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Journal of Biochemistry, ISSN 0021-924X, 03/2012, Volume 151, Issue 3, pp. 217 - 219
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2013, Volume 15, Issue 5, pp. 481 - 490
Journal Article
Molecular Cell, ISSN 1097-2765, 11/2012, Volume 48, Issue 3, pp. 353 - 364
MicroRNAs typically function at the level of posttranscriptional gene silencing within the cytoplasm; however, increasing evidence suggests that they may also... 
NF-Y | MESSENGER-RNA TRANSLATION | OXIDATIVE STRESS | UNFOLDED PROTEIN RESPONSE | CAENORHABDITIS-ELEGANS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | INITIATION FACTOR-2-ALPHA KINASE | ENDOPLASMIC-RETICULUM STRESS | MAMMALIAN-CELLS | TUMOR-GROWTH | CELL BIOLOGY | NIH 3T3 Cells | Transcription Factor CHOP - genetics | Phosphorylation | eIF-2 Kinase - metabolism | Humans | Cell Survival - genetics | Apoptosis - genetics | MicroRNAs - metabolism | Endoplasmic Reticulum Stress - genetics | Promoter Regions, Genetic - genetics | Embryo, Mammalian - metabolism | DNA-Binding Proteins - metabolism | Fibroblasts - metabolism | eIF-2 Kinase - genetics | Cells, Cultured | Gene Expression Regulation | Activating Transcription Factor 4 - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Transcription Factors - metabolism | Animals | Activating Transcription Factor 4 - metabolism | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Cell Line, Tumor | Thapsigargin - pharmacology | Fibroblasts - cytology | Mice | MicroRNAs - genetics | HeLa Cells | Histones - metabolism | Methylation | Transcription Factor CHOP - metabolism | MicroRNA | Nuclear radiation | Analysis | Oncology, Experimental | Genes | Genetic transcription | Diabetes | Research | Cancer | Index Medicus
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, pp. e39586 - e39586
Endoplasmic reticulum (ER) stress-induced apoptosis has been implicated in various neurodegenerative diseases including Parkinson Disease, Alzheimer Disease... 
MITOCHONDRIAL APOPTOSIS | TRANSCRIPTION FACTOR CHOP | UNFOLDED PROTEIN RESPONSE | INDUCED APOPTOSIS | KINASE | BIOLOGY | GENE-EXPRESSION | SYMPATHETIC NEURONS | DEATH | ENDOPLASMIC-RETICULUM STRESS | BH3-ONLY PROTEINS | Transcription Factor CHOP - genetics | Apoptosis - drug effects | Neurons - cytology | Bcl-2-Like Protein 11 | Forkhead Transcription Factors - metabolism | Telencephalon - drug effects | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | Neurons - metabolism | Neurons - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - physiology | Proto-Oncogene Proteins - metabolism | Cell Survival - drug effects | Tumor Suppressor Proteins - metabolism | Telencephalon - metabolism | Telencephalon - cytology | Membrane Proteins - genetics | Proto-Oncogene Proteins - genetics | Forkhead Transcription Factors - genetics | Apoptosis Regulatory Proteins - metabolism | Animals | Signal Transduction - drug effects | Tunicamycin - pharmacology | Signal Transduction - physiology | Mice | Apoptosis - physiology | Forkhead Box Protein O3 | Transcription Factor CHOP - metabolism | Endoplasmic Reticulum Stress - physiology | Nervous system diseases | Parkinson's disease | Huntington's chorea | Neurons | Tumor proteins | Protein kinases | Apoptosis | Phosphorylation | Transcription factors | Transcription | p53 Protein | Tunicamycin | Homology | AKT protein | CCAAT/enhancer-binding protein | Kinases | Proteins | Signal transduction | Mitochondria | Cell activation | Cell growth | Cascades | Rodents | Forkhead protein | Alzheimer's disease | Movement disorders | Deoxyribonucleic acid--DNA | FOXO3 protein | Stresses | Neurodegenerative diseases | Mortality | Gene expression | Stress | Neurological diseases | Pathology | Signaling | Cell death | Endoplasmic reticulum | CHOP protein | BIM protein | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 07/2010, Volume 299, Issue 1, pp. 236 - 243
Endoplasmic reticulum (ER) stress-mediated apoptosis is a key feature of hepatocyte cytotoxicity by saturated free fatty acids (FFA). This lipoapoptosis is... 
c-Jun | Activator protein-1 | p53 upregulated modulator of apoptosis | Nonalcoholic steatohepatitis | CAAT/enhancer binding homologous protein | BH3-only proteins | Hepatic steatosis | Endoplasmic reticulum stress | MITOCHONDRIAL APOPTOSIS | PHYSIOLOGY | PROTEIN | INDUCED APOPTOSIS | activator protein-1 | FATTY LIVER-DISEASE | ER STRESS | ENDOPLASMIC-RETICULUM STRESS | endoplasmic reticulum stress | hepatic steatosis | nonalcoholic steatohepatitis | HEPATOCYTE LIPOAPOPTOSIS | PANCREATIC BETA-CELLS | GASTROENTEROLOGY & HEPATOLOGY | BCL-2 FAMILY | Palmitic Acid - metabolism | Transcription Factor CHOP - genetics | Up-Regulation | Phosphorylation | Fatty Liver - pathology | Transcription Factor AP-1 - genetics | Humans | Transcriptional Activation | Endoplasmic Reticulum - metabolism | Hepatocytes - pathology | Hepatocytes - metabolism | RNA, Messenger - metabolism | Transcription Factor AP-1 - metabolism | Transfection | RNA Interference | Apoptosis Regulatory Proteins - genetics | Binding Sites | Fatty Liver - genetics | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | Oleic Acid - metabolism | Fatty Liver - metabolism | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Apoptosis Regulatory Proteins - metabolism | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | Transcription Factor CHOP - metabolism | Apoptosis | Physiological aspects | Genetic aspects | Gene expression | Binding proteins | Properties | Endoplasmic reticulum | Proteins | Ribonucleic acid--RNA | Fatty acids | Liver | Gastroenterology | Index Medicus | enhancer binding homologous protein | Liver and Biliary Tract | CAAT
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2010, Volume 107, Issue 41, pp. 17698 - 17703
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 06/2016, Volume 231, Issue 6, pp. 1321 - 1333
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2010, Volume 30, Issue 50, pp. 16938 - 16948
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2013, Volume 288, Issue 45, pp. 32343 - 32356
Although tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) has shown efficacy in a phase 2 clinical trial, development of resistance to TRAIL by... 
Reactive Oxygen Species (ROS) | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | MEDIATED APOPTOSIS | Limonoid | ANTITUMOR-ACTIVITY | Cancer Therapy | MAP Kinases (MAPKs) | Death Receptor | Trail | MELIA-AZADIRACHTA L | CHOP | CHOLANGIOCARCINOMA CELLS | SURVIVAL PROTEINS | NF-KAPPA-B | UP-REGULATION | APO2L/TRAIL-INDUCED APOPTOSIS | Anticancer Drug | COLON-CARCINOMA | Apoptosis | Neoplasms - metabolism | Transcription Factor CHOP - genetics | Reactive Oxygen Species - metabolism | Humans | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Tumor Suppressor Protein p53 - genetics | Dose-Response Relationship, Drug | TNF-Related Apoptosis-Inducing Ligand - pharmacology | Neoplasms - genetics | Antineoplastic Agents - pharmacology | Receptors, TNF-Related Apoptosis-Inducing Ligand - genetics | Gene Expression Regulation, Neoplastic - drug effects | Apoptosis Regulatory Proteins - biosynthesis | Response Elements | Cell Survival | Tumor Suppressor Protein p53 - metabolism | Down-Regulation - drug effects | Neoplasms - drug therapy | Up-Regulation - drug effects | Limonins - pharmacology | MAP Kinase Signaling System - drug effects | Cell Line, Tumor | Neoplasms - pathology | Transcription Factor CHOP - metabolism | Index Medicus | Signal Transduction
Journal Article