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Journal Article
Cell Cycle, ISSN 1538-4101, 08/2012, Volume 11, Issue 16, pp. 3019 - 3035
We have previously shown that a loss of stromal Cav-1 is a biomarker of poor prognosis in breast cancers. Mechanistically, a loss of Cav-1 induces the... 
autophagy | paracrine signaling | myofibroblast | cancer associated fibroblast | the field effect | cancer metabolism | "Pied-Piper of Hamelin" | mitophagy | autocrine signaling | tumor stroma | TGF beta | aerobic glycolysis | oxidative stress | Oxidative stress | Autocrine signaling | Myofibroblast | Tumor stroma | Paracrine signaling | Cancer associated fibroblast | Mitophagy | Autophagy | Cancer metabolism | The field effect | Aerobic glycolysis | Pied-Piper of Hamelin | SMOOTH MUSCLE ACTIN | TGF- beta | METASTASIS | CELL BIOLOGY | BREAST-CANCER | CAVEOLIN-1 EXPRESSION | EPITHELIAL-CELLS | MICROENVIRONMENT | TRANSFORMING GROWTH-FACTOR-BETA-1 | Immunohistochemistry | Receptors, Transforming Growth Factor beta - genetics | Oxidative Stress | Stromal Cells - pathology | Coculture Techniques | Humans | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | Breast Neoplasms - metabolism | Cellular Reprogramming | Mitochondria - genetics | Cell Transformation, Neoplastic - genetics | Female | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Autocrine Communication | Paracrine Communication | Lactic Acid - metabolism | Stromal Cells - metabolism | Oxidative Phosphorylation | Protein-Serine-Threonine Kinases - genetics | Caveolin 1 - genetics | Mitochondria - metabolism | Mitochondria - pathology | Fibroblasts - pathology | Cell Transformation, Neoplastic - metabolism | Caveolin 1 - metabolism | Gene Expression Regulation, Enzymologic | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Breast Neoplasms - pathology | Mice, Nude | Cell Line, Tumor | Glycolysis | Ligands | Mice | Neovascularization, Pathologic - metabolism | Transforming Growth Factor beta - metabolism | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2015, Volume 35, Issue 4, pp. 911 - 917
Objective-Studies of mice with mild Marfan syndrome (MFS) have correlated the development of thoracic aortic aneurysm (TAA) with improper stimulation of... 
aortic aneurysm | receptor, angiotensin, type 1 | losartan | transforming growth factor β | Marfan syndrome | PATHOGENESIS | TGF-BETA | type 1 | transforming growth factor beta | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | receptor, angiotensin | Aortic Aneurysm, Thoracic - genetics | Phosphorylation | Aortic Aneurysm, Thoracic - prevention & control | Humans | Aortic Rupture - metabolism | Mice, 129 Strain | Angiotensin II Type 1 Receptor Blockers - pharmacology | Marfan Syndrome - drug therapy | Aortic Aneurysm, Thoracic - pathology | Time Factors | Mice, Mutant Strains | Aortic Rupture - prevention & control | Transforming Growth Factor beta - antagonists & inhibitors | Aorta, Thoracic - drug effects | Microfilament Proteins - genetics | Aorta, Thoracic - pathology | Disease Models, Animal | Transforming Growth Factor beta - immunology | Fibrillin-1 | Mice, Inbred C57BL | Losartan - pharmacology | Smad2 Protein - metabolism | Aorta, Thoracic - metabolism | Antibodies, Neutralizing - pharmacology | Aortic Rupture - pathology | Fibrillins | Disease Progression | Marfan Syndrome - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Aortic Aneurysm, Thoracic - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Aortic Rupture - genetics | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Mutation | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2013, Volume 19, Issue 8, pp. 1047 - 1053
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2007, Volume 104, Issue 9, pp. 3432 - 3437
Journal Article
Cancer Research, ISSN 0008-5472, 11/2009, Volume 69, Issue 21, pp. 8317 - 8325
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2012, Volume 14, Issue 7, pp. 717 - 726
The stability and membrane localization of the transforming growth factor-beta (TGF-beta) type I receptor (T beta RI) determines the levels of TGF-beta... 
SIGNAL-TRANSDUCTION | ACTIVATION | METASTASIS | CELL-MIGRATION | GROWTH | TARGETS | MESENCHYMAL TRANSITION | CANCER PROGRESSION | E3 UBIQUITIN LIGASE | DEPENDENT DEGRADATION | CELL BIOLOGY | Oncogene Proteins - genetics | Receptor, Transforming Growth Factor-beta Type I | Phosphorylation | Receptors, Transforming Growth Factor beta - genetics | Humans | Zebrafish - embryology | Breast Neoplasms - enzymology | Ubiquitination | Transfection | RNA Interference | Time Factors | Epithelial-Mesenchymal Transition | HEK293 Cells | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Ubiquitin Thiolesterase - metabolism | Female | Cell Membrane - metabolism | Proto-Oncogene Proteins | Proto-Oncogene Proteins c-akt - metabolism | Protein-Serine-Threonine Kinases - metabolism | Signal Transduction | Neoplasm Invasiveness | Enzyme Stability | Gene Expression Regulation | Oncogene Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Ubiquitin Thiolesterase - genetics | Mice, Knockout | Protein Transport | Ubiquitin-Specific Proteases | Animals | Breast Neoplasms - genetics | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - metabolism | Breast Neoplasms - pathology | Oncogene Proteins - deficiency | Mice | Protein Kinase Inhibitors - pharmacology | HeLa Cells | Mutation | Transforming Growth Factor beta - metabolism | Cell Movement | Index Medicus
Journal Article