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Oncogene, ISSN 0950-9232, 12/2010, Volume 29, Issue 49, pp. 6485 - 6498
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 508, Issue 1, pp. 118 - 122
Treatment of BRAF(V600E) mutant melanoma by small molecule drugs that target the BRAF or MEK kinases can be effective, but resistance develops invariably(1,2).... 
GROWTH-FACTOR RECEPTOR | BRAF INHIBITOR | RAF INHIBITION | CELLS | MULTIDISCIPLINARY SCIENCES | IMPROVED SURVIVAL | DIFFERENTIATION | C-JUN | CANCER | EXPRESSION | EGFR | Receptor, Epidermal Growth Factor - genetics | Humans | Receptor Protein-Tyrosine Kinases - biosynthesis | Cellular Senescence - drug effects | Melanoma - enzymology | Antineoplastic Agents - administration & dosage | Receptor, Platelet-Derived Growth Factor beta - genetics | Indoles - administration & dosage | Mitogen-Activated Protein Kinase Kinases - metabolism | Receptor, Epidermal Growth Factor - metabolism | Flow Cytometry | Melanoma - genetics | Female | Indoles - pharmacology | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | SOXE Transcription Factors - deficiency | Proto-Oncogene Proteins B-raf - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Receptor, Epidermal Growth Factor - biosynthesis | Gene Library | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Melanoma - pathology | Receptor Protein-Tyrosine Kinases - metabolism | Sulfonamides - pharmacology | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Protein Kinase Inhibitors - administration & dosage | Transforming Growth Factor beta - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Receptor Protein-Tyrosine Kinases - genetics | Signal Transduction - drug effects | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | RNA, Small Interfering | Transforming Growth Factor beta - metabolism | Receptor, Platelet-Derived Growth Factor beta - biosynthesis | Sulfonamides - administration & dosage | Drug Resistance, Neoplasm - drug effects | SOXE Transcription Factors - genetics | Proteins | Biopsy | Rodents | Genes | Melanoma | Mutation | Kinases | Drug resistance | Patients | Tumors | Index Medicus
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2013, Volume 123, Issue 3, pp. 1348 - 1358
Journal Article
Neuron, ISSN 0896-6273, 05/2017, Volume 94, Issue 4, pp. 759 - 773.e8
Microglia, the resident macrophages of the CNS, engage in various CNS-specific functions that are critical for development and health. To better study... 
CSF1R | phagocytosis | maturation | inflammation | neurodegeneration | TGF-β | cholesterol | transplant | microglia | astrocyte | IDENTITY | GROWTH-FACTOR-BETA | PROGENITOR-CELL | BRAIN-INJURY | RNA-SEQ | ASTROCYTE | MACROPHAGES | MOUSE | RAMIFICATION | EXPRESSION | NEUROSCIENCES | Microglia - metabolism | Transforming Growth Factor beta2 - metabolism | Humans | Transcriptome | Interleukins - metabolism | Microglia - immunology | Macrophage Colony-Stimulating Factor - pharmacology | Serum | Cell Culture Techniques | Interleukins - pharmacology | Microglia - cytology | Cell Survival - drug effects | Phagocytosis - drug effects | Microglia - drug effects | Phagocytosis - immunology | Rats | Macrophage Colony-Stimulating Factor - metabolism | Cholesterol - metabolism | Transforming Growth Factor beta2 - pharmacology | Transforming Growth Factor beta - pharmacology | Animals | Cholesterol - pharmacology | Mice | Transforming Growth Factor beta - metabolism | Astrocytes - metabolism | Culture Media, Conditioned - metabolism | Medical colleges | Bone morphogenetic proteins | Macrophages | Transforming growth factors | Gene expression | Neurophysiology | Genes | Cell culture | Cell survival | Macrophage colony-stimulating factor | Central nervous system | Homeostasis | Exposure | Survival | Cholesterol | Microglia | Phagocytes | Rodents | Morphology | Growth factors | Index Medicus
Journal Article
Journal Article
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2009, Volume 29, Issue 28, pp. 8927 - 8935
Brain injury may result in the development of epilepsy, one of the most common neurological disorders. We previously demonstrated that albumin is critical in... 
TEMPORAL-LOBE EPILEPSY | ALZHEIMERS-DISEASE | TRANSFORMING GROWTH-FACTOR-BETA-1 | IN-VIVO | NEOCORTICAL EPILEPTOGENESIS | STATUS EPILEPTICUS | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | RAT HIPPOCAMPUS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | Electric Stimulation - adverse effects | Rats, Wistar | Transforming Growth Factor beta2 - metabolism | gamma-Aminobutyric Acid - metabolism | Glutamic Acid - genetics | Ion Channels - genetics | Male | Epilepsy - etiology | Brain - metabolism | Transforming Growth Factor beta2 - immunology | Gene Expression - physiology | Statistics, Nonparametric | Dioxoles - pharmacology | Benzamides - pharmacology | Disease Models, Animal | Microarray Analysis - methods | Transforming Growth Factor beta - immunology | gamma-Aminobutyric Acid - genetics | Immunoprecipitation - methods | Albumins - metabolism | Brain - physiopathology | Gene Expression Regulation | Smad2 Protein - metabolism | Rats | Gene Expression Profiling - methods | Blood-Brain Barrier - drug effects | Inflammation - etiology | Epilepsy - complications | Action Potentials - physiology | Antibodies - pharmacology | Transforming Growth Factor beta - pharmacology | Animals | Transforming Growth Factor beta - genetics | Ion Channels - metabolism | Transforming Growth Factor beta2 - antagonists & inhibitors | Signal Transduction - physiology | Glutamic Acid - metabolism | Genome-Wide Association Study - methods | In Vitro Techniques | Epilepsy - pathology | Transforming Growth Factor beta - metabolism | Astrocytes - metabolism | Cluster Analysis | Index Medicus
Journal Article
Hepatology, ISSN 0270-9139, 12/2012, Volume 56, Issue 6, pp. 2255 - 2267
Liver cirrhosis is a predominant risk factor for hepatocellular carcinoma (HCC). However, the mechanism underlying the progression from cirrhosis to HCC... 
PROGENITOR CELLS | HEPATOCELLULAR-CARCINOMA | STEM-CELLS | HEPATOCYTES | TGF-BETA | EPIGENETIC REGULATION | PTEN | SELF-RENEWAL | TUMORIGENICITY | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Rats, Wistar | TOR Serine-Threonine Kinases - metabolism | Humans | Glycoproteins - metabolism | Liver Neoplasms, Experimental - chemically induced | Male | MicroRNAs - metabolism | Proto-Oncogene Proteins c-akt - genetics | Antigens, CD - metabolism | Epithelial Cell Adhesion Molecule | Pluripotent Stem Cells - pathology | Liver Neoplasms, Experimental - metabolism | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Diethylnitrosamine | Liver Cirrhosis - metabolism | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Antigens, Differentiation - metabolism | Liver Neoplasms, Experimental - pathology | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - metabolism | Liver - metabolism | Mice, Inbred C57BL | PTEN Phosphohydrolase - metabolism | Rats | Mice, SCID | AC133 Antigen | Cell Adhesion Molecules - metabolism | Cell Transformation, Neoplastic - metabolism | Pluripotent Stem Cells - metabolism | Thy-1 Antigens - metabolism | Transforming Growth Factor beta - pharmacology | Animals | Pluripotent Stem Cells - drug effects | Liver Cirrhosis - pathology | Mice, Inbred NOD | Mice | Cell Transformation, Neoplastic - pathology | Transforming Growth Factor beta - metabolism | Proteins | Liver cancer | Liver cirrhosis | Hepatology | Index Medicus
Journal Article