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animals (230) 230
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Brain Research, ISSN 0006-8993, 2008, Volume 1256, pp. 8 - 18
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 10/1996, Volume 93, Issue 21, pp. 11950 - 11955
.... We show herein that targeted deletion of the Brn-3a gene results in defective suckling and in uncoordinated limb and trunk movements, leading to early postnatal death. Brn-3a... 
Red nucleus | Neurons | Spinal ganglia | Habenula | Antibodies | Retina | Mice | Cellular immunity | Olivary nucleus | Trigeminal ganglion | HOMEODOMAIN | HORMONE DEFICIENCY | MUTANTS | CAENORHABDITIS-ELEGANS | PROTEIN | PIT-1 | MULTIDISCIPLINARY SCIENCES | I-POU | MUTATIONS | TRANSCRIPTION FACTOR | EXPRESSION
Journal Article
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 2009, Volume 61, Issue 6, pp. 852 - 864
.... Here we dissect the roles of the highly homologous POU-domain transcription factors Brn3a and Brn3b in retinal ganglion cell (RGC... 
CELLBIO | DEVBIO | MOLNEURO | NEURONS | VISUAL-SYSTEM | MICE | MOSAIC ANALYSIS | MOUSE RETINA | DIFFERENTIATION | DOMAIN FACTOR BRN-3B | AXON OUTGROWTH | HOMEOBOX GENE | EXPRESSION | NEUROSCIENCES | Dendrites - pathology | Locomotion - genetics | Paired Box Transcription Factors - deficiency | Homeodomain Proteins - metabolism | Repressor Proteins | Alkaline Phosphatase - metabolism | Visual Pathways - metabolism | Brain - growth & development | Brain - metabolism | Retinal Ganglion Cells - cytology | Miosis - genetics | Nystagmus, Optokinetic - genetics | Visual Pathways - pathology | Transcription Factor Brn-3A - deficiency | Gene Expression Regulation, Developmental - physiology | Animals, Newborn | Dendrites - metabolism | Retinal Ganglion Cells - physiology | Transcription Factor Brn-3B - deficiency | RNA, Untranslated | PAX6 Transcription Factor | Transcription Factor Brn-3A - metabolism | Transcription Factor Brn-3B - metabolism | Mice, Knockout | Proteins - genetics | S100 Calcium Binding Protein G - metabolism | Locomotion - radiation effects | Animals | Vision Disorders - genetics | Calbindin 2 | Axons - pathology | Mice | Eye Proteins | Dendrites - genetics | Medical colleges | Neurosciences | DNA binding proteins | Ophthalmology | Ganglion | Proteins | Transcription factors | Neurons | Light | Retina | Software | Anatomy & physiology | Experiments | Deoxyribonucleic acid--DNA | Defects
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/1999, Volume 274, Issue 21, pp. 15237 - 15244
Journal Article
Journal Article
Immunology Letters, ISSN 0165-2478, 2015, Volume 164, Issue 1, pp. 25 - 32
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 10, p. e76347
.... Methods and Findings: We now have investigated genetic interactions between Brn3 transcription factors with respect to RGC development, by crossing conventional knock-out alleles of each Brn3 gene with conditional... 
SOMATOSENSORY NEURONS | NERVOUS-SYSTEM | AMACRINE CELLS | PRIMATE RETINA | MULTIDISCIPLINARY SCIENCES | VISUAL-SYSTEM | MOUSE RETINA | DIFFERENTIATION | DOMAIN FACTOR BRN-3B | MAMMALIAN RETINA | EXPRESSION | Retina - metabolism | Retina - growth & development | Cell Count | Homeodomain Proteins - metabolism | Transcription Factor Brn-3B - genetics | Cell Survival - genetics | Male | Mice, 129 Strain | Retinal Ganglion Cells - metabolism | Retinal Ganglion Cells - cytology | Retina - cytology | Gene Expression Regulation, Developmental | Rod Opsins - metabolism | Transcription Factor Brn-3C - genetics | Female | Mice, Inbred C57BL | Transcription Factor Brn-3C - metabolism | Transcription Factor Brn-3A - metabolism | Homeodomain Proteins - genetics | Transcription Factor Brn-3B - metabolism | Mice, Knockout | Animals | Transcription Factor Brn-3A - genetics | Epistasis, Genetic | Mice | Neurofilament Proteins - metabolism | Microscopy, Fluorescence | Ganglion | Genetic aspects | DNA binding proteins | Brain | Transcription factors | Gene regulation | Retina | Melanopsin | Defects | Proteins | Developmental stages | Specifications | Rodents | Physiology | Dendrites | Cell survival | Neurons | Axonogenesis | Brn-3 protein | Gene expression | Survival | Molecular chains | Retinal ganglion cells | Brn3 gene | Lamination | Insects | Islet-1 protein | Neural networks | Morphology | Alleles | Protein expression | Adults | Combinatorial analysis
Journal Article
Cancer research (Chicago, Ill.), ISSN 0008-5472, 05/2010, Volume 70, Issue 10, pp. 3985 - 3995
A variety of genetic lesions, including chromosomal translocations, internal tandem duplications, and mutations, have been described in acute myeloid leukemia (AML... 
PROGENITOR CELLS | TRANSLOCATION | REPRESSION | ONCOLOGY | AML1-ETO FUSION PROTEIN | BRN-3A TRANSCRIPTION FACTOR | LEUKEMOGENESIS | GENE-EXPRESSION | NEURONS | DIFFERENTIATION | STEM | Chromosomes, Human, Pair 8 - genetics | Oncogene Proteins, Fusion - metabolism | Embryonic Stem Cells - cytology | Luciferases - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Molecular Sequence Data | RNA, Messenger - metabolism | Immunoenzyme Techniques | Transfection | Chromatin Immunoprecipitation | Base Sequence | Hematopoietic Stem Cells - physiology | Adult | Fetus | Cell Differentiation | Electrophoretic Mobility Shift Assay | Core Binding Factor Alpha 2 Subunit - metabolism | Leukemia, Myeloid, Acute - pathology | Liver - metabolism | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Transcription Factor Brn-3A - metabolism | Chromosomes, Human, Pair 21 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Transcription Factor Brn-3A - antagonists & inhibitors | Blotting, Western | Embryonic Stem Cells - physiology | RUNX1 Translocation Partner 1 Protein | Animals | Transcription Factor Brn-3A - genetics | Oncogene Proteins, Fusion - genetics | Fluorescent Antibody Technique | Liver - cytology | Mice | Core Binding Factor Alpha 2 Subunit - genetics | Leukemia, Myeloid, Acute - genetics | Translocation, Genetic - genetics | AML | ETO | BRN3A | transcription | myeloid | AML1
Journal Article