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Arthritis Research and Therapy, ISSN 1478-6354, 05/2015, Volume 17, Issue 1
Introduction: The transcription factor activating enhancer binding protein 2 epsilon (AP-2 epsilon) was recently shown to be expressed during chondrogenesis as... 
CHONDROCYTE HYPERTROPHY | II-COLLAGEN | RAP-DEFICIENT MICE | 13-DEFICIENT MICE | IN-VITRO | ARTICULAR-CARTILAGE | X COLLAGEN | GROWTH-PLATE | RHEUMATOLOGY | HUMAN COLLAGENASE-3 MMP-13 | TRANSCRIPTION FACTOR | Embryonic development | Gene expression | Osteoarthritis | Genes | Stem cells | Protein binding
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23483
Ap-2 transcription factors comprise a family of 5 closely related sequence-specific DNA binding proteins that play pivotal and non-redundant roles in embryonic... 
FACTOR AP-2 | DNA-BINDING | MUTANT MICE | AP-2-EPSILON | EPITHELIAL-CELLS | MOUSE EMBRYOGENESIS | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | FACTOR AP-2-GAMMA | FACTOR AP-2-BETA | POU4F3 | Oligonucleotide Array Sequence Analysis | Mesencephalon - metabolism | Evoked Potentials | Apoptosis - genetics | Gene Expression Profiling | Promoter Regions, Genetic - genetics | Transcription Factor AP-2 - metabolism | Embryo, Mammalian - metabolism | Neocortex - metabolism | In Situ Hybridization | Chromatin Immunoprecipitation | Gene Expression Regulation, Developmental | Transcription Factor Brn-3C - genetics | Neurons - physiology | Transcription Factor AP-2 - deficiency | Neurons - metabolism | Inferior Colliculi - embryology | Basic Helix-Loop-Helix Transcription Factors - genetics | Binding Sites - genetics | Reverse Transcriptase Polymerase Chain Reaction | Homeodomain Proteins - genetics | Mice, Knockout | Transcription Factor AP-2 - genetics | Animals | Mesencephalon - embryology | Embryo, Mammalian - embryology | Cell Line, Tumor | Inferior Colliculi - metabolism | Protein Binding | Neocortex - cytology | Mice | Colliculus | Chromatin | Neurosciences | Transcription factors | Immunoprecipitation | Target recognition | Mesencephalon | Brain stem | Genes | Nervous system | Biochemistry | Genomes | Acoustics | Tectum | Proteins | Embryogenesis | Rodents | Physiology | Localization | Deoxyribonucleic acid--DNA | Nucleotide sequence | Cortex | Hearing impairment | Gene expression | Embryos | Organogenesis | Embryonic growth stage | Pathology | Cell death | Mutation | Laboratory animals | Binding sites | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 2361 - 14
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 10/2008, Volume 183, Issue 1, pp. 37 - 48
The mechanisms by which mammalian epidermal stem cells cease to proliferate and embark upon terminal differentiation are still poorly understood. By... 
Transcription factors | Messenger RNA | Developmental biology | Epidermal cells | Genes | Keratinocytes | Epidermis | Skin | Mice | Gene expression | REGULATOR | C/EBP-ALPHA | XENOPUS | HAIR FOLLICLE | GENE-EXPRESSION | KERATIN GENE | STEM-CELL | PROLIFERATION | TRANSCRIPTION FACTOR AP-2-ALPHA | ACTIVATOR PROTEIN-2 | CELL BIOLOGY | Transcription Factor HES-1 | Cell Proliferation | Skin - metabolism | Transglutaminases - genetics | Receptors, Notch - genetics | Embryo, Mammalian - metabolism | Hair Follicle - abnormalities | Keratins - metabolism | Gene Expression Regulation, Developmental | Cell Cycle Proteins - genetics | Epidermis - abnormalities | Skin Transplantation | CCAAT-Enhancer-Binding Proteins - genetics | Cell Differentiation - physiology | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | Skin - embryology | Animals, Newborn | CCAAT-Enhancer-Binding Proteins - metabolism | Epidermis - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Receptors, Notch - physiology | Mice, Transgenic | Microscopy, Electron | Reverse Transcriptase Polymerase Chain Reaction | Embryo, Mammalian - abnormalities | Homeodomain Proteins - genetics | Mice, Knockout | Transcription Factor AP-2 - genetics | Animals | Transcription Factor AP-2 - physiology | Epidermis - ultrastructure | Mice, Nude | Hair Follicle - metabolism | Transglutaminases - metabolism | Signal Transduction - physiology | Keratins - genetics | DNA binding proteins | Research | Properties | Cell differentiation | Stem cells
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 15781 - 11
AP-1/sigma 1B-deficiency causes X-linked intellectual disability. AP-1/sigma 1B -/- mice have impaired synaptic vesicle recycling, fewer synaptic vesicles and... 
AMPHIPHYSIN | MULTIDISCIPLINARY SCIENCES | AUXILIN | ALPHA | MECHANISMS | ADAPTER-PROTEIN | ENDOCYTOSIS | CLATHRIN-COAT | BINDING | STONIN 2 | COGNITIVE DEFICITS | Clathrin | Phosphorylation | Transcription factors | Exocytosis | Protein turnover | Recruitment | Uncoating | Endocytosis | Molecular modelling | Plasticity (synaptic) | Isoforms | Synaptic vesicles | Coated vesicles | Hsc70 protein
Journal Article
Laboratory Investigation, ISSN 0023-6837, 04/2003, Volume 83, Issue 4, pp. 571 - 578
Journal Article
ONCOGENESIS, ISSN 2157-9024, 11/2019, Volume 8, Issue 12, pp. 1 - 17
The discovery of bladder cancer transcriptional subtypes provides an opportunity to identify high risk patients, and tailor disease management. Recent studies... 
UROTHELIAL DIFFERENTIATION | ACTIVATION | MODELS | ONCOLOGY | GENE-EXPRESSION | AP-2-GAMMA | METAPLASIA | VARIANT | PROGRESSION | EVENT | Gene silencing | Transcription factors | Recombination | Biochemical analysis | Aggressive behavior | AP-2 protein | siRNA | Bladder cancer | Lymph nodes | Metastases | Cancer
Journal Article
Oncogene, ISSN 0950-9232, 06/2001, Volume 20, Issue 26, pp. 3363 - 3375
We have previously demonstrated that the transition of melanoma to the metastatic phenotype is associated with a loss of expression of the transcription factor... 
Angiogenesis | Metastasis | Transcriptional regulation | Melanoma | Matrix metallaproteinase-2 | DNA-BINDING | matrix metallaproteinase-2 | angiogenesis | transcriptional regulation | IV COLLAGENASE GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | melanoma | E-CADHERIN | CELL BIOLOGY | SIGNAL-TRANSDUCTION | MATRIX METALLOPROTEINASES | ONCOLOGY | metastasis | PRIMARY CUTANEOUS MELANOMA | C-KIT EXPRESSION | RELATIVE EXPRESSION | GENETICS & HEREDITY | MMP-2 IMMUNOREACTIVE PROTEIN | HUMAN-MALIGNANT MELANOMA | Neoplasm Transplantation | Alternative Splicing | Humans | Transcription Factors - deficiency | Male | Transcription Factor AP-2 | DNA-Binding Proteins - deficiency | Neovascularization, Pathologic - pathology | Neoplasm Metastasis | Laminin | Transfection | Genes, Dominant | Lung Neoplasms - secondary | Culture Media | Melanoma - genetics | Melanoma, Experimental - secondary | Antigens, Neoplasm - analysis | Genes, Reporter | Matrix Metalloproteinase 2 - biosynthesis | Lung Neoplasms - blood supply | Skin Neoplasms - pathology | DNA-Binding Proteins - physiology | Melanoma, Experimental - enzymology | Transcription Factors - physiology | Lung Neoplasms - enzymology | Proteoglycans | Melanoma, Experimental - blood supply | Neoplasm Invasiveness | Melanoma, Experimental - pathology | Platelet Endothelial Cell Adhesion Molecule-1 - analysis | Lymphatic Metastasis | Melanoma - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Matrix Metalloproteinase 2 - genetics | Blotting, Northern | Animals | Mice, Nude | Collagen | Skin Neoplasms - genetics | Neovascularization, Pathologic - genetics | Mice | Mice, Inbred BALB C | Drug Combinations | Cell Movement | Infection | Development and progression | Genetic engineering | Growth
Journal Article