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Journal of Neurochemistry, ISSN 0022-3042, 06/2017, Volume 141, Issue 5, pp. 750 - 765
The retina is highly sensitive to oxidative stress because of its high consumption of oxygen associated with the phototransductional processes. Recent findings... 
AMD | light‐induced retinal degeneration | Nrf2 | retina | HO‐1 | photoreceptor | light-induced retinal degeneration | HO-1 | RETINAL DEGENERATION | OXIDATIVE STRESS | HUMAN-DISEASE | PROTECTION | BRAIN-INJURY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MACULAR DEGENERATION | NEUROSCIENCES | BLUE-LIGHT | NEUROPROTECTION | IN-VITRO | ISCHEMIA-REPERFUSION | RNA, Small Interfering - genetics | Cell Death - radiation effects | Cell Nucleolus - radiation effects | Heme Oxygenase-1 - metabolism | NF-E2 Transcription Factor - genetics | Triterpenes - pharmacology | Cytosol - drug effects | Ependymoglial Cells - radiation effects | Male | Retinal Degeneration - etiology | NF-E2 Transcription Factor - metabolism | Retinal Degeneration - prevention & control | Heme Oxygenase-1 - genetics | Triterpenes - chemistry | Retina - cytology | Time Factors | Cell Nucleolus - drug effects | Membrane Proteins - metabolism | Cell Death - drug effects | Protein Biosynthesis - radiation effects | Membrane Proteins - genetics | Light - adverse effects | RNA, Small Interfering - pharmacology | Ependymoglial Cells - drug effects | Photoreceptor Cells - radiation effects | Gene Expression Regulation - drug effects | Animals | Photoreceptor Cells - drug effects | Ependymoglial Cells - cytology | Gene Expression Regulation - radiation effects | Protein Biosynthesis - drug effects | Mice | Cytosol - radiation effects | In Vitro Techniques | Cell Line, Transformed | Neurochemistry | Chlorophyll | Oxidative stress | Enzymes | Heme oxygenase (decyclizing) | Retina | Oxygen consumption | Exposure | Nuclei | Environmental risk | Risk factors | Macular degeneration | Oxygenase | Cell death | Heme | Photoreceptors | Eye diseases | In vivo methods and tests | Nuclei (cytology) | Damage | In vitro methods and tests | Age | Apoptosis
Journal Article
Cancer Cell, ISSN 1535-6108, 02/2014, Volume 25, Issue 2, pp. 139 - 151
We report that two oncogenes coamplified on chromosome 3q26, and , cooperate to drive a stem-like phenotype in lung squamous cell carcinoma (LSCC). Protein... 
INITIATING CELLS | DETECTS FREQUENT | STEM-CELLS | PROTEIN | CANCER CELLS | EPITHELIUM | ONCOLOGY | TRANSFORMED GROWTH | PKC-IOTA | KINASE-C-IOTA | BASAL-CELLS | CELL BIOLOGY | Acyltransferases - antagonists & inhibitors | Protein Kinase C - genetics | RNA, Small Interfering - genetics | Cell Proliferation | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Lung Neoplasms - metabolism | SOXB1 Transcription Factors - antagonists & inhibitors | Lung Neoplasms - pathology | Acyltransferases - metabolism | Acyltransferases - genetics | Promoter Regions, Genetic - genetics | Immunoenzyme Techniques | SOXB1 Transcription Factors - metabolism | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Cell Transformation, Neoplastic - genetics | Isoenzymes - metabolism | Protein Kinase C - metabolism | Neoplastic Stem Cells - pathology | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization | Tumor Cells, Cultured | Real-Time Polymerase Chain Reaction | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Isoenzymes - genetics | RNA, Messenger - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Protein Kinase C - antagonists & inhibitors | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Animals | High-Throughput Nucleotide Sequencing | Mice | Cell Transformation, Neoplastic - pathology | Isoenzymes - antagonists & inhibitors | Apoptosis | Squamous cell carcinoma | Genetic aspects | Cancer
Journal Article
British Journal of Haematology, ISSN 0007-1048, 07/2009, Volume 146, Issue 1, pp. 34 - 43
Summary The effect of CMC-544, a calicheamicin-conjugated anti-CD22 monoclonal antibody, was analysed in relation to CD22 and P-glycoprotein (P-gp) in B-cell... 
P-glycoprotein | calicheamicin | monoclonal antibody | CMC-544 | CD22 | P‐glycoprotein | CMC‐544 | Calicheamicin | Monoclonal antibody | GEMTUZUMAB OZOGAMICIN | ANTIBODY-TARGETED CHEMOTHERAPY | EFFICACY | P-GLYCOPROTEIN EXPRESSION | ACUTE MYELOID-LEUKEMIA | IMMUNOCONJUGATE | THERAPY | RITUXIMAB | MONOCLONAL-ANTIBODIES | HEMATOLOGY | Lymphoma, Non-Hodgkin - immunology | Cell Count | Drug Resistance, Multiple - drug effects | Humans | Immunosuppressive Agents - therapeutic use | ATP-Binding Cassette, Sub-Family B, Member 1 - metabolism | Antibodies, Monoclonal - therapeutic use | Drug Resistance, Neoplasm | Sialic Acid Binding Ig-like Lectin 2 - immunology | Antineoplastic Agents - therapeutic use | ATP-Binding Cassette, Sub-Family B, Member 1 - antagonists & inhibitors | Dose-Response Relationship, Drug | Antibodies, Monoclonal, Humanized | Flow Cytometry | Cyclosporins - therapeutic use | Sialic Acid Binding Ig-like Lectin 2 - analysis | Tumor Cells, Cultured | Leukemia, Lymphocytic, Chronic, B-Cell - immunology | Lymphoma, Non-Hodgkin - drug therapy | Jurkat Cells | Treatment Outcome | ATP-Binding Cassette, Sub-Family B, Member 1 - analysis | Leukemia, Lymphocytic, Chronic, B-Cell - metabolism | Lymphoma, Non-Hodgkin - metabolism | Cell Line, Tumor | Quinolines - therapeutic use | Leukemia, Lymphocytic, Chronic, B-Cell - drug therapy | Cell Line, Transformed | Monoclonal antibodies | Medical colleges | Non-Hodgkin's lymphomas | Dyes and dyeing
Journal Article
Cancer Cell, ISSN 1535-6108, 2008, Volume 13, Issue 6, pp. 483 - 495
Faithful modeling of mixed-lineage leukemia in murine cells has been difficult to achieve. We show that expression of in human CD34+ cells induces acute... 
STEMCELL | CELLCYCLE | HEMATOPOIETIC-CELLS | B-CELLS | STEM-CELLS | ONCOLOGY | MLL REARRANGEMENTS | ACUTE LYMPHOBLASTIC-LEUKEMIA | ACUTE MYELOGENOUS LEUKEMIA | THERAPEUTIC TARGET | GENE-EXPRESSION PROFILES | ACUTE MYELOID-LEUKEMIA | FLT3 MUTATIONS | Translocation, Genetic | Antigens, CD34 - analysis | Myeloid-Lymphoid Leukemia Protein - metabolism | Cell Proliferation | Fetal Stem Cells - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Multipotent Stem Cells - metabolism | rac GTP-Binding Proteins - metabolism | Aneuploidy | Gene Expression Profiling | Stem Cell Transplantation | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | rac GTP-Binding Proteins - genetics | Neoplastic Stem Cells - pathology | Cell Culture Techniques | Fetal Blood - immunology | Transduction, Genetic | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Genotype | Cell Lineage | Multipotent Stem Cells - pathology | Phenotype | Environment | Mice, Inbred NOD | Mice | Fetal Stem Cells - pathology | Oncogene Proteins, Fusion - metabolism | Species Specificity | Gene Expression Regulation, Neoplastic | Neoplastic Stem Cells - immunology | Multipotent Stem Cells - immunology | Fetal Stem Cells - immunology | Intercellular Signaling Peptides and Proteins - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Leukemia, Experimental - pathology | Fetal Blood - metabolism | Mice, SCID | Cell Transformation, Neoplastic - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Animals | Myeloid-Lymphoid Leukemia Protein - genetics | Oncogene Proteins, Fusion - genetics | Chromosomes, Human, Pair 11 | Cell Transformation, Neoplastic - pathology | Cell Line, Transformed | Leukemia, Experimental - metabolism | Apoptosis | Leukemia, Myeloid, Acute - genetics | Cytogenetics | Models | Growth factors | Analysis | Leukemia | Stem cells
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2011, Volume 108, Issue 4, pp. 1397 - 1402
Tumors are often heterogeneous, being composed of multiple cell types with different phenotypic and molecular properties. Cancer stem-like cells (CSCs) are a... 
Tumor cell line | MicroRNA | Stem cells | Cell lines | Transformed cell line | Breast cancer | Cells | Cancer | Tumors | Mesenchymal stem cells | Cellular transformation | Cancer stem cells equilibrium | Inflammation | cancer stem cells equilibrium | cellular transformation | ZEB1 | REPRESSION | inflammation | MULTIDISCIPLINARY SCIENCES | MESENCHYMAL TRANSITION | IDENTIFICATION | MIR-200 FAMILY | SIGNATURE | Prostatic Neoplasms - metabolism | Receptors, Estrogen - metabolism | Humans | Male | Transplantation, Heterologous | Gene Expression Profiling | Breast Neoplasms - metabolism | Neoplasms, Experimental - pathology | Cell Differentiation - genetics | Prostatic Neoplasms - genetics | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Hyaluronan Receptors - metabolism | Neoplasms, Experimental - genetics | src-Family Kinases - metabolism | Antibodies - immunology | Neoplastic Stem Cells - pathology | Female | Interleukin-6 - metabolism | Cell Line | Prostatic Neoplasms - pathology | Receptors, Estrogen - genetics | Antineoplastic Agents, Hormonal - pharmacology | Interleukin-6 - genetics | Antibodies - pharmacology | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Interleukin-6 - immunology | Tamoxifen - pharmacology | Cell Line, Tumor | Mice | MicroRNAs - genetics | Cell Transformation, Neoplastic - drug effects | Neoplasms, Experimental - metabolism | src-Family Kinases - genetics | Care and treatment | Cancer cells | Physiological aspects | Development and progression | Genetic aspects | Research | Cell transformation | Health aspects | Interleukin-6 | Biological Sciences
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2017, Volume 19, Issue 5, pp. 530 - 541
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 02/2008, Volume 151, Issue 2, pp. 284 - 296
Journal Article