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Respiratory research, ISSN 1465-993X, 08/2018, Volume 19, Issue 1, pp. 160 - 160
...); however, mechanistic studies in animal models have produced mixed results. Reports using cell lines have investigated molecular interactions between transforming growth factor beta1 (TGF-beta 1... 
Estrogen receptor | Transforming growth factor beta1 | Lung | Estrogen | Fibrosis | Life Sciences & Biomedicine | Respiratory System | Science & Technology | Estrogens - pharmacology | Respiratory Mucosa - drug effects | Humans | Middle Aged | Cells, Cultured | Male | Dose-Response Relationship, Drug | Signal Transduction - drug effects | Receptors, Estrogen - biosynthesis | Female | Signal Transduction - physiology | Aged | Respiratory Mucosa - metabolism | Transforming Growth Factor beta1 - pharmacology | Receptors | Cellular signal transduction | Research | Transforming growth factors | Epithelial cells | Animal models | Target recognition | Calcium | Mesenchyme | Estrogens | Genes | Sex | Estrogen receptors | Smooth muscle | Males | Contraction | Proteins | Respiratory tract | Signal transduction | Pathways | Gender aspects | DNA methylation | Fibroblasts | Extracellular matrix | Growth factors | Transforming growth factor-b1 | Molecular interactions | Lung diseases | Muscles | Sex differences | Gender differences | Breast cancer | Gene expression | Ribonucleic acid--RNA | Muscle contraction | Chromatin remodeling | Studies | Signaling | Androgens | Molecular modelling | Pulmonary fibrosis | Cell lines | Epigenetics | Skin | Diabetes | Females | Prostate | Index Medicus
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 09/2018, Volume 22, Issue 9, pp. 4545 - 4549
Recently, platelet‐derived growth factors present in lysates became an extreme interest in the field of regenerative medicine such as in wound healing and as substitutes to foetal bovine serum in xeno... 
megakaryocytes | growth factors | platelet lysate | regenerative medicine | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Vascular Endothelial Growth Factor A - biosynthesis | Cell Proliferation | Humans | Insulin-Like Growth Factor I - genetics | Vascular Endothelial Growth Factor A - genetics | Platelet-Derived Growth Factor - genetics | Regenerative Medicine - methods | RNA, Messenger - biosynthesis | Platelet-Derived Growth Factor - biosynthesis | Blood Platelets - cytology | Cell Differentiation | Induced Pluripotent Stem Cells - cytology | Megakaryocytes - cytology | Megakaryocytes - metabolism | Thromboplastin - biosynthesis | Transforming Growth Factor beta1 - biosynthesis | Induced Pluripotent Stem Cells - metabolism | Gene Expression | Insulin-Like Growth Factor I - biosynthesis | Epidermal Growth Factor - biosynthesis | Epidermal Growth Factor - genetics | RNA, Messenger - genetics | Cell Extracts - chemistry | Transforming Growth Factor beta1 - genetics | Blood Platelets - metabolism | Thromboplastin - genetics | Primary Cell Culture | Transforming growth factors | Vascular endothelial growth factor | Stem cells | Cell culture | Wound healing | Transcription | Tissue engineering | Transforming growth factor | Tissue factor | Insulin-like growth factors | Insulin | Endothelial cells | Regeneration (physiology) | Proteins | Regeneration | Lysates | Epidermal growth factor | Megakaryocytes | Platelets | Growth factors | Pluripotency | Inhibitory postsynaptic potentials | Index Medicus | Short Communications | Short Communication
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, pp. e35144 - e35144
...) immunohistochemistry staining showed decreased expression of alpha-smooth muscle actin (alpha-SMA), transforming growth factor beta 1 (TGF-beta 1... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Angiotensin II - pharmacology | Interleukin-6 - genetics | Mice, Inbred C57BL | Transforming Growth Factor beta1 - metabolism | Male | Myocardium - pathology | Smad3 Protein - metabolism | Interleukin-6 - deficiency | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Fibroblasts - drug effects | Myocardium - metabolism | Fibrosis | Interleukin-6 - biosynthesis | Collagen - biosynthesis | Mice | Phosphorylation - drug effects | Transforming Growth Factor beta1 - biosynthesis | Fibroblasts - metabolism | Immunohistochemistry | Heart | Oxidative stress | Collagen (type I) | Phosphorylation | Smad protein | Cardiomyopathy | Transforming growth factor | Interleukin | Staining | Smooth muscle | Cardiovascular disease | Smad3 protein | Kinases | Macrophages | Interleukin 6 | Infusion | Immunology | Actin | Rodents | Fibroblasts | Angiotensin II | Growth factors | Heart diseases | Heart failure | Cardiovascular system | Hypertension | Cytokines | Immunoregulation | Muscles | Cardiomyocytes | Inflammation | Gene expression | Hospitals | Angiotensin | Sepsis | Laboratory animals | Index Medicus | Cardiac muscle | Antibodies | Transforming growth factor- beta 1 | Transforming growth factor- beta | cardiomyocytes | Cell activation
Journal Article
The Journal of immunology (1950), ISSN 0022-1767, 12/2013, Volume 191, Issue 12, pp. 5802 - 5806
Emerging evidence suggests a link between tumor hypoxia and immune suppression. In this study, we investigated the role of hypoxia-induced Nanog, a stemness-associated transcription factor, in immune suppression... 
Genetic Therapy | Intramolecular Oxidoreductases - immunology | Up-Regulation | Lymphopoiesis | Neoplastic Stem Cells - cytology | Tumor Escape - genetics | Neoplasm Proteins - physiology | Vaccination | Gene Expression Regulation, Neoplastic | Neoplastic Stem Cells - immunology | Tumor Microenvironment | Melanoma, Experimental - metabolism | Neoplasm Proteins - antagonists & inhibitors | T-Lymphocytes, Regulatory - immunology | Melanoma, Experimental - immunology | RNA Interference | Immunotherapy | Peptide Fragments - immunology | Neoplasm Proteins - genetics | Spheroids, Cellular | Macrophages - immunology | Transforming Growth Factor beta1 - biosynthesis | Tumor Escape - immunology | Promoter Regions, Genetic | Homeodomain Proteins - biosynthesis | Nanog Homeobox Protein | Neoplasm Proteins - biosynthesis | Melanoma, Experimental - therapy | Mice, Inbred C57BL | Transforming Growth Factor beta1 - genetics | Transforming Growth Factor beta1 - secretion | Transforming Growth Factor beta1 - physiology | Homeodomain Proteins - genetics | Animals | Melanoma, Experimental - genetics | Homeodomain Proteins - antagonists & inhibitors | Cell Line, Tumor | RNA, Small Interfering | Cell Hypoxia - genetics | Homeodomain Proteins - physiology | Neoplastic Stem Cells - enzymology | Lymphocytes, Tumor-Infiltrating - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 12/2012, Volume 287, Issue 50, pp. 41991 - 42000
Journal Article
The Journal of immunology (1950), ISSN 0022-1767, 07/2009, Volume 183, Issue 1, pp. 97 - 105
TGF-beta, together with IL-6 and IL-21, promotes Th17 cell development. IL-6 and IL-21 induce activation of STAT3, which is crucial for Th17 cell... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Protein-Serine-Threonine Kinases - deficiency | Intestinal Mucosa - metabolism | Receptors, Transforming Growth Factor beta - genetics | Interleukin-6 - antagonists & inhibitors | Transforming Growth Factor beta1 - antagonists & inhibitors | Interleukin-17 - physiology | Receptors, Transforming Growth Factor beta - physiology | Intestinal Mucosa - cytology | Cell Differentiation - genetics | Signal Transduction - immunology | Mucous Membrane - cytology | STAT3 Transcription Factor - physiology | Interleukins - physiology | Intestinal Mucosa - immunology | T-Lymphocytes, Helper-Inducer - immunology | Interleukin-6 - physiology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | T-Lymphocytes, Helper-Inducer - cytology | STAT3 Transcription Factor - metabolism | T-Lymphocytes, Helper-Inducer - metabolism | Protein-Serine-Threonine Kinases - physiology | Mice, Inbred C57BL | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Mice, Transgenic | Up-Regulation - genetics | Signal Transduction - genetics | Transforming Growth Factor beta1 - genetics | Interleukin-17 - biosynthesis | Transforming Growth Factor beta1 - physiology | Down-Regulation - genetics | Interleukins - antagonists & inhibitors | Mice, Knockout | Mucous Membrane - metabolism | Cell Differentiation - immunology | Suppressor of Cytokine Signaling 3 Protein | Animals | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Mucous Membrane - immunology | Up-Regulation - immunology | Down-Regulation - immunology | Suppressor of Cytokine Signaling Proteins - antagonists & inhibitors | Mice | Suppressor of Cytokine Signaling Proteins - biosynthesis | Receptors, Transforming Growth Factor beta - deficiency | Index Medicus | Abridged Index Medicus
Journal Article
Biomaterials, ISSN 0142-9612, 2015, Volume 77, pp. 173 - 185
Journal Article