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American journal of physiology: Gastrointestinal and liver physiology, ISSN 1522-1547, 07/2010, Volume 299, Issue 1, pp. G236 - G243
...) is also induced by ER stress and promotes apoptosis. To integrate the role of these two transcription factors in ER stress-induced apoptosis, we examined the relative contribution of CHOP and AP-1 in mediating PUMA induction by saturated FFA... 
c-Jun | Activator protein-1 | p53 upregulated modulator of apoptosis | Nonalcoholic steatohepatitis | CAAT/enhancer binding homologous protein | BH3-only proteins | Hepatic steatosis | Endoplasmic reticulum stress | Gastroenterology & Hepatology | Physiology | Life Sciences & Biomedicine | Science & Technology | Palmitic Acid - metabolism | Transcription Factor CHOP - genetics | Up-Regulation | Phosphorylation | Fatty Liver - pathology | Transcription Factor AP-1 - genetics | Humans | Transcriptional Activation | Endoplasmic Reticulum - metabolism | Hepatocytes - pathology | Hepatocytes - metabolism | RNA, Messenger - metabolism | Transcription Factor AP-1 - metabolism | Transfection | RNA Interference | Apoptosis Regulatory Proteins - genetics | Binding Sites | Fatty Liver - genetics | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | Oleic Acid - metabolism | Fatty Liver - metabolism | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Apoptosis Regulatory Proteins - metabolism | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | Transcription Factor CHOP - metabolism | Apoptosis | Physiological aspects | Genetic aspects | Gene expression | Binding proteins | Properties | Endoplasmic reticulum | Proteins | Ribonucleic acid--RNA | Fatty acids | Liver | Gastroenterology | Index Medicus | nonalcoholic steatohepatitis | enhancer binding homologous protein | activator protein-1 | Liver and Biliary Tract | endoplasmic reticulum stress | CAAT | hepatic steatosis
Journal Article
Inflammation, ISSN 0360-3997, 2/2016, Volume 39, Issue 1, pp. 366 - 374
...) and cyclooxygenase-2 (COX-2) expression in LPS-stimulated RAW264.7 macrophages. GAC also suppressed the production and mRNA expression of pro-inflammatory cytokines such as tumor necrosis factor alpha (TNF-α), interleukin-1β (IL-1β... 
Pathology | chitosan | NF-κB | Medicine & Public Health | RAW macrophage | inflammation | Rheumatology | Internal Medicine | Pharmacology/Toxicology | MAPK | AP-1 | Immunology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Nitric Oxide Synthase Type II - biosynthesis | Phosphorylation | Gallic Acid - pharmacology | Dinoprostone - biosynthesis | Tumor Necrosis Factor-alpha - genetics | JNK Mitogen-Activated Protein Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Interleukin-1beta - genetics | Transcription Factor AP-1 - metabolism | Transcription Factor RelA - genetics | Lipopolysaccharides | Cyclooxygenase 2 - biosynthesis | Transcription Factor RelA - urine | I-kappa B Kinase - metabolism | Inflammation - drug therapy | Chitosan - pharmacology | Interleukin-1beta - biosynthesis | p38 Mitogen-Activated Protein Kinases - metabolism | Chitosan - analogs & derivatives | Gallic Acid - chemistry | Macrophages - immunology | Cell Line | Nitric Oxide - biosynthesis | Interleukin-6 - genetics | Anti-Inflammatory Agents - pharmacology | Macrophages - metabolism | Animals | Anti-Inflammatory Agents - chemistry | Chitosan - chemistry | MAP Kinase Signaling System - drug effects | Transcription Factor RelA - metabolism | Interleukin-6 - biosynthesis | Mice | Enzyme Activation | Tumor Necrosis Factor-alpha - biosynthesis | Index Medicus
Journal Article
Cancer biology & therapy, ISSN 1538-4047, 07/2010, Volume 10, Issue 1, pp. 19 - 29
Journal Article
Cellular and molecular life sciences : CMLS, ISSN 1420-682X, 1/2014, Volume 71, Issue 1, pp. 143 - 163
...) with the activity of pro-inflammatory transcription factors NF-κB and AP-1, a mechanism known as transrepression... 
Life Sciences | Biochemistry, general | Selective GR modulator | c-Jun | Life Sciences, general | Glucocorticoids | Mitogen-activated protein kinase (MAPK) | Inflammation | Jun N-terminal kinase (JNK) | Biomedicine general | Cell Biology | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Receptors, Glucocorticoid - antagonists & inhibitors | Gene Expression - drug effects | Transcription Factor AP-1 - genetics | Humans | Transcriptional Activation - drug effects | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Receptors, Glucocorticoid - metabolism | Mitogen-Activated Protein Kinase 9 - genetics | Transcription Factor AP-1 - metabolism | Dexamethasone - pharmacology | RNA Interference | Phosphorylation - drug effects | Interleukin-6 - metabolism | Dual Specificity Phosphatase 1 - genetics | Promoter Regions, Genetic | Interleukin-6 - genetics | Mitogen-Activated Protein Kinase 9 - metabolism | Proto-Oncogene Proteins c-jun - genetics | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase 9 - deficiency | Mice, Knockout | Triazoles - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Proto-Oncogene Proteins c-jun - metabolism | Receptors, Glucocorticoid - genetics | Cell Line, Tumor | Dual Specificity Phosphatase 1 - metabolism | Mice | Staurosporine - pharmacology | Mitogen-Activated Protein Kinases - metabolism | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Nature immunology, ISSN 1529-2916, 05/2016, Volume 17, Issue 7, pp. 851 - 860
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 09/2009, Volume 284, Issue 36, pp. 24006 - 24016
Interleukin (IL)-23, a new member of the IL-12 family, plays a central role in the Th17 immune response and in autoimmune diseases. It is clear that activated... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | MAP Kinase Signaling System - physiology | Mitogen-Activated Protein Kinase Kinases - genetics | Transcription Factor AP-1 - genetics | Dendritic Cells - immunology | Mitogen-Activated Protein Kinase Kinases - immunology | NF-kappa B - immunology | Transcription Factor AP-1 - immunology | NF-kappa B - metabolism | Proto-Oncogene Proteins c-jun - immunology | Macrophages, Peritoneal - cytology | Transcription Factor AP-1 - metabolism | Activating Transcription Factor 2 - genetics | Mitogen-Activated Protein Kinase Kinases - metabolism | Interleukin-23 Subunit p19 - biosynthesis | Interleukin-23 Subunit p19 - genetics | Myeloid Differentiation Factor 88 - immunology | Response Elements - genetics | Dendritic Cells - metabolism | Cell Line | Proto-Oncogene Proteins c-jun - genetics | Myeloid Differentiation Factor 88 - genetics | Gene Expression Regulation - physiology | Macrophages, Peritoneal - immunology | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 4 - metabolism | Activating Transcription Factor 2 - metabolism | Response Elements - immunology | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | MAP Kinase Signaling System - drug effects | NF-kappa B - genetics | Proto-Oncogene Proteins c-jun - metabolism | Activating Transcription Factor 2 - immunology | Autoimmune Diseases | Lipopolysaccharides - pharmacology | Mice | Interleukin-23 Subunit p19 - immunology | Mutation | Macrophages, Peritoneal - metabolism | Interleukin-10 - immunology | Interleukin-10 - pharmacology | Myeloid Differentiation Factor 88 - metabolism | Index Medicus | Mechanisms of Signal Transduction
Journal Article