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Journal Article
Molecular Cell, ISSN 1097-2765, 11/2012, Volume 48, Issue 3, pp. 353 - 364
MicroRNAs typically function at the level of posttranscriptional gene silencing within the cytoplasm; however, increasing evidence suggests that they may also... 
NF-Y | MESSENGER-RNA TRANSLATION | OXIDATIVE STRESS | UNFOLDED PROTEIN RESPONSE | CAENORHABDITIS-ELEGANS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | INITIATION FACTOR-2-ALPHA KINASE | ENDOPLASMIC-RETICULUM STRESS | MAMMALIAN-CELLS | TUMOR-GROWTH | CELL BIOLOGY | NIH 3T3 Cells | Transcription Factor CHOP - genetics | Phosphorylation | eIF-2 Kinase - metabolism | Humans | Cell Survival - genetics | Apoptosis - genetics | MicroRNAs - metabolism | Endoplasmic Reticulum Stress - genetics | Promoter Regions, Genetic - genetics | Embryo, Mammalian - metabolism | DNA-Binding Proteins - metabolism | Fibroblasts - metabolism | eIF-2 Kinase - genetics | Cells, Cultured | Gene Expression Regulation | Activating Transcription Factor 4 - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Transcription Factors - metabolism | Animals | Activating Transcription Factor 4 - metabolism | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Cell Line, Tumor | Thapsigargin - pharmacology | Fibroblasts - cytology | Mice | MicroRNAs - genetics | HeLa Cells | Histones - metabolism | Methylation | Transcription Factor CHOP - metabolism | MicroRNA | Nuclear radiation | Analysis | Oncology, Experimental | Genes | Genetic transcription | Diabetes | Research | Cancer | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2013, Volume 15, Issue 5, pp. 481 - 490
Journal Article
Nature Immunology, ISSN 1529-2908, 05/2010, Volume 11, Issue 5, pp. 411 - 418
Sensors of pathogens, such as Toll-like receptors (TLRs), detect microbes to activate transcriptional programs that orchestrate adaptive responses to specific... 
UNFOLDED PROTEIN RESPONSE | PATHWAY | ER STRESS | HOST-DEFENSE | ENDOPLASMIC-RETICULUM STRESS | FRANCISELLA-TULARENSIS INFECTION | IMMUNOLOGY | LIVE VACCINE STRAIN | INFLAMMATORY RESPONSE | NEGATIVE REGULATOR | SYSTEMS BIOLOGY | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2010, Volume 30, Issue 50, pp. 16938 - 16948
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, pp. e39586 - e39586
Endoplasmic reticulum (ER) stress-induced apoptosis has been implicated in various neurodegenerative diseases including Parkinson Disease, Alzheimer Disease... 
MITOCHONDRIAL APOPTOSIS | TRANSCRIPTION FACTOR CHOP | UNFOLDED PROTEIN RESPONSE | INDUCED APOPTOSIS | KINASE | BIOLOGY | GENE-EXPRESSION | SYMPATHETIC NEURONS | DEATH | ENDOPLASMIC-RETICULUM STRESS | BH3-ONLY PROTEINS | Transcription Factor CHOP - genetics | Apoptosis - drug effects | Neurons - cytology | Bcl-2-Like Protein 11 | Forkhead Transcription Factors - metabolism | Telencephalon - drug effects | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | Neurons - metabolism | Neurons - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - physiology | Proto-Oncogene Proteins - metabolism | Cell Survival - drug effects | Tumor Suppressor Proteins - metabolism | Telencephalon - metabolism | Telencephalon - cytology | Membrane Proteins - genetics | Proto-Oncogene Proteins - genetics | Forkhead Transcription Factors - genetics | Apoptosis Regulatory Proteins - metabolism | Animals | Signal Transduction - drug effects | Tunicamycin - pharmacology | Signal Transduction - physiology | Mice | Apoptosis - physiology | Forkhead Box Protein O3 | Transcription Factor CHOP - metabolism | Endoplasmic Reticulum Stress - physiology | Nervous system diseases | Parkinson's disease | Huntington's chorea | Neurons | Tumor proteins | Protein kinases | Apoptosis | Phosphorylation | Transcription factors | Transcription | p53 Protein | Tunicamycin | Homology | AKT protein | CCAAT/enhancer-binding protein | Kinases | Proteins | Signal transduction | Mitochondria | Cell activation | Cell growth | Cascades | Rodents | Forkhead protein | Alzheimer's disease | Movement disorders | Deoxyribonucleic acid--DNA | FOXO3 protein | Stresses | Neurodegenerative diseases | Mortality | Gene expression | Stress | Neurological diseases | Pathology | Signaling | Cell death | Endoplasmic reticulum | CHOP protein | BIM protein | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 44, pp. 17008 - 17020
Vascular calcification (or mineralization) is a common complication of chronic kidney disease (CKD) and is closely associated with increased mortality and... 
vascular calcification | fatty acid | cyclin-dependent kinase (CDK) | kidney disease | vascular biology | ENDOPLASMIC-RETICULUM STRESS | SMOOTH-MUSCLE | IN-VITRO | GENE-EXPRESSION | CHOP | saturated fatty acids | vascular smooth muscle cells | atherosclerosis | RNA-POLYMERASE-II | MAMMALIAN TARGET | cardiovascular disease | BIOCHEMISTRY & MOLECULAR BIOLOGY | ER stress | endoplasmic reticulum stress (ER stress) | renal dysfunction | P-TEFB | CDK9 | ARTERIAL CALCIFICATION | CHRONIC KIDNEY-DISEASE | CELL-DIFFERENTIATION | Transcription Factor CHOP - genetics | Phosphorylation | Cyclin T - metabolism | Humans | Vascular Calcification - genetics | Male | Renal Insufficiency, Chronic - complications | Renal Insufficiency, Chronic - metabolism | Vascular Calcification - metabolism | Vascular Calcification - etiology | Renal Insufficiency, Chronic - genetics | Vascular Calcification - physiopathology | Mice, Inbred DBA | Fatty Acids - metabolism | Myocytes, Smooth Muscle - metabolism | Cyclin-Dependent Kinase 9 - genetics | Cyclin T - genetics | Activating Transcription Factor 4 - genetics | Mice, Transgenic | Renal Insufficiency, Chronic - physiopathology | Animals | Activating Transcription Factor 4 - metabolism | Endoplasmic Reticulum Stress | Protein Binding | Cyclin-Dependent Kinase 9 - metabolism | Mice | Transcription Factor CHOP - metabolism | Index Medicus | Lipids
Journal Article
Immunity, ISSN 1074-7613, 09/2014, Volume 41, Issue 3, pp. 389 - 401
Journal Article