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Journal of Cellular Physiology, ISSN 0021-9541, 03/2018, Volume 233, Issue 3, pp. 2067 - 2074
Fibrosis of the subsynovial connective tissue (SSCT) in carpal tunnel syndrome (CTS) patients is increasingly recognized as an important aspect of CTS... 
carpal tunnel syndrome | fibrosis | subsynovial connective tissue | transforming growth factor β | CANCER-CELLS | SUBSYNOVIAL CONNECTIVE-TISSUE | ATTENUATES LIVER FIBROSIS | PHYSIOLOGY | TGF-BETA | transforming growth factor beta | SYSTEMIC-SCLEROSIS | PDGF RECEPTOR | CELL BIOLOGY | RENAL FIBROSIS | GROWTH-FACTOR-BETA | RECEPTOR TYROSINE KINASES | LUNG-DISEASE | Fibrosis - drug therapy | ErbB Receptors - antagonists & inhibitors | Receptors, Platelet-Derived Growth Factor - antagonists & inhibitors | Collagen Type III - biosynthesis | Connective Tissue - pathology | Humans | Cells, Cultured | Synovial Membrane - pathology | Collagen Type III - genetics | Connective Tissue Growth Factor - biosynthesis | Plasminogen Activator Inhibitor 1 - biosynthesis | Carpal Tunnel Syndrome - pathology | Collagen Type I - biosynthesis | Collagen Type I - genetics | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Connective Tissue Growth Factor - genetics | Fibrosis - pathology | Plasminogen Activator Inhibitor 1 - genetics | Carpal Tunnel Syndrome - drug therapy | Transforming Growth Factor beta - metabolism | Synovial Membrane - cytology | Connective Tissue Cells - cytology | Fibroblasts - metabolism | Carpal tunnel syndrome | Collagen | Analysis | Genes | Gene expression | Vascular endothelial growth factor | Growth factors | Smad protein | Collagen (type I) | Platelet-derived growth factor | Epidermal growth factor receptors | Gene regulation | Overuse injuries | Collagen (type III) | Connective tissue growth factor | Patients | Vascular endothelial growth factor receptors | Polymerase chain reaction | Connective tissues | Signal transduction | Signaling | Epidermal growth factor | Down-regulation | Fibrosis | Fibroblasts | Inhibition | Subsynovial connective tissue
Journal Article
Journal of Controlled Release, ISSN 0168-3659, 05/2015, Volume 206, p. 131
Obstructive nephropathy is the leading cause of kidney disease in children. The tissue injury resulting from initial dilation precipitates a deleterious... 
Care and treatment | Chronic kidney failure | Interleukins | Immunotherapy | Hyaluronic acid | Transforming growth factors
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 07/2011, Volume 17, Issue 13, pp. 4296 - 4308
Journal Article
Journal Article
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 10/2006, Volume 24, Issue 29, pp. 4721 - 4730
Journal Article
Nature, ISSN 0028-0836, 02/2010, Volume 463, Issue 7281, pp. 676 - 680
Chronic myeloid leukaemia (CML) is caused by a defined genetic abnormality that generates BCR-ABL, a constitutively active tyrosine kinase(1). It is widely... 
CHRONIC MYELOGENOUS LEUKEMIA | MURINE MODEL | APOPTOSIS | OXIDATIVE STRESS | ABL-EXPRESSING CELLS | MULTIDISCIPLINARY SCIENCES | HEMATOPOIETIC STEM-CELLS | BCR-ABL | BONE-MARROW | MICE | TRANSCRIPTION FACTOR | Phosphorylation | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Antineoplastic Agents - therapeutic use | Cell Nucleus - metabolism | Neoplastic Stem Cells - metabolism | Forkhead Transcription Factors - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Neoplastic Stem Cells - pathology | Transforming Growth Factor beta - antagonists & inhibitors | Cell Differentiation | Proto-Oncogene Proteins c-akt - metabolism | Forkhead Transcription Factors - deficiency | Disease Models, Animal | Tumor Stem Cell Assay | Mice, Inbred C57BL | Piperazines - therapeutic use | Forkhead Transcription Factors - genetics | Imatinib Mesylate | Protein Transport | Animals | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Benzamides | Enzyme Activation | Forkhead Box Protein O3 | Transforming Growth Factor beta - metabolism | Apoptosis | Protein-Tyrosine Kinases - antagonists & inhibitors
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 02/2017, Volume 23, Issue 3, pp. 804 - 813
Purpose: Immunotherapy of high-risk neuroblastoma using the anti-GD2 antibody dinutuximab induces antibody-dependent cell-mediated cytotoxicity (ADCC).... 
BREAST-CANCER | RECEPTOR KINASE INHIBITOR | TGF-BETA | GAMMA PRODUCTION | METASTASIS | ONCOLOGY | GROWTH | TUMOR-TISSUE | ANTITUMOR-ACTIVITY | EX-VIVO | CYTOTOXICITY | Receptor, Transforming Growth Factor-beta Type I | Receptors, Transforming Growth Factor beta - genetics | Humans | Neoplasm Proteins - physiology | Male | Neoplasm Proteins - antagonists & inhibitors | Gene Expression Profiling | Receptors, Transforming Growth Factor beta - physiology | Smad2 Protein - antagonists & inhibitors | Quinolines - pharmacology | RNA, Messenger - biosynthesis | Protein Processing, Post-Translational - drug effects | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Female | Phosphorylation - drug effects | Neuroblastoma - pathology | Transforming Growth Factor beta1 - biosynthesis | Pyrazoles - pharmacology | Killer Cells, Natural - transplantation | Specific Pathogen-Free Organisms | Immunotherapy, Adoptive | Protein-Serine-Threonine Kinases - physiology | Antibodies, Monoclonal - pharmacology | RNA, Messenger - genetics | Protein-Serine-Threonine Kinases - genetics | Smad2 Protein - metabolism | Transforming Growth Factor beta1 - genetics | Transforming Growth Factor beta1 - physiology | Antineoplastic Agents, Immunological - pharmacology | Drug Synergism | Protein-Serine-Threonine Kinases - biosynthesis | Xenograft Model Antitumor Assays | Receptors, Transforming Growth Factor beta - biosynthesis | Animals | RNA, Neoplasm - biosynthesis | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Cell Line, Tumor | RNA, Neoplasm - genetics | Mice, Inbred NOD | Mice | Neuroblastoma - metabolism | Cytotoxicity, Immunologic | Phosphorylation | Smad protein | Severe combined immunodeficiency | Toxicity | Cytotoxicity | Activation | Neuroblastoma | Blood | Neuroblastoma cells | NKG2 antigen | Receptors | Smad2 protein | Immunotherapy | Xenografts | Bone marrow | Plasmas | CD226 antigen | Natural killer cells | Perforin | Antibody-dependent cell-mediated cytotoxicity | Cell survival | Transforming growth factor-b1 | Risk groups | Gene expression | Experimental design | Cell lines | TRAIL protein | Bone | Tumors | Cancer | TGFβR1 inhibitor | dinutuximab | adoptive cell therapy | galunisertib | immunotherapy
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 11/2011, Volume 17, Issue 21, pp. 6754 - 6765
Journal Article
Journal Article