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PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0121246 - e0121246
Background Epithelial cell death is a major contributor to fibrogenesis in the lung. In this study, we sought to determine the function of mitochondria and... 
PARKIN | RECRUITMENT | ACTIVATION | DISEASES | MULTIDISCIPLINARY SCIENCES | DAMAGED MITOCHONDRIA | DEGRADATION | MITOPHAGY | AUTOPHAGY | PINK1-DEFICIENT MICE | PROMOTES | Protein Kinases - metabolism | Cell Line | Gene Expression Regulation, Enzymologic - drug effects | Pulmonary Alveoli - pathology | Reactive Oxygen Species - metabolism | Epithelial Cells - drug effects | Humans | Epithelial Cells - pathology | Male | Pulmonary Fibrosis - pathology | Bleomycin - adverse effects | Mitochondria - drug effects | Mitochondria - pathology | Membrane Potential, Mitochondrial - drug effects | Animals | Mitochondrial Degradation - drug effects | Pulmonary Fibrosis - enzymology | Pulmonary Fibrosis - chemically induced | Mice | Cell Death - drug effects | Transforming Growth Factor beta1 - pharmacology | Cell death | Analysis | Fibrosis | Bone morphogenetic proteins | Respiratory tract diseases | Mitochondrial DNA | B cells | Transforming growth factors | Oxidative stress | Flow cytometry | Epithelial cells | Lung | Homeostasis | Critical care | Kinases | Autophagy | Experiments | Medical schools | Western blotting | Depolarization | Mitochondria | Bleomycin | Rodents | Animal tissues | Alveoli | Growth factors | Transforming growth factor-b1 | Mortality | Lung diseases | Gene expression | Electron microscopy | Medicine | Cytometry | Hospitals | Transmission electron microscopy | Pulmonary fibrosis | Lungs | Womens health | PTEN-induced putative kinase | Laboratory animals | Alzheimers disease | Immunofluorescence | PTEN protein | Apoptosis | Index Medicus
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 02/2016, Volume 13, Issue 1, pp. 37 - 37
Journal Article
Biotechnology and Bioengineering, ISSN 0006-3592, 09/2012, Volume 109, Issue 9, pp. 2357 - 2368
Bioprinting as a promising but unexplored approach for cartilage tissue engineering has the advantages of high throughput, digital control, and highly accurate... 
photoplymerization | cartilage tissue engineering | inkjet printing | chondrocyte | hydrogel | extracellular matrix | Cartilage tissue engineering | Extracellular matrix | Inkjet printing | Hydrogel | Chondrocyte | Photoplymerization | CELLS | SERUM-FREE MEDIUM | HUMAN ARTICULAR CHONDROCYTES | HUMAN EAR CHONDROCYTES | CARTILAGE | IN-VITRO | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | EXPANSION | POLY(ETHYLENE GLYCOL) HYDROGELS | GENE-EXPRESSION | PDGF-BB | Chondrocytes - cytology | Humans | Transforming Growth Factor beta1 - metabolism | Fibroblast Growth Factor 2 - pharmacology | Extracellular Matrix - metabolism | Polyethylene Glycols - chemistry | Methacrylates - chemistry | Photochemical Processes | Tissue Scaffolds - chemistry | Biotechnology - methods | Chondrocytes - drug effects | Hydrogel, Polyethylene Glycol Dimethacrylate | Fibroblast Growth Factor 2 - metabolism | Chondrocytes - metabolism | Transforming Growth Factor beta1 - pharmacology | Tissue Engineering - methods | Extracellular Matrix - drug effects | Cells, Cultured | Polymerization | Proteoglycans - metabolism | Transforming Growth Factor beta1 - genetics | Cartilage - metabolism | Analysis of Variance | Fibroblast Growth Factor 2 - genetics | Histocytochemistry | Biological products | Tissue engineering | Genes | Bone morphogenetic proteins | Fibroblast growth factors | Gene expression | Cartilage | Cell growth | Histology | Gender | Cells | Index Medicus | Human | Bioengineering | Nucleation | Three dimensional printing | Electrochemical machining | Differentiation | Density
Journal Article
Circulation Research, ISSN 0009-7330, 07/2011, Volume 109, Issue 3, pp. e14 - e26
RATIONALE:Systemic sclerosis (SSc) is characterized by widespread microangiopathy, fibrosis, and autoimmunity. Despite the lack of angiogenesis, the expression... 
antiangiogenic agents | systemic sclerosis | peripheral vascular diseases | vascular endothelial growth factor A | angiogenesis | scleroderma | Phosphorylation - physiology | Gene Expression - drug effects | Humans | Transforming Growth Factor beta1 - metabolism | Culture Media, Conditioned - pharmacology | Ischemia - genetics | Vascular Endothelial Growth Factor A - metabolism | RNA-Binding Proteins | Phosphoproteins - metabolism | Vascular Endothelial Growth Factor A - genetics | Gene Expression - physiology | Scleroderma, Systemic - physiopathology | Dermis - blood supply | Neovascularization, Pathologic - physiopathology | Phosphorylation - drug effects | Nuclear Proteins - genetics | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Transforming Growth Factor beta1 - pharmacology | Scleroderma, Systemic - metabolism | Cells, Cultured | Scleroderma, Systemic - genetics | Ischemia - metabolism | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Alternative Splicing - physiology | Nuclear Proteins - metabolism | Transforming Growth Factor beta1 - genetics | Ischemia - physiopathology | Phosphoproteins - genetics | Vascular Endothelial Growth Factor B - pharmacology | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Endothelial Cells - cytology | Serine-Arginine Splicing Factors | Neovascularization, Pathologic - genetics | Signal Transduction - physiology | Vascular Endothelial Growth Factor B - genetics | Neovascularization, Pathologic - metabolism | Index Medicus
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 03/2012, Volume 23, Issue 5, pp. 781 - 791
Journal Article
Cancer Research, ISSN 0008-5472, 12/2010, Volume 70, Issue 23, pp. 9621 - 9630
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2010, Volume 207, Issue 8, pp. 1589 - 1597
Uncontrolled extracellular matrix production by fibroblasts in response to tissue injury contributes to fibrotic diseases, such as idiopathic pulmonary... 
MEDICINE, RESEARCH & EXPERIMENTAL | RESPONSES | PHOSPHATASE | INJURY | TENSIN HOMOLOG | MECHANISMS | GROWTH-FACTOR | IMMUNOLOGY | MYOFIBROBLAST DIFFERENTIATION | EXPRESSION | CANCER | MICRORNA | Oligonucleotides - genetics | Pulmonary Fibrosis - therapy | Idiopathic Pulmonary Fibrosis - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Actins - metabolism | MicroRNAs - metabolism | Pulmonary Fibrosis - genetics | Idiopathic Pulmonary Fibrosis - metabolism | Actins - genetics | Antisense Elements (Genetics) - therapeutic use | Collagen - genetics | Lung - metabolism | Phosphorylation - drug effects | Smad7 Protein - genetics | Extracellular Matrix Proteins - metabolism | Transforming Growth Factor beta1 - pharmacology | Fibroblasts - metabolism | Smad7 Protein - metabolism | Cell Line | Lung - pathology | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Smad2 Protein - metabolism | Mice, Transgenic | Pulmonary Fibrosis - pathology | Transforming Growth Factor beta1 - genetics | Fibroblasts - pathology | Fibronectins - metabolism | Collagen - metabolism | Animals | Fibroblasts - drug effects | Antisense Elements (Genetics) - genetics | Idiopathic Pulmonary Fibrosis - pathology | Pulmonary Fibrosis - chemically induced | Bleomycin - pharmacology | Fibronectins - genetics | Mice | MicroRNAs - genetics | Index Medicus | Brief Definitive Report
Journal Article
Circulation Research, ISSN 0009-7330, 08/2010, Volume 107, Issue 3, pp. 418 - 428
Journal Article
Oncogene, ISSN 0950-9232, 01/2015, Volume 34, Issue 3, pp. 290 - 302
Activation of myofibroblast rich stroma is a rate-limiting step essential for cancer progression. The responsible factors are not fully understood, but TGF... 
Stroma | TGFβ | Exosomes | Fibroblast | Prostate | CELLS | TGF-BETA | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | stroma | INDUCTION | CELL BIOLOGY | BREAST-CANCER | HUMAN PROSTATE-CANCER | fibroblast | ONCOLOGY | REACTIVE STROMA | exosomes | GROWTH | GENETICS & HEREDITY | LUNG FIBROBLASTS | prostate | TGF beta | EXPRESSION | Exosomes - metabolism | Prostatic Neoplasms - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Immunoblotting | Male | Transplantation, Heterologous | rab GTP-Binding Proteins - genetics | Heparitin Sulfate - metabolism | Gene Knockdown Techniques | Myofibroblasts - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Prostatic Neoplasms - genetics | Stromal Cells - drug effects | Cell Differentiation | Transforming Growth Factor beta1 - pharmacology | rab GTP-Binding Proteins - metabolism | Prostatic Neoplasms - pathology | Human Umbilical Vein Endothelial Cells - drug effects | Heparitin Sulfate - pharmacology | Stromal Cells - metabolism | Cells, Cultured | Myofibroblasts - drug effects | Animals | Mice, Nude | Cell Line, Tumor | rab27 GTP-Binding Proteins | Fibroblasts | Genetic aspects | Cell organelles | Transforming growth factors | Health aspects | Carcinogenesis | Angiogenesis | Protein expression | Cellular biology | Prostate cancer | Index Medicus
Journal Article
Journal Article