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Journal Article
Bioconjugate Chemistry, ISSN 1043-1802, 03/2018, Volume 29, Issue 3, pp. 813 - 823
Current strategies for treating autoimmunity involve the administration of broad-acting immunosuppressive agents that impair healthy immunity. Intravenous... 
EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | BONE-MARROW | BIOCHEMICAL RESEARCH METHODS | CHEMISTRY, ORGANIC | INDUCTION | CHEMISTRY, MULTIDISCIPLINARY | TRANSPLANTATION | DELIVERY | TOLEROGENIC NANOPARTICLES | IN-VIVO | IMMUNE TOLERANCE | Polyglactin 910 - therapeutic use | Encephalomyelitis, Autoimmune, Experimental - immunology | Transforming Growth Factor beta - chemistry | Antigens - administration & dosage | Nanoconjugates - therapeutic use | Antigens - therapeutic use | Transforming Growth Factor beta - therapeutic use | Female | Polyglactin 910 - chemistry | Polyglactin 910 - administration & dosage | Immune Tolerance - drug effects | Immunologic Factors - chemistry | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Mice, Inbred C57BL | Cells, Cultured | Immunologic Factors - administration & dosage | Nanoconjugates - administration & dosage | Antigens - chemistry | Animals | Transforming Growth Factor beta - administration & dosage | Multiple Sclerosis - immunology | Mice | Nanoconjugates - chemistry | Immunologic Factors - therapeutic use | Multiple Sclerosis - drug therapy | Animal models | Multiple sclerosis | Intravenous administration | Encephalomyelitis | Lymphocytes T | Cobalt | Immunity | Immunosuppressive agents | Nanoparticles | Lymphocytes | Bone marrow | Growth factors | Antigens | Phenotypes | Immune response | Cytokines | Transforming growth factor-b1 | Dendritic cells | Immunoregulation | Immunomodulation | Inflammation | Immunological tolerance | Experimental allergic encephalomyelitis | Immune systems | Silver | Conjugation | Poly(lactide-co-glycolide) | Index Medicus
Journal Article
Journal Article
Biomaterials, ISSN 0142-9612, 2011, Volume 32, Issue 26, pp. 6183 - 6193
Journal Article
Biochemical Journal, ISSN 0264-6021, 08/2012, Volume 445, Issue 3, pp. 403 - 411
N-linked 21ycosylation is a critical determinant of protein structure and function, regulating processes such as protein folding, stability and localization,... 
Type II transforming growth factor β receptor (TβRII) | N-linked glycosylation | Cell surface transport | Transforming growth factor β sensitivity (TGF-β sensitivity) | Transforming growth factor β signalling (TGF-β signalling) | transforming growth factor beta signalling (TGF-beta signalling) | OLIGOSACCHARIDES | MEMBRANE | STABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHENOTYPE | MECHANISMS | type II transforming growth factor beta receptor (T beta RII) | transforming growth factor beta sensitivity (TGF-beta sensitivity) | CELL-SURFACE TRANSPORT | BIOSYNTHESIS | TUMOR-SUPPRESSOR | cell surface transport | PROTEINS | EXPRESSION | Receptors, Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - chemistry | Humans | Molecular Sequence Data | Glycosylation - drug effects | HEK293 Cells | Conserved Sequence | Cell Membrane - metabolism | Protein-Serine-Threonine Kinases - metabolism | Recombinant Proteins - metabolism | Amino Acid Sequence | Cell Line | Alkaloids - pharmacology | Mutagenesis, Site-Directed | Protein-Serine-Threonine Kinases - genetics | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Hep G2 Cells | Sequence Homology, Amino Acid | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Protein-Serine-Threonine Kinases - chemistry | HeLa Cells | Transforming Growth Factor beta - metabolism | Amino Acid Substitution | Asparagine - chemistry | Index Medicus | transforming growth factor β sensitivity (TGF-β sensitivity) | KIF, kifunensine | dNG, deficient N-linked glycosylation | TβRII, type II TGF-β receptor | WT, wild-type | Tun, tunicamycin | FBS, fetal bovine serum | TGF-β, transforming growth factor β | Endo H, endoglycosidase H | HEK, human embryonic kidney | DAPI, 4′,6-diamidino-2-phenylindole | type II transforming growth factor β receptor (TβRII) | PNGase F, peptide N-glycosidase F | PEI, polyethyleneimine | GlcNAc, N-acetylglucosamine | transforming growth factor β signalling (TGF-β signalling) | TβRI, type I TGF-β receptor | ER, endoplasmic reticulum | PDI, protein disulfide-isomerase | RT, room temperature
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 01/2015, Volume 35, Issue 1, pp. 213 - 226
Background/Aims: Hypoxia is a basic pathological challenge that is associated with numerous cardiovascular disorders including aberrant cardiac remodeling.... 
Original Paper | Cardiac fibrosis | Cardiac fibroblasts | Hypoxia | Transforming growth factor β receptor I | MicroRNAs | PHYSIOLOGY | MYOFIBROBLAST | MECHANISMS | ATRIAL-FIBRILLATION | CELL BIOLOGY | RECEPTOR 1 | RENAL FIBROSIS | HYPERTROPHY | GROWTH-FACTOR-BETA | DISEASE | ACUTE MYOCARDIAL-INFARCTION | Transforming growth factor beta receptor I | EXPRESSION | Receptor, Transforming Growth Factor-beta Type I | Up-Regulation | Receptors, Transforming Growth Factor beta - genetics | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - chemistry | Transforming Growth Factor beta1 - metabolism | Oligonucleotides, Antisense - metabolism | Male | MicroRNAs - metabolism | Smad3 Protein - metabolism | Cell Hypoxia | Myocardial Infarction - pathology | Collagen - genetics | Myocardial Infarction - etiology | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Fibrosis - genetics | Down-Regulation | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Rats | Myocardium - pathology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardium - cytology | Collagen - metabolism | Animals | Receptors, Transforming Growth Factor beta - metabolism | Fibroblasts - cytology | Protein-Serine-Threonine Kinases - chemistry | Cell Movement | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2009, Volume 106, Issue 37, pp. 15679 - 15683
Transforming growth factor-β (TGF-β) elicits its signals through two transmembrane serine/threonine kinase receptors, type II (TβRII) and type I receptors. It... 
Molecules | Receptors | Bleaching | Imaging | HeLa cells | Fluorescence | Cell membranes | Dimers | Dimerization | Monomers | Serine/threonine kinase receptor | Subunit stoichiometry | ACTIVATION | MULTIDISCIPLINARY SCIENCES | subunit stoichiometry | serine/threonine kinase receptor | MICROSCOPY | PLASMA-MEMBRANE | EGFR | SIGNAL-TRANSDUCTION | INHIBITION | SURFACE | PROTEINS | EXPRESSION | LIVING CELLS | Receptor, Epidermal Growth Factor - genetics | Receptors, Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - chemistry | Humans | Green Fluorescent Proteins - genetics | Recombinant Fusion Proteins - metabolism | Cell Membrane - chemistry | Receptor, Epidermal Growth Factor - metabolism | Protein Structure, Quaternary | Cell Membrane - metabolism | Green Fluorescent Proteins - chemistry | Cell Membrane - drug effects | Protein-Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - pharmacology | Cell Line | Green Fluorescent Proteins - metabolism | Biophysical Phenomena | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | Recombinant Fusion Proteins - chemistry | Receptor, Epidermal Growth Factor - chemistry | Receptors, Transforming Growth Factor beta - metabolism | Recombinant Fusion Proteins - genetics | Ligands | Protein-Serine-Threonine Kinases - chemistry | HeLa Cells | Microscopy, Fluorescence | Index Medicus | Biological Sciences | serine | threonine kinase receptor
Journal Article
Journal Article
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 06/2017, Volume 42, Issue 1, pp. 357 - 372
Endothelial-to-mesenchymal transition (EndMT) plays significant roles under various pathological conditions including cardiovascular diseases, fibrosis, and... 
Endothelial-to-mesenchymal transition | MALAT1 | SMAD3 | Endothelial progenitor cells | miR-145 | TGFBR2 | Receptors, Transforming Growth Factor beta - genetics | Endothelial Progenitor Cells - cytology | MicroRNAs - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - chemistry | Humans | Transforming Growth Factor beta1 - metabolism | Collagen Type III - metabolism | Epithelial-Mesenchymal Transition - drug effects | MicroRNAs - metabolism | Smad3 Protein - metabolism | Smad3 Protein - genetics | Receptor, Transforming Growth Factor-beta Type II | RNA Interference | Base Sequence | 3' Untranslated Regions | Protein-Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - pharmacology | Endothelial Progenitor Cells - metabolism | Collagen Type I - metabolism | Signal Transduction | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | RNA, Long Noncoding - genetics | Down-Regulation - drug effects | Sequence Alignment | Receptors, Transforming Growth Factor beta - metabolism | Smad3 Protein - chemistry | MicroRNAs - genetics | Protein-Serine-Threonine Kinases - chemistry | Antagomirs - metabolism | RNA, Long Noncoding - antagonists & inhibitors | RNA, Long Noncoding - metabolism | RNA, Small Interfering - metabolism | Studies | Liver cancer | Cell growth | MicroRNAs | Rodents | Smooth muscle | Metastasis | Growth factors | Cell adhesion & migration | Index Medicus
Journal Article
Structure, ISSN 0969-2126, 11/2017, Volume 25, Issue 11, pp. 1740 - 1750.e2
A major cause of visual impairment, corneal dystrophies result from accumulation of protein deposits in the cornea. One of the proteins involved is... 
extracellular matrix protein | cysteine-rich CROPT domain | multi-domain protein | pathological mutants | crystal structure | corneal dystrophy | fasciclin FAS1 domain | GENOTYPE-PHENOTYPE CORRELATIONS | KERATO-EPITHELIN MUTATIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMYLOID DEPOSITS | STRUCTURE ALIGNMENT | CELL BIOLOGY | BIOPHYSICS | STRUCTURE VALIDATION | MACROMOLECULAR CRYSTALLOGRAPHY | CHINESE PATIENTS | BIGH3 GENE-MUTATIONS | STRUCTURE PREDICTION | GRANULAR DYSTROPHY | Protein Aggregates | Humans | Integrins - chemistry | Corneal Dystrophies, Hereditary - genetics | Integrins - genetics | Crystallography, X-Ray | Transforming Growth Factor beta - chemistry | Integrins - metabolism | Corneal Dystrophies, Hereditary - pathology | Cloning, Molecular | HEK293 Cells | Protein Interaction Domains and Motifs | Binding Sites | Protein Aggregation, Pathological - genetics | Extracellular Matrix Proteins - metabolism | Recombinant Proteins - metabolism | Amino Acid Sequence | Protein Conformation, alpha-Helical | Extracellular Matrix Proteins - chemistry | Gene Expression | Genetic Vectors - chemistry | Extracellular Matrix Proteins - genetics | Genetic Vectors - metabolism | Models, Molecular | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Corneal Dystrophies, Hereditary - metabolism | Sequence Homology, Amino Acid | Sequence Alignment | Transforming Growth Factor beta - genetics | Protein Conformation, beta-Strand | Protein Binding | Mutation | Transforming Growth Factor beta - metabolism | Protein Aggregation, Pathological - metabolism | Index Medicus
Journal Article