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2011, Volume 186, Issue 8, pp. 4794 - 4804
Endosomal TLRs play an important role in innate immune response as well as in autoimmune processes. In the therapy of systemic lupus erythematosus,... 
IMMUNOSTIMULATORY CPG-OLIGODEOXYNUCLEOTIDES | CELLS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | ACTIVATION | DNA | COMPLEXES | TOLL-LIKE RECEPTORS | H+-ATPASE | IFN-ALPHA INDUCER | IMMUNOLOGY | NUCLEIC-ACIDS | Humans | Toll-Like Receptor 9 - genetics | Toll-Like Receptor 3 - antagonists & inhibitors | Endosomes - metabolism | Quinolines - pharmacology | Chloroquine - pharmacology | Quinacrine - pharmacology | Transfection | Hydrogen-Ion Concentration - drug effects | HEK293 Cells | Toll-Like Receptors - antagonists & inhibitors | Toll-Like Receptor 3 - genetics | Culture Media - chemistry | Imidazoles - metabolism | Toll-Like Receptor 8 - agonists | Binding, Competitive | Toll-Like Receptor 7 - agonists | Toll-Like Receptor 7 - genetics | Cell Line | Antimalarials - metabolism | Toll-Like Receptor 7 - metabolism | Toll-Like Receptor 9 - antagonists & inhibitors | Cytokines - metabolism | Cells, Cultured | Imidazoles - pharmacology | Nucleic Acids - metabolism | Toll-Like Receptor 3 - metabolism | Toll-Like Receptors - agonists | Antimalarials - pharmacology | Quinolines - metabolism | Animals | Toll-Like Receptors - genetics | Culture Media - pharmacology | Toll-Like Receptor 8 - genetics | Endosomes - chemistry | Toll-Like Receptor 9 - metabolism | Toll-Like Receptor 8 - metabolism | Serum - chemistry | Index Medicus | Abridged Index Medicus
Journal Article