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Journal Article
Clinical Cancer Research, ISSN 1078-0432, 02/2017, Volume 23, Issue 3, pp. 804 - 813
Purpose: Immunotherapy of high-risk neuroblastoma using the anti-GD2 antibody dinutuximab induces antibody-dependent cell-mediated cytotoxicity (ADCC).... 
BREAST-CANCER | RECEPTOR KINASE INHIBITOR | TGF-BETA | GAMMA PRODUCTION | METASTASIS | ONCOLOGY | GROWTH | TUMOR-TISSUE | ANTITUMOR-ACTIVITY | EX-VIVO | CYTOTOXICITY | Receptor, Transforming Growth Factor-beta Type I | Receptors, Transforming Growth Factor beta - genetics | Humans | Neoplasm Proteins - physiology | Male | Neoplasm Proteins - antagonists & inhibitors | Gene Expression Profiling | Receptors, Transforming Growth Factor beta - physiology | Smad2 Protein - antagonists & inhibitors | Quinolines - pharmacology | RNA, Messenger - biosynthesis | Protein Processing, Post-Translational - drug effects | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Female | Phosphorylation - drug effects | Neuroblastoma - pathology | Transforming Growth Factor beta1 - biosynthesis | Pyrazoles - pharmacology | Killer Cells, Natural - transplantation | Specific Pathogen-Free Organisms | Immunotherapy, Adoptive | Protein-Serine-Threonine Kinases - physiology | Antibodies, Monoclonal - pharmacology | RNA, Messenger - genetics | Protein-Serine-Threonine Kinases - genetics | Smad2 Protein - metabolism | Transforming Growth Factor beta1 - genetics | Transforming Growth Factor beta1 - physiology | Antineoplastic Agents, Immunological - pharmacology | Drug Synergism | Protein-Serine-Threonine Kinases - biosynthesis | Xenograft Model Antitumor Assays | Receptors, Transforming Growth Factor beta - biosynthesis | Animals | RNA, Neoplasm - biosynthesis | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Cell Line, Tumor | RNA, Neoplasm - genetics | Mice, Inbred NOD | Mice | Neuroblastoma - metabolism | Cytotoxicity, Immunologic | Phosphorylation | Smad protein | Severe combined immunodeficiency | Toxicity | Cytotoxicity | Activation | Neuroblastoma | Blood | Neuroblastoma cells | NKG2 antigen | Receptors | Smad2 protein | Immunotherapy | Xenografts | Bone marrow | Plasmas | CD226 antigen | Natural killer cells | Perforin | Antibody-dependent cell-mediated cytotoxicity | Cell survival | Transforming growth factor-b1 | Risk groups | Gene expression | Experimental design | Cell lines | TRAIL protein | Bone | Tumors | Cancer | Index Medicus | TGFβR1 inhibitor | dinutuximab | adoptive cell therapy | galunisertib | immunotherapy
Journal Article
Allergy, ISSN 0105-4538, 01/2010, Volume 65, Issue 1, pp. 109 - 116
To cite this article: Aceves SS, Newbury RO, Chen D, Mueller J, Dohil R, Hoffman H, Bastian JF, Broide DH. Resolution of remodeling in eosinophilic esophagitis... 
budesonide | remodeling | transforming growth factor-beta (TGFβ) | eosinophilic esophagitis | pediatric | Transforming growth factor-beta (TGFβ) | Budesonide | Remodeling | Pediatric | Eosinophilic esophagitis | TRIAL | ALLERGY | transforming growth factor-beta (TGF beta) | ASTHMA | IMMUNOLOGY | FLUTICASONE | CHILDREN | Fibrosis - drug therapy | Glucocorticoids - administration & dosage | Vascular Cell Adhesion Molecule-1 - drug effects | Humans | Child, Preschool | Male | Eosinophilia - pathology | Esophagitis - genetics | Esophagitis - drug therapy | Transforming Growth Factor beta1 - drug effects | Female | Mucous Membrane - drug effects | Eosinophilia - drug therapy | Child | Transforming Growth Factor beta1 - biosynthesis | Promoter Regions, Genetic | Eosinophilia - etiology | Genetic Predisposition to Disease | Administration, Oral | Transforming Growth Factor beta1 - genetics | Esophagitis - immunology | Mucous Membrane - pathology | Mucous Membrane - immunology | Fibrosis - etiology | Adolescent | Vascular Cell Adhesion Molecule-1 - biosynthesis | Polymorphism, Single Nucleotide | Budesonide - administration & dosage | Administration, Topical | Esophagitis | Bone morphogenetic proteins | Corticosteroids | Transforming growth factors | Analysis | Biopsy | Esophagus | Steroids | Children & youth | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, pp. e35144 - e35144
Interleukin-6 (IL-6) is an important cytokine participating in multiple biologic activities in immune regulation and inflammation. IL-6 has been associated... 
VASCULAR FIBROSIS | HYPERTROPHY | MYOCARDIAL-INFARCTION | INFLAMMATION | BIOLOGY | RECEPTOR | MYOFIBROBLASTS | DYSFUNCTION | INTERLEUKIN-6 | HYPERTENSION | TRANSFORMING GROWTH FACTOR-BETA | Angiotensin II - pharmacology | Interleukin-6 - genetics | Mice, Inbred C57BL | Transforming Growth Factor beta1 - metabolism | Male | Myocardium - pathology | Smad3 Protein - metabolism | Interleukin-6 - deficiency | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Fibroblasts - drug effects | Myocardium - metabolism | Fibrosis | Interleukin-6 - biosynthesis | Collagen - biosynthesis | Mice | Phosphorylation - drug effects | Transforming Growth Factor beta1 - biosynthesis | Fibroblasts - metabolism | Immunohistochemistry | Heart | Oxidative stress | Collagen (type I) | Phosphorylation | Smad protein | Cardiomyopathy | Transforming growth factor | Interleukin | Staining | Smooth muscle | Cardiovascular disease | Smad3 protein | Kinases | Macrophages | Interleukin 6 | Infusion | Immunology | Actin | Rodents | Fibroblasts | Angiotensin II | Growth factors | Heart diseases | Culture | Heart failure | Cardiovascular system | Hypertension | Cytokines | Immunoregulation | Muscles | Cardiomyocytes | Inflammation | Gene expression | Hospitals | Angiotensin | Sepsis | Laboratory animals | Index Medicus | Cardiac muscle | Antibodies | Transforming growth factor- beta 1 | Transforming growth factor- beta | cardiomyocytes | Cell activation
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 2008, Volume 590, Issue 1, pp. 400 - 408
Journal Article
Journal Article
Journal Article
Cryobiology, ISSN 0011-2240, 04/2017, Volume 75, pp. 151 - 153
The biological mechanism underlying cryosurgical treatment of keloids remains unclear. Transforming growth factor-beta1 (TGF-β1) has been implicated in the... 
Cryosurgery | Keloid | Transforming growth factor-beta1 | Immunohistochemistry | Keloid - metabolism | Humans | Middle Aged | Female | Male | Keloid - surgery | Transforming Growth Factor beta1 - biosynthesis | Medicine, Experimental | Medical research | Keloids | Bone morphogenetic proteins | Transforming growth factors | Surgery | Index Medicus
Journal Article
Thorax, ISSN 0040-6376, 05/2013, Volume 68, Issue 5, pp. 410 - 420
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e54001 - e54001
Background: Histone deacetylase (HDAC) inhibitors are promising anti-fibrosis drugs; however, nonselective inhibition of class I and class II HDACs does not... 
GROWTH-FACTOR-BETA | SOLID TUMORS | OBSTRUCTIVE NEPHROPATHY | MULTIDISCIPLINARY SCIENCES | CARDIAC-HYPERTROPHY | INTERSTITIAL FIBROSIS | INJURY | ANGIOTENSIN-II | CHRONIC KIDNEY-DISEASE | FACTOR RECEPTOR | MOLECULAR-MECHANISMS | Kidney - pathology | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Smad3 Protein - metabolism | Ureteral Obstruction - metabolism | Leukocytes - immunology | Kidney - metabolism | Receptor, Epidermal Growth Factor - metabolism | Benzamides - therapeutic use | Benzamides - pharmacology | Phosphorylation - drug effects | Ureteral Obstruction - drug therapy | Protein-Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - biosynthesis | Fibroblasts - metabolism | Pyridines - therapeutic use | Collagen Type I - metabolism | Kidney - drug effects | Histone Deacetylases - metabolism | Fibroblasts - pathology | Fibronectins - metabolism | Ureteral Obstruction - immunology | Gene Expression Regulation - drug effects | Acetylation - drug effects | Animals | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Fibroblasts - drug effects | Ureteral Obstruction - pathology | Fibrosis | Histone Deacetylase Inhibitors - pharmacology | Leukocytes - drug effects | Cell Proliferation - drug effects | Histone Deacetylase Inhibitors - therapeutic use | Mice | Pyridines - pharmacology | Histones - metabolism | Fibronectins | Muscle proteins | Growth factors | Collagen | Analysis | Drugs | Histone deacetylase | Phosphorylation | Smad protein | Animal models | Collagen (type I) | Nephrology | Transcription | Syngeneic grafts | Transforming growth factor-b | Smooth muscle | Activation | Kinases | Fibronectin | Medical schools | Epidermal growth factor | Actin | Rodents | Cell cycle | Fibroblasts | Ureter | Inhibition | Kidneys | Transforming growth factor-b1 | Epidermal growth factor receptors | Pulmonary arteries | Fibrils | Muscles | Medicine | Signaling | Hospitals | Kidney diseases | Cancer | Index Medicus
Journal Article