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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2016, Volume 311, Issue 4, pp. H871 - H880
We previously reported that endoplasmic reticulum (ER) stress is induced in the subfornical organ (SFO) and the hypothalamic paraventricular nucleus (PVN) of... 
Heart failure | Brain | Sympathetic activity | Hypothalamic paraventricular nucleus | Mitogen-activated protein kinase | Subfornical organ | Endoplasmic reticulum stress | heart failure | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | INDUCED PHOSPHORYLATION | MYOCARDIAL-INFARCTION | ER STRESS | subfornical organ | KINASE | RATS | sympathetic activity | KAPPA-B | brain | endoplasmic reticulum stress | hypothalamic paraventricular nucleus | mitogen-activated protein kinase | UNFOLDED PROTEIN RESPONSE | PERIPHERAL VASCULAR DISEASE | UP-REGULATION | Cholagogues and Choleretics - pharmacology | Tumor Necrosis Factor-alpha - genetics | Heart Failure - physiopathology | Male | NF-KappaB Inhibitor alpha - genetics | Peptidyl-Dipeptidase A - drug effects | Interleukin-1beta - genetics | Sympathetic Nervous System - physiopathology | RNA, Messenger - metabolism | Activating Transcription Factor 6 - genetics | Subfornical Organ - drug effects | Brain - metabolism | Heat-Shock Proteins - genetics | Inflammation - metabolism | Receptor, Angiotensin, Type 1 - genetics | Cyclooxygenase 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Real-Time Polymerase Chain Reaction | Echocardiography | Signal Transduction | Rats | Cyclooxygenase 2 - drug effects | Heart Failure - metabolism | Rats, Sprague-Dawley | Blotting, Western | Brain - drug effects | Tumor Necrosis Factor-alpha - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Endoplasmic Reticulum Stress | Paraventricular Hypothalamic Nucleus - metabolism | Infusions, Intraventricular | Mitogen-Activated Protein Kinases - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Interleukin-1beta - drug effects | Sympathetic Nervous System - drug effects | Mitogen-Activated Protein Kinase 1 - drug effects | X-Box Binding Protein 1 - drug effects | Sympathetic Nervous System - metabolism | Transcription Factor RelA - genetics | Activating Transcription Factor 6 - drug effects | Mitogen-Activated Protein Kinase 3 - drug effects | Taurochenodeoxycholic Acid - pharmacology | Peptidyl-Dipeptidase A - genetics | Renin-Angiotensin System | Receptor, Angiotensin, Type 1 - drug effects | RNA, Messenger - drug effects | Subfornical Organ - metabolism | Heat-Shock Proteins - drug effects | Activating Transcription Factor 4 - genetics | Activating Transcription Factor 4 - drug effects | NF-KappaB Inhibitor alpha - drug effects | Paraventricular Hypothalamic Nucleus - drug effects | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | Transcription Factor RelA - drug effects | X-Box Binding Protein 1 - genetics | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Cellular signal transduction | Endoplasmic reticulum | Health aspects | Mitogen-activated protein kinases | Cardiovascular Neurohormonal Regulation
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2015, Volume 24, Issue 1, pp. 178 - 187
Summary Objective To determine whether mandibular condylar cartilage degradation induced by experimentally abnormal occlusion could be ameliorated via systemic... 
Rheumatology | Strontium | Cartilage | Subchondral bone | NBD (NEMO-binding domain) peptide | Temporomandibular joint | Osteoarthritis | Dental occlusion | TGF-BETA | CHONDROCYTES | BLOCKS OSTEOCLASTOGENESIS | RHEUMATOLOGY | TRABECULAR BONE | SUBCHONDRAL BONE LOSS | RANELATE | INHIBITION | TEMPOROMANDIBULAR-JOINT | ORTHOPEDICS | NF-KAPPA-B | KNEE OSTEOARTHRITIS | Immunohistochemistry | RANK Ligand - metabolism | Collagen Type II - drug effects | Tumor Necrosis Factor-alpha - genetics | ADAM Proteins - drug effects | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Proteoglycans - drug effects | Collagen Type II - metabolism | Cartilage, Articular - metabolism | Interleukin-1beta - metabolism | Malocclusion | Receptor Activator of Nuclear Factor-kappa B - metabolism | Aggrecans - drug effects | Real-Time Polymerase Chain Reaction | ADAMTS5 Protein | NF-KappaB Inhibitor alpha | Proteoglycans - metabolism | I-kappa B Proteins - drug effects | Aggrecans - metabolism | ADAM Proteins - metabolism | Tumor Necrosis Factor-alpha - drug effects | Strontium - pharmacology | Mice | Osteoprotegerin - metabolism | ADAM Proteins - genetics | Interleukin-1beta - drug effects | Tumor Necrosis Factor-alpha - metabolism | I-kappa B Proteins - metabolism | I-kappa B Proteins - genetics | Receptor Activator of Nuclear Factor-kappa B - drug effects | Transcription Factor RelA - genetics | RANK Ligand - drug effects | Mandibular Condyle - pathology | Osteoprotegerin - genetics | Female | Mandibular Condyle - drug effects | Osteoprotegerin - drug effects | RNA, Messenger - drug effects | Mice, Inbred C57BL | Aggrecans - genetics | Osteoclasts - metabolism | Collagen Type II - genetics | Peptides - pharmacology | RANK Ligand - genetics | Animals | Transcription Factor RelA - metabolism | Receptor Activator of Nuclear Factor-kappa B - genetics | Transcription Factor RelA - drug effects | Cartilage, Articular - drug effects | Mandibular Condyle - metabolism | Dental Occlusion | Osteoclasts - drug effects | Peptides
Journal Article
American Journal of Obstetrics and Gynecology, ISSN 0002-9378, 07/2018, Volume 219, Issue 1, pp. 113.e1 - 113.e9
Journal Article
Journal of Dental Research, ISSN 0022-0345, 4/2012, Volume 91, Issue 4, pp. 407 - 412
Journal Article
Journal of Endodontics, ISSN 0099-2399, 2014, Volume 40, Issue 5, pp. 640 - 647
Abstract Introduction Mineral trioxide aggregate (MTA) has been widely used in clinical apexification and apexogenesis. However, the effects of MTA on the stem... 
Endocrinology & Metabolism | Dentistry | dentinogenesis | odontoblast | stem cell | nuclear factor kappa B | Apical papilla | PROGENITOR CELLS | OSTEOBLAST DIFFERENTIATION | ROOT | OSTEOGENIC DIFFERENTIATION | PROLIFERATION | CALCIUM HYDROXIDE | MESSENGER-RNA | DENTISTRY, ORAL SURGERY & MEDICINE | TNF-ALPHA PROMOTES | DENTAL-PULP CELLS | IMMATURE PERMANENT TEETH | Up-Regulation | Phosphoproteins - drug effects | Calcium - metabolism | Humans | Sialoglycoproteins - drug effects | Quinoxalines - pharmacology | Young Adult | Calcium Compounds - pharmacology | Silicates - pharmacology | Tooth Apex - cytology | NF-kappa B - antagonists & inhibitors | Odontogenesis - drug effects | Dentinogenesis - drug effects | Osteogenesis - drug effects | Cells, Cultured | I-kappa B Proteins - drug effects | Imidazoles - pharmacology | Alkaline Phosphatase - drug effects | Extracellular Matrix Proteins - drug effects | Cell Shape - drug effects | Oxides - pharmacology | Core Binding Factor Alpha 1 Subunit - drug effects | Signal Transduction - drug effects | Cell Differentiation - drug effects | Adolescent | Osteocalcin - drug effects | Stem Cells - drug effects | Transcription Factor RelA - drug effects | Root Canal Filling Materials - pharmacology | Aluminum Compounds - pharmacology | Cell Proliferation - drug effects | Drug Combinations | NF-kappa B - drug effects | Medical colleges | Stem cells
Journal Article
Brain Research, ISSN 0006-8993, 2009, Volume 1273, pp. 129 - 137
Abstract Adiponectin (APN), a circulating adipose-derived hormone regulating inflammation and energy metabolism, has beneficial actions on cardio- and... 
Neurology | Cerebral ischemia/reperfusion | Cytokines | Nuclear factor-κB | Anti-inflammation | Adiponectin | Nuclear factor-kappa B | HUMAN CEREBROSPINAL-FLUID | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | STROKE | OBESITY | IN-VIVO | NITRIC-OXIDE | INFLAMMATORY RESPONSE | RECEPTORS | EXPRESSION | HYPOTHALAMUS | Infarction, Middle Cerebral Artery - physiopathology | Injections, Intravenous | Active Transport, Cell Nucleus - physiology | Reperfusion Injury - drug therapy | Brain Ischemia - metabolism | Male | Cytoprotection - physiology | Infarction, Middle Cerebral Artery - metabolism | Anti-Inflammatory Agents - metabolism | Neuroprotective Agents - metabolism | Matrix Metalloproteinase 9 - metabolism | Encephalitis - physiopathology | Neuroprotective Agents - pharmacology | Encephalitis - drug therapy | Encephalitis - metabolism | Cytoprotection - drug effects | Adiponectin - metabolism | Infarction, Middle Cerebral Artery - drug therapy | Reperfusion Injury - metabolism | Immunoglobulin G - drug effects | Disease Models, Animal | Adiponectin - pharmacology | Peroxidase - drug effects | Cytokines - metabolism | Anti-Inflammatory Agents - pharmacology | Rats | Brain Ischemia - physiopathology | Rats, Sprague-Dawley | Animals | Cytokines - drug effects | Transcription Factor RelA - metabolism | Active Transport, Cell Nucleus - drug effects | Brain Ischemia - drug therapy | Reperfusion Injury - physiopathology | Matrix Metalloproteinase 9 - drug effects | Transcription Factor RelA - drug effects | Immunoglobulin G - metabolism | Peroxidase - metabolism | Registered nurses | Interleukins | Anti-inflammatory drugs | Peptide hormones | Immunoglobulin G | Tumors
Journal Article
Endocrinology, ISSN 0013-7227, 12/2015, Volume 156, Issue 12, pp. 4741 - 4754
Journal Article
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 2017, Volume 89, pp. 660 - 672
Journal Article
by Long, X and Li, Y and Qiu, S and Liu, J and He, L and Peng, Y
Translational psychiatry, ISSN 2158-3188, 03/2016, Volume 6, Issue 3, pp. e757 - e757
Chronic opioid abusers are more susceptible to bacterial and viral infections, but the molecular mechanism underlying opioid-induced immunosuppression is... 
HEROIN | IMMUNE-SYSTEM | GENE | CHRONIC MORPHINE | BUPRENORPHINE | PSYCHIATRY | INNATE | TNF-ALPHA | MICRORNAS | EXPRESSION | WITHDRAWAL | Tumor Necrosis Factor-alpha - metabolism | Phosphoproteins - drug effects | Humans | Middle Aged | NF-kappa B - immunology | Analgesics, Opioid - pharmacology | Opioid-Related Disorders - metabolism | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | Monocytes - immunology | Cyclic AMP Response Element-Binding Protein - drug effects | Phosphoproteins - metabolism | Case-Control Studies | Young Adult | Signal Transduction - immunology | Analgesics, Opioid - adverse effects | Tumor Necrosis Factor-alpha - immunology | Cyclic AMP Response Element-Binding Protein - immunology | Interleukin-10 - metabolism | Transcription Factor RelA - immunology | Adult | Female | Immunologic Deficiency Syndromes - immunology | Heroin - pharmacology | Cyclic AMP Response Element-Binding Protein A - metabolism | Morphine - pharmacology | MicroRNAs - immunology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Immunologic Deficiency Syndromes - chemically induced | Opioid-Related Disorders - immunology | Monocytes - drug effects | Tumor Necrosis Factor-alpha - drug effects | Transcription Factor RelA - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Cyclic AMP Response Element-Binding Protein A - immunology | MicroRNAs - drug effects | Immunologic Deficiency Syndromes - metabolism | Transcription Factor RelA - drug effects | Cyclic AMP Response Element-Binding Protein A - drug effects | MicroRNAs - genetics | In Vitro Techniques | Interleukin-10 - immunology | NF-kappa B - drug effects | Original
Journal Article