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FEBS Letters, ISSN 0014-5793, 04/2015, Volume 589, Issue 9, pp. 1040 - 1047
Cartilage generation and degradation are regulated by miRNAs. Our previous study has shown altered expression of miR-193b in chondrogenic human adipose-derived... 
MiR-193b | TGF-beta | Chondrogenesis | TNF-alpha | Osteoarthritis | BIOCHEMISTRY & MOLECULAR BIOLOGY | OSTEOGENIC DIFFERENTIATION | MICRORNAS | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | GROWTH-FACTOR-BETA | BIOPHYSICS | REPRESSES CELL-PROLIFERATION | ARTICULAR-CARTILAGE | CHONDROCYTE PROLIFERATION | SOX9 | EXPRESSION | Tumor Necrosis Factor-alpha - metabolism | Chondrocytes - cytology | Interleukin-1beta - pharmacology | Phosphorylation | Receptors, Transforming Growth Factor beta - genetics | Chondrogenesis - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | 3' Untranslated Regions - genetics | Adipose Tissue - cytology | Smad3 Protein - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Cell Differentiation - genetics | Smad3 Protein - genetics | Mesenchymal Stromal Cells - cytology | RNA Interference | Chondrocytes - metabolism | SOX9 Transcription Factor - metabolism | Mesenchymal Stromal Cells - drug effects | Gene Expression | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Proteoglycans - metabolism | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Animals | Receptors, Transforming Growth Factor beta - metabolism | Cell Differentiation - drug effects | Mice | MicroRNAs - genetics | Transforming Growth Factor beta2 - genetics | SOX9 Transcription Factor - genetics | Core Binding Factor Alpha 1 Subunit - genetics | Proteoglycans - genetics | Transforming growth factors | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e78935 - e78935
Background: An important phenomenon observed in glioma metabolism is increased aerobic glycolysis in tumor cells, which is generally referred to as the Warburg... 
GLIOBLASTOMA | MALIGNANCY | MULTIDISCIPLINARY SCIENCES | THROMBOSPONDIN-1 | MATRIX METALLOPROTEINASE-2 | LATENT TGF-BETA | PROTEINS | EXPRESSION | BINDING | CANCER STEM-CELLS | MOTILITY | L-Lactate Dehydrogenase - metabolism | Apoptosis - drug effects | Transforming Growth Factor beta2 - metabolism | Humans | Transcriptional Activation - drug effects | Apoptosis - genetics | Cell Movement - genetics | Glioma - metabolism | Glioma - genetics | RNA Interference | Isoenzymes - metabolism | Glioma - pathology | Thrombospondin 1 - genetics | Tumor Cells, Cultured | Lactic Acid - pharmacology | Enzyme-Linked Immunosorbent Assay | Isoenzymes - genetics | Lactic Acid - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Cell Movement - drug effects | L-Lactate Dehydrogenase - genetics | Cell Line, Tumor | Cell Proliferation - drug effects | Thrombospondin 1 - metabolism | Transforming Growth Factor beta2 - genetics | Gliomas | Physiological aspects | Development and progression | Genetic aspects | Research | Transforming growth factors | Cell migration | Boyden chamber | Brain cancer | Glioblastoma | Smooth muscle | Neurosurgery | Proteins | Angiogenesis | Proteolysis | Glioma cells | Extracellular matrix | Inhibition | Growth factors | Enzyme-linked immunosorbent assay | Thrombospondin | Transforming growth factor-b1 | Cytokines | Tumor cells | siRNA | Metabolism | Gene expression | Beta2 protein | Latency | Neurology | Immunosuppression | Sodium | Ribonucleic acids | Glycolysis | Hypoxia | Lactic acid | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 05/2017, Volume 94, Issue 4, pp. 759 - 773.e8
Microglia, the resident macrophages of the CNS, engage in various CNS-specific functions that are critical for development and health. To better study... 
CSF1R | phagocytosis | maturation | inflammation | neurodegeneration | TGF-β | cholesterol | transplant | microglia | astrocyte | IDENTITY | GROWTH-FACTOR-BETA | PROGENITOR-CELL | BRAIN-INJURY | RNA-SEQ | ASTROCYTE | MACROPHAGES | MOUSE | RAMIFICATION | EXPRESSION | NEUROSCIENCES | Microglia - metabolism | Transforming Growth Factor beta2 - metabolism | Humans | Transcriptome | Interleukins - metabolism | Microglia - immunology | Macrophage Colony-Stimulating Factor - pharmacology | Serum | Cell Culture Techniques | Interleukins - pharmacology | Microglia - cytology | Cell Survival - drug effects | Phagocytosis - drug effects | Microglia - drug effects | Phagocytosis - immunology | Rats | Macrophage Colony-Stimulating Factor - metabolism | Cholesterol - metabolism | Transforming Growth Factor beta2 - pharmacology | Transforming Growth Factor beta - pharmacology | Animals | Cholesterol - pharmacology | Mice | Transforming Growth Factor beta - metabolism | Astrocytes - metabolism | Culture Media, Conditioned - metabolism | Medical colleges | Bone morphogenetic proteins | Macrophages | Transforming growth factors | Gene expression | Neurophysiology | Genes | Cell culture | Cell survival | Macrophage colony-stimulating factor | Central nervous system | Homeostasis | Exposure | Survival | Cholesterol | Microglia | Phagocytes | Rodents | Morphology | Growth factors | Index Medicus
Journal Article
Cancer Cell, ISSN 1535-6108, 2009, Volume 15, Issue 4, pp. 315 - 327
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2009, Volume 29, Issue 28, pp. 8927 - 8935
Brain injury may result in the development of epilepsy, one of the most common neurological disorders. We previously demonstrated that albumin is critical in... 
TEMPORAL-LOBE EPILEPSY | ALZHEIMERS-DISEASE | TRANSFORMING GROWTH-FACTOR-BETA-1 | IN-VIVO | NEOCORTICAL EPILEPTOGENESIS | STATUS EPILEPTICUS | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | RAT HIPPOCAMPUS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | Electric Stimulation - adverse effects | Rats, Wistar | Transforming Growth Factor beta2 - metabolism | gamma-Aminobutyric Acid - metabolism | Glutamic Acid - genetics | Ion Channels - genetics | Male | Epilepsy - etiology | Brain - metabolism | Transforming Growth Factor beta2 - immunology | Gene Expression - physiology | Statistics, Nonparametric | Dioxoles - pharmacology | Benzamides - pharmacology | Disease Models, Animal | Microarray Analysis - methods | Transforming Growth Factor beta - immunology | gamma-Aminobutyric Acid - genetics | Immunoprecipitation - methods | Albumins - metabolism | Brain - physiopathology | Gene Expression Regulation | Smad2 Protein - metabolism | Rats | Gene Expression Profiling - methods | Blood-Brain Barrier - drug effects | Inflammation - etiology | Epilepsy - complications | Action Potentials - physiology | Antibodies - pharmacology | Transforming Growth Factor beta - pharmacology | Animals | Transforming Growth Factor beta - genetics | Ion Channels - metabolism | Transforming Growth Factor beta2 - antagonists & inhibitors | Signal Transduction - physiology | Glutamic Acid - metabolism | Genome-Wide Association Study - methods | In Vitro Techniques | Epilepsy - pathology | Transforming Growth Factor beta - metabolism | Astrocytes - metabolism | Cluster Analysis | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2017, Volume 37, Issue 11, pp. 2102 - 2102
OBJECTIVE—The role of TGF-β (transforming growth factor-β) signaling in abdominal aortic aneurysm (AAA) formation is controversial. Others reported that... 
aneurysm | smooth muscle | SERVICES TASK-FORCE | ATHEROSCLEROSIS | RECEPTOR | mice | macrophages | TGF-beta | DISRUPTION | angiotensin II | ANEURYSM MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | aorta | SMOOTH-MUSCLE-CELLS | APOE(-/-) MICE | HEMATOLOGY | E-DEFICIENT MICE | PROMOTES | PROGRESSION | Protein-Serine-Threonine Kinases - deficiency | Tunica Media - pathology | Aortic Aneurysm, Abdominal - chemically induced | Receptors, Transforming Growth Factor beta - genetics | Aortic Aneurysm, Abdominal - prevention & control | Muscle, Smooth, Vascular - metabolism | Transforming Growth Factor beta1 - antagonists & inhibitors | Transforming Growth Factor beta2 - metabolism | Aortic Aneurysm, Thoracic - prevention & control | Transforming Growth Factor beta1 - metabolism | Male | Aortic Aneurysm, Thoracic - chemically induced | Transforming Growth Factor beta3 - metabolism | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Adventitia - metabolism | Receptor, Transforming Growth Factor-beta Type II | Aortic Aneurysm, Thoracic - pathology | Tunica Media - metabolism | Transforming Growth Factor beta - antagonists & inhibitors | Female | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Vascular Remodeling - drug effects | Angiotensin II | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Aorta, Thoracic - pathology | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Severity of Illness Index | Genetic Predisposition to Disease | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Aorta, Thoracic - metabolism | Transforming Growth Factor beta3 - antagonists & inhibitors | Mice, Knockout | Antibodies - pharmacology | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Signal Transduction - drug effects | Aortic Aneurysm, Thoracic - metabolism | Transforming Growth Factor beta2 - antagonists & inhibitors | Adventitia - pathology | Transforming Growth Factor beta - metabolism | Receptors, Transforming Growth Factor beta - deficiency | Index Medicus | growth factor | Animal Models of Human Disease | Vascular Disease | Genetically Altered and Transgenic Models | Aortic Dissection | genetically altered mice
Journal Article
Nature Genetics, ISSN 1061-4036, 07/2012, Volume 44, Issue 8, pp. 916 - 921
Journal Article